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1 r humanized (ex-GF mice colonized with human fecal microbiota).
2 protocol for 16S ribosomal RNA sequencing of fecal microbiota.
3 ffects of co-habitation on skin than oral or fecal microbiota.
4 itivity to colitis and skewed composition of fecal microbiota.
5 ative PCR to monitor and quantify changes in fecal microbiota.
6 ing clinical challenge mostly originating in fecal microbiota.
7 agents that relatively spare the indigenous fecal microbiota.
8 gi, with mixed effects on rumen bacteria and fecal microbiota.
9 ice with colitis, via TLR4, and alters their fecal microbiota.
10 ficant alterations in the composition of the fecal microbiota.
11 is associated with differences in the human fecal microbiota.
12 es and in fecal preparations of mixed murine fecal microbiota.
13 an expansion of the genus Allobaculum in the fecal microbiota.
16 th MaR, but not GaM, significantly decreased fecal microbiota abundance and diversity, and expanded t
17 assic approaches, we found no differences in fecal microbiota abundance or composition between patien
18 s and pectinase modulated the composition of fecal microbiota after in vitro fermentation, increasing
24 levels of tryptophan and composition of the fecal microbiota, analyzed by 16S ribosomal DNA amplicon
26 amily member 3 alpha gene (REG3A) alters the fecal microbiota and affects development of colitis in m
33 te temporal changes in the concentrations of fecal microbiota and short-chain fatty acids (SCFAs) in
34 dietary protein increased the density of the fecal microbiota and the severity of colitis in SPF mice
35 to 12 mo alters the 16S configuration of the fecal microbiota as read out by amplicon sequence varian
39 crolimus, and mycophenolate mofetil modified fecal microbiota at the family level in each experimenta
41 crobial responses were tested using in vitro fecal microbiota batch fermentations of five donors.
42 n was associated with the composition of the fecal microbiota, but not with constipation or colonic t
45 iltrates from donor stool (FFT), rather than fecal microbiota, can be sufficient to restore normal st
46 results in reduced IL-18 levels and altered fecal microbiota characterized by expanded representatio
48 oos and hosted a strikingly low diversity of fecal microbiota compared to individuals born in natural
49 We did not find significant differences in fecal microbiota composition among patients with differe
52 g 16S ribosomal gene sequencing, we assessed fecal microbiota composition and diversity, as measured
55 rked differences in the small intestinal and fecal microbiota composition between mice housed on clea
62 ed the impact of initial alpha-diversity and fecal microbiota composition on beta-diversity instabili
64 e adjusted for confounding factors, we found fecal microbiota composition to be associated with devel
66 ces in asthma characteristics, food diaries, fecal microbiota composition, and levels of serum inflam
67 .Fecal and serum menaquinone concentrations, fecal microbiota composition, and plasma and fecal cytok
69 lly, delta-infected GSH exhibited an altered fecal microbiota composition, marked by increased inflam
71 ssed nutrient intake, food-related behavior, fecal microbiota composition, microbial fermentation, an
72 eral and adipose tissue insulin sensitivity, fecal microbiota composition, plasma and fecal SCFA, ene
75 ndry and metagenomics to show that the human fecal microbiota consists largely of taxa and predicted
76 from healthy donors or were given autologous fecal microbiota (control); each transplant was administ
78 and B-diversity of the 16S configuration of fecal microbiota did not differ between the 2 groups.
