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1 function mutations in the gene encoding this facilitative glucose transporter.
2 nd as dehydroascorbic acid (DHA) through the facilitative glucose transporters.
3 rbic acid is taken up by these cells through facilitative glucose transporters.
4 oxidized form, dehydroascorbic acid, through facilitative glucose transporters.
5 e movement of glucose into cells through the facilitative glucose transporters.
6 ascorbic acid (DHA), enters mitochondria via facilitative glucose transporter 1 (Glut1) and accumulat
7         Here, we show that expression of the facilitative glucose transporter 1 (GLUT1) is induced by
8 icated expression of marker proteins such as facilitative glucose transporter 1 and claudin-5 in fres
9         Altered glucose reabsorption via the facilitative glucose transporter 2 (GLUT2) during diabet
10                                          The facilitative glucose transporter-3 (GLUT 3) and hexokina
11 y bind and inactivate the insulin-responsive facilitative glucose transporter 4 (GLUT4).
12 suppresses the glucose transporter-1 (GLUT1) facilitative glucose transporter 49-66% in preimplantati
13 ion matched the sequence for the human GLUT3 facilitative glucose transporter, a high-velocity-high-a
14    Subcellular targeting and the activity of facilitative glucose transporters are likely to be regul
15 u and increased transport of DHA through the facilitative glucose transporters at the cell membrane.
16 is novel protein to members of the mammalian facilitative glucose transporter family (GLUT), we refer
17 Mutations in SLC2A10/GLUT10, a member of the facilitative glucose transporter family, are associated
18 tence of additional members of the mammalian facilitative glucose transporter family.
19 ate channel of this and other members of the facilitative glucose transporter family.
20                         Glut1 is the primary facilitative glucose transporter for the retina.
21 g the exocytic trafficking rate of the GLUT4 facilitative glucose transporter from intracellular stor
22       Insulin stimulates the movement of the facilitative glucose transporter glucose transporter-4 (
23 d muscle cells by regulating delivery of the facilitative glucose transporter, glucose transporter is
24 ve correlation between the expression of the facilitative glucose transporter Glut-1 and FDG accumula
25 ated extracellular glucose concentrations on facilitative glucose transporter (GLUT) expression in ra
26  mainly by insufficient expression levels of facilitative glucose transporter (GLUT)1 in up to 50% of
27 GLUT5, a fructose-transporting member of the facilitative glucose transporter (GLUT, SLC2) family, is
28 ucose efflux from tubules to interstitium by facilitative glucose transporters (GLUT).
29                 We report that a decrease in facilitative glucose transporter (GLUT1) expression and
30                        The human erythrocyte facilitative glucose transporter (Glut1) is predicted to
31 sly, we demonstrated a critical role for the facilitative glucose transporter, Glut1, in the regulati
32                                          The facilitative glucose transporter, GLUT1, is expressed on
33  interaction with purified human erythrocyte facilitative glucose transporter, GLUT1.
34 -dependent activation and recruitment of the facilitative glucose transporter GLUT2 to the brush-bord
35 s oocyte expression, we investigated whether facilitative glucose transporters GLUT2 and GLUT5-12 tra
36                  Our hypothesis was that the facilitative glucose transporter, GLUT2, could act as a
37 ity of fat and muscle cells to sequester the facilitative glucose transporter GLUT4 in an intracellul
38 e in fat and muscle is mediated by the major facilitative glucose transporter Glut4.
39  the regulation of glucose transport via the facilitative glucose transporter (GLUT4) and glycogen sy
40 ese hamster ovary cells transfected with the facilitative glucose transporter, GLUT4, we identified t
41                            The expression of facilitative glucose transporters (Gluts) 1, 3, and 4 wa
42 lucose transporters in the body, the passive facilitative glucose transporters (GLUTs) and the second
43 ross mammalian cell membranes is mediated by facilitative glucose transporters (GLUTs) embedded in li
44 ium-coupled glucose transporters (SGLTs) and facilitative glucose transporters (GLUTs) in glucose hom
45  in most cells, vitamin C enters through the facilitative glucose transporters (GLUTs) in the form of
46 (PNBs) via carrier-mediated transport by the facilitative glucose transporters (GLUTs).
47    Glucose transporter 3 (GLUT3) is the main facilitative glucose transporter in neurons.
48 enetic inactivation, we targeted the primary facilitative glucose transporter in the retina, Glut1, a
49 in B, indicating the direct participation of facilitative glucose transporters in the transport of ox
50  glucose permeation pathway within the GLUT1 facilitative glucose transporter is hypothesized to be f
51                                    The GLUT4 facilitative glucose transporter is recruited to the pla
52                  The trafficking of GLUT4, a facilitative glucose transporter, is examined in transfe
53                                   The murine facilitative glucose transporter isoform 3 (Glut 3) is d
54                                   The murine facilitative glucose transporter isoform 3 is developmen
55                                              Facilitative glucose transporter isoform 4 (GLUT4) in ra
56                                    The GLUT4 facilitative glucose transporter mediates insulin-depend
57 orbic acid than neutrophils, related to more facilitative glucose transporters on the monocyte cell m
58 pparent K(m) for substrate transport through facilitative glucose transporters on the plasma membrane
59 the total amount of GLUT4 protein or related facilitative glucose transporters present in skeletal mu
60 ake is highly sensitive to the levels of the facilitative glucose transporter protein, GLUT4.
61 ips between FDG uptake and the expression of facilitative glucose transporters, the sizes of populati
62      Both the GLUT1 and GLUT3 high-affinity, facilitative glucose transporters were expressed in GT1(