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1            At triage, 30.7% of patients were febrile, 17.3% had a respiratory rate greater than 24 br
2 irect effectiveness (surveillance study) was febrile acute respiratory illness in an unvaccinated hou
3 nfirmed influenza in a vaccinated child with febrile acute respiratory illness, analysed in the modif
4 ways at clinical presentation would identify febrile adults at risk of death.
5                                       Of 507 febrile adults, 32 died (6.3%) within 28 days of present
6 agement of shock, but increased mortality in febrile African children in the FEAST trial.
7 n those with DHF or dengue fever (DF) during febrile and critical phases.
8 disability with delayed speech, a history of febrile and/or non-febrile seizures, and a wide-based, s
9                       At presentation he was febrile at 40.1 degrees C but hemodynamically stable.
10 including aphthae, gastrointestinal disease, febrile attacks, and small-vessel vasculitis characteris
11  septic shock, 207 of 378 (55%; 50-60%) were febrile by history or measurement.
12                                       Of all febrile cases in the study, DENV-positive C cases accoun
13 nalyze changes by month to the proportion of febrile cases prescribed a blood culture compared with t
14 y passive case detection, with management of febrile cases.
15  from blood and 132 from fecal samples) from febrile children <5 years of age were studied.
16  investigated autoantibody (AAb) profiles in febrile children (<= 5 years) admitted to a hospital in
17 l 2018, data were prospectively collected on febrile children aged 0-18 years presenting to 12 EDs in
18 otic and broad-spectrum prescriptions in all febrile children are lacking.
19 ) to decide on antibiotic prescription among febrile children at risk of pneumonia has not been teste
20 e included EDs are not representative of all febrile children attending EDs in that country.
21 cterize dengue virus (DENV) infections among febrile children enrolled in a pediatric cohort study wh
22  was conducted to estimate the proportion of febrile children hospitalized at the study hospitals.
23                       Clinical management of febrile children in these settings is difficult given th
24  the e-POCT tool was non-inferior to that of febrile children managed by a validated electronic algor
25 to determine whether the clinical outcome of febrile children managed by the e-POCT tool was non-infe
26                           From 2008 to 2016, febrile children or children with a history of fever age
27 ppropriateness of antibiotic prescription in febrile children visiting EDs in Europe.
28 prescription rate of antibiotics is high for febrile children visiting the emergency department (ED),
29 arried out with plasma samples obtained from febrile children with confirmed bacterial infection (n =
30                      In the resuscitation of febrile children, albumin and saline boluses can cause r
31                                       In all febrile children, antibiotic prescriptions were appropri
32 inguishing bacterial from viral infection in febrile children, to facilitate effective clinical manag
33 ntibiotics and broad-spectrum antibiotics in febrile children.
34                               KD and matched febrile control sera did not demonstrate differences in
35 mperature, blood pressure, and heart rate in febrile critically ill patients.
36 s arthropod-borne viral pathogens that cause febrile disease in humans.
37 ratyphi A, is the leading cause of bacterial febrile disease in South Asia.
38 (CHIKV), a mosquito-borne alphavirus, causes febrile disease, muscle and joint pain, which can become
39  an emerging vector-borne pathogen causing a febrile disease.
40 oding human nucleoporin NUP214 causing acute febrile encephalopathy.
41        The primary outcome was time to first febrile episode or death from all causes within the firs
42 st 12 weeks of therapy significantly reduced febrile episodes and deaths compared with placebo withou
43 ity and age, altering the risk of developing febrile episodes and suggesting that host immunity plays
44                               95 (19%) first febrile episodes or deaths occurred in 489 patients in t
45 levated temperature conditions that simulate febrile episodes, especially at the beginning of the par
46 rations of HRP2 were substantially higher in febrile HRP2-positive patients than in afebrile HRP2-pos
47                           In addition, a new febrile hyperinflammatory Kawasaki-like syndrome (also k
48 y hospitalizations with acute respiratory or febrile illness (ARFI) and clinician-ordered real-time r
49 lizations for acute respiratory infection or febrile illness (ARFI) and influenza-associated ARFI amo
50 ies: healthy control, nonfebrile DENV, other febrile illness (OFI), and apparent DENV using multivari
51 T) are widely used to treat undifferentiated febrile illness (UFI).
52 tober 2017 met study criteria for confirmed (febrile illness and culture positivity or >=four-fold ri
53 V) is a mosquito-borne RNA virus that causes febrile illness and debilitating arthralgia in humans.
54 ungo virus infection presented with an acute febrile illness and died rapidly from massive hemorrhage
55 determinants of healthcare seeking for acute febrile illness and enteric fever risk in these communit
56 d by neurologic regression in the setting of febrile illness and other stressors.
