ライフサイエンスコーパス: failure to

1 coverage for a transplant due to a patient's failure to abstain from drinking, 273 cases involved inc

2 fn1 and Mfn2, spermatogenesis arrests due to failure to accomplish a metabolic shift during meiosis.

3 -values as evidence for the null hypothesis, failure to account for forms of multiple testing that ar

4 Use of Medicare data and failure to account for indirect costs, downstream revenu

5 These include failure to account for intrinsic efficacy and ceiling ef

6 This is due, at least in part, to a failure to account for neurobiological heterogeneity.

7 Failure to account for variation in individual risk lead

8 als from the viscera (or both), leading to a failure to accurately update beliefs about the body.

9 The primary outcome was failure to achieve a clinically functional AVF within 8

10 Induction failure was defined as failure to achieve a partial response (PR) by the end of

11 Failure to achieve a PSA50 was associated with shorter p

12 ts were 18-70 years old with TRD, defined as failure to achieve a satisfactory response (e.g., less t

13 For failure to achieve an improvement in global IBS symptoms

14 o achieve three or more CSBMs per week or or failure to achieve an increase of one or more CSBM per w

15 n best spirometry does not predict survival, failure to achieve FVC>80% predicted during the first ye

16 Based on an endpoint of failure to achieve improvement in global IBS symptoms at

17 Failure to achieve kidney recovery after immune checkpoi

18 e hypothesized to be strong risk factors for failure to achieve or maintain a suppressed HIV viral lo

19 d strengthen clinical prediction of risk for failure to achieve or maintain HIV RNA suppression in CS

20 over to the other mapping arm was defined as failure to achieve the primary end point.

21 Based on an endpoint of failure to achieve three or more complete spontaneous bo

22 ere superior to placebo, according to either failure to achieve three or more CSBMs per week or or fa

23 ted to strategic corrections of task errors (failures to achieve task goals).

24 sion and stalled fork recovery, along with a failure to activate CHK1 phosphorylation.

25 This difference correlates with the failure to activate high levels of expression of interfe

26 of SMAD1/5 phosphorylation and a concomitant failure to activate Lef1, Fgf8 and Wnt4, thus explaining

27 ion immediately following weaning leads to a failure to activate medial prefrontal cortex neurons pro

28 damage response protein TopBP1, resulting in failure to activate the ATR-Chk1 pathway despite increas

29 ulates aspects of limb development, and that failure to activate this developmental program results i

30 s of participants or site anomalies, such as failures to adhere to study protocols that could negativ

31 A failure to adjust expectations sufficiently when an envi

32 was limited by an overall lack of research, failure to adjust for several key confounding factors, a

33 This failure to adjust resulted in these drought-stressed tre

34 back-driven coexistence, partly because of a failure to analyse pairwise PSF, the metric directly lin

35 t whereas criminal justice professionals see failures to appear as relatively unintentional, laypeopl

36 These interventions reduce failures to appear by 13 to 21% and lead to 30,000 fewer

37 s were limited by narrow selection criteria, failure to apply reference standard tests consistently,

38 nt for this disappointing outcome, including failure to appreciate disease heterogeneity, inappropria

39 Failure to appropriately account for the grouped nature

40 e circuit on target preferring neurons and a failure to appropriately discount the activity of neuron

41 urface forces apparatus (SFA) allow adhesive failure to be distinguished from cohesive failure and sh

42 ted to our hospital with decompensated heart failure to be evaluated for a heart transplant.

43 ta collection systems, and identify modes of failure to both quantify and qualify network performance

44 Failure to bypass these obstacles results in genome inst

45 Failure to clear antigens causes CD8(+) T cells to becom

46 severity of these infections combined with a failure to clearly identify the causative pathogen resul

47 , with partial closure in 33 (28%) cases and failure to close in 18 (15%) cases.

48 real-time show that this defect is due to a failure to closely align DNA ends.

49 A failure to complete MiDAS at FRAXA leads to severe locus

50 The main limitation of the study was failure to conduct MMR germline sequencing for the whole

51 Failure to confirm these infections retrospectively if a

52 odel of agonism are frequently confounded by failure to consider the assumptions of the model.

