ライフサイエンスコーパス: family environment

1 th affective psychopathology, cognition, and family environment.

2 clinical characteristics, and aspects of the family environment.

3 of several demographic parameters as well as family environment.

4 nce is attributable to the effects of shared family environment.

5 eatment and emotional abuse in the childhood family environment.

6 tering, and emotional abuse in the childhood family environment.

7 otectiveness and authoritarianism was due to family environment.

8 tial influence of both genetic variation and family environment.

9 may reflect influence of demography and the family environment.

10 nd prevent possible depressogenic effects of family environment.

11 both early neuromotor deficits and unstable family environments.

12 ral problems have been associated with their family environments.

13 of common variants but other factors such as family environments.

14 logy, socioeconomic status (SES), school and family environment; 10 child psychopathological scales;

15 ndirect genetic effects mediated through the family environment(4) also play a role.

16 In addition, an emotionally abusive family environment accentuated the decrements in mental

17 h their precise matching for age, the common family environment and background environmental variatio

18 hood demonstrated associations between risky family environment and CVH remained significant for thos

19 ome-wide association study analyses, such as family environment and early childhood experiences.

20 by synthesizing the wealth of literature on family environment and limbic development, specifically

21 o quantify the association between childhood family environment and longitudinal cardiovascular healt

22 review the more comprehensive literature on family environment and neurobehavioral outcomes in pedia

23 Both an emotionally abusive family environment and the interaction of an emotionally

24 Stressful early family environments and neuroticism were significantly a

25 pmental delays among children separated from family environments and placed in orphanages or other in

26 cy implications in that improving school and family environments and promoting local economic develop

27 contribution of at least 40% from the common family environment, and a difference in the genetic cont

28 ren are exposed to childhood maltreatment in family environments, and another in which children are e

29 problems, parental substance misuse, adverse family environments, and high-risk child maltreatment-re

30 se, maternal mental health problems, adverse family environments, and high-risk presentations of chil

31 tween the brain structure of children, their family environments, and their behavioral problems.

32 haracteristics of students, and the home and family environment are in the net effect category.

33 substantial literature base has established family environment as a significant predictor of neurobe

34 blished that genes and aspects of the shared family environment both play important roles in shaping

35 First, aspects of the family environment, but not the wider neighbourhood cont

36 in early life and entry into a high-quality family environment can support more normative trajectori

37 history of substance use disorder, disturbed family environment, childhood parental loss, low self-es

38 dered: 1) childhood (genetic risk, disturbed family environment, childhood sexual abuse, and childhoo

39 rompt us to speculate that both genetics and family environment contribute to acquisition of EBV infe

40 sult solely from genetic factors or does the family environment contribute?

41 ty was 14.5% +/- 4.0% (P < .001), and common family environment contributed 81.0% +/- 4% (P < .001) t

42 Shared (family) environment did not seem to play a role.

43 nce use disorders, parental loss, vulnerable family environment), early-adolescence (self-esteem, soc

44 hese conditions relates to sociocultural and family environments, especially a gulf between how women

45 r genetic factors, modelled alongside common family environment, explained phenotypic variance in alc

46 By contrast, neuroticism, early family environment, female gender and not being coupled

47 idence suggesting the relevance of the early family environment for launching public health intervent

48 in the US and globally that assures adequate family environments for children.

49 esults demonstrate that without a supportive family environment, gender identity development increase

50 Family environment generally declined in importance over

51 this older age, the influence of the shared family environment had disappeared, and only aspects of

52 e, any effects that host genes and the early family environment have on the presence of specific bact

53 e sex, US medical education, difficult early family environment, history of major depression, lower b

54 rs for spouses to determine whether a common family environment in adulthood plays a role in disease

55 plain this familial aggregation; the role of family environment in generalized anxiety disorder is un

56 Child-specific aspects of the family environment, including mothers' child-feeding pra

57 While the family environment, including parental religiosity, shap

58 n all major dimensions of mental health, the family environment is an important influence on only int

59 However, the degree to which family environment matters varies, such that countries w

60 tantially influenced by genetic factors, but family environment may also play a role.

61 Examination of aspects of the family environment may provide insight into increases in

62 We investigated whether aspects of the family environment mediated genetic effects on academic

63 ffects' model fits the data better than the 'family-environments' model.

64 After controlling for genes and family environment, no significant differences existed b

65 2) whether there were any differences in the family environment of adolescents with JPFS compared wit

66 ease prevention must be directed also to the family environment of the developing child.

67 twins, suggesting a negligible effect of the family environment on these outcomes.

68 l control in feeding, an aspect of nonshared family environment, on daughters' eating and relative we

69 R, 1.01 [95% CI, 1.00-1.03]; P = .03), early family environment (OR, 1.03 [95% CI, 1.01-1.05]; P < .0

70 problems, parental substance misuse, adverse family environments, or high-risk presentations of maltr

71 ved illness severity and vulnerability), and family environment (parental support and parent-child co

72 Data on academic achievement and family environments (parenting, home environments, and g

73 The addition of the family-environment pathway provided a good fit and showe

74 disorders, family composition, and childhood family environments perceived as violent and lacking coh

75 found that socioeconomic status, rather than family environment, played a more important role in modi

76 with prior results in studies of women, the family environment plays a role in twin resemblance for

77 he results of the current study suggest that family environment plays a significant role in the forma

78 Family environment probably has an impact on risk for ag

79 the characteristics of social relationships, family environment, quality of schools and activities, f

80 Risky Family environment (range, 7-28) was assessed at year 15

81 s, we summarize the extant literature on the family environment's role in neurobehavioral sequelae in

82 Inventory, Children's Depression Inventory, Family Environment Scale (FES), Sensitivity Temperament

83 anxiety and eating disorders, the so-called family environment, special issues raised by twin studie

84 assessed included depression, anxiety, early family environment, stressful life experiences, medical

85 Expressed emotion (EE) is a measure of the family environment that has been demonstrated to be a re

86 cts core psychopathological processes in the family environment that link putatively separate psychia

87 ctors, including genetic liability and early family environment, that are shared by identical twins w

88 ined the contribution of genetics and shared family environment to chronic pain by spouse, sibling, a

89 The neural mechanisms linking family environment to neurobehavioral outcomes have, how

90 Family environment was an important influence on interpe

91 ohol and other drugs and many aspects of the family environment were assessed at 7 different ages, pr

92 Second, family environments were more robustly linked to noncogn

93 ; 95% CI 0.57-1.04); estimates for improving family environments were similar (0.59; 0.43-0.80).

94 can account for GxE interactions and shared family environment, which in many cases are not explaine

95 nd the interaction of an emotionally abusive family environment with the various maltreatment types h