ライフサイエンスコーパス: faecium

1 astrointestinal (carriage) populations of E. faecium.

2 e Clostridium sp. 7 2 43FAA and Enterococcus faecium.

3 ia such as Escherichia coli and Enterococcus faecium.

4 synthetic urine also containing Enterococcus faecium.

5 aureus and vancomycin-resistant Enterococcus faecium.

6 licating a role for CcpA in the growth of E. faecium.

7 casei, Lactococcus lactis, and Enterococcus faecium.

8 nized with vancomycin-resistant Enterococcus faecium.

9 for 45 of 46 cultures with E. faecalis or E. faecium.

10 uence of esp on the transport behavior of E. faecium.

11 in the effects of esp on the retention of E. faecium.

12 aureus and vancomycin-resistant Enterococcus faecium.

13 were taken at intervals and cultured for E. faecium.

14 ureus, and vancomycin-resistant Enterococcus faecium.

15 vancomycin-resistant strains of Enterococcus faecium.

16 avancin have different modes of action in E. faecium.

17 cedes invasive infection due to Enterococcus faecium.

18 ween vancomycin-resistant E. faecalis and E. faecium.

19 ty against vancomycin-resistant Enterococcus faecium.

20 ree) were also tested for the presence of E. faecium.

21 n, such as vancomycin-resistant Enterococcus faecium.

22 ge of quinupristin-dalfopristin-resistant E. faecium.

23 in-dalfopristin resistance in human fecal E. faecium.

24 29 cultures, all of which were Enterococcus faecium.

25 reus, Enterococcus faecalis and Enterococcus faecium.

26 ene is associated with reduced density of E. faecium.

27 both E. faecalis and multidrug-resistant E. faecium.

28 E strains (vancomycin-resistant Enterococcus faecium [0], methicillin-resistant Staphylococcus aureus

29 , Escherichia coli (11.8%), and Enterococcus faecium (11.4%).

30 Enterococcus faecalis (48%), Enterococcus faecium (14%), Enterococcus mundtii (13%), and Enterococ

31 Enterococcus faecium 664.1H1 is multiply antibiotic resistant and mer

32 upport for the tetracycline resistance of E. faecium 664.1H1 was characterized.

33 this gene is located on the chromosome of E. faecium 664.1H1, on a novel conjugative transposon.

34 63%, 63%, 63%, and 56%, respectively, for E. faecium, 87%, 83%, 98%, and 80%, respectively, for E. fa

35 We discovered that Enterococcus faecium (E. faecium), a ubiquitous commensal bacterium, and its secr

36 the vancomycin-binding site) in Enterococcus faecium, a leading antibiotic-resistant pathogen.

37 For E. faecium, a susceptible dose-dependent (SDD) breakpoint o

38 ses are nanomolar inhibitors of Enterococcus faecium AAC(6')-Ii.

39 ococci are Enterococcus faecalis, whereas E. faecium accounts for 5 to 10% of isolates.

40 ally and in combination against Enterococcus faecium, Acinetobacter baumannii and Klebsiella pneumoni

41 to 2002, we evaluated the percentages of E. faecium among all enterococcal isolates and the percenta

42 Both the percentage of E. faecium among the enterococci and the proportion of vanc

43 gdom was obtained and cultured to isolate E. faecium, ampicillin-resistant E. faecium (AREfm), and VR

44 eria were MDR, including 95% of Enterococcus faecium and 55% of Enterobacteriaceae; 82% of deep SSIs

45 s, such as vancomycin-resistant Enterococcus faecium and carbapenem-resistant Klebsiella pneumoniae,

46 t Acm has contributed to the emergence of E. faecium and CC17 in nosocomial infections.

47 identifying patients colonized with both E. faecium and E. faecalis, a feature useful for infection

48 s of the recovery of vancomycin-resistant E. faecium and E. faecalis, the sensitivity and PPV were as

49 ferentiate vancomycin-resistant Enterococcus faecium and Enterococcus faecalis (VRE).