79 ently, here we demonstrate that the phyla of fecal microbiota differ substantially between L1-KO mice
82 here has been growing interest in the use of fecal microbiota for the treatment of patients with chro
86 tics and subsequently transplanted them with fecal microbiota from Casp1(-/-) mice based on a cohousi
87 ht to investigate whether transplantation of fecal microbiota from drug-free patients with schizophre
88 aised under standard conditions; exposure of fecal microbiota from L2-IL1B mice fed the HFD to L2-IL1
91 ecal microbiota from the mice which received fecal microbiota from patients with constipation also up
95 The results revealed that transplantation of fecal microbiota from schizophrenic patients into antibi
98 TMOP were transmissible by transplanting the fecal microbiota from TMOP-treated mice, indicating that
100 from different studies of the Western adult fecal microbiota generally clustered by study, and the 1
102 lonized with the patients' and the controls' fecal microbiota, highlighting 78 differentially enriche
103 015), and gamma-proteobacteria domination of fecal microbiota (HR, 2.64; 95% CI, 1.10-5.65; P = 0.031
105 This study compared the structure of the fecal microbiota in 29 mestizo children aged 7-18 years,
106 provide novel insights into the role of the fecal microbiota in cardio-metabolic disease with clear
110 his study, we determine the structure of the fecal microbiota in mothers and neonates according to ma
112 Anti-TNF therapy shifted the diversity of fecal microbiota in patients with IBD, but not with rheu
115 ubstituting antibiotics, this study assessed fecal microbiota in pigs from different dietary treatmen
116 icrobiota are altered in MS by comparing the fecal microbiota in relapsing remitting MS (RRMS) (n = 3
117 one prebiotic formula on the same inoculated fecal microbiota in two frequently used batch systems: p
118 es from the mice transplanted with patients' fecal microbiota increased both kynurenic acid synthesis
119 to colonize GF mice shows a direct effect of fecal microbiota independent of active liver inflammatio
120 In conclusion, the structure of the infant fecal microbiota is affected by the maternal H. pylori s
122 s support the notion that disruptions to the fecal microbiota may help explain the observed effects o
123 ntries, which correlates with alterations in fecal microbiota (microflora) and widespread use of anti
125 emerging under ciprofloxacin pressure in the fecal microbiota, no proof of selection of quinolone-res
127 A similar shift in the composition of the fecal microbiota occurred after a few months in TG mice
128 iple antimicrobial resistance genes), in the fecal microbiota of 147 mothers and their children sampl
130 acterize bacterial strain composition in the fecal microbiota of 37 U.S. adults sampled for up to 5 y
131 rtium of bacterial strains cultured from the fecal microbiota of a 6-month-old stunted Malawian infan
132 nce during the first 3 wk of life within the fecal microbiota of an infant born at 28-wk gestation.
133 sequencing, plasma/urine metabolomes and the fecal microbiota of Angus steers grazing non-toxic or E+
136 ly, these data suggest that abundance in the fecal microbiota of facultative anaerobes, such as S. Ty
139 in (150 mg 4 times daily for 10 days) on the fecal microbiota of healthy humans for a period of 1 yea
140 al 16S ribosomal RNA gene sequences from the fecal microbiota of humans and 59 other mammalian specie
143 Here we analyzed circadian rhythms in the fecal microbiota of mice using deep sequencing, and foun
147 reduced Clostridia abundance distinguish the fecal microbiota of pre-IBD patients from IBS patients.
148 lic consequences and specific effects on the fecal microbiota of protein and zinc deficiency were pro
157 ne pre-treatment rectal gene expression, and fecal microbiota profiles, in 206 pediatric UC patients
159 significantly disturbed composition whereas fecal microbiota recovered a similar composition to cont
160 on deficiency altered the composition of the fecal microbiota, reduced mucosal barrier function, and
162 roach begins by identifying uncultured human fecal microbiota samples that transmit immune phenotypes
168 gnotobiotic mice colonized with a synthetic fecal microbiota, StrainR2 resolves strain abundances wi
169 NA-based analysis of the T-bet(-/-)Rag2(-/-) fecal microbiota suggest that the structure of the endog
171 impact on the viability and diversity of the fecal microbiota that is recovered in the laboratory.