57 riates were explored as risk factors for RSV febrile illness and RSV pneumonia or hospitalization.
58 help improve interpretation of the burden of febrile illness and shape policy on fever case managemen
59  >=four-fold rise in SAT titre) or probable (febrile illness and single SAT titre >=160) brucellosis.
60                 Kawasaki disease is an acute febrile illness and systemic vasculitis of unknown aetio
61                Pacific Coast tick fever is a febrile illness associated with the bite of Dermacentor
62 ikely to be taken to the study hospitals for febrile illness at all sites.
63                           Dengue is an acute febrile illness caused by dengue virus (DENV) and a majo
64 havirus that causes explosive epidemics of a febrile illness characterized by debilitating arthralgia
65 dardised surveillance of blood culture-based febrile illness in 13 African sentinel sites with previo
66 an genotype ZIKAV caused an outbreak of mild febrile illness in 2007 in Yap State, Federated States o
67 aria-endemic settings, accurate diagnosis of febrile illness in children is challenging.
68 s (CCHFV) is a tick-borne pathogen causing a febrile illness in humans, which can progress to hemorrh
69 of 250,1.2%), the agent of eschar-associated febrile illness in humans.
70 esistant infection among patients with acute febrile illness in India.
71 ion, we estimate incidence of RSV-associated febrile illness in the first 6 months of life and identi
72 sease are major causes of invasive bacterial febrile illness in the sampled locations, most commonly
73 7 plasma samples collected 2-12 months after febrile illness in Western and coastal Kenya (1993-2016)
74 ciated with infectious mononucleosis (IM), a febrile illness in which patients have high circulating
75 rizing healthcare-seeking patterns for acute febrile illness is critical for generating population-ba
76 it neurological regression in the setting of febrile illness or infection.
77 management of children with undifferentiated febrile illness outside of malaria are not well understo
78 tissues and initially causes a self-limiting febrile illness similar to dengue.
79          Mayaro virus (MAYV) causes an acute febrile illness similar to that produced by chikungunya
80 kungunya fever (CHIKF), a self-limited acute febrile illness that can progress to chronic arthralgic
81 ever is often self-limiting, presenting as a febrile illness that can result in atypical pneumonia.
82 hi A, frequently presents as a nonlocalizing febrile illness that is difficult to distinguish from ot
83 causes human monocytic ehrlichiosis (HME): a febrile illness that may progress to fatal sepsis with m
84              For most children, malaria is a febrile illness that resolves with time, but in ~1% of c
85 k, and surgical sites who had a diagnosis of febrile illness that was either suspected or blood cultu
86 ldren aged 2-59 months presenting with acute febrile illness to 9 outpatient clinics in Dar es Salaam
87 ic illness ranges from a mild, self-limiting febrile illness to one manifested by plasma leakage that
88 the PERFORM (Personalised Risk assessment in Febrile illness to Optimise Real-life Management across
89               Human disease ranges from mild febrile illness to severe fatal neurologic infection.
90 -Congo haemorrhagic fever (CCHF) is a severe febrile illness transmitted by Hyalomma ticks in endemic
91     Plasmodium falciparum infections lead to febrile illness unless the host has sufficient immunity,
92       History of tick bites and tick-related febrile illness were assessed as part of a case-control
93                          Subsequent peaks of febrile illness were found to incorporate smaller outbre
94                 Participants presenting with febrile illness were tested for virologically confirmed
95                 Participants presenting with febrile illness were tested for virologically confirmed
96 ease following VEEV infection manifests as a febrile illness with flu-like symptoms, which can progre
97 kungunya virus (CHIKV) infection is an acute febrile illness with polyarthralgia and arthritis.
98  Among 1524 patients hospitalized with acute febrile illness, 133 isolates were found among 115 patie
99 ptomatic dengue patients experience an acute febrile illness, 5-20% progress to severe infection asso
100        We evaluated spatial heterogeneity in febrile illness, care seeking, and enteric fever inciden
101  a blood culture compared with the burden of febrile illness, stratified by hospital.