50 epidermidis and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay al

51 w-spectrum antibacterial activity against E. faecium and exhibited metabolic stability with low intri

52 cluding human pathogens such as Enterococcus faecium and Listeria monocytogenes.

53 rity) were vancomycin-resistant Enterococcus faecium and meticillin-resistant Staphylococcus aureus.

54 th the exception of 2 isolates, Enterococcus faecium and Nocardia exalbida, all the other 725 (99.7%)

55 l impact on colonization and virulence in E. faecium and possibly other Gram-positive bacterial speci

56 e and drug-resistant strains of Enterococcus faecium and Staphylococcus aureus.

57 of E. faecalis strains (unlike Enterococcus faecium and streptococci) have a single pilus locus.

58 vanA and vanB], 93 vancomycin-susceptible E. faecium) and epidemiological data were collected.

59 iae, Enterococcus faecalis, and Enterococcus faecium) and three associated genetic resistance determi

60 us 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 Staphylococcus aureus isolates using (i

61 imipenem was 91% for E. faecalis, 98% for E. faecium, and 87% for other enterococci.

62 us aureus, vancomycin-resistant Enterococcus faecium, and beta-lactam-resistant Klebsiella pneumoniae

63 ccurate detection of Staphylococcus spp., E. faecium, and E. faecalis and its ability to ascertain me

64 three additional C. coli, one additional E. faecium, and one C. jejuni also developed resistance whe

65 ccus species, 633 strains of E. faecalis, E. faecium, and other enterococci isolated from blood cultu

66 er/platinum combination against Enterococcus faecium, and silver/copper combination against Acinetoba

67 rsed MRSA, vancomycin-resistant Enterococcus faecium, and Staphylococcus epidermidis biofilms.

68 antibiotic-resistant hospital-associated E. faecium are often replaced by clade B strains once patie

69 isolate E. faecium, ampicillin-resistant E. faecium (AREfm), and VREfm.

70 their involvement has been implicated in E. faecium as well.

71 d epidemiological and genomic analysis of E. faecium associated with bloodstream infection and isolat

72 Genomes were also available for E. faecium associated with bloodstream infections in 15 pat

73 walls of vancomycin-susceptible Enterococcus faecium (ATTC 49624).

74 es of Enterococcus faecalis and Enterococcus faecium bacteremia and 9 cases of Streptococcus pneumoni

75 i surface protein (esp) found in Enterococci faecium, Bacteroides HF183, adenoviruses (AVs), and poly

76 country, indicating frequent movement of E. faecium between regions that rarely share hospital patie

77 ycin MICs of 3-4 microg/mL in the initial E. faecium blood isolate predicted microbiological failure

78 ited Kingdom identified 342 patients with E. faecium bloodstream infection over 7 years.

79 ibiotic for multidrug-resistant Enterococcus faecium bloodstream infections (BSIs).

80 took whole-genome sequencing (WGS) of 495 E. faecium bloodstream isolates from 2001-2011 in the Unite

81 t study that included adult patients with E. faecium BSI for whom initial isolates, follow-up blood c

82 We postulate that patients with E. faecium BSIs exhibiting daptomycin MICs of 3-4 microg/mL

83 (VM) errors (4/14) with enterococci (all E. faecium) but none (0/22) with staphylococci.

84 lfopristin resistance was absent in human E. faecium, but 56% of conventional poultry isolates were q

85 llin-imipenem tested with E. faecalis and E. faecium by BMD was >/=94% but was </=90% for other enter

86 he fecal shedding of daptomycin-resistant E. faecium by up to 80-fold.

87 illin- and vancomycin-resistant Enterococcus faecium C68 in a mouse model.

88 ization by vancomycin-resistant Enterococcus faecium C68 in a mouse model.

89 ), at doses that promoted colonization by E. faecium C68.

