ライフサイエンスコーパス: fecal

1 However, fecal acetic acid concentration was the highest in RS pa

2 ctomy revealed different concentrations of 4 fecal and 5 plasma BAs (p <= 0.05).

3 lower-respiratory tract specimens as well as fecal and blood samples from 180 patients with confirmed

4 quencing and comparative genomic analyses of fecal and blood specimens from recipients of allogeneic

5 This study aimed to determine the fecal and mucosa-associated bacterial composition along

6 There is evidence from both fecal and mucosa-associated microbial studies that patie

7 An oxalate oxidase assay measured fecal and P(ox) concentrations.

8 0 mL differences were observed for composite fecal and sewage samples (n = 147) by the LinRegPCR appr

9 We collected fecal and urine samples from 80 patients with IBS (Rome

10 asally or intradermally, eliciting serum and fecal antibody responses against CfaE and LTB, as well a

11 rs and H. pylori infection was determined by fecal antigen test.

12 Intriguingly, inulin fortification reduced fecal ATNC (p = 0.03) and FeNO (p = 0.04) concentrations

13 These data suggest fluctuations in total fecal BA levels could provide the basis for the first pr

14 Changes in fecal bacteria were assessed by taxa-specific quantitati

15 purred a Bacteroides-dominated population of fecal bacteria.

16 They identified associations between fecal bacterial BA metabolism and specific microbiota, e

17 Fecal bacterial profile differed between IBS subtypes, w

18 et al. investigated BA metabolism, including fecal BAs, serum BAs, and FGF19, in patients and control

19 Excessive fecal bile acid (BA) loss causes symptoms in a large pro

20 Furthermore, fecal bile acids are reduced in pregnant Fxr(-/-) mice.

21 Body weights were measured, and fecal, blood, and liver tissues were collected and analy

22 iations of asthma with the measured level of fecal butyrate (OR = 0.28 (0.09-0.91), P = 0.034), bacte

23 , toxigenic culture, clinical diagnosis, and fecal calprotectin levels.

24 ort the use of inflammatory markers, such as fecal calprotectin to detect SIBO.

25 me immunoassay (EIA), toxigenic culture, and fecal calprotectin were performed.

26 s of S100A8-A9 in fecal samples (also called fecal calprotectin) from newborns and during infancy, an

27 rement of serum C-reactive protein (CRP) and fecal calprotectin.

28 ol-forming microbes have significantly lower fecal cholesterol levels and lower serum total cholester

29 This farming system drove surges in fecal coliform bacteria, disturbing amphibian skin bacte

30 The inactivation of indigenous fecal coliforms (FC) in SEW was also measured.

31 ellent potential for real field detection of fecal coliforms.

32 s) (aOR: 1.12; 95% CI: 1.04, 1.23), elevated fecal concentration of alpha-1 antitrypsin (aOR: 4.82; 9

33 evealed that inulin-enrichment increased the fecal concentration of short-chain fatty acids (SCFAs).

34 udy in mice, metabolomics revealed increased fecal concentrations of microbially derived propionate a

35 igated the relationships between groundwater fecal contamination and different environmental paramete

36 ify areas where groundwater is most prone to fecal contamination and prioritize monitoring activities

37 d dye tracer tests provide evidence of human fecal contamination in the private wells studied, sugges

38 Fecal contamination may be introduced at the water sourc

39 This study used data on fecal contamination of drinking water, food, soil, hands

40 with poor sanitation conditions that enable fecal contamination of water distribution systems.

41 tive E. coli to humans and livestock through fecal contamination of water, public areas and agricultu

42 To identify the level of human fecal contamination, E25 sterol congeners were measured

43 Fecal content of apparent total N-nitroso compounds (ATN

44 sectional study the Groningen Defecation and Fecal Continence questionnaire was completed by a repres

45 nsumption, nest scores, sucrose consumption, fecal corticosterone and blood for hematology were colle

46 Fecal DNA from 2 cohorts (1 Norwegian and 1 German), in

47 se questions, we performed 16S sequencing on fecal DNA samples from thirty-nine bighorn sheep across

48 We used mark-recapture analyses for fecal DNA sampling data, spatially-explicit capture-reca

49 Fecal DNA sampling was the most expensive, although this

50 Calprotectin was measured and fecal DNA was sequenced using shotgun metagenomics and a

51 re obtained from mothers and infants and the fecal DNA was sequenced.