53 tical and experimental evidence showing that failure to consider the assumptions of the operational m

54 We describe how the failure to consider the complex systems that characteriz

55 of statistics in nursing research, including failure to consider the effects of multiple testing, ina

56 have been inconsistent, possibly because of failure to consider the time-to-event nature of the outc

57 n times than controls, possibly due to their failure to construct value-based or internal evidence du

58 y INR and PLT count), without an increase in failure to control bleed, failure to prevent rebleed, an

59 Failure to control bleed, failure to prevent rebleeds, a

60 ciated with each hour until antibiotics, and failure to control for large potential confounders inclu

61 d functionality and subsequently mediate the failure to control M. tuberculosis infection in macropha

62 Failure to control occult retinal vasculitis adequately

63 Either a failure to convert ribulose-5-phosphate to R5P or a bloc

64 Failure to cooperate in the provision or preservation of

65 ion results in attenuation of these CRRs and failure to correct glucose deficit, constituting a poten

66 currently incurable ciliopathy caused by the failure to correctly establish or maintain cilia-depende

67 at the expenditure differences resulted from failure to correctly estimate the respiratory quotient (

68 imulation by SARS-CoV-2 alone but detect the failure to counteract STAT1 phosphorylation upon IFN-I p

69 itals were included, of whom 630 (10.7%) had failure to cure (hospital variation [5.5%-19.1%]).

70 This study aimed to describe failure to cure in terms of incidence, hospital variatio

71 Failure to cure is a composite outcome measure that coul

72 spitals had statistically significant higher failure to cure percentages, whereas 2 had lower percent

73 The failure to cure rate after salvage surgery was 27.6% in

74 The incidence of failure to cure was 10.7%.

75 Association of baseline characteristics with failure to cure was analyzed using multivariable logisti

76 Failure to cure was defined as: 1) no surgical resection

77 cant hospital variation in the percentage of failure to cure, improvement is needed.

78 d resection before 2014 were associated with failure to cure.

79 51 salvage esophagectomy patients, 32.5% had failure to cure.

80 and non-constipated individuals, especially failure to defecate (Delta = 41.2%).

81 luded in the Rome IV criteria, such as daily failure to defecate and an average duration of straining

82 Failure to defend the lower limit of plasma triglyceride

83 Failure to degrade B-type Cyclins during anaphase preven

84 ation of the more abundant piRNA resulted in failure to degrade maternally deposited transcripts in t

85 Failure to demonstrate ACM benefit should not diminish a

86 s directed against people who use drugs, and failures to deploy evidence-based therapies for opioid a

87 his approach relies on negative evidence - a failure to detect a homolog of a query gene.

88 ation, impaired flow-induced dilatation, and failure to detect beta3 integrin S-sulfhydration, all of

89 ' years represent real pauses in breeding or failures to detect breeding can be difficult, posing pro

90 can lead to both false positive results and failures to detect genuine associations.

91 aps inattention to behavioral state leads to failures to detect movement-driven activity [8].

92 immune responses and may be the result of a failure to develop immune memory to infection, pointing

93 esponses following vaccination can result in failure to develop protective immunity leaving individua

94 animal models is often complicated by their failure to develop the end-stage complications which cha

95 Their failure to develop tolerance may be explained by their h

96 eciation of family history often result in a failure to diagnose ADTKD.

97 included mismanagement of glaucoma (20.3%), failure to diagnose glaucoma (17.4%), failure to diagnos

98 0.3%), failure to diagnose glaucoma (17.4%), failure to diagnose or mismanagement of angle-closure gl

99 mon scenarios leading to litigation included failure to diagnose or mismanagement of glaucomatous dis

100 ) from mouse progenitor epithelia results in failure to differentiate key pancreatic lineages: acinar

101 osis-susceptible population characterized by failure to differentiate, whereas successful differentia

102 er patients (52%) developed refractory heart failure to disabling New York Heart Association function

103 In contrast, discrimination errors (i.e., failure to discriminate similar stimuli) were associated

104 5 mum) in the absence of MatP because of its failure to displace MukBEF from the 800 kbp replication

105 site analysis is referred to as "prophylaxis failure" to distinguish from the pre-specified sensitivi

106 is critical to maintaining homeostasis, and failure to do so contributes to the burden of chronic in

107 Failure to do so could result in gaps in transparency, s

108 m a single, round nucleus after mitosis, and failures to do so can compromise genomic integrity.

