ライフサイエンスコーパス: fasting

1 leavage in response to PPARalpha agonism and fasting.

2 ess induced by the dietary transition during fasting.

3 fasting and 1 week after 4-week intermittent fasting.

4 d with the levels before 4-week intermittent fasting.

5 ne proteins 1 week after 4-week intermittent fasting.

6 ase to maintain low insulin secretion during fasting.

7 activation of glucocorticoid signaling upon fasting.

8 tains a high level of O-GlcNAcylation during fasting.

9 urodegeneration and resistance to death upon fasting.

10 due to neurodegeneration, and is lethal upon fasting.

11 n of large amounts of unprocessed foods, and fasting.

12 d with the levels before 4-week intermittent fasting.

13 ds, the major fuel used by beta-cells during fasting.

14 This study analysed fasting (8 h) blood samples from an obese, normoglycemic

15 ulation (7.7% vs. 3.6%, p<0.001), especially fasting acid exposure (6.0% vs.1.3%, p<0.001).

16 During fasting, activation of the nuclear receptor peroxisome p

17 VLCD yielded a pronounced increase in fasting acylcarnitine levels, whereas RYGB, both immedia

18 e end of 4th week during 4-week intermittent fasting and 1 week after 4-week intermittent fasting.

19 Moreover, both fasting and a fasting-mimicking diet prevent tamoxifen-i

20 broblast growth factor 21 (FGF21) after 24-h fasting and after COLD were highly correlated and associ

21 and SU+GLP-1, respectively, and 4) increased fasting and arginine-induced glucagon levels compared wi

22 th EBL, RQ decreased on average by 9% during fasting and by 4% during HFOF but increased by 4% during

23 reduced by 88 +/- 16% (n = 5) after 24 h of fasting and by 76 +/- 22% (n = 5) at 1 h after intraperi

24 hanges in mouse liver, both before and after fasting and during glucagon infusion.

25 Fasting and excess of allergen, but not NSAID and exerci

26 Serial MRI scans obtained after fasting and hourly for 4 h following meal ingestion were

27 ladaptive signaling by triggering a state of fasting and hypoxia mimicry, which includes activation o

28 l for the organismal survival upon prolonged fasting and maintaining systemic homeostasis under metab

29 16 represses hepatic lipid catabolism during fasting and may thus participate in the preservation of

30 feeding state, with reduced levels following fasting and normalized by refeeding.

31 Ten patients underwent fasting and post-prandial concurrent manometry and pH fo

32 Furthermore, higher fasting and postprandial amino acid removal were observe

33 Fasting and postprandial large, medium, and small TRLPs,

34 GWAS of fasting and postprandial serum TG at 150 minutes were pe

35 In a cross-sectional study, to compare fasting and postprandial triglyceride-rich lipoprotein p

36 bute to the paradoxical association of lower fasting and postprandial TRLP subfractions despite insul

37 ned as the change in 24-h RQ from EBL during fasting and standard overfeeding (STOF) (50% carbohydrat

38 carbohydrate, 30% fat) and then during 24-h fasting and three 200% overfeeding diets in a crossover

39 owed selective activation to food cues after fasting and to social cues after isolation; these respon

40 s known to increase FFA uptake in the heart (fasting) and BAT [cold exposure and injection with the b

41 Previous work found that aging, fasting, and immobilization promote skeletal muscle atro

42 7% with HIV infection, decreased by 28% with fasting, and increased by 49% with a high-fat meal.

43 t was scanned under standard diet, overnight fasting, and ketogenic diet conditions.

