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1 itis) to severely invasive (e.g. necrotizing fasciitis).
2  etc.) to life-threatening (e.g. necrotizing fasciitis).
3 thickness > 4 mm being suggestive of plantar fasciitis.
4 irulence factors in our model of necrotizing fasciitis.
5 host interactions underlying GAS necrotizing fasciitis.
6 e-threatening infections such as necrotizing fasciitis.
7 he molecular pathogenesis of GAS necrotizing fasciitis.
8 A) is a rarely reported cause of necrotizing fasciitis.
9 tening invasive disease, such as necrotizing fasciitis.
10  of diseases from pharyngitis to necrotizing fasciitis.
11 s; average, 49.9 years) with chronic plantar fasciitis.
12 fibrils but a severely destructive monocytic fasciitis.
13 for previous cases of C albicans necrotizing fasciitis.
14 d invasive infections, including necrotizing fasciitis.
15 e model of group A streptococcal necrotizing fasciitis.
16 d organ failure, with or without necrotizing fasciitis.
17 ction in a murine model of human necrotizing fasciitis.
18  valuable in assessing suspected necrotizing fasciitis.
19  LINE-1 insertion associated with keloid and fasciitis.
20  compare them with patients with necrotizing fasciitis.
21 ce phenotype in a mouse model of necrotizing fasciitis.
22 ning invasive infections such as necrotizing fasciitis.
23  severely ill than patients with necrotizing fasciitis.
24 (ABC), and the related benign lesion nodular fasciitis.
25 e in a nonhuman primate model of necrotizing fasciitis.
26 ed virulence in a mouse model of necrotizing fasciitis.
27 ococcal toxic shock syndrome and necrotizing fasciitis.
28 sses ranging from pharyngitis to necrotizing fasciitis.
29 an primate experimental model of necrotizing fasciitis.
30 mmon pathological conditions such as plantar fasciitis.
31 such as toxic shock syndrome and necrotizing fasciitis.
32 ce phenotype in a mouse model of necrotizing fasciitis.
33 g invasive infections, including necrotizing fasciitis.
34 animal models of pharyngitis and necrotizing fasciitis.
35 human primates with experimental necrotizing fasciitis.
36 ed virulence in a mouse model of necrotizing fasciitis.
37 literature on this rare cause of necrotizing fasciitis.
38 e-threatening infections such as necrotizing fasciitis.
39 sed with caution in the diagnosis of plantar fasciitis.
40 rimate models of pharyngitis and necrotizing fasciitis.
41 s anginosus constellatus causing necrotizing fasciitis.
42 mary isolate from a patient with necrotizing fasciitis.
43 ess virulent in a mouse model of necrotizing fasciitis.
44 ns is an extremely rare cause of necrotizing fasciitis.
45 ), and subsequent development of necrotizing fasciitis (1 of 8).
46 e were performed on 28 patients with plantar fasciitis; 17 had spondylarthropathy (SpA)-associated di
47 comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02), especiall
48 with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), while median (
49 unger and had a higher degree of necrotizing fasciitis (56% vs 14%).
50 h toxic shock syndrome (55%) and necrotizing fasciitis (58%).
51                                      Plantar fasciitis accounts for more than 1 million patient visit
52  This review highlights three areas: plantar fasciitis, Achilles tendinitis, and carpal tunnel syndro
53 rep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating syndrome).
54                                  Necrotizing fasciitis, also known as the flesh-eating disease, is a
55 t these cells at high levels in eosinophilic fasciitis, an orphan disease characterized by inflammati
56  deviation, 46.3 years +/- 8.7) with plantar fasciitis and 50 feet of 50 asymptomatic volunteers (27
57 ician in confirming the diagnosis of plantar fasciitis and also in assessing the response to treatmen
58 in a high clinical suspicion for necrotizing fasciitis and distinguish it from more common forms of c
59 riage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in human blood
60                                  Necrotizing fasciitis and myonecrosis caused by invasive infection w
61                                  Necrotizing fasciitis and myositis are devastating infections charac
62                                  Necrotizing fasciitis and myositis caused by group A streptococci (G
63 eptococcal toxic shock syndrome, necrotizing fasciitis and myositis, and the post-infection sequelae
64 s including cellulitis, superficial and deep fasciitis and necrotizing fasciitis, pyomyositis, infect
65 individuals with TRAPS is due to a monocytic fasciitis and not to myositis.
