ライフサイエンスコーパス: fatigability

1 for the influence of metabolic thresholds on fatigability.

2 ation complained of mild muscle weakness and fatigability.

3 the degree of which is referred to cognitive fatigability.

4 ability, which explains the patient's muscle fatigability.

5 ere NMJs dismantlement, muscle weakness, and fatigability.

6 ated EOMs did not show evidence of increased fatigability.

7 Treatment had no effect on muscle fatigability.

8 atients showed increased tendency for muscle fatigability.

9 extrafusal slow muscle fibres and increased fatigability.

10 % CI -0.40 to 0.48) and a moderate effect on fatigability (6 min walk test: ES 0.45, 95% CI -0.18 to

11 SUR2 loss-of-function causes fatigability and cardiac dysfunction in mice, and reduce

12 standing individual differences in cognitive fatigability and developing interventions for clinical c

13 wk of age, compounded with heightened muscle fatigability and exercise intolerance.

14 ntensity-duration relationship, and compared fatigability and recovery between sexes following interm

15 gest that NOR-1 expression may reduce muscle fatigability and that NOR-1 drives myoglobin expression

16 uing exercise protocol were used to quantify fatigability and the local muscle hemodynamic profile.

17 seizures, spasticity, short stature, muscle fatigability and weakness.

18 is an important mechanism underpinning motor fatigability and, potentially, also pathological fatigue

19 mal forces and showed only a 10% increase in fatigability, and no signs of oxidative damage or apopto

20 e-related loss in power output and increased fatigability are unresolved.

21 y and severe domains, with the mechanisms of fatigability assessed via non-invasive neurostimulation,

22 The muscular weakness and fatigability associated with myasthenia gravis are engen

23 osine triphosphate (ATP) levels and a higher fatigability at the neuromuscular junction during high e

24 ting for metabolic thresholds when comparing fatigability between sexes, whilst emphasising the notio

25 roximal muscles, without short-term clinical fatigability but with marked variation in strength over

26 women, but that the age-related increase in fatigability cannot be explained by an increased sensiti

27 n and women, but the age-related increase in fatigability cannot be explained by an increased sensiti

28 igh lean mass, 60% lower power and a greater fatigability compared to young adult muscles.

29 clinically unnecessary given the absence of fatigability, diurnal variation, or generalized weakness

30 h (Pt) and tetanic (Po) tensions, as well as fatigability during 5 secs of nerve stimulation at 50, 1

31 between males and females, the mechanisms of fatigability during CP-matched exercise above and below

32 between males and females, the mechanisms of fatigability during critical power-matched exercise are

33 n relationship during cycling, then assessed fatigability during critical power-matched exercise with

34 ata suggest that the age-related increase in fatigability during dynamic exercise has a bioenergetic

35 e mechanisms for the age-related increase in fatigability during dynamic exercise remain elusive.

36 s in muscle power output and the increase in fatigability during dynamic exercise remain elusive.

37 explanatory factor for the sex difference in fatigability during intermittent, isometric contractions

38 Motor fatigability emerges when demanding tasks are executed o

39 ation class II-III patients with EI and high fatigability exhibited significantly faster rates of exe

40 the first to test whether sex differences in fatigability exist when exercise intensity is normalised

41 These data show that a sex difference in fatigability exists even when exercise is matched for cr

42 (MVC); however, whether a sex difference in fatigability exists when exercise is prescribed relative

43 ative capacity compared with healthy and low-fatigability HF patients, suggesting that SM metabolism

44 ar function and reducing muscle weakness and fatigability in a dose-dependent manner.

45 , we report that improved motor function and fatigability in ambulatory type 3 SMA patients and mouse

46 fects in glycolytic metabolism and increased fatigability in dystrophic muscle may be caused in part

47 slowing') to study the central component of fatigability in healthy adults.

48 ies occur and contribute to EI and increased fatigability in HF patients with reduced or preserved ej

49 tal muscle RyR have been linked to increased fatigability in HF.

50 udy aimed to determine whether the increased fatigability in old adults during dynamic exercise is as

51 These data suggest that the increased fatigability in old adults during dynamic exercise is pr

52 pose of this study was to compare quadriceps fatigability in patients with varying severity of chroni

53 strates the potential to improve fatigue and fatigability in RRMS.

54 ) transport compromised and locomotor muscle fatigability is exacerbated with a combined net effect o

55 rEF and HFpEF patients with EI and increased fatigability manifest early, rapid exercise-induced decl

56 s, suggesting that the improvement in muscle fatigability might be due to a process of adaptation rat

57 Immobilization reduced Pt, Po, fatigability, muscle mass, and fiber cross-sectional are

58 ate decline than did HFrEF patients with low fatigability (New York Heart Association class I), despi

59 1) absolute muscular strength and power; (2) fatigability of limb muscles as a measure of relative pe

60 Mice have fatigability of limb muscles, electrophysiological evide

61 he loss of nNOS contributes to the increased fatigability of mdx mice, a model of DMD.

62 The increased fatigability of skalpha2(-/-) muscles is reproduced in c

63 Our aim was to determine the fatigability of thenar motor units paralysed chronically

64 vestigated whether similarities in diaphragm fatigability persist under acute hypoxic conditions.

65 The age-related increase in fatigability (reduction in mechanical power) of the knee

66 Fatigability (reductions in mechanical power) of the kne

67 An energetic plantar flexion exercise fatigability test and magnetic resonance spectroscopy we

68 chronic paralysis did not limit the range of fatigability typically found for thenar units, only its

69 Furthermore, quadriceps fatigability was evaluated in 12 patients by measuring u

70 situ after repeated stimulation showed that fatigability was improved in the treated TA.

71 Fatigue and fatigability were measured at three visits (weeks 0, 3 a

72 smission and metabolic diseases cause muscle fatigability, which is characterised by failure to susta