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1 than women without knee OA (P < 0.04 at the femoral neck).
2 at the lumbar spine (P = 0.038), but not the femoral neck.
3 cartilage of patients with a fracture of the femoral neck.
4 oflavones, except for a modest effect at the femoral neck.
5 to 3.7 percentage points; P < 0.001) at the femoral neck.
6 0.7% (CI, -1.5% to 0.01%; P = 0.081) at the femoral neck.
7 on for impending pathologic fractures of the femoral neck.
8 nd 1.6% (CI, 0.4% to 2.8%; P = 0.008) at the femoral neck.
9 eatment groups had an increase in BMD at the femoral neck.
10 lder age groups, especially at the spine and femoral neck.
11 not a major feature of normal ageing of the femoral neck.
12 lacement for OA or following fracture to the femoral neck.
13 d not experience significant declines at the femoral neck.
14 Bone loss began by the midtwenties at the femoral neck.
15 over the age of 50 have osteoporosis of the femoral neck.
16 cular eccentricity and stresses in the human femoral neck.
17 d with increased bone mineral density of the femoral neck.
18 rence in standardized BMD of the ipsilateral femoral neck.
19 s associated with increased bone loss at the femoral neck.
20 ivity level, and bone mineral density at the femoral neck.
21 esses anterior, posterior, and medial to the femoral neck.
22 core in the lumbosacral spine, total hip, or femoral neck.
23 Similar trends were observed in the hip and femoral neck.
24 S) for impending pathologic fractures of the femoral neck.
25 multiple radial images along the axis of the femoral neck.
26 rochanter (0.026 +/- 0.006 g/cm2, P <0.001), femoral neck (0.022 +/- 0.006 g/cm2, P <0.001), total hi
28 time at multiple anatomical sites, including femoral neck (-0.08%/year per one interquartile increase
29 itamin D and placebo groups were as follows: femoral neck, +0.50+/-4.80 and -0.70+/-5.03 percent, res
30 for age and sex (lumbar spine, +0.7 +/- 1.6; femoral neck, -0.1 +/- 1.1; total hip, 0.0 +/- 1.1).
31 have increased (lumbar spine, -0.2 +/- 1.6; femoral neck, -0.6 +/- 1; total hip, -0.6 +/- 1.1; match
32 (mean Z scores: lumbar spine, -0.4 +/- 1.6; femoral neck, -0.7 +/- 1.1; total hip, -0.7 +/- 1.1).
33 difference, -0.08, 95% CI -0.13 to -0.02 for femoral neck; -0.09, 95% CI -0.15 to -0.03 for lumbar sp
34 placebo at the lumbar spine (5.4% vs 1.1 %), femoral neck (1.6% vs -1.2%), femoral trochanter (3.3% v
35 fidence interval, -2.18 to -1.01; P < 0.001; femoral neck, -1.20%; 95% confidence interval, -1.69 to
38 at the spine (3.96%; 95% CI: 4.86%, 3.06%), femoral neck (2.39%; 95% CI: 3.61%, 1.17%), total hip (1
39 the lumbar spine (17 +/- 3%), total hip and femoral neck (24 +/- 3% and 20 +/- 4%, respectively).
40 ad, 8.51-8.73 GPa vs 9.32-9.67 GPa; P = .04; femoral neck, 3.11-3.72 GPa vs 4.39-4.82 GPa; P = .04; W
42 e estrogen had BMD increases of 2.6% for the femoral neck; 3.6%, total hip; 2.8%, spine; and 1.2%, to
43 o 0.04 (P < 0.00001; 95% CI: 0.02, 0.05) and femoral neck (4 RCTs, n = 524) to 0.03 (P < 0.05; 95% CI
44 erve transection to a level posterior to the femoral neck (8-27 cm) depending on the length of the st
45 al, 8.1 to 12.4 percent), 5.4 percent at the femoral neck (95 percent confidence interval, 3.5 to 7.4
46 sociated with fracture risk independently of femoral neck aBMD and the Fracture Risk Assessment Tool
50 bular dysplasia, pistol grip deformity, wide femoral neck, altered femoral neck-shaft angle, appear t
53 0 or less at the lumbar spine, total hip, or femoral neck and -3.5 or more at each of the three sites
54 with daily cola intake was 3.7% lower at the femoral neck and 5.4% lower at Ward's area than of those
56 X-ray absorptiometry at the lumbar spine and femoral neck and by peripheral quantitative computed tom
57 evere CAL was associated with low BMD of the femoral neck and deleterious clinical dental parameters
58 was no association between bone loss at the femoral neck and fractures regardless of bisphosphonate
59 onide was associated with loss of BMD at the femoral neck and lumbar spine after 3 years of treatment
60 Consistent results were seen for change in femoral neck and lumbar spine BMD and across a range of
63 of -0.29 (-0.44 to -0.15, p < 0.001) at the femoral neck and of -0.25(-0.45 to -0.05, p = 0.015) at
64 reases bone mineral density in the spine and femoral neck and reduces risk of vertebral fracture.
