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1 s, 37 had bilateral and three had unilateral facial weakness.
2  of disease and degree of ocular, bulbar and facial weakness.
3                          Four cases also had facial weakness.
4 ance, loss of taste, visual disturbance, and facial weakness.
5 years; 24 females, 16 males) with congenital facial weakness: 38 presented at a Moebius Syndrome Conf
6 finger and foot drop, scapular winging, mild facial weakness, Achilles tendon contractures, and dimin
7                        Mild progressive left facial weakness also developed a few weeks after initial
8 MK-CFZS as a congenital myopathy with marked facial weakness and additional clinical and pathologic f
9  a disorder with congenital, non-progressive facial weakness and limited abduction of one or both eye
10 sistent bulbar involvement, including marked facial weakness and tongue muscle wasting.
11 ations such as ptosis and ophthalmoplegia or facial weakness, and links myasthenic disorders with dys
12 tients presented with total ophthalmoplegia, facial weakness, and myopathy.
13 hat no specific clinical features, including facial weakness, are associated with vaccination-related
14 genital fibrosis of the extraocular muscles, facial weakness, developmental delay and possible periph
15  diagnosis of congenital ophthalmoplegia and facial weakness, even without clinical skeletal myopathy
16 des horizontal gaze abnormalities, deafness, facial weakness, hypoventilation, vascular malformations
17  patients, which persisted in 3 at 4 months; facial weakness in 3 patients, which persisted in 1 at 4
18 g with severe congenital ophthalmoplegia and facial weakness in the setting of only mild skeletal myo
19                                   Congenital facial weakness is present in a heterogeneous group of c
20 terns of ocular motor deficits in congenital facial weakness may assist with further delineation of a
21 rtical movements in subjects with congenital facial weakness may suggest involvement of ocular motor
22 ly progressive proximal and distal weakness, facial weakness, nasal speech, swallowing difficulties,
23                                              Facial weakness occurred in 50% of seropositive particip
24 eriods of weeks or months, with little or no facial weakness or ptosis and no ophthalmoplegia.
25 reas 20.1% present incomplete phenotype with facial weakness or scapular girdle weakness, 6.7% displa
26                                              Facial weakness was more common in SRP-IgG-positive pati
27 linically, the patients' ophthalmoplegia and facial weakness were far more significant than their hyp
28      In addition, the frequency of bilateral facial weakness, which typically occurs in <20% of GBS c