ライフサイエンスコーパス: fail to stimulate

1 Monomeric IgG2aj failed to stimulate.

2 ells bearing boVbetaBTB35, which SEC(bovine) failed to stimulate.

3 , HU-210, and Delta(9)-tetrahydrocannabinol, failed to stimulate [(35)S]GTP gamma S binding in CB(1)(

4 yrrolidine dicarboxylate and dihydrokainate) failed to stimulate [3H]D-aspartate efflux but did inhib

5 ctivation of Stat1 and Stat3 (up to 1 h) but fail to stimulate a second phase of response and do not

6 In contrast, POT1 failed to stimulate a bacterial 3'-5'-helicase.

7 anti-NK1.1 inhibited the lytic activity and failed to stimulate a calcium response in cells expressi

8 ound to HLA-DR1, but three of these peptides failed to stimulate a CD4(+) T lymphocyte line which rec

9 lpha-dihydrotestosterone, and dexamethasone, failed to stimulate a change in mRNA levels.

10 of Shigella to the T84-cell apical membrane failed to stimulate a directed PMN transepithelial migra

11 lation of NG2(+) glial cell in older animals failed to stimulate a similar repopulation response, pos

12 In contrast, infection with RESTV failed to stimulate a strong host response in infected m

13 umor cells) remain within brain tissue, thus failing to stimulate a systemic immune response.

14 use the mutant defective in DNA binding also fails to stimulate Abf1 ARS1 DNA-binding activity, our r

15 es, a catalytically defective mutant of SopB failed to stimulate actin cytoskeleton rearrangements an

16 We also found that the agonist pramipexole failed to stimulate activation of Akt in PC12-D2R cells,

17 aidic acid (a trans-isomer of linoleic acid) failed to stimulate adhesion to collagen IV, suggesting

18 CR/ABL mutants deficient in PI-3k activation failed to stimulate Akt kinase, a recently identified PI

19 Treatment of ki/ki platelets with thrombin failed to stimulate Akt phosphorylation, resulting in po

20 insulin could actually induce apoptosis and failed to stimulate Akt/GSK-3beta phosphorylation.

21 Human MSC fail to stimulate allogeneic PBMC or T-cell proliferatio

22 e presence of an anticodon-derived stem-loop failed to stimulate aminoacylation of the minihelix.

23 Although most of the suppressor mutants failed to stimulate amplification of genomes encoding th

24 highest activity and an 11mer linear CpG DNA failed to stimulate an immune response.

25 b was a genuine antagonist of CCR5, since it failed to stimulate an increase in intracellular calcium

26 the RPA mutant lacking the C terminus of p34 failed to stimulate an XPA-DNA interaction, and (ii) the

27 c Northwest of Canada and the United States, fails to stimulate an effective immune response in other

28 ed the ability to bind to RhoA, while K1321A failed to stimulate and to bind to RhoA.

29 In Lrg1(-/-) explants, however, IL-6 failed to stimulate angiogenesis and vessels exhibited i

30 but not SLP-76(-/-) platelets (P <.01), and failed to stimulate annexin V binding to either SLP-76(+

31 Immunization with Gal+/+ xenogeneic cells failed to stimulate anti-Gal antibody production in mixe

32 Murine MIP-1 beta, which fails to stimulate any biologic functions in vascular sm

33 Remarkably, BMP4 infusion failed to stimulate aortic NADPH oxidases, increase bloo

34 a and receptor activator of NF-kappaB ligand fail to stimulate AP-1 and NF-kappaB binding to DNA in c

35 es in which whole-cell electrophile flooding fails to stimulate ARE induction prior to causing cytoto

36 ed a mutated, nonfunctional insulin receptor failed to stimulate ARF activation.

37 Serum failed to stimulate association of 14-3-3 with these mut

38 that is defective in nucleolar localization, failed to stimulate ATF5 polyubiquitination and was unab

39 In striking contrast, doxorubicin fails to stimulate autophagy in A375 AC-null cells, whic

40 protein dimerization, since monomeric Gal-1 fails to stimulate axonal re-growth.