44 shes, carbohydrate loading, and avoidance of fasting are key components of successful colorectal ERAS

45 Fasting-associated changes in PKA signaling are attenuat

46 Collectively, our data suggest that fasting-associated ketosis and the ketogenic effects of

47 whole-room indirect calorimeter during 24-h fasting at thermoneutrality (24 degrees C) and during en

48 ted serum samples before 4-week intermittent fasting, at the end of 4th week during 4-week intermitte

49 After overnight fasting, BAT lacking ALK7 showed increased expression of

50 We also analyzed fasting blood for markers of inflammation, glucose and i

51 or body fat mass (BFM) with blood pressure, fasting blood glucose (FBG), and urinary kidney injury m

52 elafin levels are inversely correlated with fasting blood glucose and hemoglobin A1c levels in men w

53 s glucose tolerance, insulin sensitivity and fasting blood glucose in diet-induced obesity (DIO) and

54 ipocyte proliferation, hepatic inflammation, fasting blood glucose level, and glucose intolerance, co

55 pha2 subunits exhibited significantly higher fasting blood glucose levels and produced more glucose t

56 f ABA extract in the DIO model and increased fasting blood glucose levels.

57 ed gluconeogenesis, glucose intolerance, and fasting blood glucose levels.

58 systolic and diastolic blood pressure (BP), fasting blood glucose, glycated hemoglobin (HbA1c), trig

59 In T2D, FKBPL was negatively correlated with fasting blood glucose, HbA1c and diastolic blood pressur

60 econdary outcomes (including blood pressure, fasting blood glucose, lipids) were assessed after 4 yea

61 t being associated with liver enzymes or non-fasting blood glucose.

62 In the morning, a fasting blood sample was taken, and whole-blood platelet

63 ne was measured in a central laboratory from fasting blood samples and categorized as <=0.5 nmol/L, >

64 tiety hormonal profiles were determined from fasting blood samples collected prior to intervention.

65 Fasting blood samples were used to measure ornithine lev

66 nd methylmalonic acid (MMA) were assessed in fasting blood samples.

67 s had higher body mass index, transaminases, fasting blood sugar, triglyceride, low density lipoprote

68 Autophagy is activated by prolonged fasting but cannot overcome the ensuing hepatic lipid ov

69 eding strategy of high energy transfer while fasting, but we anticipate that mothers exhibiting a lac

70 , urinary albumin/creatinine ratio (ACR) and fasting C-peptide.

71 PIGR), and 1 week after 4-week intermittent fasting (CALU, CALR, IGFBP4, SEMA4B) compared with the l

72 e end of 4th week during 4-week intermittent fasting (CALU, INTS6, KIT, CROCC, PIGR), and 1 week afte

73 SRGN), and 1 week after 4-week intermittent fasting (CAMP, PLAC1) compared with the levels before 4-

74 on causes social craving, similar to the way fasting causes hunger.

75 dy, known cohorts with available measures of fasting circulating phylloquinone (vitamin K-1) and conf

76 trient and environmental adaptations such as fasting, cold, or exercise.

77 gy stress was additionally exacerbated under fasting conditions and by AMPK deficiency in hepatocytes

78 In non-fasting conditions, results showed that Scia-induced inh

79 ize this human phenotype identified by acute fasting conditions, we analyzed changes in 24 EE and sle

80 ed GSIS or prevented insulin secretion under fasting conditions.

81 ) active warm-up protocols; (3) intermittent fasting conditions; (4) warming-up while listening to mu

82 24-h energy expenditure (24 EE) during 24-h fasting defines a "thriftier" metabolic phenotype prone

83 ch remained relatively hyperleptinemic while fasting, did not exhibit fasting-induced reductions in t

84 Across the range of doses, fasting DNL was reduced by up to 90% (P = 0.003).

85 Elevated fasting esophageal acid exposure mediated symptoms.

86 1c) 5.5 +/- 0.2% [37 +/- 3 mmol/mol]) during fasting euglycemia followed by a 60-min +5.5 mmol/L hype

87 e expression during the normal physiological fasting-feeding cycle in nutrient-sensitive and -insensi

88 function exhibit circadian rhythmicity with fasting-feeding.

89 urage further exploration of the benefits of fasting/FMD in cancer therapy.