109 del in which Lmo2-induced T-ALL results from failure to downregulate Ldb1/Lmo2-nucleated transcriptio

110 alyses suggested that errors followed from a failure to encode the target stimulus in both AC and dlP

111 drawbacks including drug resistance and the failure to eradicate infection highlight the need to ide

112 multiple meiotic prophase defects including failure to establish homolog pairing, inappropriate load

113 phorylation of the SAF-A DNA-binding domain; failure to execute this pathway leads to accumulation of

114 Moreover, this failure to exhibit apoptotic responses was not associate

115 ce that respond to such training, and with a failure to exhibit LTP after aggression training.

116 Failure to expand pancreatic beta-cells in response to m

117 This failure to 'explain away' connections occurs when there

118 lower expression of Fgf10 and a concomitant failure to express genes required for limb proliferation

119 Although water treatment strategies, failure to follow regulations, and unethical behavior we

120 ied of CRC vs 53.2% of cancer-free patients; failure to follow-up on abnormal results occurred in 8.1

121 g to spermatid decapitation as a result of a failure to form a stable head-tail linkage.

122 R) results from incomplete NCC migration and failure to form an appropriate ENS.

123 The failure to form synapses can cause the destabilization a

124 ng one of these muteins, we demonstrate that failure to form the NSC in FOP results in more severe di

125 ignment of the OF margins in Nf2 mutants and failure to fuse properly, resulting in persistent colobo

126 yo, and knockout of these ligands leads to a failure to gastrulate.

127 Obstacles to PROMs use include failure to generate actionable data, implementation obst

128 distinct types of errors: failures-to-stop, failures-to-go, and incorrect choices.

129 oustic stimuli, prey-capture deficits, and a failure to habituate to acoustic stimuli, are replicated

130 these neurons, whose activity underlies the failure to habituate.SIGNIFICANCE STATEMENT Habituation

131 R(i) mutant CTCF, which in turn results in a failure to halt cohesin-mediated extrusion.

132 ir molecular composition and structure and a failure to identify PNN-specific targets.

133 enge in the treatment of glioblastoma is the failure to identify the cancer invasive area outside the

134 trials and negative results may reflect the failure to implement the intervention.

135 Failure to include both sexes and to address age in mech

136 nonpathogenic Bacillus species, resulting in failure to induce a robust host response, which may cont

137 them resistant to insulin signals marked by failure to induce Akt2 phosphorylation.

138 mediated down-regulation of cAMP, subsequent failure to induce Death-Inducing Signaling Complex (DISC

139 These defects were associated with failure to induce IL-23 and IL-6, two key IL-22 inducers

140 development efforts, largely because of the failure to induce potent antibody responses targeting co

141 TA system have been lost, such as following failure to inherit a plasmid harbouring a TA system.

142 Failure to inhibit replication initiation indeed causes

143 itory saccade control.SIGNIFICANCE STATEMENT Failures to inhibit automatic saccadic responses are a h

144 was used to identify factors associated with failure to initiate ART rapidly under treat all.

145 , followed by severe developmental delay and failure to initiate gastrulation by embryonic day 7.5.

146 preterm birth, slow progress of labour, and failure to initiate labour.