90 correlated with mutations in the putative E. faecium capsular polysaccharide (cps) biosynthetic locus

91 or more stool samples, 40 (40%) developed E. faecium carriage after admission based on culture, compa

92 ococcus faecium (VREfm) is the driver for E. faecium carriage in hospitalized patients, which, in tur

93 For E. faecium, categorical agreement was poor between the refe

94 ly US AMP isolates were clonally related, E. faecium CC17 isolates have been circulating in the Unite

95 We suggest that actively dividing E. faecium cells have three zones of unique peptidoglycan p

96 d significantly enhance the attachment of E. faecium cells onto the surface of silica sands and thus

97 of colony-forming units per gram) by the E. faecium clades at any time point (P > .05).

98 This gene is highly enriched in E. faecium clinical isolates and absent in commensal isolat

99 These results indicate that E. faecium clinical isolates express transmissible factors

100 sistant Enterococci faecalis and Enterococci faecium clinical isolates revealed that the nature and r

101 ify Acinetobacter baumannii and Enterococcus faecium co-association on multiple surfaces, and demonst

102 e demonstrated a significant reduction in E. faecium collagen adherence by affinity-purified anti-Acm

103 deleted from the chromosome of Enterococcus faecium D344R in all possible combinations in order to i

104 of a large genomic region from Enterococcus faecium D344R in which the sequence from "joint" regions

105 lin-resistant, Tn916-containing Enterococcus faecium D344R.

106 conjugants derived from matings that used E. faecium D344SRF as a recipient strain colonized mouse ga

107 Correspondingly, E. faecalis, but not E. faecium, degrades the glycan on RNase B during growth.

108 eficient Lactococcus lactis and Enterococcus faecium did not enhance biofilm formation as was observe

109 We discovered that Enterococcus faecium (E. faecium), a ubiquitous commensal bacterium,

110 ive staphylococci, Enterococcus faecalis, E. faecium, E. avium, E. durans, E. casseliflavus, and E. g

111 luding the species Enterococcus faecalis, E. faecium, E. casseliflavus, and E. gallinarum.

112 . faecalis, E. mundtii, E. casseliflavus, E. faecium, E. hirae, E. avium, and E. durans, respectively

113 e identified 2 gene clusters in Enterococcus faecium each encoding a homolog of Gls24 (Gls33 and Gls2

114 Enterococcus faecium encodes a triple-specific aaPGS (RakPGS) that ut

115 four distinct surface carbohydrates from E. faecium endocarditis isolate Tx16, shown previously to b

116 ffinity-purified anti-Acm antibodies from E. faecium endocarditis patient sera, suggesting that Acm m

117 antibodies to Acm were present in all 14 E. faecium endocarditis patient sera.

118 on of VanA- and VanB-containing Enterococcus faecium (ENFM) and Enterococcus faecalis (ENFS) isolates

119 aused by Staphylococcus aureus, Enterococcus faecium, Enterococcus faecalis, Klebsiella pneumoniae, P

120 ncluding Enterococcus faecalis, Enterococcus faecium, Enterococcus gallinaraum, and Enterococcus dura

121 ously characterized aaPGSs, the Enterococcus faecium enzyme used an expanded repertoire of amino acid

122 Strains of Enterococcus faecium express a cell wall-anchored protein, Acm, which

123 fibronectin-binding protein of Enterococcus faecium (fnm), a homologue of Streptococcus pneumoniae p

124 of morphine-withdrawn mice were Enterococcus faecium followed by Klebsiella pneumoniae.

125 n-exposed group, daptomycin resistance of E. faecium from the off-target population was on average 50

126 d this idea in a mouse model of Enterococcus faecium gastrointestinal tract colonization.