52 xplained by overall reproductive stage or by fecal estrogen (fE) and progesterone (fP) concentrations

53 Total urinary and fecal excretion of catabolites accounted for <5% of each

54 d was higher (p < 0.001, p < 0.001), and the fecal excretion of deoxycholic (p < 0.03, p < 0.02) and

55 The ratio between urinary and fecal excretion was also altered in antibiotic treated a

56 IgA was found only in the vaginal washes and fecal extracts of p.o.-immunized animals.

57 Fecal fat content, fat absorption, chymotrypsin levels,

58 intake, comparable activity, and equivalent fecal fat.

59 rly adversity, adult social bonds, and adult fecal glucocorticoid hormone concentrations (a measure o

60 All 22 infants tested negative for fecal H. pylori at 15 days of age, but those born vagina

61 atients with AAs or CRC more accurately than fecal hemoglobin concentration alone.

62 C) of 0.93, compared with an AUC of 0.67 for fecal hemoglobin concentration alone.

63 Most importantly, younger age fecal IgA -abundance and -nAg reactivity of lupus-prone

64 Overall, these observations show that fecal IgA features, nuclear antigen reactivity particula

65 The fecal immunochemical test (FIT) is the second most commo

66 completion in adults with abnormal screening fecal immunochemical test (FIT) results.

67 We performed a prospective study of fecal immunochemical test (FIT) screening among African

68 ce, or workup due to positive results from a fecal immunochemical test or signs or symptoms of CRC, a

69 strategy in organized programs involves the fecal immunochemical test, which is limited by low sensi

70 Primary colonoscopy and fecal immunochemical testing (FIT) are considered first-

71 67) from patients with positive results from fecal immunochemical tests in a CRC screening program.

72 ohort study of Bangladeshi children, greater fecal immunoglobulin A, but not plasma immunoglobulin G,

73 obstructed defecation syndrome (ODS) in 40%, fecal incontinence (FI) in 22%, combination of ODS and F

74 ed prospectively from patients implanted for fecal incontinence (FI) in 7 French centers between Janu

75 ifferent pathways to children's ingestion of fecal indicator bacteria and if ingestion decreased with

76 icated good water quality with low levels of fecal indicator bacteria; however, the collection and an

77 os, the potential advantages of a crAssphage fecal indicator, and the potential influence of site-spe

78 ) is a tool to evaluate the use of candidate fecal indicators to signify a health risk from enteric p

79 ajor depressive disorder exhibited increased fecal interleukin 17A, SAA, and SFB levels.

80 In our model, the combination of fecal level of MIR421, MIR27a-3p, and hemoglobin identif

81 Fecal levels of butyrate and ammonia decreased during th

82 High fecal levels of S100 proteins, from 30 days to 1 year of

83 genomic sequencing and relationship to human fecal markers (HFMs; crAssphage, enterococci) and anthro

84 ntration of pathogen genes and host-specific fecal markers.

85 y the proximal colon, which encapsulates the fecal material including the microbiota, and a minor for

86 Nonarthropod invertebrate biomass and fecal material were also distinguishable from plastic, t

87 within terrestrial invertebrate biomass and fecal material.

88 Here we set out to describe fecal metabolite composition in healthy participants in

89 g TwinsUK cohort datasets consisting of 1116 fecal metabolites and 16s rRNA microbiome from 786 indiv

90 g were performed on feces, whereas urine and fecal metabolites were analyzed by gas chromatography an

91 acteria was correlated with the levels of 18 fecal metabolites, and levels of these metabolites diffe

92 Fecal metabolome analysis can be used to distinguish pat

93 Fecal metabolome profiles could discriminate patients wi

94 Various behaviors, gut microbiota, and fecal metabolome were assessed at 90 days of age.

95 nges in microbial function, reflected in the fecal metabolome, appear to be more precise indicators o

96 ncing, both stress and Test diet altered the fecal metabolome/microbiome.

