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1 n, mesenteric panniculitis, and encapsulated fat necrosis.
2 th acute abdominal pain diagnosed as primary fat necrosis.
3 raphy) concentrations were measured in human fat necrosis.
4 to properly resolve, resulting in extensive fat necrosis.
5 Both benign lesions represented areas of fat necrosis.
6 had no residual cancer; specimens contained fat necrosis.
7 A spectrum of US findings is associated with fat necrosis.
8 terized by acute inflammatory infiltrate and fat necrosis.
9 PNLIP, PNLIPRP2, and CEL were increased in fat necrosis.
10 ile acid concentrations higher than in human fat necrosis.
11 oleate because linoleic acid is increased in fat necrosis.
12 9%-15.80% vs 11.92%; 95% CI, 11.63%-12.21%), fat necrosis (8.26%; 95% CI, 7.27-9.38 vs 4.05%; 95% CI,
17 re reported in the xenon group: subcutaneous fat necrosis and transient desaturation during the MRI.
19 ic band (representing edema, hemorrhage, and fat necrosis), and hypoechoic areas suggestive of fibros
20 perforation, bowel ischemia, intraabdominal fat necrosis, and miscellaneous processes such as endome
21 consisting of pancreatic tissue destruction, fat necrosis, and systemic elevations in inflammatory re
25 , sclerosing adenosis, chronic inflammation, fat necrosis, fibrotic breast tissue, and scar tissue, w
27 struction (odds ratio = 1.48; P < 0.001) and fat necrosis in those with autologous reconstruction (od
34 contracture (five [12%] of 41 patients]) and fat necrosis (one [2%] patient) requiring surgical inter
36 of AP induction (serum amylase and lipase), fat necrosis, pancreatic necrosis, and multisystem organ
37 range 20-70 years) diagnosed with abdominal fat necrosis (primary omental infarct) on CT imaging bet
38 ancreatic lipases in SAP-associated visceral fat necrosis, the inflammatory response, local injury, a