102                Infection with WNV results in febrile illness, which can progress to severe neurologic
103 supportive treatment to dengue patients with febrile illness, which is difficult to diagnose clinical
104 15, for occurrence of acute undifferentiated febrile illness, with clinical and laboratory data avail
105 and Southeast Asia and a rare cause of acute febrile illness, Zika virus (ZIKAV) arose from obscurity
106 nfants and children who suffered episodes of febrile illness-induced neurodegeneration or cardiac fai
107 and cephalosporins) typically used for acute febrile illness.
108 upils, and diffuse anhidrosis 7 days after a febrile illness.
109 c use in primary care patients with an acute febrile illness.
110 ons about healthcare seeking in the event of febrile illness.
111 an arthropod-borne emerging pathogen causing febrile illness.
112  in one) neurological deterioration during a febrile illness.
113  Arboviral infections are frequent causes of febrile illness.
114 diomyopathy with lactic acidosis following a febrile illness.
115 VFV infection mainly include a self-limiting febrile illness.
116 othelial parameters between dengue and other febrile illness.
117 of the genus Flavivirus and can cause severe febrile illness.
118 to potential coinfection with a non-malarial febrile illness.
119  known to circulate for decades causing mild febrile illness.
120 chment area to characterize care seeking for febrile illness.
121  responsible for sporadic outbreaks of acute febrile illness.
122 0.85; 95% CI, 0.81 to 0.90), and nonmalarial febrile illnesses (1122 vs. 1424 episodes; incidence rat
123 matology can be easily confounded with other febrile illnesses (e.g., dengue fever and leptospirosis)
124 sts (RDTs) for common acute undifferentiated febrile illnesses are often used inappropriately.
125 milar adjustments may be indicated for other febrile illnesses for which QT-interval-prolonging medic
126            Using prevalence data of 5 common febrile illnesses from India and Cambodia, and performan
127 ial to transform clinical diagnosis of acute febrile illnesses in resource-limited settings.
128 reatment of patients with non-specific acute febrile illnesses is not considered.
129                                        Acute febrile illnesses such as dengue and chikungunya, which
130 mprove the clinical outcome of children with febrile illnesses while reducing antibiotic use through
131  for the diagnosis of acute undifferentiated febrile illnesses with presently available tests, which
132 e the disease into differential diagnosis of febrile illnesses with unknown etiology.
133 suspected enteric fever, controls with other febrile illnesses, and healthy controls in Dhaka, Bangla
134 gue and other pathogens that can cause acute febrile illnesses, the search for secondary pathogens wi
135  be infected with other co-circulating acute febrile illnesses.
136 TREM-1 at clinical presentation can identify febrile individuals at risk of all-cause febrile mortali
137 the course of medical care of 0- to 3-mo-old febrile infants (n = 913) and subsequently archived at -
138 mmonly used or optimal thresholds identified febrile infants 60 days or younger with IBIs with high a
139         Clinicians often risk stratify young febrile infants for invasive bacterial infections (IBIs)
140 in reaction-based testing for SARS-CoV-2 for febrile infants in an outbreak setting.
141 eported successful treatment of a child with febrile infection-related epilepsy syndrome (FIRES), a s
142                     Dengue fever is an acute febrile infectious disease caused by dengue virus (DENV)
143                Immunisation reduced maternal febrile influenza-like illness with an overall efficacy
144 l presentation and management of a cohort of febrile Kenyan children at five hospital/clinic sites fr
145                             Virus-associated febrile lower respiratory tract infections (fLRIs) durin
146 cMSP33D7 had a significantly reduced risk of febrile malaria (adjusted hazard ratio, 0.36 [95% confid
147 mia was associated with an increased risk of febrile malaria in children of all ages.
148 itemia was associated with a reduced risk of febrile malaria in older children (> 3 years), while in
149 ally distinct from those of individuals with febrile malaria in the transmission season, coinciding w
150 es previously shown to be protective against febrile malaria in this same cohort were significantly a
151 her probability of children not experiencing febrile malaria.
152 itemia in determining the risk of developing febrile malaria.
153 itemia in determining the risk of developing febrile malaria.
154 ver 11325 child-years of follow-up to detect febrile malaria.
155 thout conferring enhanced protection against febrile malaria.
156  .30-.73; P = .0008) with protection against febrile malaria.
157 ntigens were associated with protection from febrile malaria.
158  production in some individuals during acute febrile malaria.
159 ify febrile individuals at risk of all-cause febrile mortality.
160  25 [51%]), anaemia (25 [47%] and 24 [49%]), febrile neutropaenia (17 [32%] and 21 [43%]), and pneumo
161 a (51%), infections (24%), anemia (12%), and febrile neutropenia (10%).
