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1 constipation, cholelithiasis, short stature, failure to thrive).
2 pmental delay, progressive microcephaly, and failure to thrive).
3 al retardation, conductive hearing loss, and failure to thrive.
4  2 yr of age, who presented with vomiting or failure to thrive.
5 mes that conspire to create the phenotype of failure to thrive.
6 ted patients diagnosed with juvenile ALS and failure to thrive.
7  in infancy with refractory constipation and failure to thrive.
8  malabsorption, intestinal inflammation, and failure to thrive.
9  including hypotonia, suckling deficits, and failure to thrive.
10  with lymphoproliferation, autoimmunity, and failure to thrive.
11 l heart disease, skeletal abnormalities, and failure to thrive.
12 rized by neonatal-onset watery diarrhoea and failure to thrive.
13 rrent fevers, interstitial lung disease, and failure to thrive.
14 ein-losing enteropathy, hypoalbuminemia, and failure to thrive.
15 lor, and lethargy; chronic FPIES can lead to failure to thrive.
16 ading to irreversible kidney dysfunction and failure to thrive.
17  to deteriorated eczema, severe diarrhea and failure to thrive.
18 ated with severe neurological regression and failure to thrive.
19 , moderate to severe developmental delay and failure to thrive.
20 pecially if accompanied by systemic signs of failure to thrive.
21 ntion of seizure activity or amelioration of failure to thrive.
22            These defects result primarily in failure to thrive.
23 trolyte imbalance, recurrent infections, and failure to thrive.
24 y P14, when they began exhibiting ataxia and failure to thrive.
25 us cause severe Cbl deficiency and the noted failure to thrive.
26 s [28.1%]; OR, 1.87; 95% CI, 1.02-3.42), and failure to thrive (215 patients [22.6%]; OR, 1.62; 95% C
27  who presented with intractable diarrhea and failure to thrive after in utero exposure to pembrolizum
28    Classical symptoms of CS patients include failure to thrive and a severe neuropathology characteri
29             This multisystem disorder causes failure to thrive and accelerated atherosclerosis leadin
30 ions and gastrointestinal disorders, such as failure to thrive and delayed gastric emptying, together
31 gastrointestinal (GI) disease because of the failure to thrive and early death from malnutrition in i
32  mice lacking alpha but not beta suffer from failure to thrive and early mortality.
33 alities, hypotonia, intellectual disability, failure to thrive and feeding problems.
34 y different; one presented in childhood with failure to thrive and hypertrophic cardiomyopathy, and t
35 A exhibit metabolic abnormalities, including failure to thrive and increased diabetes risk.
36 as severe psychomotor retardation, seizures, failure to thrive and intolerance to wheat and dairy pro
37 f Maf factors results in a neonatal-specific failure to thrive and loss of macromolecular nutrient up
38 beginning at postnatal day 5 with subsequent failure to thrive and median survival of 35 d.
39  usually presents in the newborn period with failure to thrive and metabolic crisis leading to coma o
40 This paper examines the medical etymology of failure to thrive and proposes a rational approach to ev
41 d muscular hyper- and hypotonia, spasticity, failure to thrive and short stature, feeding difficultie
42 ile hypotonia, and irritability, followed by failure to thrive and short stature.
43 eated infections), 7 (fungal infections), 5 (failure to thrive) and 4 (>=2 pneumonias in 1 year) were
44 ons are common and include chronic diarrhea, failure to thrive, and abdominal distention; however, ex
45 ce, a substantial attenuation in the initial failure to thrive, and an increase in life span.
46 ss behavioural phenotype corresponded with a failure to thrive, and death in homozygote atg13 nulls w
47 ns included inadequate caloric intake (85%), failure to thrive, and feeding difficulties.
48 a on infections, gastrointestinal illnesses, failure to thrive, and hospital readmission in the first
49  resulting in sparse hair, male infertility, failure to thrive, and hydrocephaly, the anemia is the f
50                             We describe PAP, failure to thrive, and increased GM-CSF levels in two si
51 ed endothelial nitric oxide (ENOS) activity, failure to thrive, and kidney insufficiency.
52 n and thereby result in persistent acidosis, failure to thrive, and nephrocalcinosis.