127 erred DNA into a second recipient strain (E. faecium GE-1), which also colonized mice in significantl

128 ates, are part of the hospital-associated E. faecium genogroup referred to as clonal complex 17 (CC17

129 The Enterococcus faecium genogroup, referred to as clonal complex 17 (CC1

130 E. faecium has been categorized as belonging to three clade

131 uggest that hydrogen peroxide produced by E. faecium has cytotoxic effects and highlight the utility

132 solation of multidrug-resistant Enterococcus faecium has dramatically reduced the therapeutic alterna

133 Enterococcus faecium has recently emerged as an important cause of no

134 Here we show E. faecium has unique peptidoglycan composition and remodel

135 Antimicrobial-resistant E. coli and E. faecium have become significant pathogens in oncology.

136 promote gastrointestinal colonization by E. faecium have not been identified.

137 ncidence of multidrug-resistant Enterococcus faecium hospital infections has been steadily increasing

138 e important nosocomial pathogen Enterococcus faecium in a hospital system where resistance to daptomy

139 portant for the pathogenesis of Enterococcus faecium in a rat infective endocarditis model.

140 opristin, a drug for vancomycin-resistant E. faecium in humans.

141 and dissemination of hospital-associated E. faecium in the UK&I and provide evidence for WGS as an i

142 s, Bifidobacterium bifidum and Streptococcus faecium) in capsules, twice a day for 30 days.

143 ge of enterococci that were identified as E. faecium increased from 12.7 to 22.2% (P < 0.001) and the

144 nd the proportion of vancomycin-resistant E. faecium increased significantly over this 10-year period

145 Six patients (3.4%) developed an invasive E. faecium infection from their own gut-colonizing strain,

146 the potential for streptogramin-resistant E. faecium infection in humans.

147 ts of a glycoconjugate vaccine to prevent E. faecium infection.

148 Enterococcus faecalis and Enterococcus faecium infections are increasingly difficult to treat d

149 articular, vancomycin-resistant Enterococcus faecium infections have been increasing in frequency, re

150 hirty-seven of 41 sera from patients with E. faecium infections showed reactivity with recombinant Ac

151 of 10 genetically related VRE (Enterococcus faecium) infections was discovered.

152 ration approval for the treatment of VRE (E. faecium) infections, namely, linezolid and quinupristin/

153 Enterococcus faecium is a common cause of nosocomial infections, of w

154 Enterococcus faecium is a multidrug-resistant opportunist causing dif

155 E. faecium is able to produce hydrogen peroxide by using gl

156 Enterococcus faecium is an important cause of hospital-associated inf

157 s, non-susceptibility to DAP by Enterococcus faecium is correlated frequently with a mutation in LiaR

158 triazolo[1,5-a]pyrimidines active against E. faecium is reported herein.

159 secreted antigen A (SagA) from Enterococcus faecium is sufficient to protect Caenorhabditis elegans

160 ization by antibiotic-resistant Enterococcus faecium is the first step in a process that can lead to

161 ion of transpeptidation by oritavancin in E. faecium is the result of the large number of secondary b

162 ity of a colonization-proficient clinical E. faecium isolate (C68) to transfer colonizing ability to

163 udy in which the daptomycin resistance of E. faecium isolated from rectal swabs from daptomycin-expos

164 Here we analyzed 90 E. faecium isolates (99% acm(+)) and found that the Acm pro

165 utic alternatives because the majority of E. faecium isolates are resistant to ampicillin and vancomy

166 Among 481 E. faecium isolates belonging to the hospital-adapted clade

167 53 clinical and geographically diverse US E. faecium isolates dating from 1971 to 1994, we determined

168 United Kingdom and Ireland (UK&I) and 11 E. faecium isolates from a reference collection.

169 vatE was present in 38% of E. faecium isolates from patients and none from vegetarians

170 revious studies showed that some clinical E. faecium isolates produce a cell wall-anchored collagen a

171 es were frequently present in 30 clinical E. faecium isolates studied; one of these, acm, has been st

172 erococcal isolates and the percentages of E. faecium isolates that were vancomycin resistant.