65 E involves displacement between the proximal femoral neck and the femoral head at the level of the op
66 cant increase of bone mineral density at the femoral neck and the trochanteric region (13% and 15%, r
67 isone therapy, bone mineral densities of the femoral neck and the Ward triangle did not increase sign
69 cent change in bone mineral densities at the femoral neck and total hip at 24 months and at all three
71 ge or older, with normal BMD (T score at the femoral neck and total hip, -1.00 or higher) or osteopen
73 uted the incidence rates of fractures of the femoral neck and trochanteric region of the proximal fem
74 atients underwent baseline BMD measurements (femoral neck and/or lumbar spine) using dual x-ray absor
75 0(-12) for lumbar spine and p=1.9x10(-4) for femoral neck) and an increased risk of both osteoporotic
76 0(-10) for lumbar spine and p=3.3x10(-8) for femoral neck) and increased risk of osteoporosis (OR 1.2
77 t the lumbar spine, 2.5+/-0.4 percent at the femoral neck, and 2.0+/-0.2 percent for the total body (
80 es in bone mineral density at the total hip, femoral neck, and distal third of the radius at all time
85 ation between stimulant use and total femur, femoral neck, and lumbar spine bone mineral content (BMC
86 (average T scores and Z scores) of the hip, femoral neck, and lumbar spine of IgE-CMA patients were
87 absorptiometric examinations of the forearm, femoral neck, and lumbar spine were performed by 11 tech
90 nd bone mineral density of the lumbar spine, femoral neck, and proximal radius were measured by dual
92 also observed at the total hip, total body, femoral neck, and the predominantly cortical one-third r
93 ture had lower aBMD at the spine, total hip, femoral neck, and the ultradistal radius, the last havin
94 ptiometry, was assessed at the lumbar spine, femoral neck, and total femur (grams per square centimet
95 reater decreases in BMD at the lumbar spine, femoral neck, and total hip from the end of year 1 (mean
96 ineral density of the total body, total hip, femoral neck, and trabecular bone of the lumbar spine al
97 l density from baseline at the lumbar spine, femoral neck, and trochanter by 1% to 4% and in the tota
100 in bone mineral density at the total hip and femoral neck, as well as transitory increases in bone-fo
101 ents at the level of the greater trochanter, femoral neck, base of the femoral neck, and level of the
104 For bone mineral density T scores at the femoral neck, biomechanical CT analysis was highly corre
105 ; 95% CI, 0.94-0.94 g/cm2; P = .03) and mean femoral neck BMC (4.34 g; 95% CI, 4.13-4.57 g vs 4.59 g;
106 13.38 g; 95% CI, 13.25-13.51 g; P = .03) and femoral neck BMD (0.87 g/cm2; 95% CI, 0.74-0.83 g/cm2 vs
107 intakes were associated with maintenance of femoral neck BMD (FN-BMD) in men (dark fish + tuna, dark
109 ink/d of alcohol had a significantly greater femoral neck BMD (P = 0.008) and lumbar spine BMD (P = 0
110 The beneficial effect of current HT use on femoral neck BMD appeared to be greater in women with hi
112 the hypothesis that a major QTL controlling femoral neck BMD is located on chromosome 1p36.2-p36.3,
113 PD patients had lower hip, lumbar spine and femoral neck BMD levels compared with healthy controls;
118 d hazard ratios (HRs) for 1-unit decrease in femoral neck BMD T score in women with DM were 1.88 (95%
120 multipoint LOD score of 3.53 for linkage of femoral neck BMD to a quantitative trait locus (QTL) loc
121 for the A593-T598-C620 haplotype (n=85) had femoral neck BMD values 5.7% lower than those who did no
123 t of women in the lowest tertile (p = 0.02); femoral neck BMD was 12% greater in the highest versus t
126 , supplement-induced increases in spinal and femoral neck BMD were lost within 2 y of supplement disc
128 letal and sexual maturity, anthropometry and femoral neck BMD Z-score to control confounding effects.