53  delirium, dehydration, falls and fractures, failure to thrive, and pressure ulcers.
54 henotype, infant-onset recurrent infections, failure to thrive, and protracted diarrhea.
55 cessive condition characterized by seizures, failure to thrive, and rapid, progressive neurodegenerat
56 al mutilation, cerebral leukoencephalopathy, failure to thrive, and recurrent metabolic acidosis with
57 ient presented with delayed cord separation, failure to thrive, and sepsis.
58 d Mut(ko/ki) mice survive post-weaning, show failure to thrive, and show increased methylmalonic acid
59 sy including infantile spasms, irritability, failure to thrive, and stereotypic hand movements.
60 for seizures (AOR, 1.58; 95% CI, 1.01-2.46), failure to thrive (AOR, 1.99; 95% CI, 1.36-2.93), trauma
61 ep apnea in infants has been associated with failure to thrive, behavioral deficits, and sudden infan
62  infections, gastrointestinal illnesses, and failure to thrive between IBD cases and matched controls
63 erized by elevation of glycine, seizures and failure to thrive, but glycine reduction often fails to
64 r, neutrophilic dermatitis/panniculitis, and failure to thrive, but without obvious primary immunodef
65 perimental group, 11 of 11 mice demonstrated failure to thrive by day 13; 5 deaths occurred between d
66  multiple organ systems, encompassing severe failure to thrive, cardiac anomalies including hypertrop
67 eumonia, autoimmunity, chronic diarrhea, and failure to thrive compared to CTLA-4 insufficiency (n =
68 f VEGF is associated with (1) a high rate of failure to thrive/death and (2) formation of endothelial
69                  We describe a new case with failure to thrive, developmental delay, lactic acidosis
70 A2 who exhibited global developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy,
71 uterine growth retardation, dehydration, and failure to thrive due to a lack of normal insulin secret
72 spectrum disorder, developmental regression, failure-to-thrive, exercise intolerance/fatigue) was ass
73 atic G6PC activity up to 98% and resulted in failure to thrive, fasting hypoglycemia, hypertriglyceri
74              Elevated sweat chloride levels, failure to thrive (FTT), and lung disease are characteri
75                 Infected mice had a relative failure to thrive, gaining weight significantly more slo
76 idge-Ropers syndrome (BRS), characterized by failure to thrive, global developmental delay, feeding p
77 ndividuals show prenatal growth restriction, failure to thrive, global developmental delay, intracere
78 F1 gene in a 2-year-old girl presenting with failure to thrive, global neurodevelopmental regression,
79 premature aging, including low birth weight, failure to thrive, graying and loss of hair, reduced ski
80                  Despite this heterogeneity, failure to thrive has had its own international Classifi
81 r that presents during infancy, resulting in failure to thrive, hepatomegaly, and hepatic failure, an
82           Tat expression in the brain caused failure to thrive, hunched gesture, tremor, ataxia, and
83 nine XSCID share similar features, such as a failure to thrive, hypogammaglobulinemia, an absent T ce
84 use an early onset multisystem disorder with failure to thrive, immunodeficiency and neurological sym
85 peatedly cited as prevalent in patients with failure to thrive: impaired physical functioning, malnut
86 ficiently treated dRTA can cause rickets and failure to thrive in children, osteomalacia in adults, n
87  PWS is characterized by severe hypotonia, a failure to thrive in infancy and, on emerging from infan
88 onatal respiratory depression, hypotonia and failure to thrive in infancy, followed by hyperphagia an
89 racterized by severe fat malabsorption and a failure to thrive in infancy.
90  role in inflammation and contributes to the failure to thrive in this mouse model of CF.
91 ter of these infants meet the definition of 'failure to thrive' in the first year of life.
