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1 racterized by the sudden onset of unilateral facial paralysis.
2  interactions and external support following facial paralysis.
3 an result in significant morbidity including facial paralysis.
4  causing acute unilateral lower motor neuron facial paralysis.
5 dition characterized by congenital bilateral facial paralysis.
6 rvical dystonia (2), focal weakness (2), and facial paralysis (1).
7 ent patient education about the diagnosis of facial paralysis; (2) lack of appropriate care coordinat
8  of appropriate care coordination related to facial paralysis; (3) changes in physical and emotional
9 se to explore the potential safety signal of facial paralysis after COVID-19 vaccination.
10 iences of individuals who developed complete facial paralysis after surgical treatment of vestibular
11 ith adults aged 25 to 70 years who developed facial paralysis after treatment for vestibular schwanno
12 atient was left with a sequela of peripheral facial paralysis and 2 patients with sensorineural heari
13 ical blindness/retinal detachment, temporary facial paralysis and maternal death, were adopted.
14 s coarse movement modulation persisted after facial paralysis and thus was not due to sensory reaffer
15  in intellectual and behavioral impairments, facial paralysis, and/or later-onset axonal sensorimotor
16 s in physical and emotional health following facial paralysis; and (4) changes in social interactions
17 feeling that the education and management of facial paralysis by their clinicians was inadequate.
18 rsonality alterations, language disorder and facial paralysis have all been apportioned differing lev
19          It is well-known that patients with facial paralysis have reduced quality of life, severe ps
20 used to treat the early stages of idiopathic facial paralysis (i.e., Bell's palsy), but their effecti
21             As for the hoxb-1 mutations, the facial paralysis observed in mice homozygous for the hox
22 e syndrome, 50% were found to have bilateral facial paralysis on examination.
23                 In this qualitative study of facial paralysis, patients express, in their own words,
24 gressive behaviour, facial dysmorphism, left facial paralysis, post-axial polydactyly, and for the fi
25 hat survived to 3 weeks of age showed marked facial paralysis similar to, but more severe than, that
26 s preserved, and no abnormalities suggesting facial paralysis were noted.
27     Injury to the facial nerve can result in facial paralysis with major functional, social, and psyc