66 he role of SpyCEP in S. pyogenes necrotizing fasciitis and respiratory tract infection in mice using
67  fibroblasts from patients with eosinophilic fasciitis and scleromyxedema were studied as well as PBM
68 ious invasive infections such as necrotising fasciitis and sepsis.
69 vere invasive infections such as necrotizing fasciitis and streptococcal toxic shock syndrome.
70 a variety of diseases, including necrotizing fasciitis and toxic shock syndrome, were analyzed.
71 sive human infections, including necrotizing fasciitis and toxic shock syndrome.
72 e infections in humans including necrotizing fasciitis and toxic shock syndrome.
73 h invasive infections, including necrotizing fasciitis and toxic shock syndrome.
74 etigo to severe outcomes such as necrotizing fasciitis and toxic shock syndrome.
75  infections worldwide, including necrotizing fasciitis and toxic shock syndrome.
76 o invasive conditions, including necrotizing fasciitis and toxic shock syndrome.
77 n infections to life-threatening necrotizing fasciitis and toxic shock syndrome.
78 ve infections, including sepsis, necrotizing fasciitis and toxic shock.
79 specific association between GAS necrotising fasciitis and trauma.
80 that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngitis) diseases.
81                      Morton neuroma, plantar fasciitis, and Achilles tendinopathy are foot and ankle
82                      Morton neuroma, plantar fasciitis, and Achilles tendinopathy are painful foot an
83 kness, clostridial gas gangrene, necrotizing fasciitis, and acute crush injury.
84 s, acute kidney injury, perineal necrotizing fasciitis, and hyperkalemia.
85 carditis, necrotizing pneumonia, necrotizing fasciitis, and septicemia.
86 a; stretching and foot orthotics for plantar fasciitis; and eccentric strengthening exercises for Ach
87 coccal bacteremia, myositis, and necrotizing fasciitis are presented and compared with those of Strep
88       The diagnosis and treatment of plantar fasciitis are reviewed; nonsurgical treatments remain th
89  toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive manifestations
90 itis, impetigo, toxic shock, and necrotizing fasciitis, as well as the postinfection sequelae rheumat
91 characteristics and outcomes of eosinophilic fasciitis-associated monoclonal gammopathy.
92 as been a very uncommon cause of necrotizing fasciitis, but we have recently noted an alarming number
93                         Diagnosis of plantar fasciitis can be easily verified by ultrasonography with
94 ed protease activity and reduced necrotizing fasciitis capacity.
95 ificantly underrepresented among necrotizing fasciitis cases has a unique frameshift mutation that tr
96                                  Necrotizing fasciitis caused by C albicans is extremely rare in the
97                                  Necrotizing fasciitis caused by community-associated MRSA is an emer
98 coccal toxic shock syndrome, and necrotizing fasciitis) caused by six distinct M types of GAS serocon
99 lysis of primary clinical samples of nodular fasciitis confirmed the activation of a Jak1-STAT3 gene
100 developed into rapidly spreading necrotizing fasciitis despite antimicrobial therapy and surgical deb
101 ing a cascade of events, resulting in severe fasciitis, destruction of tissues, and subsequent rheuma
102 two cases of fatal monomicrobial necrotizing fasciitis due to Acinetobacter baumannii, an unusual fin
103                                  Necrotising fasciitis due to invasive group A streptococcus (iGAS) i
104                                  Necrotizing fasciitis due to Vibrio vulnificus may result in overwhe
105             The pathogenesis of eosinophilic fasciitis (EF) and morphea is poorly understood.
106                     Importance: Eosinophilic fasciitis (EF) is a connective tissue disorder in which
107 om pharyngitis (strep throat) to necrotizing fasciitis (flesh-eating disease).
108 sease, including pharyngitis and necrotizing fasciitis (flesh-eating syndrome).
109  associated with decreased human necrotizing fasciitis ("flesh-eating disease").