41 imulate macrophages and dendritic cells, but fail to stimulate B cells.

42 PET-CT in every volunteer, whereas ephedrine failed to stimulate BAT.

43 Furthermore, the NO donors failed to stimulate bile flow in mutant TR- rats in whic

44 ruitment of the MAPK p38delta (SAPK4), while failing to stimulate binding of JNK, the preferred kinas

45 nds nucleic acid; however, protein substrate fails to stimulate binding.

46 ability to transport AI-2 (lsr null mutant) failed to stimulate biofilm formation.

47 o 11,12-EET, because both 8,9- and 14,15-EET failed to stimulate BK channels.

48 defective EDR1 hepatoma cells, dexamethasone failed to stimulate C/EBP alpha and p21 protein expressi

49 atidylserine (dihexanoyl-phosphatidylserine) failed to stimulate C1P transfer.

50 um from cells expressing prepro[P1]PTH(1-84) failed to stimulate cAMP production despite similar PTH

51 Furthermore, activation of AHR by Way-169916 fails to stimulate canonical DRE-driven AHR-mediated CYP

52 d increase in caspase-3 activity, while IL-1 fails to stimulate caspase-3 activity.

53 The mutant with a replaced AP-1-binding site failed to stimulate CAT expression in an oxidation-sensi

54 t to outer surface protein A, B. burgdorferi failed to stimulate CD14-transfected Chinese hamster ova

55 and MRL lpr/lpr mice, while nonspecific APCs failed to stimulate CD4 T cells.

56 r histocompatibility complex (MHC) antigens, failed to stimulate CD4(+) and CD8(+) T-cell allorespons

57 49) also has RNA triphosphatase activity but fails to stimulate Ceg1 in vitro and is lethal when expr

58 dient but, unlike the wt protein, the mutant failed to stimulate cell migration across a model endoth

59 treatment with the moderate dose of estrogen failed to stimulate cell proliferation, and a decrease i

60 By contrast, cocaine fails to stimulate cell death processes reflecting parth

61 that activation of either chimeric receptor failed to stimulate cellular proliferation.

62 endogenous PKA activity, and phorbol esters failed to stimulate CFTR channels trapped into either th

63 tide (ASBNP) was synthesized and unlike BNP, failed to stimulate cGMP in vascular cells or vasorelax

64 to stimulate cytolytic activity in CTLs also failed to stimulate chemokine release.

65 While IgG1 anti-CD3 MAbs fail to stimulate chimpanzee T cells, IgG2a anti-CD3 MAb

66 C2 (Pro120Ser) fails to bind cholesterol and fails to stimulate cholesterol transfer from NPC1(NTD) t

67 Because the uncaging of Ca2+ fails to stimulate CICR in the absence of cAMP-elevating

68 l(-) current and a Ca(2+)-mobilizing agonist failed to stimulate Cl(-) efflux, requirements for fluid

69 Nuclear-specific overexpression of GSK-3beta failed to stimulate CM differentiation.

70 estricted Ca(2+) signals generated by NMDARs failed to stimulate CREB-dependent transcription.

71 acterium-infected osteoblasts, peptidoglycan failed to stimulate CXCL10 secretion.

72 ith these residues mutated, GCAP1 completely failed to stimulate cyclase activity but still bound Ret

73 ic domain truncated after the GFFKR sequence failed to stimulate cyclin E/cdk2 activation or entry in

74 The D58G/D65G double mutation also failed to stimulate CYP2C19-catalyzed (S)-mephenytoin 4-

75 und to appropriate HLA class I molecules but failed to stimulate cytolytic activity in CTLs also fail

76 mparable degrees; however, unlike LPS, SMase failed to stimulate detectable interferon activity.