90 asting plasma insulin [FPI]), and adipocyte (fasting free fatty acid x FPI).

91 These findings suggest that intermittent fasting from dawn to sunset actively modulates the respe

92 Fasting from dawn to sunset for four weeks also induced

93 o test the anticancer effect of intermittent fasting from dawn to sunset in metabolic syndrome, we co

94 studies are needed to test the intermittent fasting from dawn to sunset in the prevention and treatm

95 s evaluating pre and post-procedure hormones fasting ghrelin, postprandial GLP-1, postprandial PYY, a

96 n, postprandial GLP-1, postprandial PYY, and fasting GIP levels were included.

97 nesses) and HbA1c z-scores with dysglycemia (fasting glucose >=6.1 mmol/L with 2-hour glucose >=7.8 m

98 g/mL) higher leptin and tended to have lower fasting glucose (-0.8 mmol/L; -1.8, 0.2 mmol/L, nonsigni

99 s in leptin (-0.7 ng/mL; -2.1, 0.8 ng/mL) or fasting glucose (0.2 mmol/L; -0.5, 0.9 mmol/L) in men ex

100 model identified the combination of cT1-AST-fasting glucose (cTAG) as far superior to any individual

101 of two traits for diabetes diagnosis, serum fasting glucose (FG) and glycated hemoglobin (HbA(1c)),

102 Women without DM who had fasting glucose (FG) and insulin (FI) data for >=2 visit

103 uding LDL cholesterol, triacylglycerol (TG), fasting glucose (FG), glycated hemoglobin (HbA1c), insul

104 I was associated with lower risk of elevated fasting glucose (HR: 0.80, 95% CI 0.70-0.92, P-trend = 0

105 s assessing pre-hypertension and an impaired fasting glucose (IFG) and their combined effects on the

106 IR was computed as fasting insulin (mIU/L) x fasting glucose (mmol/L)/22.5.

107 ng/mL), lack of outdoor exercise, increased fasting glucose and a family history of PCOS in at least

108 dence supporting causal associations between fasting glucose and cancer.

109 cid (UA) and diabetes-related traits such as fasting glucose and glycated hemoglobin.

110 th the M-value and inversely associated with fasting glucose and HbA(1c) (P < 0.05), whereas BRS was

111 ose of LES consumed, cointervention type, or fasting glucose and insulin levels.

112 rofile, but resulted in reductions in BP and fasting glucose concentration and in improvements in ins

113 ulin sensitivity increased at 3 and 6 mo and fasting glucose concentration declined at 6 mo (-2.67; 9

114 nsitivity (1/fasting insulin concentration), fasting glucose concentration, and lipid profile and to

115 e serially increased with an increase in the fasting glucose in a dose-dependent manner, but not with

116 erence, and 1.6% (95% CI: - 0.6, 3.8) higher fasting glucose in fully adjusted models.

117 the pathway between early-life nutrition and fasting glucose in women.

118 lucose and lipid metabolism, associated with fasting glucose level (P = 1.80 x 10(-8)).

119 PM2.5 exposure during trimester 2 increased fasting glucose level by 0.85% (95% CI: 0.41, 1.29).

120 ipoprotein (HDL) cholesterol level, impaired fasting glucose level, type 2 diabetes mellitus, hyperte

121 Supporting NHANES analyses showed that fasting glucose levels of obese adults were inversely re

122 low-density lipoprotein cholesterol levels, fasting glucose levels, and adiposity at 12 to 24 months

123 found stress to be associated with increased fasting glucose levels, especially among those who resid

124 iculum (ER) stress related gene expressions, fasting glucose levels, insulin sensitivity and restored

125 tors of insulin sensitivity, and measures of fasting glucose metabolism.

126 ts did not differ by type or dose of LES, or fasting glucose or insulin levels.

127 insulin sensitivity as well as with impaired fasting glucose production in Atp7b (-/-) mice.