110 ssue provides a clinical overview of plantar fasciitis focusing on prevention, diagnosis, treatment,
111 cent reports have cited cases of necrotising fasciitis following non-combat-related injuries or in th
112 sherman with rapidly progressive necrotizing fasciitis from Photobacterium (Vibrio) damsela infection
113                                  Necrotizing fasciitis has conventionally been associated with the st
114                    Historically, necrotising fasciitis has often, but not exclusively, been associate
115 ds to show how the management of necrotising fasciitis has progressed in parallel with prevailing sci
116 recapitulate key features of ABC and nodular fasciitis; however, the identity of USP6's relevant subs
117  occurred in 29 children (5.8%), necrotizing fasciitis in 12 children (2.4%); 10 children (2.0%) died
118 or ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnoses in 47 pati
119  we present a case of multifocal necrotizing fasciitis in a healthy adult patient, secondary to Haemo
120 port a case of Vibrio vulnificus necrotizing fasciitis in a patient with previously undiagnosed chron
121 an unusually severe case of MRSA necrotizing fasciitis in a previously undiagnosed AIDS patient.
122 r group A Streptococcus to cause necrotizing fasciitis in mice and nonhuman primates.
123 gh index of suspicion for fungal necrotizing fasciitis in the setting of wound infection and merits a
124 Current therapeutic regimens for necrotizing fasciitis include surgical debridement and treatment wit
125  Real-time sonoelastography can show plantar fasciitis, increase diagnostic performance of B-mode US,
126 egulates the AdcR regulon during necrotizing fasciitis infection in WT mice but not in S100a9 (-/-) m
127       We describe a rare case of necrotizing fasciitis involving Candida albicans, an organism that h
128  12.30; 95% CI, 4.14-52.70), and necrotizing fasciitis (IRR, 26.10; 95% CI, 5.14-475.00).
129                                  Necrotizing fasciitis is a life-threatening infection requiring urge
130 rome (strep TSS) with associated necrotizing fasciitis is a rapidly progressive process that kills 30
131                       Periocular necrotizing fasciitis is a rare but potentially devastating disease,
132                                  Necrotising fasciitis is a rare, but potentially fatal, soft-tissue
133  potential devastation caused by necrotizing fasciitis is also reviewed, since this diagnosis is easi
134 tinent to S. marcescens-mediated necrotizing fasciitis is also reviewed.
135                  Retroperitoneal necrotizing fasciitis is an uncommon soft tissue infection that is o
136  of magnetic insoles for the pain of plantar fasciitis is lacking.
137                                  Necrotizing fasciitis is often confused for cellulitis at initial pr
138 re suggests that survival of retroperitoneal fasciitis is possible with prompt debridement and antibi
139                                      Plantar fasciitis is the most common cause of heel pain in adult
140  fasciitis (NF), also known as pseudosarcoma fasciitis, is defined as a benign, self-limiting, vascul
141 e, epidemic toxic oil syndrome, eosinophilic fasciitis, localized forms of scleroderma, keloid, and t
142 nd Laboratory Risk Indicator for Necrotizing Fasciitis (LRINEC) score in diagnosis of necrotizing sof
143 s, Laboratory Risk Indicator for Necrotizing Fasciitis (LRINEC) score, imaging results, microbiology
144 een in these 5 cases, periocular necrotizing fasciitis may cause severe visual loss more often than p
145 icantly decreased virulence in a necrotizing fasciitis model of infection.
146 n in group A streptococcal (GAS) necrotizing fasciitis/myonecrosis often necessitates extensive debri
147  [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5]) were incl
148 l and intraoperative findings of necrotizing fasciitis, necrotizing myositis, or both.
149 ty in vivo and recapitulated the necrotizing fasciitis-negative phenotype of the DeltamtsR mutant str
150 th invasive infections including necrotizing fasciitis (NF) and express a potent DNase (Sda1).
151  toxic shock syndrome (STSS) and necrotizing fasciitis (NF) cases.