77 However, opsonized C. neoformans failed to stimulate detectable release of interleukin 10

78 FCS1/RFCS2 formed in solution; however, they failed to stimulate DNA synthesis by a cognate DNA polym

79 Alone, poly IC fails to stimulate DNA damage in islets isolated from PK

80 LEF-3 was added prior to the polymerase, it failed to stimulate DNApol replication on a singly prime

81 5-HT also fails to stimulate egg laying in ser-7(tm1325), ser-1(ok

82 HERF-1 null kidney tissue; however, dopamine failed to stimulate either cAMP accumulation or PKC acti

83 al ERK and p38 kinase activity, whereas FMLP failed to stimulate either mitogen-activated protein kin

84 Androgens failed to stimulate either the growth of ebp1 transfecta

85 We now present evidence that taxol fails to stimulate either apoptosis or phosphorylation o

86 metholone ejaculated, oxymetholone generally failed to stimulate ejaculation above the levels of the

87 Moreover, aldosterone fails to stimulate ENaC acutely, suggesting that Ang II

88 ermore, culture supernatants from such cells failed to stimulate endothelial cell growth in vitro.

89 uPAR-expressing cells failed to stimulate engulfment of viable cells, suggesti

90 athophysiology of the disease, BMP2 and BMP4 failed to stimulate eNOS phosphorylation in PAECs isolat

91 Furthermore, BMP2 and BMP4 failed to stimulate eNOS phosphorylation when BMPRII was

92 nt on serum constituents because ERR alpha 1 fails to stimulate eNOS promoter-dependent luciferase ac

93 but signaling-defective EphB1 point mutants failed to stimulate ephrin-B1 dependent attachment.

94 oupling to biological responses, Neuregulin4 fails to stimulate ErbB4 coupling to biological response

95 e CRT null mouse embryonic fibroblasts (MEF) fail to stimulate ERK phosphorylation in response to TSP

96 Direct activation of Epac1 failed to stimulate ERK activity in starved cells, sugge

97 pathway without inducing PTH1R endocytosis, failed to stimulate ERK1/2 activity.

98 -34) (10(-8) m), PTH-(1-31), or 8-bromo-cAMP failed to stimulate ERKs, whereas treatment with phorbol

99 ithin SSB's C terminus produce variants that fail to stimulate ExoI activity, whereas the SSB-Ct pept

100 transgenic flies, this mutant dCAMTA variant failed to stimulate expression of dFbxl4 and rescue the

101 Further, IGF-I failed to stimulate F-actin reorganization and phosphory

102 MC3-R and MC4-R antagonist SHU9119 not only failed to stimulate food intake at the same doses as HS0

103 bertal Tg glands, reduced ERalpha expression failed to stimulate formation of the ERalpha/IRS-1 compl

104 s well as angiotensin II, and phorbol ester, fail to stimulate forward Na+-H+ exchange in adult hyper

105 tly, variants detected at both epitopes also failed to stimulate fresh uncultured cells while index p

106 Insulin failed to stimulate FTase in cells expressing the chimer

107 Although FEN acts via beta(2)-ARs, it fails to stimulate G(i)-/NO signalling and preferentiall

108 h facilitates microsomal G6P uptake by G6PT, fails to stimulate G6P uptake in P(i)-loaded G6PT-proteo

109 r the other cyclase receptors, nevertheless, failed to stimulate GC-G expressed in transient or stabl

110 hibit glucagon secretion at high glucose and failed to stimulate glucagon secretion at very low gluco

111 n and an inhibition of insulin secretion but fails to stimulate glucagon secretion.

112 torage because a kinase-inactive (M721) EGFR failed to stimulate glucose incorporation into glycogen

113 Peripheral injection of NMDA failed to stimulate GnRH/LH release in prepubertal or go

114 Moreover, human and mouse VAT failed to stimulate growth in soft of agar in cells defi

115 spho-CREB, JUN/FOS, GATA-1, Pit-1, and EKLF, failed to stimulate HAT activity.