128 bers in Southern Israel who had at least one fasting glucose test during an MO period and at least on

129 ancy BMI, each 1-mmol/L increase in maternal fasting glucose was associated with higher SD scores for

130 sulin was measured by chemiluminescence, and fasting glucose was measured with the enzymatic colorime

131 ults showed that levels of cT1, AST, GGT and fasting glucose were all good predictors of NAS >= 4 and

132 se tolerance) and/or fasting state (impaired fasting glucose) or by intermediate HbA(1c) levels.

133 esterol, HDL cholesterol, triglycerides, and fasting glucose) to dietary fat.

134 Maternal GDM, fasting glucose, 1-h, and 2-h glucose concentrations fol

135 enome-wide significant associations for T2D, fasting glucose, and fasting insulin, comprising 65, 43,

136 tatistically significant, for triglycerides, fasting glucose, and non-HDL cholesterol.

137 ressure, waist circumference, triglycerides, fasting glucose, and non-high-density lipoprotein (non-H

138 n analyzing the combination effect of BP and fasting glucose, cancer risks were serially increased wi

139 and triglycerides and 0.2 mmol/l higher non-fasting glucose, compared with mothers of AGA offspring.

140 Laboratory data including serum fasting glucose, haemoglobin A1c levels, creatinine leve

141 Glucose metabolism parameters including fasting glucose, insulin and homeostasis model of assess

142 ths-6.5 years, and ages 6.5-11.5 years) with fasting glucose, insulin, insulin resistance, beta-cell

143 ), but otherwise no changes were observed in fasting glucose, insulin, ketones, and renal function.

144 nergy expenditure, respiratory quotient, and fasting glucose, insulin, total and high-density lipopro

145 d physical activity on 1-year change in BMI, fasting glucose, triglycerides, and HDL cholesterol in i

146 worse triglycerides-, HDL-, blood pressure-, fasting glucose- and hemoglobin A1C values.

147 g T2DM in individuals with isolated impaired fasting glucose.

148 tions for individuals with isolated impaired fasting glucose.

149 roteins which have been previously linked to fasting glycaemic traits and insulin resistance in genom

150 that Lmna (LCS/LCS) animals normalize their fasting glycemia by both increasing insulin secretion an

151 a consequence of the progressive increase in fasting glycemia induced by insulin resistance in the pr

152 75 and liraglutide combined therapy improved fasting glycemia upon short-term treatment and a chronic

153 articipants can be ascribed to the increased fasting gut peptides.

154 Prolonged postoperative fasting has been the traditional model of care following

155 Intermittent fasting has grown in popularity as a weight loss strateg

156 impact health, while caloric restriction and fasting have putative benefits for disease prevention an

157 This group was characterized by fasting hyperglucagonemia, hyperaminoacidemia, and no lo

158 the propensity for diet-induced obesity and fasting hyperglycemia in adulthood.

159 olerance tests and ex vivo analyses revealed fasting hyperglycemia, glucose intolerance, reduced sens

160 markedly attenuated glucose utilization and fasting hyperglycemia.

161 ally exploited as an alternative way to halt fasting hyperinsulinemia and the progression of type 2 d

162 Fasting hyperinsulinemia precedes the development of typ

163 is primarily a hyperglycemic agent driven by fasting/hypoglycemia and highlight the recent advances t

164 terregulatory hormone to insulin, induced by fasting/hypoglycemia to raise blood glucose through acti

165 Intermittent fasting (IF) and Paleolithic (Paleo) diets produce weigh

166 (EI) and introducing periods of intermittent fasting (IF) exert important metabolic effects.

167 Intermittent fasting (IF) is a promising dietary intervention for all

168 ircuits, we tested if these genes respond to fasting in mice hypothalami, which highlighted the diffe

169 ponses and efficient metabolic adaptation to fasting in vivo.