147 The failure to initiate MSCI is linked to complete meiotic a

148 Suboptimal adherence, which includes failure to initiate pharmacotherapy, to take medications

149 ning deficit in high AQ scorers, namely, the failure to integrate social context to adapt one's belie

150 Odds of experiencing therapeutic inertia (failure to intensify treatment within 12 months of HbA1c

151 Diabetes Mellitus, or Prediabetes, and Heart Failure]) to investigate the cardiac effects of empaglif

152 rgued that psychosis may emerge because of a failure to learn sensory statistics, resulting in an imp

153 FrEF) and a recent hospitalization for heart failure to liraglutide versus placebo.

154 1 is inactive during mitosis, reflecting its failure to localize to lysosomes due to CDK1-dependent R

155 A failure to maintain meiosis can result in highly aberran

156 ence or telomere length but is a result of a failure to maintain progenitor cell populations.

157 Failure to maintain these physiologic rhythms invariably

158 If so, then a failure to manage for hybridization will exacerbate the

159 he verbal nature of existing hypotheses, the failure to mechanistically link interacting ecological a

160 ated transcriptional activity resulting in a failure to mediate wild-type cellular responses includin

161 Failure to meet 30by3 had increased odds of mortality (o

162 his recessive disorder is characterized by a failure to metabolize cobalamin into adenosyl- and methy

163 on > 255 min, "nonhybrid" esophagectomy, and failure to mobilize patients within 24 h from surgery we

164 We demonstrated that failure to mount a coherent physiologic response to simp

165 Failure to mount this nuclear mechanoresponse results in

166 Failure to normalize preop or postop CA19-9 was associat

167 There were no postoperative complications or failure to observe the study protocol.

168 uptake to mount a proper immune response and failure to obtain sufficient nutrients or engage the app

169 ge various B cell and T cell tumors, because failure to open DNA hairpins and accumulation of chromos

170 MENT Infertility affects 15%-20% of couples; failure to ovulate is a common cause.

171 Although a failure to persist often has ecological causes, key gaps

172 Failure to precisely distinguish malignant from healthy

173 However, it has been critiqued for its failure to predict a broader range of community properti

174 Failure to predict and understand the causes of preterm

175 pressure, and number of extrapulmonary organ failures to predict ICU outcome at 24 hours of acute res

176 ntification of "at-risk" patient cohorts for failure to present or delays to treatment.

177 haviour is important for patients with heart failure to prevent disease progression.

178 out an increase in failure to control bleed, failure to prevent rebleed, and mortality.

179 Failure to control bleed, failure to prevent rebleeds, and mortality between the t

180 ul for studying recovery mechanisms, but its failure to produce a substantial ischemic penumbra weake

181 vation has been linked to reduced virulence, failure to produce biofilms, and impaired intercellular

182 Fanconi anaemia (FA), a disease resulting in failure to produce blood cells and a predisposition to c

183 ion of B cells and thereby contribute to the failure to produce neutralizing antibodies.

184 The age-related failure to produce oligodendrocytes from oligodendrocyte

185 d Y. pestis bacterial burden in the lung and failure to progress into the lethal proinflammatory phas

186 rust were: inability to follow instructions, failure to progress, and unsafe manoeuvres.

187 speed of cell intercalation, resulting in a failure to promote full elongation of the body axis.

188 s abolished on all chromosomes, leading to a failure to properly congress or orient chromosomes in me

189 tance use disorders (SUDs) may result from a failure to properly encode rules guiding situationally a

190 d in individuals with SUDs may result from a failure to properly encode states because of drug-induce

191 Specifically, the failure to properly replicate, resect, and/or form appro

192 Conversely, the novel measure unmasked failure to provide defect-free chemoprophylaxis in 18% (

193 this study were to: (1) measure the rate of failure to provide defect-free postoperative venous thro

194 ) chemoprophylaxis, (2) identify reasons for failure to provide defect-free VTE chemoprophylaxis, and

195 Conversely, failure to provide T-CPR in this manner results in preve

196 l vein in 110 patients with or without heart failure to quantify the uptake and release of 277 metabo

197 Failure to reabsorb the primary cilium induces cellular

198 Failure to reach 30by3 was associated with increased odd

199 ed the contribution of adhesive and cohesive failure to rebleeding in a rat model of femoral artery i

200 rcoming several significant barriers such as failure to recapitulate the human TME in animal models a