173 terococcus faecalis, and 88% of Enterococcus faecium isolates were interpreted as susceptible by Etes

174 VR E. faecium isolates were less genetically diverse than vanc

175 The increasing frequency of Enterococcus faecium isolates with multidrug resistance is a serious

176 occus faecalis isolates, 30 of 58 (51.7%) E. faecium isolates, 1 of 1 E. raffinosus isolate, 0 of 4 E

177 -dalfopristin resistance were assessed in E. faecium isolates, and resistance genes were identified b

178 ium populations, we have now assessed 433 E. faecium isolates, including 264 isolates from human clin

179 ecium strain TX82 and all other sequenced E. faecium isolates.

180 n resistance and 1 for nitrofurantoin, in E. faecium isolates.

181 for the reference method, especially for E. faecium isolates.

182 R E. faecalis isolates and none of the VR E. faecium isolates.

183 in most E. faecalis but not in Enterococcus faecium isolates.

184 determined the structure of the activated E. faecium LiaR protein at 3.2A resolution and, in combinat

185 A comparison of the E. faecalis LiaR, E. faecium LiaR, and the LiaR homolog from Staphylococcus a

186 tal contamination by the hospital-adapted E. faecium lineage were hyperendemic in our study populatio

187 nization levels by strains from different E. faecium lineages: clade B, part of the healthy human mic

188 cereus, Enterococcus faecalis, Enterococcus faecium, Listeria monocytogenes, Streptococcus pneumonia

189 zolid- and vancomycin-resistant Enterococcus faecium (LR-VRE) in solid organ transplant recipients re

190 resistant, vancomycin-resistant Enterococcus faecium (LRVRE).

191 faecalis (green colonies) from Enterococcus faecium (mauve colonies) on the basis of chromogenic sub

192 Using E. faecium-mediated killing of the nematode worm Caenorhabd

193 c strain overproducing catalase prevented E. faecium-mediated killing.

194 ing that bisected the wild-type Enterococcus faecium MIC distribution.

195 g/mL), and vancomycin-resistant Enterococcus faecium (MIC(50) 2.9 +/- 0.8 microg/mL).

196 ococcus aureus: MIC: 16 mug/mL; Enterococcus faecium: MIC: 32 mug/mL) and Harungana madagascariensis

197 agascariensis (S. aureus: MIC: 32 mug/mL; E. faecium: MIC: 32 mug/mL) stem bark.

198 ion caused by a DAP-susceptible Enterococcus faecium (minimum inhibitory concentration, 3 microg/mL)

199 c-Lys could lyse the ampicillin-resistant E. faecium mutant with 3-->3 L-Lys(3)-D-Asn-L-Lys(3) bridge

200 hylococcus aureus (n = 14), and Enterococcus faecium (n = 14).

201 olid were highly active against Enterococcus faecium (n = 267) globally (100% and 98% susceptible, re

202 pathogens: vancomycin-resistant Enterococcus faecium (n=19), methicillin-resistant Staphylococcus aur

203 The frequency of E. faecium occurrence increased from 6% in the dry period t

204 gene in 81 cultures containing Enterococcus faecium or E. faecalis was 100% sensitive and specific.

205 ients presented here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by bo

206 as either vancomycin-resistant Enterococcus faecium or Enterococcus faecalis, based on distinct colo

207 s consideration of vancomycin-susceptible E. faecium Our findings reveal the evolution and disseminat

208 S permease BepA is directly implicated in E. faecium pathogenesis.

209 he first factor shown to be important for E. faecium pathogenesis.

210 a secondary binding interaction with the E. faecium peptidoglycan.

211 d-Ala-d-Ala binding pocket still binds to E. faecium peptidoglycan.

212 rated platinum and gold against Enterococcus faecium, platinum against Klebsiella pneumoniae and plat

213 lence of the other 14 genes among various E. faecium populations, we have now assessed 433 E. faecium

214 indicator of toxicity, we determined that E. faecium produces hydrogen peroxide at levels that cause

215 we identify and characterize 6 Enterococcus faecium proteins containing the WxL domain which, by rev

216 reviously, we have shown that 2 Enterococcus faecium proteins, the secreted antigen A and the peptidy

217 in-dalfopristin (Q-D)-resistant Enterococcus faecium (QDREF) isolates were isolated from humans, turk

218 ceftriaxone, or amoxicillin against DAP-R E. faecium R497 using established in vitro and in vivo mode

219 [CI], 6.20-8.76) and vancomycin-resistant E. faecium (rate ratio, 2.27, 95% CI, 2.03-2.53).