170 Instead, p75NTR is required for fasting-induced activation of neurons within the arcuate

171 verall, our data indicate that physiological fasting-induced downregulation of GRK2 in the liver is k

172 Here, we report fasting-induced Fibroblast Growth Factor-21 (FGF21) sign

173 ation of lipid metabolism and contributes to fasting-induced free fatty acid mobilization.

174 represents the main mechanism implicated in fasting-induced GRK2 degradation in the liver in vivo.

175 the activin receptor ALK7 in BAT resulted in fasting-induced hypothermia due to exaggerated catabolic

176 nd found that nesfatin-1 fully abolishes the fasting-induced increase in the reward value of sucrose.

177 perleptinemic while fasting, did not exhibit fasting-induced reductions in temperature.

178 Here we show that entry into mouse torpor, a fasting-induced state with a greatly decreased metabolic

179 rons are required for the full expression of fasting-induced torpor in both female and male mice, the

180 d syndrome led to the discovery of the novel fasting-induced, glucogenic, and orexigenic hormone name

181 l, 0.06 mmol/L (-0.07 to 0.2), p = 0.37; and fasting insulin (log), -0.06 mU/L (-0.19 to 0.07), p = 0

182 HOMA-IR was computed as fasting insulin (mIU/L) x fasting glucose (mmol/L)/22.5.

183 Further, improvements in fasting insulin and insulin resistance have also been re

184 blood pressure (BP), insulin sensitivity (1/fasting insulin concentration), fasting glucose concentr

185 However, it is unclear whether fasting insulin hypersecretion is a primary driver of in

186 1.3 rs1333049 risk allele together with high fasting insulin levels benefitted from bariatric surgery

187 Genetically predicted fasting insulin levels were positively associated with c

188 henotype (e.g. higher waist-to-hip ratio and fasting insulin levels, but lower body fat).

189 Elevated levels of fasting insulin release and insufficient glucose-stimula

190 Fasting insulin was elevated in mice infused with pregna

191 t associations for T2D, fasting glucose, and fasting insulin, comprising 65, 43, and 13 single nucleo

192 ong with the previously identified predictor fasting insulin, modifies the preventive effect of baria

193 Secondary outcomes included fasting insulin-like growth factor 1, lipids, glucose, i

194 pulsive binge feeding separated by prolonged fasting intervals, increasing NAD-dependent deacetylase

195 Fasting is another physiological stress that elicits wel

196 Finally, the physiological role of CT-1 in fasting is confirmed by the impaired food restriction-in

197 eating is recommended, whereby intermittent fasting is done for 12 to 16 h each day.

198 Fasting large, medium, and very small TRLPs negatively c

199 ducts for 12 wk showed beneficial effects on fasting LDL cholesterol and endothelial function compare

200 The modified diet attenuated the rise in fasting LDL cholesterol observed with the control diet (

201 nsulin, and glucagon concentrations at their fasting level.

202 plasma glucose concentration clamped at the fasting level; and 3) repeat EGP measurement with inhibi

203 Both reflect the induction of a fasting-like and hypoxia-like transcriptional paradigm t

204 n mouse hepatocytes and obese mice elicits a fasting-like gene expression profile, improves glucose m

205 may be related to their ability to induce a fasting-like paradigm, which triggers the activation of

206 econd and third trimesters for evaluation on fasting lipids levels.

207 remely high rates while undergoing prolonged fasting, making lactation a tremendously energy demandin

208 Waist circumference and fasting metabolic laboratory parameters were also measur

209 rk Study 077 evaluated changes in weight and fasting metabolic parameters in HIV-uninfected individua

210 ween arms were found for change in weight or fasting metabolic parameters overall or for subgroups.

211 ice constitutively expressing adropin and in fasting mice treated acutely with adropin peptide.