97 /or dietary prebiotics (Test diet) alter the fecal metabolome; and explored if these changes were rel

98 ts have significant differences in urine and fecal metabolomes and fecal microbiome vs control indivi

99 Fecal metabolomes, but not microbiomes, could distinguis

100 ls; these were accompanied by differences in fecal metabolomes.

101 ediatory relationships between traits of the fecal metagenome, disease markers, and risk exposures.

102 Conclusion: Fecal microbial colonization from patients with cirrhosi

103 Fecal microbial community changes are associated with nu

104 Campylobacter infection, linear growth, and fecal microbial community features in a prospective birt

105 The fecal microbial composition was analyzed by Genetic Anal

106 on, both OS and RS patients exhibited higher fecal microbial diversity than NS controls.

107 After long-term PPI exposure, the fecal microbial profile was altered and showed similarit

108 leted and in germ-free mice with and without fecal microbial transfer, provided evidence for a role o

109 Fecal microbial transplant (FMT) after antibiotics impro

110 patients with high Candida responded best to fecal microbial transplant (FMT).

111 y of metabolites that can be transferred via fecal microbial transplant into mice is identified.

112 nation (probiotic and prebiotic) altered the fecal microbiome but did not reduce liver fat content or

113 We assessed fecal microbiome composition and function using shotgun

114 Changes in fecal microbiome composition and metabolites were associ

115 Fecal microbiome composition was evaluated by sequencing

116 glioma development and Temozolomide (TMZ) on fecal microbiome in mice and humans.

117 The fecal microbiome in patients with moderate alcoholic hep

118 f liver fibrosis, and the composition of the fecal microbiome in patients with NAFLD.

119 We analyzed the fecal microbiome of control, SL/vulnerable, and LL/resil

120 Similar to humans, the fecal microbiome of dogs may be useful in diagnosing dis

121 l study investigated the contribution of the fecal microbiome to influence host physiology in two Ind

122 ces of PN that were attenuated by concurrent fecal microbiome transplantation (FMT).

123 fferences in urine and fecal metabolomes and fecal microbiome vs control individuals, independent of

124 samples analyzed, the alpha-diversity of the fecal microbiome was unchanged among subjects after init

125 ected at the start and end of the study, the fecal microbiome were analyzed by 16S ribosomal DNA sequ

126 Moreover, intestinal FAs and fecal microbiome were analyzed.

127 n, whereas associations between TMAO and the fecal microbiome were assessed by permutational multivar

128 zyme-linked immunosorbent assay and analyzed fecal microbiomes by 16S sRNA gene sequencing.

129 nces in network connections between diet and fecal microbiomes compared with control individuals; the

130 ice compared with feces from wild-type mice; fecal microbiomes of mice given GDNF were similar to tho

131 In an analysis of serum metabolites and fecal microbiomes of patients hospitalized with cirrhosi

132 We profiled the fecal microbiomes of patients with and without IBS to id

133 depth impact gene-centric analysis of human fecal microbiomes when using DIAMOND, an alignment tool

134 S100-knockout mice had alterations in their fecal microbiomes, with higher abundance of Enterobacter

135 -day mortality and were also associated with fecal microbiomes.

136 The aim is to investigate fecal microbiota and metabolites at different ages in in

137 n child health and might modulate changes in fecal microbiota and metabolites.

138 We characterized changes in the fecal microbiota and metabolome to identify the mechanis

139 We simultaneously measured the fecal microbiota and multiple environmental and host-rel

140 to 12 mo alters the 16S configuration of the fecal microbiota as read out by amplicon sequence varian

141 We did not find significant differences in fecal microbiota composition among patients with differe

142 Fecal microbiota composition and inferred function, feca

143 Differences in fecal microbiota composition between groups were analyze

144 Analysis of fecal microbiota composition by 16S ribosomal RNA gene s

145 One year after surgery, the fecal microbiota composition differed between CD patient

146 The role of the fecal microbiota composition for the postoperative disea

147 The fecal microbiota composition of CD patients, analyzed by

148 Analysis of fecal microbiota composition showed an increase in bacte

149 e adjusted for confounding factors, we found fecal microbiota composition to be associated with devel

150 In this study, we investigated if the fecal microbiota composition, determined by a high throu

151 ns in energy balance, blood metabolomics and fecal microbiota composition.

