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1 function mutations in the gene encoding this facilitative glucose transporter.
2 nd as dehydroascorbic acid (DHA) through the facilitative glucose transporters.
3 rbic acid is taken up by these cells through facilitative glucose transporters.
4 oxidized form, dehydroascorbic acid, through facilitative glucose transporters.
5 e movement of glucose into cells through the facilitative glucose transporters.
6 ascorbic acid (DHA), enters mitochondria via facilitative glucose transporter 1 (Glut1) and accumulat
8 icated expression of marker proteins such as facilitative glucose transporter 1 and claudin-5 in fres
12 suppresses the glucose transporter-1 (GLUT1) facilitative glucose transporter 49-66% in preimplantati
13 ion matched the sequence for the human GLUT3 facilitative glucose transporter, a high-velocity-high-a
14 Subcellular targeting and the activity of facilitative glucose transporters are likely to be regul
15 u and increased transport of DHA through the facilitative glucose transporters at the cell membrane.
16 is novel protein to members of the mammalian facilitative glucose transporter family (GLUT), we refer
17 Mutations in SLC2A10/GLUT10, a member of the facilitative glucose transporter family, are associated
21 g the exocytic trafficking rate of the GLUT4 facilitative glucose transporter from intracellular stor
23 d muscle cells by regulating delivery of the facilitative glucose transporter, glucose transporter is
24 ve correlation between the expression of the facilitative glucose transporter Glut-1 and FDG accumula
25 ated extracellular glucose concentrations on facilitative glucose transporter (GLUT) expression in ra
26 mainly by insufficient expression levels of facilitative glucose transporter (GLUT)1 in up to 50% of
27 GLUT5, a fructose-transporting member of the facilitative glucose transporter (GLUT, SLC2) family, is
31 sly, we demonstrated a critical role for the facilitative glucose transporter, Glut1, in the regulati
34 -dependent activation and recruitment of the facilitative glucose transporter GLUT2 to the brush-bord
35 s oocyte expression, we investigated whether facilitative glucose transporters GLUT2 and GLUT5-12 tra
37 ity of fat and muscle cells to sequester the facilitative glucose transporter GLUT4 in an intracellul
39 the regulation of glucose transport via the facilitative glucose transporter (GLUT4) and glycogen sy
40 ese hamster ovary cells transfected with the facilitative glucose transporter, GLUT4, we identified t
42 lucose transporters in the body, the passive facilitative glucose transporters (GLUTs) and the second
43 ross mammalian cell membranes is mediated by facilitative glucose transporters (GLUTs) embedded in li
44 ium-coupled glucose transporters (SGLTs) and facilitative glucose transporters (GLUTs) in glucose hom
45 in most cells, vitamin C enters through the facilitative glucose transporters (GLUTs) in the form of
48 enetic inactivation, we targeted the primary facilitative glucose transporter in the retina, Glut1, a
49 in B, indicating the direct participation of facilitative glucose transporters in the transport of ox
50 glucose permeation pathway within the GLUT1 facilitative glucose transporter is hypothesized to be f
57 orbic acid than neutrophils, related to more facilitative glucose transporters on the monocyte cell m
58 pparent K(m) for substrate transport through facilitative glucose transporters on the plasma membrane
59 the total amount of GLUT4 protein or related facilitative glucose transporters present in skeletal mu
61 ips between FDG uptake and the expression of facilitative glucose transporters, the sizes of populati