212 our data indicate that the combination of a fasting-mimicking diet and vitamin C represents a promis

213 esults support further clinical studies of a fasting-mimicking diet as an adjuvant to oestrogen thera

214 cer receiving oestrogen therapy, cycles of a fasting-mimicking diet cause metabolic changes analogous

215 Moreover, both fasting and a fasting-mimicking diet prevent tamoxifen-induced endomet

216 The fasting-mimicking diet selectivity reverses vitamin C-in

217 Fasting-mimicking diets delay tumor progression and sens

218 distinctive metabolic manifestations of this fasting mimicry are enhanced gluconeogenesis and ketogen

219 HVs had significantly higher fasting motility indexes [106 +/- 13 arbitrary units (a.

220 glucagon in mice, demonstrating that neither fasting- nor SGLT2i-induced ketosis is altered by interr

221 We report here that fasting or calorie restriction protocols in C57BL6 mice

222 nutritional stress, such as during prolonged fasting or cold exposure, organisms need to balance the

223 enriched in liver and further increases with fasting or high-fat diet (HFD).

224 ipants (n = 40) experienced 10 h of mandated fasting or total social isolation.

225 is a hormone secreted by the stomach during fasting periods and acts through its receptor, the growt

226 gliflozin and LY2409021 individually lowered fasting PG compared with placebo, and the combination fu

227 acebo, and the combination further decreased fasting PG.

228 y and identified 134 metabolites in maternal fasting plasma at 26-28 weeks of gestation.

229 Furthermore, no significant differences in fasting plasma glucose (2.9%; 95% CI: -0.4, 6.3%; P = 0.

230 body mass index (BMI), blood pressure (BP), fasting plasma glucose (FPG), and type 2 diabetes (T2D).

231 coverage practices had 0.2 mmol/L lower mean fasting plasma glucose and 0.9% lower cardiovascular ris

232 creened for DM using glycated hemoglobin and fasting plasma glucose at TB treatment and after 3 month

233 t, excessive gestational weight gain, raised fasting plasma glucose during pregnancy, short breastfee

234 expression in WAT inversely correlated with fasting plasma glucose in both obese mice and humans.

235 ary risks, high systolic blood pressure, and fasting plasma glucose in ranked attributable proportion

236 ng laboratory glycated hemoglobin (HbA1c) or fasting plasma glucose in TB patients.

237 Fasting plasma glucose increased from 94 +/- 2 to 142 +/

238 The performance in prediction of fasting plasma glucose level was measured using 100 boot

239 od that was superior to clinical parameters (fasting plasma glucose, 2-hour plasma glucose).

240 ded lack of measurement of lipid components, fasting plasma glucose, and visceral fat, and there migh

241 mass index [BMI] as behavioral CVH metrics; fasting plasma glucose, total cholesterol, and blood pre

242 patic (basal endogenous glucose production x fasting plasma insulin [FPI]), and adipocyte (fasting fr

243 he microbiota was positively correlated with fasting plasma insulin.

244 The patients showed significantly greater fasting plasma levels of glucose and insulin and HOMA-IR

245 hol consumption interactions associated with fasting plasma lipid levels.

246 ing and Third Generation cohorts, who had 85 fasting plasma proteins measured using Luminex xMAP plat

247 The study utilized fasting plasma samples collected from a well-characteriz

248 Women provided non-fasting plasma samples during 3 prenatal study visits (a

249 iling covering >1000 lipids was performed on fasting plasma samples from participants 6-9 week postpa

250 Research in Genomic Epidemiology consortium, fasting plasma triglycerides and high- and low-density l

251 e end of 4th week during 4-week intermittent fasting (POLK, CD109, CAMP, NIFK, SRGN), and 1 week afte

252 GFBP-5) and 1 week after 4-week intermittent fasting (PRKCSH), and an anti-aging proteome response by

253 ce imaging after overnight cold exposure and fasting produced a significant increase in AlexaFFA upta

254 number of hypoglycemic events, the ratio of fasting proinsulin to C-peptide over time, and response

255 e found that the stimulation of autophagy on fasting protects Lgr5(+)ISC against DNA damage and cell

256 Short-term fasting protects tumor-bearing mice against the toxic ef

257 both groups, the lactating women exhibited a fasting rate of endogenous glucose production (EGP) that

258 We show here that suckling-weaning and fasting-refeeding transitions in rodents are associated

259 ed and refed mice, however the impact of the fasting-refeeding-cycle on pol III function has not been

260 o physiochemical stimuli, they are among the fasting replicating bacteria studied, capable of respond

261 Comparison of the fasting response in wild-type and IQGAP1-null mice revea

262 l transduction pathways that drive the liver fasting response.