152 ht to investigate whether transplantation of fecal microbiota from drug-free patients with schizophre

153 The fecal microbiota from mothers and their babies differed

154 The results revealed that transplantation of fecal microbiota from schizophrenic patients into antibi

155 TMOP were transmissible by transplanting the fecal microbiota from TMOP-treated mice, indicating that

156 of wheat and ATIs on severity of colitis and fecal microbiota in mice.

157 es from the mice transplanted with patients' fecal microbiota increased both kynurenic acid synthesis

158 to colonize GF mice shows a direct effect of fecal microbiota independent of active liver inflammatio

159 In conclusion, the structure of the infant fecal microbiota is affected by the maternal H. pylori s

160 s support the notion that disruptions to the fecal microbiota may help explain the observed effects o

161 sequencing, plasma/urine metabolomes and the fecal microbiota of Angus steers grazing non-toxic or E+

162 pyridoxamine did not produce alterations in fecal microbiota of mice.

163 reduced Clostridia abundance distinguish the fecal microbiota of pre-IBD patients from IBS patients.

164 E+ grazing affected the fecal microbiota profile; +THI conditions modulated the

165 AN fecal microbiota showed a compositional predominance of

166 We applied our strategy to a fecal microbiota transplant (FMT) donor stool using mult

167 Fecal microbiota transplant (FMT) reverses the course of

168 mall intestinal cell shedding, whereas adult fecal microbiota transplant alone had no effect.

169 rial to assess the safety and feasibility of fecal microbiota transplantation (FMT) and reinduction o

170 We investigated the safety of weekly oral fecal microbiota transplantation (FMT) capsules from hea

171 e CDI, fidaxomicin for first recurrence, and fecal microbiota transplantation (FMT) for subsequent re

172 Our understanding and utilization of fecal microbiota transplantation (FMT) has jump-started

173 se and replacement of the microbiota through fecal microbiota transplantation (FMT) is a promising ap

174 Fecal microbiota transplantation (FMT) is used commonly

175 We tested the effect of fecal microbiota transplantation (FMT) on obesity, and p

176 Fecal microbiota transplantation (FMT) targeting gut mic

177 e analyze data from two published studies of fecal microbiota transplantation (FMT), finding that hig

178 el diseases (IBDs), where clinical trials of fecal microbiota transplantation have shown some efficac

179 Taken together these results suggest that fecal microbiota transplantation may be a treatment opti

180 responses by treatment with either a healthy fecal microbiota transplantation or defined commensal ba

181 mother-infant pairs and patients undergoing fecal microbiota transplantation to evaluate the pattern

182 such as probiotics, prebiotics, antibiotics, fecal microbiota transplantation, and gene manipulation

183 ow that p-crAssphage can be acquired through fecal microbiota transplantation.

184 representation of these 14 duodenal taxa in fecal microbiota was significantly different from that i

185 Diversity of fecal microbiota, assessed by Shannon index, did not dif

186 We carried out 16S-based profiling of fecal microbiota, assessed the expression of bile acid m

187 lonized with the patients' and the controls' fecal microbiota, highlighting 78 differentially enriche

188 itivity to colitis and skewed composition of fecal microbiota.

189 ice with colitis, via TLR4, and alters their fecal microbiota.

190 Candida was the most abundant genus in the fecal mycobiota of the two alcohol groups, whereas genus

191 availability) influence the ratios at which fecal nutrients are supplied to plants.

192 either undergoing an immunological test for fecal occult blood or colonoscopy.

193 olorectal tests was defined by a record of a fecal occult blood test in the past 2 years, flexible si

194 re less likely than nonphysicians to undergo fecal occult blood testing and were more likely to under

195 ing volatile associated with both floral and fecal odors-by a set of 36 tested odorants.

196 es that compared intestinal microbiota (from fecal or colonic or ileal tissue samples) among patients

197 y outcome was difference in specific taxa in fecal or intestinal tissue samples from patients with IB

198 on infection that is transmitted through the fecal-oral route, shed in the stool of infected individu

199 ose contact and generally occurs through the fecal-oral route.

200 preventable viral disease transmitted by the fecal-oral route.