92 velopmental disorder characterized by severe failure to thrive, intellectual disability, and facial d
93                                    The term "failure to thrive" is frequently used to describe older
94 sorder resulting in seizures, hypotonia, and failure to thrive, is due to inherited loss-of-function
95 cal form of celiac disease, characterized by failure to thrive, is still the most frequent presentati
96 uited for investigating nutritionally based "failure-to-thrive" issues, particularly regarding the lo
97 ylation imprints at Ndn and Mkrn3 and suffer failure to thrive leading to a fully penetrant neonatal
98 ogressively develop HGPS symptoms, including failure to thrive, lipodystrophy, vascular smooth muscle
99 n impairment, pontocerebellar abnormalities, failure to thrive, liver dysfunction, lower extremity ed
100  the clinical presentation of CD can include failure to thrive, malnutrition, and distension in juven
101 en with SMS and include infantile hypotonia, failure to thrive, mental retardation, autistic features
102 on of patients with PYCR2 mutations included failure to thrive, microcephaly, craniofacial dysmorphis
103 ures were skin manifestations (n = 14, 93%), failure to thrive (n = 10, 67%), recurrent infections (n
104 ons and intra-abdominal abscess (n = 75) and failure to thrive (n = 38).
105 ment were cerebrovascular accident (n = 80), failure to thrive (n = 71), other central nervous system
106 ), rectal haemorrhage (n=1), dyspnoea (n=1), failure to thrive (n=1), and interstitial lung disease (
107 ung cancer (n=4100), dementia (n=40,084), or failure to thrive (n=42,950) between 2003 and 2014.
108 o adverse events (one cardiac arrest and one failure to thrive), neither of which were treatment-rela
109 endently increased for comorbid diagnoses of failure to thrive, neurodevelopmental delay, cardiopulmo
110  report a proband presenting with hypotonia, failure to thrive, nystagmus, and abnormal brain MRI fin
111 t include severe neurodevelopmental defects, failure to thrive, ocular abnormalities, and defects in
112 r intestinal inflammation, food allergy, and failure to thrive, often necessitating nutritional suppl
113 if they had conditions affecting growth (eg, failure to thrive or cystic fibrosis), any acute or chro
114 nodeficiency in a patient who presented with failure to thrive, persistent EBV viremia and hepatitis,
115 mb2(-/-) mice are responsible for the severe failure to thrive phenotype, and that renal replacement
116 ia, hypokalemic alkalosis, low birth weight, failure to thrive, poor growth, and in many cases nephro
117 d colons, nutritional defects, and a general failure to thrive, rarely surviving beyond weaning.
118 mmon problem which can manifest as vomiting, failure to thrive, recurrent pneumonias, asthma, sinusit
119          PTLS is characterized by hypotonia, failure to thrive, reduced body weight, intellectual dis
120 onates had severe malabsorptive diarrhea and failure to thrive, required prolonged parenteral nutriti
121 ive fumaric aciduria in early childhood with failure to thrive, seizures, developmental delay, mental
122  deaths and infants presenting at birth with failure to thrive, severe salt-wasting crises associated
123  clinical features of these patients include failure to thrive, short stature, feeding difficulties,
124 th -3.4 SDS; weight -2.1 SDS) presented with failure to thrive, short stature, severe hypocalcemia an
125  age of one with severe infantile-onset IBD, failure to thrive, skin rash, and perirectal abscesses r
126 t a valid diagnostic entity, but encompasses failure to thrive, stunting secondary to chronic malnutr
127  the mouse p locus that is associated with a failure-to-thrive syndrome, in addition to diminished pi
128     Live-born FX(-/-) mice exhibit postnatal failure to thrive that is suppressed with a fucose-suppl
129 cribe unrelated individuals with ichthyosis, failure to thrive, thrombocytopenia, photophobia, and pr
130 op a neurological phenotype characterized by failure to thrive, tremor, and gait ataxia.
131 ma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous mutations in ABCA12
132 ma, hypotrichosis, severe nail dystrophy and failure to thrive, two heterozygous mutations in ABCA12
133 ic SLC34A1 variant carriers showed polyuria, failure to thrive, vomiting, constipation, hypercalcemia
134 s characterized by severe hypercalcemia with failure to thrive, vomiting, dehydration, and nephrocalc
135                    Patients with weight loss/failure to thrive were more likely to receive dietitian
136          We describe three infants, two with failure to thrive, who had dehydration and diarrhea with
137 re normal at birth but exhibited progressive failure to thrive, whole-body wasting, and ataxia and di

 
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