162                    There were 29 episodes of febrile neutropenia (10%).
163 , were neutropenia (340 [15%] vs 328 [15%]), febrile neutropenia (112 [5%] vs 142 [6%]), and leucopen
164 rade 3 or worse were neutropenia (20 [30%]), febrile neutropenia (12 [18%]), and thrombocytopenia (si
165 sed neutrophil count (31 [12%] vs 27 [11%]), febrile neutropenia (14 [6%] vs 16 [6%]), diarrhoea (12
166 re than 28 days (32%), infections (20%), and febrile neutropenia (14%).
167 of 37 patients), neutropenia (15 [41%]), and febrile neutropenia (15 [41%]) in the pazopanib group, a
168  thrombocytopenia (30%), hypertension (15%), febrile neutropenia (15%), and lung infection (11%).
169  common grade 3 or 4 adverse events included febrile neutropenia (16 [41%] of 39), neutropenia (12 [3
170  8 [3%]), bone pain (16 [5%] vs 5 [2%]), and febrile neutropenia (16 [5%] vs 9 [3%]).
171 reatment-related serious adverse events were febrile neutropenia (18 patients [7%]), sepsis or septic
172 he most common grade 3-4 adverse events were febrile neutropenia (22 [66%]), bloodstream infections (
173 gher with ramucirumab than with placebo were febrile neutropenia (24 [9%] of 258 patients in the ramu
174 ll 285 serious adverse events recorded, were febrile neutropenia (27 [17%] of 155 serious adverse eve
175 utropenia (46%), thrombocytopenia (38%), and febrile neutropenia (29%).
176  adverse events (>=10% in either group) were febrile neutropenia (31 [42%] vs 28 [39%]), decreased wh
177 grade 3/4 toxicities were neutropenia (62%), febrile neutropenia (35%), and peripheral sensory neurop
178 ing infection (16.9% v 10.7%, respectively), febrile neutropenia (35.0% v 17.7%, respectively), mucos
179 onhematologic toxicities with regimen M were febrile neutropenia (39.2%) and infections (21.6%).
180  events, including incidence and severity of febrile neutropenia (41 [18%] patients in the A+CHP grou
181 ommon grade 3 or greater adverse events were febrile neutropenia (42%), thrombocytopenia (38%), and W
182  neutropenia (124 [60%] of 207 patients) and febrile neutropenia (48 [23%]), whereas in the placebo g
183 hematologic toxicities during induction were febrile neutropenia (55%) and sepsis (35%).
184  group), neutropenia (47 [15%] vs 92 [30%]), febrile neutropenia (57 [18%] vs 34 [11%]), leucopenia (
185 e 3/4 neutropenia (45.8% v 21.5%; P < .001), febrile neutropenia (6.3% v 3.7%; P = .019), and diarrhe
186 s irrespective of relation to treatment were febrile neutropenia (97 [39%] of 252), anaemia (61 [24%]
187 nts occurring in 5% or more of patients were febrile neutropenia (98 [39%] of 252; five related to tr
188 ), pneumonia (five [5%] and five [11%]), and febrile neutropenia (five [5%] and six [13%]).
189 ea (five [2%]) in the quizartinib group, and febrile neutropenia (five [5%]), sepsis or septic shock
190 r more patients) were pneumonia (five [6%]), febrile neutropenia (five [6%]), pulmonary embolism (thr
191 nt (IEAT) in oncohematological patients with febrile neutropenia (FN) and its impact on mortality.
192 y group compared with pneumonia (four [4%]), febrile neutropenia (four [4%]), anaemia (three [3%]), a
193  vs 28 [6%]; grade 4, 98 [20%] vs 77 [15%]), febrile neutropenia (grade 3, 52 [10%] vs 31 [6%]; grade
194 th grades 3 or 4 neutropenia (n=79, 47%) and febrile neutropenia (n=49, 29%).
195 naemia (11 [25%]), leukopenia (seven [16%]), febrile neutropenia (seven [16%]), and pneumonia (seven
196 openia (eight [16%]), anaemia (seven [14%]), febrile neutropenia (six [12%]), and leucopenia (six [12
197  patients), thrombocytopenia (14 [17%]), and febrile neutropenia (ten [12%]).
198 vents were increased lipase (three [7%]) and febrile neutropenia (three [7%]).
199  neutropenia (three [9%] of 35 patients) and febrile neutropenia (three [9%]) in the control group.
200 up (P = 0.07); events were related mainly to febrile neutropenia and infection.