263 or metabolic function linked to glucagon and fasting responses, but were not the canonical direct tra

264 ating insulin levels and liver expression of fasting-responsive cAMP-dependent protein kinase A (PKA)

265 miRNAs, using RNA sequencing technology, in fasting samples from the baseline visit (<100 days from

266 In conclusions, increased BP status or the fasting serum glucose level status were associated with

267 We included all fasting serum glucose tests taken between 2007-2014, of

268 ice in: body composition (lean or fat mass); fasting serum insulin; HbA1c; glucose dynamics during gl

269 Key safety endpoints were change in fasting serum lipid concentrations, the incidence of adv

270 The primary outcome was fasting serum trimethylamine-N-oxide (TMAO).

271 Metabolomic profiling of fasting serum was performed using a global, untargeted a

272 sed LC-MS to quantify SCFA concentrations in fasting serum, collected at baseline and the end of each

273 f the heart and BAT after exposure to CL and fasting showed a significant increase in AlexaFFA uptake

274 The decrease in fasting small-bowel motility noted in CD participants ca

275 Furthermore, the serum fasting sorbitol levels in patients were dramatically in

276 al state (impaired glucose tolerance) and/or fasting state (impaired fasting glucose) or by intermedi

277 idual reflux events in most patients (90% in fasting state and 40% post-prandial).

278 od samples were obtained during the day - at fasting state and before and after two standardized exer

279 ver metabolic responses were measured in the fasting state and during a two-step (10 and 20 mU/m(2)/m

280 The fasting state is associated with biochemical and behavio

281 Clinical biomarkers were assessed at fasting state, while untargeted lipidomics was undertake

282 mportance of standardized sampling times and fasting states when metabolite biomarkers are used.

283 analyses adjusted for gender, age, smoking, fasting status, and lipid-modifying medication were perf

284 xpression Omnibus data sets, we confirm that fasting suppresses liver adropin expression in lean C57B

285 rs for 24 h was associated with an increased fasting T1AC (>75% of values >90th centile of the normal

286 GWAS of fasting TG and postprandial serum TG at 150 minutes resu

287 to postprandial TG metabolism independent of fasting TG concentrations, resulting in smaller increase

288 variation of postprandial TG independent of fasting TG, we calculated the TG response at 150 minutes

289 predicted TG at 150 minutes as a function of fasting TG.

290 Greater decrease in 24 EE during fasting (thriftier phenotype) was associated with less i

291 The liver plays a key role during fasting to maintain energy homeostasis and euglycemia vi

292 loss of Bmal1 response to feeding abolished fasting-to-feeding metabolic fuel switch from fatty acid

293 observed in median changes from baseline in fasting total cholesterol (14 mg/dL vs 10 mg/dL; p=0.034

294 x 10(-14)) for each one-percent decrement in fasting triglyceride concentrations, i.e., h(2) +/- SE w

295 g full downregulation of hepatic GRK2 during fasting using adenovirus-driven overexpression of this k

296 e end of 4th week during 4-week intermittent fasting (VPS8, POLRMT, IGFBP-5) and 1 week after 4-week

297 onic pump inhibitors, exercise, alcohol, and fasting) were considered.

298 Examples include the idea that fasting when feverish ("starving a fever") can increase

299 on alters food intake during refeeding after fasting, whereas long-term leptin treatment reduces fat

300 umber of hours per day (usually 4-10 h), and fasting (with zero-calorie beverages) for the remaining