116 e discovered a mutant form of IE1 (YL2) that fails to stimulate HCMV infection while retaining 30 to

117 The RNA helicase dead YTHDC2 (E332Q) mutant failed to stimulate HCV translation initiation.

118 In contrast, FacRPA1 fails to stimulate helicase activity to the same extent

119 ctivation of counterregulation, hypoglycemia failed to stimulate hepatic glycogen breakdown or activa

120 Significantly, partial hepatectomy failed to stimulate hepatocytes to proliferate in p21 tr

121 tion is specific because other RecQ homologs fail to stimulate hExo1.

122 on of T cells in spleen cell suspensions but failed to stimulate highly purified T cells unless these

123 In contrast, T. gondii infection failed to stimulate HIV-1 transcription in tissues of tw

124 ontaining the entire human enhancer homology failed to stimulate human renin promoter activity in tra

125 nsity to form helical structures in TFE also failed to stimulate I(SC).

126 cAMP signaling from the plasma membrane and fails to stimulate iCa(2+) release and recruit beta-arre

127 e type 2 IL-4R and mimics many IL-4 effects, failed to stimulate IFN-gamma production and, in most ex

128 In PKR null cell lines, pIC failed to stimulate IKK activity compared to cells from

129 reover, corneas of galectin-3-deficient mice failed to stimulate IL-1beta after wounding.

130 (dnTCF4) or HCT116 cells with silenced Snail failed to stimulate IL1beta production in macrophages, d

131 s the B7-CD28 costimulatory pathway, DC that failed to stimulate in primary MLR induced markedly augm

132 ecombinant antigens ESAT-6, MPB83, and MPB64 failed to stimulate in vivo DTH in cattle that had been

133 experimentally-induced apposition in molars failed to stimulate increased CC formation in Sost(-/-)

134 FN-gamma, for in IL-12-/- mice egg injection fails to stimulate increased production of either of the

135 cles that readily promote sterol loading but fail to stimulate inflammatory activation.

136 ed from iPLA2beta-null mice, virus infection fails to stimulate iNOS mRNA accumulation and protein ex

137 onpregnant mice and that sEVs from GDM women fail to stimulate insulin secretion and cause exacerbate

138 pletion, leucine, not alpha-ketoisocaproate, failed to stimulate insulin release.

139 ce of nitrendipine and thapsigargin, glucose failed to stimulate insulin release.

140 m of the non-AM-ester of 8-pCPT-2'-O-Me-cAMP failed to stimulate insulin secretion and was a weak act

141 Pilocarpine also fails to stimulate insulin secretion and, instead, antag

142 Curiously, glycerol fails to stimulate insulin secretion, even though it has

143 persisting isolate LCMV clone 13 (CL13) also failed to stimulate interleukin-6 (IL-6) in macrophages.

144 evoid of ligand-dependent downregulation and failed to stimulate intracellular calcium release, cell

145 IP3 failed to stimulate intracellular or plasma membrane (PM

146 oA mutated so that it cannot be geranylated, failed to stimulate invasion.

147 S-1 (Ser-312) and ERK phosphorylation, IGF-I failed to stimulate IRS-1 (Tyr-612) or Akt phosphorylati

148 as reported that in vitro acetylation of p53 fails to stimulate its DNA binding to large DNA fragment

149 IK potently induced NF-kappaB activation, it failed to stimulate JNK activation.

150 uit Raf-1 to the membrane efficiently and so fail to stimulate kinase activity.

151 Compound 14 failed to stimulate locomotor activity in C57BL/6J mice

152 ddition to cultured RAP-deficient adipocytes failed to stimulate LPL secretion in the medium, suggest

153 ted with dexamethasone (10 microM) for 24 hr failed to stimulate luciferase activity, whereas cells c

154 Whilst ATP alone fails to stimulate LX ligation activity, addition of XLF

155 IFN-gamma itself failed to stimulate lymphocyte proliferation and lymphok

156 ic small arteries of anaesthetized rats, EFS failed to stimulate major dilatation via sensory-motor n

157 e tyrosine kinase Syk, both m1 and m2 mAChRs failed to stimulate MAPK kinase and MAPK.