201                                              Febrile neutropenia and sensory neuropathy incidences we
202                        Prophylaxis prevented febrile neutropenia and systemic infection.
203                      One patient experienced febrile neutropenia but was able to complete paclitaxel
204     An antibiotic cycling-based strategy for febrile neutropenia effectively reduced carbapenem use,
205 and the incidence of grade 4 neutropenia and febrile neutropenia favored pazopanib.
206 ed after paclitaxel only if patients had had febrile neutropenia in a prior cycle or at investigator
207 pulmonary fibrosis in group B and one due to febrile neutropenia in group C.
208 he most common grade 3-4 adverse events were febrile neutropenia in seven (14%) patients, decreased n
209                                              Febrile neutropenia occurred in 16% of cycles, and tumor
210 ignancy unit: monthly antibiotic cycling for febrile neutropenia that included cefepime (+/- metronid
211                            Treatment-related febrile neutropenia was reported in 10% of patients.
212                              Neutropenia and febrile neutropenia were key secondary end points.
213  11 (10%) patients, of which neutropenia and febrile neutropenia were the most common (five [5%] pati
214 11%] events), and anaemia and neutropenia or febrile neutropenia were the most frequent grade 3 or wo
215 event in the subcutaneous daratumumab group (febrile neutropenia) and four in the intravenous group (
216  A single serious adverse event (ie, grade 4 febrile neutropenia) was reported.
217 f 270 patients; nine [3%] vs no patients had febrile neutropenia), infections (86 [31%] vs 48 [18%]),
218 duced frequency and severity of neutropenia, febrile neutropenia, and infections.
219 imumab plus platinum-etoposide group (death, febrile neutropenia, and pulmonary embolism [n=2 each];
220 0%) included nausea, diarrhea, constipation, febrile neutropenia, fatigue, hypokalemia, decreased app
221                 Three serious adverse events-febrile neutropenia, intestinal perforation, and cholang
222 vents included one (3%) patient with grade 3 febrile neutropenia, one (3%) patient with grade 4 hyper
223 emia and neutropenia; 10 patients (9.3%) had febrile neutropenia.
224  most common noninfectious reactions include febrile nonhemolytic transfusion reactions, allergic tra
225                                       In 937 febrile, nonsevere neutropenia episodes, frequencies of
226 safely reduces unnecessary antibiotic use in febrile, nonseverely neutropenic pediatric patients with
227 s (>30 min); or with clusters of two or more febrile or afebrile seizures within a 24-h period were a
228 urrent prolonged (>10 min) febrile seizures; febrile or afebrile status epilepticus (>30 min); or wit
229        The association between the number of febrile outpatients and the proportion prescribed blood
230            Samples from 797 afebrile and 457 febrile outpatients from two provinces in Angola with kn
231                        UMT sensitivity among febrile patients (for whom the test was indicated) was 8
232 n southeast Asia, antibiotic prescription in febrile patients attending primary care is common, and a
233                                           In febrile patients attending primary care, testing for CRP
234 and identify risk factors for brucellosis in febrile patients from a pastoralist community of Tanzani
235 re defined by positive RDT and controls were febrile patients from the same clinic with a negative RD
236      In this case-control study, we enrolled febrile patients presenting to outpatient departments at
237  we searched for methanogens in the blood of febrile patients using specific tools.
238    Surveillance for arboviral diseases among febrile patients was performed at an emergency health un
239 ted as causing archaemia and endocarditis in febrile patients who are coinfected by bacteria.
240                    Malaria case counts among febrile patients within IRS areas was lower post- compar
241                                           In febrile patients, only a small proportion of infections
242 antigens did not differ between afebrile and febrile patients.
243 aria cases or sub-optimal case management of febrile patients.
244                        We report a series of febrile pediatric patients with acute heart failure pote
245         Data were prospectively collected on febrile pediatric patients with cancer with a central ve
246                                Management of febrile pediatric patients with cancer with an absolute
247 had been cultivated in MHB experienced short febrile periods (median, 3.2 days), limited weight loss
248 ad been cultivated in BHI experienced longer febrile periods (median, 5.5 days) and greater weight lo
249 for determination of malarial etiology among febrile persons.
250 e time-points, apparent already in the early febrile phase (1.29 vs 1.75; P = .012).
251                                              Febrile-range hyperthermia worsens and hypothermia mitig
252 ere judged to be related to vaccination (one febrile reaction and one anaphylaxis) and one possibly r
253 -triggered anaphylaxis, acute cardiovascular/febrile reactions).