201 Rotavirus, a diarrheal pathogen spread via fecal-oral transmission, is typically characterized by a

202 ing was negative, raising the possibility of fecal-oral transmission.

203 stocystis sp. to patients was assessed on 16 fecal patient samples, pre- and post-FMT, by PCR and sub

204 elerated disease and reduced bacteria in the fecal pellets.

205 Fecal pollution at beaches can pose a health risk to rec

206 ated with corresponding reductions in bovine fecal prevalence of ciprofloxacin-resistant E. coli.

207 owever, functional analysis showed increased fecal proteolytic and elastase activity before UC onset.

208 years; mean weight loss, 8.3 kg) provided a fecal sample that was processed into aFMT by frozen, opa

209 A fecal sample was provided by 319 children aged 5 y.

210 We studied levels of S100A8-A9 in fecal samples (also called fecal calprotectin) from newb

211 Fecal samples (n = 234) were collected over time during

212 lysis were measured in an independent set of fecal samples (n = 767) from patients with positive resu

213 ioperative changes in microbial diversity of fecal samples and the skin.

214 (abundance) of strains in serially collected fecal samples and their transcriptional responses to dif

215 A low level of S100 proteins in infants' fecal samples associated with development of sepsis and

216 yses were used to identify bacterial taxa in fecal samples at ages 6, 12, 18, and 24 months (N = 928)

217 We collected fecal samples at key stages of EAE progression and quant

218 into the recording strain in human clinical fecal samples can be extensive and is driven by differen

219 e quantified by sequencing 16S rRNA genes in fecal samples collected at 6, 12, 18, and 24 months.

220 ost-vaccine introduction, rotavirus positive fecal samples collected between 2011 and 2016 from child

221 en had Campylobacter present in asymptomatic fecal samples during the follow-up period.

222 develop gram-negative infection), with 4,768 fecal samples for analysis.

223 We obtained fecal samples from 13 children with new-onset CD after 6

224 ed the microbial taxonomic composition of 91 fecal samples from 15 females (n = 16 cycling, n = 36 pr

225 We cultured bacteria from serially collected fecal samples from a healthy infant; 34 sequenced strain

226 d the global metabolome of 231 plasma and 97 fecal samples from a large cohort of children with ASD a

227 e-transcription polymerase chain reaction in fecal samples from a subset of patients included in the

228 A8-A9 during the first 3 months of life than fecal samples from adults; levels decreased to adult lev

229 dition, we obtained 21 plasma samples and 27 fecal samples from age-matched healthy children living i

230 studies reported 16S rRNA gene sequences of fecal samples from HIV+ patients.

231 We observed dysbiosis in fecal samples from Hol(Tg/Tg) mice compared with feces f

232 Fecal samples from infants born by cesarean delivery had

233 iling, shotgun metagenomics and SCFAs in 153 fecal samples from non-kidney stone (NS) controls, patie

234 before administration of donor material than fecal samples from nonresponders (P = .04) and distinct

235 IR27a-3p were confirmed to be upregulated in fecal samples from patients with advanced neoplasms.

236 lusion, intestinal viral taxa are altered in fecal samples from patients with AH and associated with

237 Fecal samples from responders had higher diversity of mi

238 Fecal samples from term infants, but not preterm infants

239 ctrometric analysis of carbonyl compounds in fecal samples identified signatures associated with mice

240 ne the microbial composition and function in fecal samples obtained from a cohort of healthy individu

241 16S V3 and V4 gene sequencing results from fecal samples of patients tested positive for C. diffici

242 Analysis of fecal samples revealed that HFD-fed mice exhibited a red

243 tgun metagenomes of DNA extracted from human fecal samples sequenced using the Illumina platform shou

244 sis of the V4 region of the 16S rRNA gene in fecal samples shows maternal carriage of Prevotella copr

245 of a stabilization device to preserve canine fecal samples under various storage conditions simulatin