220 Enterococcus faecium recently evolved from a generally avirulent comm

221 nsfer colonizing ability to noncolonizing E. faecium recipient strains.

222 nd 3.1 x 10(1) CFU/mL or g of E. coli and E. faecium, respectively) sprout and water samples tested p

223 Deletion of ahyD in E. faecium resulted in increased formation of Ala-PG and Ly

224 significant ESKAPE pathogens: E nterococcus faecium, S taphylococcus aureus, K lebsiella pneumoniae,

225 We have shown previously that Enterococcus faecium SagA has broad-spectrum binding to extracellular

226 436 S. aureus, 1362 K. pneumoniae and 348 E. faecium samples, ROC curves demonstrate that the conserv

227 ST6 (n = 3), and ST185 (n = 1), which are E. faecium sequence types belonging to clonal complex 5 (CC

228 ich will inform the future translation of E. faecium sequencing into routine outbreak detection and i

229 emic clones ( E. coli ST131 and Enterococcus faecium ST17) were identified for the first time by mult

230 tification of ESKAPE pathogens (Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, A

231 Mortality was also analyzed for Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, A

232 organisms (MDROs), and ESKAPE (Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, A

233 he most common ESKAPE bacteria (Enterococcus faecium, Staphylococcus aureus, Klebsiella pneumoniae, P

234 us spp., Enterococcus faecalis, Enterococcus faecium, Staphylococcus spp., Staphylococcus aureus, Sta

235 icacious against a DAP-tolerant Enterococcus faecium strain (HOU503).

236 coccus pneumoniae pavA, in the genomes of E. faecium strain TX82 and all other sequenced E. faecium i

237 oxicillin was efficacious against a DAP-R E. faecium strain, and pbp5 alleles may be important contri

238 one Campylobacter coli and one Enterococcus faecium strain, while these strains plus three additiona

239 For penicillin, three of five E. faecium strains but none of five Enterococcus faecalis s

240 We previously showed that some E. faecium strains express a cell wall-anchored collagen ad

241 ferred protection against E. faecalis and E. faecium strains in a mouse infection model.

242 ombinations against DAP-resistant (DAP-R) E. faecium strains is unknown.

243 olates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within 4 and 8 h of pre-en

244 We previously showed that E. faecium strains with daptomycin minimum inhibitory conce

245 Overall, our results suggested that E. faecium strains with esp could display lower mobility wi

246 e used cautiously against DAP-susceptible E. faecium strains with minimum inhibitory concentrations >

247 mobility within saturated sand packs than E. faecium strains without esp.

248 aller segments were detected in Enterococcus faecium strains.

249 es to intestinal colonization in clinical E. faecium strains.

250 bserved against different E. faecalis and E. faecium strains.

251 esistance development in four out of five E. faecium strains; however, increased resistance was obser

252 ococcus, Enterococcus faecalis, Enterococcus faecium, Streptococcus agalactiae, Escherichia coli, Kle

253 aureus, Enterococcus faecalis, Enterococcus faecium, Streptococcus pyogenes, Streptococcus agalactia

254 Rectal or fecal samples for E. faecium testing were obtained from 567 newly admitted ho

255 o 22.2% (P < 0.001) and the proportion of E. faecium that was vancomycin resistant increased from 28.

256 is and 4 isolates of vancomycin-resistant E. faecium that were not recovered by BEAV.

257 also show that CcpA affects the growth of E. faecium, that an intact ccpA gene is important for full

258 ver, all the Emp pilins are important for E. faecium to cause infection in the urinary tract.