172 systematic compositional differences in the fecal microbiota that occurred with age and between West
174 scuss how probiotics, prebiotics, synbiotic, fecal microbiota transfer, polyphenols, specific diets,
178 We aimed to investigate the outcomes of fecal microbiota transplant (FMT) for relapsing CDI usin
180 pose of this article is to review the use of fecal microbiota transplant (FMT) in the treatment of pe
186 ulate various microbial communities, such as fecal microbiota transplant and bioreactor systems' opti
187 ehavioral symptoms in children with ASD, and fecal microbiota transplant could transform the dysbioti
192 wel cleanse, a stomach-acid suppressant, and fecal microbiota transplant, and observed significant im
193 orientations of some invertons diverge after fecal microbiota transplant, potentially as a result of
195 cacy and safety of diet-modulated autologous fecal microbiota transplantation (aFMT) for treatment of
196 We intend to review the most recent data on fecal microbiota transplantation (FMT) and critically di
197 rial to assess the safety and feasibility of fecal microbiota transplantation (FMT) and reinduction o
198 cal evaluation of the safety and efficacy of fecal microbiota transplantation (FMT) as a potential th
200 current Clostridium difficile infection with fecal microbiota transplantation (FMT) at a tertiary ref
202 We investigated the safety of weekly oral fecal microbiota transplantation (FMT) capsules from hea
207 fficile infection (CDI) guidelines recommend fecal microbiota transplantation (FMT) for persons with
208 se recovery is apparent given the success of fecal microbiota transplantation (FMT) for recurrent CDI
209 e CDI, fidaxomicin for first recurrence, and fecal microbiota transplantation (FMT) for subsequent re
210 ral case series have reported the effects of fecal microbiota transplantation (FMT) for ulcerative co
211 tudies suggest that weight may be altered by fecal microbiota transplantation (FMT) from a lean donor
218 cerative colitis (UC) patients responsive to fecal microbiota transplantation (FMT) have reduced phag
219 round: To date, evidence for the efficacy of fecal microbiota transplantation (FMT) in recurrent Clos
223 se and replacement of the microbiota through fecal microbiota transplantation (FMT) is a promising ap
235 difficile infections (rCDI) are treated with fecal microbiota transplantation (FMT) provided by healt
237 expression is regulated by the microbiota as fecal microbiota transplantation (FMT) rescues antibioti
239 the reported promising results of open-label fecal microbiota transplantation (FMT) therapy in patien
241 rs of antibiotic-resistant microbes and that fecal microbiota transplantation (FMT) would reduce the
243 roaches to modifying this ecosystem, such as fecal microbiota transplantation (FMT), are being develo
244 e analyze data from two published studies of fecal microbiota transplantation (FMT), finding that hig
245 difficile infections (rCDI) are treated with fecal microbiota transplantation (FMT), using feces prov
248 n, 15.4% vs vancomycin, 25.3%; P = .005) and fecal microbiota transplantation (response rates of 83%-
249 most current and best methods for performing fecal microbiota transplantation and summarize clinical
250 es, with recent clinical trials highlighting fecal microbiota transplantation as a strategy to enhanc
251 a diverse intestinal microbial population by fecal microbiota transplantation attenuates disease and
252 developed and higher quality data to support fecal microbiota transplantation for treating recurrent
253 udy reported higher than 80% success rate of fecal microbiota transplantation for treatment of recurr
254 ready been applied to the clinic, such as in fecal microbiota transplantation for treatment of recurr
255 el diseases (IBDs), where clinical trials of fecal microbiota transplantation have shown some efficac
256 ive therapies like probiotics, curcumin, and fecal microbiota transplantation in the management of mi
262 Taken together these results suggest that fecal microbiota transplantation may be a treatment opti
264 robes using nonabsorbable antibiotics and by fecal microbiota transplantation of dysbiotic cecal cont
265 responses by treatment with either a healthy fecal microbiota transplantation or defined commensal ba
266 mother-infant pairs and patients undergoing fecal microbiota transplantation to evaluate the pattern
269 such as probiotics, prebiotics, antibiotics, fecal microbiota transplantation, and gene manipulation
270 unities for treatment of other diseases with fecal microbiota transplantation, based on findings from
285 ygen species (ROS) in bacterial cultures and fecal microbiota using 2',7'-dichlorofluorescein diaceta
290 enters, the diversity of early pretransplant fecal microbiota was lower in patients than in healthy c
291 representation of these 14 duodenal taxa in fecal microbiota was significantly different from that i
299 w that the abundances of Escherichia coli in fecal microbiota were significantly higher in young chil
300 lthy donors and those who received their own fecal microbiota, which may be due to limited numbers.