158 In addition, flagellin-null mutants failed to stimulate matrilysin expression in cultured ce

159 Daily administration of recombinant Epo fails to stimulate melanoma growth in vivo, but the trea

160 timulate NF-kappa B-dependent transcription, failed to stimulate membrane translocation of PKC.

161 At 48 h, infected MPhis (I-MPhis) failed to stimulate MHC-II-restricted T cells but not MH

162 Although activated Rit fails to stimulate mitogen-activated protein kinase/extr

163 Moreover, epidermal growth factor (EGF) failed to stimulate MMP-9 expression, cell invasion, and

164 Although IFN-alpha2a failed to stimulate monocyte cytokine secretion, IFN-kap

165 Further, IGF-I failed to stimulate motility of the cells, in which S6K1

166 since a DEF-1 mutant lacking the GAP domain failed to stimulate motility.

167 based vaccines induced MUC1-specific Abs but failed to stimulate MUC1-specific T cells.

168 Insulin treatment also failed to stimulate muscle cytochrome C oxidase activity

169 stent with an insufficient APC function, HSC failed to stimulate naive OT-II TCR transgenic CD4(+) T

170 over, diabetic-derived Gr-1(+)CD11b(+) cells fail to stimulate neovascularization in vivo and have ab

171 Additionally, a GABA(B)R agonist failed to stimulate neutrophil superoxide burst.

172 or mutants lacking a functional death domain failed to stimulate NF-kappa B, while phosphatidylcholin

173 Lipopolysaccharide failed to stimulate NF-kappaB activation in rip1-/-MyD88

174 In contrast, Tax1 mutant M22, which fails to stimulate NF-kappa B-dependent transcription, f

175 Interestingly, HMGB1 failed to stimulate NFkappaB translocation to the nucleu

176 ctivity and increased surface expression but failed to stimulate NHE3 activity or increase surface ex

177 ha, interferon-gamma, and lipopolysaccharide fail to stimulate nitric oxide formation by RINm5F cells

178 AT(2) antagonist PD123319 (1 microm), Ang II failed to stimulate NO (0.1 +/- 0.1 fluorescence units/m

179 ansduced with dominant negative Akt1, Ang II failed to stimulate NO (0.1 +/- 0.2 fluorescence units/m

180 luorescence units/min (p < 0.001; n = 5) but failed to stimulate NO production in THALs isolated from

181 eted FGFR1 (TK-), which did not bind to RSK1 failed to stimulate nuclear RSK1 activity or RSK1 activa

182 In contrast, the kinase inhibitor Lapatinib fails to stimulate nuclear accumulation of the receptor

183 Ang II failed to stimulate O. in TALs from p47(phox) -/- mice (

184 pressing dominant-negative PKC alpha, Ang II failed to stimulate O2.

185 r-tyrosyl phosphorylation of FRS-2 alpha but fails to stimulate or potentiate either FGF-2-induced Er

186 were overexpressed in medium with salt, urea failed to stimulate osmotic stress response.

187 wever, overexpression of C/EBPalpha or c-Fos failed to stimulate osteoclastogenesis in the mutant cel

188 A mutant CBP lacking the N terminus failed to stimulate p53-dependent transactivation.

189 M/ATR kinases and the phosphorylation mutant fails to stimulate p53 ubiquitination.

190 This mutant failed to stimulate PAK and JNK activity but still induc

191 ilar approach, we demonstrate that cytokines fail to stimulate peroxynitrite generation by rat islets

192 5-HT fails to stimulate pharyngeal pumping and the firing of

193 Moreover, Tsc1 deletion failed to stimulate phospho-Ser-302 or other putative S6

194 established criteria for a PITP in vitro and fails to stimulate phosphoinositide production in vivo.