259 TS, significantly impaired the ability of E. faecium to colonize the murine intestinal tract during a

260 nstrate that Fnm affects the adherence of E. faecium to fibronectin and is important in the pathogene

261 acquisition has favored the adaptation of E. faecium to nosocomial environments and/or patients.

262 ed surveillance and control of nosocomial E. faecium transmission and infection.

263 dy, we provide a comprehensive picture of E. faecium transmission in an entire sampled patient popula

264 We identified E. faecium transposon insertion mutants with altered C. ele

265 urified against these subsegments reduced E. faecium TX2535 strain collagen adherence up to 73 and 50

266 ld decrease (P < 0.05) in the adhesion of E. faecium TX82 to fibronectin.

267 onpiliated DeltaempABC::cat derivative of E. faecium TX82 was attenuated in biofilm formation and in

268 strain of daptomycin-resistant Enterococcus faecium, using a murine model of peritonitis.

269 e no reports experimentally demonstrating E. faecium virulence determinants.

270 oniae (6), vancomycin-resistant Enterococcus faecium (VRE FCM) (16), vancomycin-susceptible Enterococ

271 Vancomycin-resistant Enterococcus faecium (VRE) is a leading cause of hospital-acquired in

272 Vancomycin-resistant Enterococcus faecium (VRE) is a major cause of nosocomial infections.

273 guide anti-vancomycin-resistant Enterococcus faecium (VRE) therapy.

274 the gut by vancomycin-resistant Enterococcus faecium (VRE), a leading cause of hospital-acquired infe

275 us (MRSA), vancomycin-resistant Enterococcus faecium (VRE), and beta-lactam-resistant Klebsiella pneu

276 us (MRSA), vancomycin-resistant Enterococcus faecium (VRE), Escherichia coli SMS-3-5, and Pseudomonas

277 pansion of vancomycin-resistant Enterococcus faecium (VRE), Klebsiella pneumoniae, and Escherichia co

278 (MRSA) and vancomycin-resistant Enteroccocus faecium (VRE).

279 Vancomycin-resistant Enterococcus faecium (VREF) was isolated from six pigs but not from h

280 (MRSA) and vancomycin-resistant Enterococcus faecium (VREF) with MIC values of 1.4 and 2.2 mug mL(-1)

281 , vancomycin-resistant Enterococcus faecalis/faecium (VREfc/VREfm), and ciprofloxacin-resistant Esche

282 ructure of vancomycin-resistant Enterococcus faecium (VREfm) in Latin America (LATAM).

283 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of healthcare-associa

284 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of nosocomial infecti

285 Vancomycin-resistant Enterococcus faecium (VREfm) is an important cause of healthcare-asso

286 mission of vancomycin-resistant Enterococcus faecium (VREfm) is the driver for E. faecium carriage in

287 including vancomycin-resistant Enterococcus faecium (VREfm).

288 stant Enterococcus faecalis and Enterococcus faecium, VRESelect, was compared to bile esculin azide a

289 llin-imipenem tested with E. faecalis and E. faecium was >/=98% and was 92% for other enterococci; CA

290 E. faecium was isolated from 105 patients, 65 vegetarians,

291 of VR isolates, belonging to esp-positive E. faecium, was revealed.

292 esis of Gram-positive bacteria, including E. faecium We previously demonstrated that a nonpiliated De

293 ction with antibiotic-resistant Enterococcus faecium We used a mouse GIT colonization model to test d

294 ighly related vanA-positive and -negative E. faecium, which implies that control of vancomycin-resist

295 encoded immediately adjacent to rakPGS in E. faecium, which is responsible for the hydrolysis of aa-P

296 These effects do not occur in Enterococcus faecium, which lacks the respiratory chain pathway.

297 he disparity in the transport behavior of E. faecium with and without esp could limit the effectivene

298 ycin use in poultry selects for Enterococcus faecium with cross-resistance to quinupristin-dalfoprist

299 lica sands and thus lower the mobility of E. faecium within sand packs.

300 to investigate the transport of Enterococcus faecium within saturated quartz sands.