201 ndomly assigned patients with advanced heart failure to receive either the centrifugal-flow pump or t

202 preoperative visual acuity (VA) (P = 0.001), failure to receive panretinal photocoagulation within 2

203 Failure to receive signal 2 within a defined time window

204 However, failure to recognize adenomas (vs hyperplastic polyps),

205 s historically a disease of miners; however, failure to recognize and control the risk associated wit

206 ic derangement, and human factors, including failure to recognize and reluctance to manage the failed

207 tive results typically prompt revaccination, failure to recognize presumptive immunity in individuals

208 eral deficiency in producing ATP, but rather failure to recoup the ATP cost of glycolysis and diversi

209 enter (MTOC) linker protein, C-NAP1, and the failure to recruit MTOC components and liquid-like spind

210 the RA model, efficacy was achieved despite failure to reduce autoantibodies.

211 Failure to regenerate a normal EC monolayer in response

212 ficantly reduced proliferation and selective failure to regenerate cone photoreceptors.

213 including issues regarding potential loss by failure to regrow, contamination on transfer, mix up and

214 Failure to reinitiate ART within 18 months was common in

215 ustment for multiple comparisons resulted in failure to reject the null hypothesis (adjusted p = 0.08

216 s, cirrhosis, HBeAg negative at baseline and failure to remain in virological remission were associat

217 mutations produce hypomorphic alleles whose failure to remain within the ER significantly reduces bu

218 ions in maternal and fetal blood spaces, and failure to remodel the maternal spiral arteries.

219 re and cancer susceptibility stemming from a failure to remove DNA crosslinks and other chromosomal l

220 The failure to remove protein aggregates, damaged organelles

221 tes has been challenged because of a claimed failure to replicate our findings (Landegren, 2019).

222 ence is a consequence of seemingly pervasive failures to replicate previous research.

223 Failure to report the results of a clinical trial can di

224 antly impaired GC formation, associated with failure to repress immune synapse genes linked to GC exi

225 this review, we discuss the consequences of failure to reprogram histone methylation during three cr

226 ion between centers' volume and incidence of failure to rescue (FTR) following liver resection for he

227 Outcomes were failure to rescue (FTR), mortality, number of subsequent

228 uld reduce postoperative mortality (POM) and failure to rescue (FTR).

229 to trigger an escalation of care to prevent failure to rescue and eventually poor outcome.

230 morbidity-mortality composite end point, or failure to rescue.

231 ve mortality (POM), major morbidity (MM) and failure-to-rescue (FTR) after cytoreductive surgery (CRS

232 Repeated failure to resolve chronic wounds compromises the reserv

233 ecent studies link chronic inflammation with failure to resolve early inflammation, a process operate

234 olves pathologic inflammation and subsequent failure to resolve fibrosis in response to epithelial in

235 Failure to resolve internalized fluid impairs the tissue

236 Failure to resolve mtDNA breaks leads to mitochondrial d

237 TOP1ccs are highly cytotoxic and a failure to resolve them underlies the pathology of neuro

238 Failure to resolve tubular injury after U-IRI results in

239 ive glucocorticoid signaling associated with failure to respond to checkpoint blockade in both precli

240 s due to its metastatic propensity, frequent failure to respond to chemotherapy, and lack of alternat

241 e in tubular cells were each associated with failure to respond to treatment.

242 zation; and 3) recognition and management of failure to restore oxygenation and reduce the risk of ca

243 distinct behavioral impairments, including a failure to retrieve stimulus value, a reduced task engag

244 ted to routine use of antibiotics because of failure to return an adequate biomarker result.

245 stent and accurate dose monitoring; however, failure to return one or both badges, reversal of badges

246 Failure to screen, or failure to screen at appropriate intervals, occurred in

247 Failure to screen was associated with fewer visits to pr

248 Failure to screen, or failure to screen at appropriate i

249 , attributed to a lack of pain sensation and failure to seek care for minor injuries.