195 f in WT Raptor in a PKA-dependent manner but failed to stimulate phosphorylation of the PKA motif in

196 GH failed to stimulate phosphorylation or activation of Jun

197 of 1,25(OH)2D3 to isolated colonic membranes failed to stimulate PI hydrolysis, but required secoster

198 Integrin alpha2beta1 failed to stimulate PI3Kbeta in platelets from phospholi

199 uces expression of MSH in keratinocytes, but fails to stimulate pigmentation in the absence of functi

200 3-silenced (Sh-Pdia3) cells, 1,25(OH)(2)D(3) failed to stimulate PKC and PGE(2) responses; in Pdia3-o

201 Furthermore, ET-1 failed to stimulate PLA2 activity measured in the cytoso

202 the effects, as a mutant lacking this region failed to stimulate plasminogen activation, although a s

203 mediated inhibition of adenylyl cyclase, but failed to stimulate PLC activity as did wild-type SSTR2.

204 the mutagenesis-defective pol30-113 mutant, fails to stimulate Pol zeta(4) activity, providing an ex

205 Finally, TZDs failed to stimulate PPARgamma activation and adipocyte d

206 Heat-inactivated C. pneumoniae failed to stimulate production of these proteins by all

207 specific inhibition of EGFR or MEK, and GRP failed to stimulate proliferation in EGFR-deficient cell

208 DCs from MB49-bearing female mice fail to stimulate proliferative and IFN-gamma-producing

209 oocytes and embryos revealed that enhancers failed to stimulate promoters prior to formation of a tw

210 biosis associated with sensitization to food fails to stimulate protective tolerogenic pathways, lead

211 A "kinase-dead" EGFR failed to stimulate Rab5a function.

212 in RFS-1, which abolish filament remodeling, fail to stimulate RAD-51 strand exchange activity, demon

213 BRC-2 alone, lacking the DNA-binding domain, fail to stimulate RAD-51-mediated D-loop formation and d

214 A binding and tetramerization mutants of Rta fail to stimulate RBP-Jk DNA binding.

215 F. novicida and F. tularensis subspecies failed to stimulate reactive oxygen species production i

216 Consistent with this, Ang II failed to stimulate renal medullary prostaglandin E(2) a

217 Hyperglycemia failed to stimulate Rho-kinase activity and PAI-1 expres

218 hoGEF and LARG (leukemia-associated RhoGEF), fails to stimulate Rho-dependent transcriptional activat

219 e depletion also increases SGK1 activity but fails to stimulate ROMK channels and K secretion.

220 A) bound effectively to nuclear RSK1, but it failed to stimulate RSK1.

221 tivated ras mutant in Balb/c-3T3 fibroblasts failed to stimulate S phase entry in the absence of plas

222 sence of heregulin, elevation of cAMP levels failed to stimulate Schwann cell proliferation.

223 e previously shown that viable F. tularensis fails to stimulate secretion of proinflammatory cytokine

224 In macrophages from MyD88(-/-) mice, RBP4 fails to stimulate secretion of tumor necrosis factor, I

225 The ligand-coated 6-micron beads also fail to stimulate significant degranulation of RBL-2H3 c

226 show that maximally activated P2X1 receptors failed to stimulate significant aggregation but could am

227 The 0.1-mg simvastatin dose failed to stimulate significant bone growth.

228 Infusion of insulin alone or TGFalpha alone failed to stimulate significant DNA synthesis.

229 is incubated with serum and then washed also failed to stimulate significant TNF-alpha production by

230 ologous single-stranded DNA-binding proteins fail to stimulate similarly the helicase activity of BLM

231 1 were protected from CTL-mediated lysis and failed to stimulate specific memory T-cell responses to

232 Cytokines, however, failed to stimulate sphingomyelin metabolism.

233 However, the complex fails to stimulate splicing activity.

234 ogenic glycoprotein tumor Ags, such as MUC1, fail to stimulate strong immune responses.