246 Fecal samples were collected at 3 (n = 16, 12, and 14, r

247 Fecal samples were collected at enrollment and at 7, 28,

248 Fecal samples were collected at the start and end of the

249 Serial fecal samples were collected from premature infants with

250 Fecal samples were collected longitudinally and analyzed

251 Fecal samples were evaluated for bacterial composition p

252 Virus-like particles from fecal samples were fractionated using differential filtr

253 Fecal samples were obtained from healthy subjects as wel

254 A total of 161 pre-diet fecal samples were sequenced from a discovery cohort (n

255 An additional 157 fecal samples were sequenced from the discovery cohort a

256 Fecal samples were serially collected and evaluated via

257 unannotated small protein families from five fecal samples, more than doubling the number of small pr

258 ces of bacteriomes and viromes from the same fecal samples, the host bacteria-phage associations are

259 16S ribosomal RNA sequencing of a series of fecal samples, we found that genetic predisposition to p

260 uring 48-h anaerobic incubation with healthy fecal samples.

261 ommunity and applied to five different human fecal samples.

262 - 4.2 compared with 11 +/- 1.9 umol/g) in AN fecal samples.

263 Bacterial strains were isolated from fecal samples.

264 al transcribed spacer amplicon sequencing of fecal samples.

265 Independent of breastfeeding status at fecal sampling, infant antibiotic exposure or other cova

266 icrobiota composition and inferred function, fecal SCFA concentration, gastrointestinal (GI) symptoms

267 complemented with targeted quantification of fecal SCFAs, bile acids, and functional microbial genes.

268 on, pancreatic fat, intramyocellular lipids, fecal SCFAs, blood pressure, or 24-h heart rate variabil

269 Using an oral route of inoculation, and fecal shedding as a marker for GI colonization, we showe

270 positively associated with increased cattle fecal shedding of ciprofloxacin-resistant E. coli.

271 djunctive cholestyramine therapy reduced the fecal shedding of daptomycin-resistant E. faecium by up

272 The median duration of fecal shedding was 28 days (95% CI, 19-28).

273 Fecal short chain fatty acids (SCFAs) were measured usin

274 explanation for increased hepatic IR injury, fecal short-chain fatty acids butyrate and propionate le

275 toms delivered offspring who exhibited lower fecal sIgA concentrations especially in later infancy.

276 Very low fecal sIgA concentrations were more common in infants of

277 n and stress symptom trajectories and infant fecal sIgA concentrations.

278 e of infection as measured by Kato-Katz (KK) fecal smears.

279 d that HFD-fed mice exhibited a reduction in fecal species richness, and that TCS further diminished

280 C-9 P45 showed reduced rotavirus shedding in fecal specimens and did not induce diarrhea compared to

281 Fecal specimens were collected from recipients of allo-H

282 vaccine candidates that prevent disease and fecal spread of enteric CoVs, including SARS-CoV-2.

283 Fecal ST131 and FQREC isolates resembled contemporaneous

284 necessity to investigate the ecotoxicity of fecal sterols in mammals, and consequent implications fo

285 oronavirus recovery (bovine coronavirus) and fecal strength (pepper mild mottle virus) controls.

286 Microbiome samples from fecal swabs were obtained at weaning (Wean), at mid-test

287 hartic preparations for CT colonography with fecal tagging can improve patient comfort but may result

288 y compared with conventional CT at different fecal tagging levels in vitro.

289 nography phantom, especially with suboptimal fecal tagging.

290 Assays for these miRNAs might be added to fecal tests for the detection of CRC or AAs.

291 cut-off value of 4 U/ml, the sensitivity of fecal tM2-PK test was 100% and the specificity was 68%,

292 Fecal transfer approach and administration of oleic acid

293 ogical studies and studies of the effects of fecal transfer in germ-free mice.

294 We performed fecal transplant gavage 3 days after middle cerebral art

295 ter stroke, aged stroke mice receiving young fecal transplant gavage had less behavioral impairment,

296 tabolomics analysis demonstrating that young fecal transplants contained much higher SCFA levels and

297 A2M 8 +/- 3 mm compared with CON 15 +/- 3mm; fecal urgency: A2M 4 +/- 1 compared with CON 10 +/- 3 mm

298 ion by 16S ribosomal RNA gene sequencing and fecal/urinary metabolites by 1H-NMR spectroscopy was com

299 AN participants had lower quantities of fecal/urinary metabolites than RA participants and metab

300 In a study of fecal viromes from patients with NAFLD and control indiv