250 Selective failure to shed the most abundant SUB2 substrate, MSP1,

251 Loss of Dnmt3a and Dnmt3b resulted in failure to silence developmental genes.

252 are found throughout vertebrate genomes, and failure to silence their activation can have deleterious

253 However, the failure to simulate tropical forests under extreme condi

254 We also show that failure to simultaneously account for all three effects

255 The primary outcome was failure to spontaneously pass the gestational sac within

256 tematic processes in formulating guidelines, failure to state conflicts of interest, and lack of cons

257 Failure to stimulate postprandial LCN2 in individuals wi

258 were predictive of distinct types of errors: failures-to-stop, failures-to-go, and incorrect choices.

259 e idea that movement corrections driven by a failure to successfully achieve movement goals underpin

260 iescence compared to log phase, resulting in failure to survive during starvation.

261 fatty acid (FA) oxidation, which causes the failure to switch to glycolysis.

262 Failure to synthesize IP8 (via Asp1 IPP kinase mutation)

263 sult from people trying to explain their own failure to take action to combat inequality.

264 edication usage, poor symptom perception and failure to take responsibility, and positive factors wer

265 I argue that failure to take the alignment between verbal and statist

266 dicate the onset and propagation of adhesion failure to the evolution of piezo-resistivity in varying

267 plants, soft sensors could expose faults and failures to the operator.

268 Although failure to thermoregulate spermatogenetic tissue and pro

269 ung cancer (n=4100), dementia (n=40,084), or failure to thrive (n=42,950) between 2003 and 2014.

270 constipation, cholelithiasis, short stature, failure to thrive).

271 ient presented with delayed cord separation, failure to thrive, and sepsis.

272 erized by elevation of glycine, seizures and failure to thrive, but glycine reduction often fails to

273 n impairment, pontocerebellar abnormalities, failure to thrive, liver dysfunction, lower extremity ed

274 t include severe neurodevelopmental defects, failure to thrive, ocular abnormalities, and defects in

275 r intestinal inflammation, food allergy, and failure to thrive, often necessitating nutritional suppl

276 nodeficiency in a patient who presented with failure to thrive, persistent EBV viremia and hepatitis,

277 d colons, nutritional defects, and a general failure to thrive, rarely surviving beyond weaning.

278 clinical features of these patients include failure to thrive, short stature, feeding difficulties,

279 cribe unrelated individuals with ichthyosis, failure to thrive, thrombocytopenia, photophobia, and pr

280 ntion of seizure activity or amelioration of failure to thrive.

281 SC expansion, defective cell cycle exit, and failure to transition efficiently from the proliferative

282 aphase I but then display a highly penetrant failure to transition to anaphase I.

283 Motivational anhedonia, the failure to translate positive reinforcements into future

284 ingly beset by chronic illness and prominent failures to translate research from "bench to bedside."

285 ical F-actin to plasma membrane results in a failure to transmit actomyosin force to cell membrane, c

286 sodic or chronic migraine who had documented failure to two to four classes of migraine preventive me

287 tion and persistence, often as a result of a failure to undergo apoptosis when tissue repair is compl

288 d confounding in observational research, and failure to understand risks and benefits of treatments w

289 e and suggest future work to capture dynamic failure to unravel mechanisms that account for convoluti

290 contexts in working memory and, ultimately, failure to update overfamiliar prior beliefs.

291 ion of reduced fatty acid availability and a failure to upregulate carbohydrate metabolism, essential

292 epithelial regeneration was paralleled by a failure to upregulate IkappaBa, the molecule that termin

293 ation of post-publication correspondence and failure to use >=1 alternative search strategy.

294 t is possible that getting lost represents a failure to use distal cues to get oriented in space.

295 Specifically, we provide evidence for a failure to use the strength of others' disconfirming opi

296 The failure to use the Wells Scores in all cases does not ne

297 cally and determined factors associated with failure to vaccinate using spatial and multivariate logi

298 Failure to visualize PGs could lead to accidental damage

299 erred from an acute care hospital because of failure to wean from mechanical ventilation and who were

300 e respiratory distress syndrome, followed by failure to wean ventilation and post-extracorporeal memb