235 p38 activity, and heterotypic cross-linking failed to stimulate synergistic activation of either ERK

236 ranscription 3 (STAT3) signaling because gAd failed to stimulate system A in cells in which STAT3 had

237 three NS3358-375 and one NS3505-521 variants failed to stimulate T cell proliferation, and two other

238 eptide was unable to bind I-Ak molecules and failed to stimulate T cells in the absence of intracellu

239 Immature DCs in healthy arteries failed to stimulate T cells, but DCs in PMR arteries cou

240 In MAPTA-loaded hepatocytes, CPT-cAMP failed to stimulate TC uptake, failed to increase cytoso

241 The gamma W72A mutant failed to stimulate TFIID-DNA binding (D-A complex) but

242 s II-restricted altered peptide ligands that fail to stimulate the circulating T lymphocyte repertoir

243 urons in all three areas in intact males but fail to stimulate the magnocellular division of the medi

244 nt antigens arise in HIV-1(+) patients which fail to stimulate the T cell antigen receptor of HLA cla

245 In the Uvhog1 mutant, NaCl treatment failed to stimulate the accumulation of sorbitol and gly

246 In contrast, anti-CD3 antibody failed to stimulate the binding of F-HA in Jurkat transf

247 -deficient cell line in which phorbol esters failed to stimulate the diffusion of integrin.

248 -dependent increase in intracellular calcium failed to stimulate the extracellular signal-regulated p

249 a-subunit gene expression 3-fold, whereas it failed to stimulate the gene cyclooxygenase-2, which was

250 Endothelin-3 failed to stimulate the incorporation of [3H]thymidine o

251 Unlike Tat, NF-kappaB failed to stimulate the integrated transcriptionally sil

252 Further, infection failed to stimulate the memory B cell compartment in pre

253 to induce macrophage proteinase expression, failed to stimulate the phosphorylation of MAPK(erk1/2).

254 hed to fibronectin-coated dishes, calcitonin failed to stimulate the phosphorylation of paxillin and

255 tor, were added to cultured macrophages, but failed to stimulate the production of macrophage inflamm

256 However, activated Rac in non-adherent cells failed to stimulate the Rac effector PAK.

257 l activity of an NFAT-IL-2 reporter gene, it failed to stimulate the transcriptional or DNA-binding a

258 In addition, ZnG UvrA failed to stimulate the UvrB DNA damage-associated ATPas

259 nt with familial platelet disorder with AML, fails to stimulate the ELA2 promoter in vitro, and bone

260 dc2 in granule neurons, activity deprivation fails to stimulate the expression of E2F-target genes th

261 In the absence of p38 activity, EGF fails to stimulate the ubiquitin ligase Cbl or ubiquitin

262 t mammary (Rama) 27 fibroblasts, although it failed to stimulate their proliferation.

263 activity in antisense expressing clones, but failed to stimulate their proliferation.

264 However, the TrkC kinase insert forms fail to stimulate these events.

265 omes from miR-214-depleted endothelial cells failed to stimulate these processes.

266 These two beta-glucans failed to stimulate TNF-alpha in Dectin-1 (beta-glucan r

267 ut is unable to bind topoisomerase IIIalpha, fails to stimulate topoisomerase activity.

268 , since other single strand binding proteins failed to stimulate transfer.

269 e exponential replication over time, Schu S4 failed to stimulate transforming growth factor beta, int

270 tant Y131E, which may mimic phosphotyrosine, failed to stimulate transient DNA replication, and genom

271 2+) mobilization and ERK phosphorylation but failed to stimulate TrkA phosphorylation, NFAT activatio

272 omains from other receptors, EGFR and FGFR1, failed to stimulate TrkB phosphorylation.

273 lutathione S-transferase-TRP in solution but failed to stimulate TRP binding to DNA.

274 Agrin, however, fails to stimulate tyrosine phosphorylation of MuSK that

275 timulation of JAB1 siRNA-transfected RA FLSs failed to stimulate ubiquitination of TRAF2.

276 iophage T4 gene 32 protein (gp32) completely failed to stimulate WRN helicase to unwind long DNA dupl

277 receptor (IGF1R) but, in the absence of FSH, fails to stimulate YXXM phosphorylation of IRS1 (insulin