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1 lized growth zone (LoGZ) to regulate growth (feather buds).
2 esenchymal cells required to begin to form a feather bud.
3 pressed in the developing chick limb bud and feather bud.
4 ablished; the BDNF in dermis is localized to feather buds.
5 HH, NCAM, Tenascin-C) were characteristic of feather buds.
6 ectoderm results in the ectopic formation of feather buds.
7 nvolved in establishing the A-P asymmetry of feather buds.
8 o cartilage rudiments and then in developing feather buds.
9 (without feathers), FGFs can also induce new feather buds.
10 ion and a region with an increased number of feather buds.
11 ce variations in tissue mechanics to promote feather budding.
12 donor dermis induces host epidermis to form feather buds according to the spatial pattern and timeta
13 w along the dorsal midline, with rows of new feather buds added sequentially in a spreading wave.
14 s characteristic of the middle and posterior feather buds and suggest that P-D elongation of vertebra
16 ng parallel of molecular profiles in the A-P feather buds and the ventral-dorsal (V-D) Drosophila app
17 n the dermis and epidermis of the developing feather buds and their expression is induced in embryoni
21 ture allowed the sequential emergence of new feather buds at specific spatial locations relative to t
22 ional insights into pattern formation in the feather bud can be inferred from the effects of altered
24 e interfered with Shh signaling during early feather bud development and observed a dramatic change i
26 elopment and is expressed in early stages of feather bud development though its role has not been def
28 ic mechanism plays a primary role in hair or feather bud development, we are beginning to discover th
39 ts message is predominantly expressed in the feather bud epithelium, and the protein is enriched in t
45 in induction of Notch-1 and-2 and a loss of feather buds from the embryo in either large or small pa
46 he chick skin leads to both feather loss and feather bud fusions, suggesting that DLX proteins play a
48 J activity would enable the emergence of new feather buds if the local environment were competent and
51 rphogenetic protein (BMP) signaling from the feather bud inhibits bud formation in the adjacent inter
52 d tenascin, molecules that are important for feather bud initiation as well as bud outgrowth and morp
53 rstanding of morphogenetic processes such as feather bud initiation, spacing, orientation, elongation
56 rizing activity," localized in the posterior feather bud, is necessary and sufficient to mediate the
57 ted in the growth and differentiation of the feather buds, little is known about how the discrete pat
59 Delta-1 expressing cells differentiate into feather buds more quickly than normal and inhibit their
60 ceptor impair the epithelial contribution to feather bud morphogenesis, while the dermal contribution
61 inhibiting this transcription factor alters feather bud number and size in a stage-specific manner.
62 lant culture enables direct visualization of feather bud or scale formation and facilitate functional
64 he midline, FGFs led to fusion of developing feather buds, representing FGFs' ability to expand feath
66 -mesenchymal signaling interactions generate feather buds that are neatly arrayed in space and time.
68 ure system to interfere with EphA4 levels in feather buds using anti-sense oligonucleotides, demonstr
69 In feather tracts, short, wide, and curled feather buds with abnormal morphology and random orienta
70 In apteric and scale-producing regions, new feather buds with normal-appearing follicle sheaths, der