254 inate between anaphylaxis and cardiovascular/febrile reactions, ROC curve analysis revealed a reasona
255 ic prior viral infections against subsequent febrile respiratory illness (FRI).
256                         We conclude that the febrile response is dependent on local release of PGE2 o
257                    These data imply that the febrile response is dependent on the local release of PG
258       Mammary treatment with LPS resulted in febrile response that was detectable in TN goats, but wa
259 hermoregulatory neurons, is critical for the febrile response to peripheral inflammation.
260 PGE2 level in the brain do not influence the febrile response.
261 ines and attenuated systemic circulatory and febrile responses, likely reflecting decreased systemic
262 ing for epilepsy type, sex, site, history of febrile seizure, and age at last observation.
263 tinct acute encephalopathy syndromes, simple febrile seizures (14), other seizures (16), acute ataxia
264 pilepsy was lower in patients with prolonged febrile seizures (14.3%, 6.3-29.4) and survivors of acut
265 ts from an epilepsy GWAS meta-analysis and a febrile seizures (FS) GWAS are significantly more enrich
266  a subset of children experiencing prolonged febrile seizures (FSs), the most common type of childhoo
267  was raised in individuals with a history of febrile seizures (hazard ratio [HR] 1.12, 95% CI 1.08-1.
268 tinct acute encephalopathy syndromes, simple febrile seizures (n = 14), other seizures (n = 16), acut
269 ased with a growing number of admissions for febrile seizures (p<0.0001) and with later onset of chil
270  were categorised according to occurrence of febrile seizures and epilepsy, before entering the follo
271 g of KCC2, found previously in patients with febrile seizures and epilepsy, has been demonstrated to
272 articipant (2.9%, 0.5-14.5) in the prolonged febrile seizures group developed temporal lobe epilepsy
273                                              Febrile seizures occurred after dose 1 of MMR vaccine at
274 included associations of SCN1A with "complex febrile seizures" (HP: 0011172; p = 2.1 x 10(-5)) and "f
275          43 148 individuals had a history of febrile seizures, 10 355 had epilepsy, and 1696 had both
276 ayed speech, a history of febrile and/or non-febrile seizures, and a wide-based, spastic, and/or stif
277 ry clinical discharge diagnoses (eg, sepsis, febrile seizures, dehydration, or other non-respiratory
278 drawal, age at onset of epilepsy, history of febrile seizures, number of seizures before remission, a
279                   Children with epilepsy and febrile seizures-with and without concomitant epilepsy-a
280 itive problems in individuals suffering long febrile seizures.
281 resenting with recurrent prolonged (>10 min) febrile seizures; febrile or afebrile status epilepticus
282 nce of megalencephaly and ventriculomegaly, (febrile) seizures and autism-like behaviour.
283 n patient from New Caledonia presenting with febrile splenomegaly,weight loss, life-threatening autoi
284 o three months apart and are under long-term febrile surveillance to detect dengue by serotype-specif
285 b) Mass Screen and Treat (MSAT) classifed by febrile, symptomatic infections, and (c) Mass Test and T
286 opheles mosquitoes and presents with generic febrile symptoms that are challenging to diagnose clinic
287 with arthritogenic alphaviruses results in a febrile syndrome characterized by debilitating musculosk
288 d culture-confirmed enteric fever from other febrile syndromes.
289                                          The febrile Th2 switch was IL4 independent, but a gamma-secr
290 Vs) since TRPV1/TRPV4 antagonism blocked the febrile Th2 switch, while TRPV1 agonists mediated a Th2
291 patient recuperation starting from the early febrile to the defervescence and convalescent stages of
292         Dividing UTIs into three categories; febrile upper UTI (acute pyelonephritis), lower UTI (cys
293                          A single episode of febrile UTI is often caused by a virulent Escherichia co
294 ations of atypical and recurrent episodes of febrile UTI should focus on urinary tract abnormalities,
295                                           No febrile UTI were recorded in the WOCA group vs. 9 (45.0%
296                                           No febrile UTIs were recorded in the WOCA group versus 9 (4
297                Secondary outcomes: number of febrile UTIs, number of hospitalisations, WOCA tolerance
298            Secondary outcomes were number of febrile UTIs, number of hospitalizations, WOCA tolerance
299 Forest disease (KFD) is a tick-borne, acute, febrile viral illness endemic in southern India.
300 reatment after a neutropenic patient becomes febrile, whether and how to modify the initial treatment

 
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