152                                  Necrotizing fasciitis (NF) caused by flesh-eating bacteria is associ
153     Shock frequently complicates necrotizing fasciitis (NF) caused by group A Streptococcus (GAS) or
154 report described a human case of necrotizing fasciitis (NF) caused by mixed infection with 4 Aeromona
155  toxic shock syndrome (STSS) and necrotizing fasciitis (NF) express numerous virulence factors, inclu
156                                  Necrotizing fasciitis (NF) is a destructive skin and soft tissue inf
157                                  Necrotizing fasciitis (NF) is a rapidly progressive, life-threatenin
158 r from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock syndrome (ST
159                                      Nodular fasciitis (NF), also known as pseudosarcoma fasciitis, i
160 spectrum of infections including necrotizing fasciitis (NF).
161 escribes mortality predictors of necrotizing fasciitis (NF).
162 sease, mastoiditis, myocarditis, necrotizing fasciitis, nontraumatic intracranial hemorrhage, orbital
163 on surgery, 2 patients developed necrotizing fasciitis of a lower extremity that required amputation,
164      We report the first case of necrotizing fasciitis of the chest wall due to infection with S. mar
165 re infections that can result in necrotizing fasciitis or even death in otherwise healthy adults outs
166 r from a wound in a patient with necrotizing fasciitis or streptococcal toxic shock syndrome between
167 sites or wounds in patients with necrotizing fasciitis or streptococcal toxic shock syndrome.
168 or from a wound in patients with necrotizing fasciitis or streptococcal toxic shock syndrome.
169 erosis was defined when cutaneous sclerosis, fasciitis, or joint contracture was first documented in
170 te recovered from a patient with necrotizing fasciitis, or with isogenic gene replacement mutants def
171  to address whether CLI improves necrotizing fasciitis outcome by modulating virulence factors of CLI
172 ouse infectious disease model of necrotizing fasciitis, PAD4(-/-) mice are more susceptible to bacter
173 nt species of bacteria can cause necrotising fasciitis, perhaps the most widely known is group A stre
174 ent of patients with periorbital necrotizing fasciitis (PONF) through an observational retrospective
175 ollected from a patient with GAS necrotizing fasciitis post-IVIG infusions markedly inhibited the mit
176                                              Fasciitis precedes and frequently accompanies muscle nec
177 perficial and deep fasciitis and necrotizing fasciitis, pyomyositis, infectious bursitis, infectious
178        Chromosomal translocations in nodular fasciitis result in USP6 overexpression, leading to tran
179  We report 5 cases of periocular necrotizing fasciitis resulting in severe vision loss, 3 of which re
180 usative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine protease SpeB.
181 eptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis with shock) w
182 empirical treatment of suspected necrotizing fasciitis should include antibiotics predictably active
183  and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%] vs 4 [12.9%
184  a murine model of streptococcal necrotizing fasciitis, the activated partial thromboplastin times we
185 ts of the clinical management of necrotising fasciitis; these treatment approaches all originate from
186  on admission than patients with necrotizing fasciitis, they have more chronic comorbidities and most
187    During the invasive infection necrotizing fasciitis, this is in part due to the GAS protease SpeB
188 h cut-off of > 4 mm as diagnostic of plantar fasciitis, this study had a sensitivity of 96%, specific
189                                              Fasciitis was observed in 18 of 30 biopsies (60%).
190  44 clinically diagnosed patients of plantar fasciitis were compared to 50 normal volunteers.
191              Sixty-two patients with plantar fasciitis were randomized to a placebo controlled trial
192 ients with pathologically proved necrotizing fasciitis were reviewed retrospectively for fascial thic
193 cluding toxic shock syndrome and necrotizing fasciitis, which are both associated with significant mo
194 cteria can result in devastating necrotizing fasciitis, which can progress to septicemia.
195 sions of linear scleroderma and eosinophilic fasciitis, which can result in considerable morbidity.
196 oon as possible to patients with necrotizing fasciitis, while our in vitro studies emphasize that a h
197 rly diagnosis of retroperitoneal necrotizing fasciitis, wide and repeated debridement, broad-spectrum
198 sembling both systemic sclerosis and diffuse fasciitis, with severe loss of motion and flexion contra
199 xic shock syndrome cases and one necrotizing fasciitis without shock, were treated with i.v. infusion

 
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