130 dary endpoints comprised the dose effects on femoral neck BMD, falls, circulating calciotropic hormon
131 s total hip bone mineral density (BMD), with femoral neck BMD, lumbar spine BMD, and lumbar spine tra
140 to vegetable protein intake have more rapid femoral neck bone loss and a greater risk of hip fractur
143 ry, we compared lumbar spine, total hip, and femoral neck bone mineral density (BMD) in 581 HIV-posit
145 strong relation (r = 0.71) between increased femoral neck bone mineral density and increased serum 25
148 nd associated with a significant increase in femoral neck bone mineral density; vascular calcificatio
150 xifene increased bone mineral density in the femoral neck by 2.1 % (60 mg) and 2.4% (120 mg) and in t
151 various sites, bone density measured at the femoral neck by dual-energy x-ray absorptiometry is the
152 was associated with reduced bone loss in the femoral neck compared with controls (mean +/- SD -0.29+/
153 ws thinning of the underloaded superolateral femoral neck cortex arises from the failure of walking t
154 range of non-destructive metrics to measure femoral neck cortical bone stiffness at the millimetre l
155 ty, as measured by micro-CT, correlated with femoral neck cortical bone's elastic modulus and ultimat
157 lcitriol group), and the mean density at the femoral neck decreased by 6.2 percent (P=0.001 for compa
161 ral density change at both the spine and the femoral neck, even after accounting for prolactin levels
163 sis at baseline at the lumbar spine (LS) and femoral neck (FN) was 17.6% and 7.2%, respectively.
165 tiles and mean BMD at the lumbar spine (LS), femoral neck (FN), femoral trochanter (FT), and femoral
166 0%; n = 5) but no effect on total hip (TH), femoral neck (FN), or total body BMD or bone biomarkers.
167 mposition and whole-body, lumbar spine (LS), femoral neck (FN), trochanter, and Ward's triangle (WT)
169 populations, we built assessment models for femoral neck (FN)-fracture prediction and BMD value pred
172 bone mineral density at total femur (TFBMD), femoral neck (FNBMD), lumbar spine (LSBMD), and physicia
173 ndom-effects models for the lumbar spine and femoral neck for all studies providing isoflavones as ag
174 .2%, -1.2%) of a z score was observed at the femoral neck for each unit increase in BMD z score at ba
176 on in hip arthroplasty, especially following femoral neck fracture in the elderly, associated with su
177 50 years of age or older and had a displaced femoral neck fracture to undergo either total hip arthro
179 s and from lesion-free control subjects with femoral neck fracture was assessed by measuring malondia
180 CS for consolidation of impending pathologic femoral neck fracture with a mean follow-up of 533 days
181 ables discrimination of patients at risk for femoral neck fracture, and our results show the potentia
182 isplaced femoral neck fracture, nondisplaced femoral neck fracture, intertrochanteric fracture, previ
183 ng boxes and classified as normal, displaced femoral neck fracture, nondisplaced femoral neck fractur
187 h there was a trend toward a reduced risk of femoral neck fractures in subjects with severe radiograp
188 the hemiarthroplasty treatment of displaced femoral neck fractures in the absence of contraindicatio
189 s of diabetes mellitus, vertebral fractures, femoral neck fractures, and hip fractures were 2-5 times
190 d with cartilage obtained from patients with femoral neck fractures, the expression of both miR-140-5
191 pendently ambulating patients with displaced femoral neck fractures, the incidence of secondary proce
194 % in women with baseline osteoporosis at the femoral neck (>2.5 SDs below the normal young adult mean
195 trend for higher annual losses of BMD at the femoral neck; however, within the raloxifene group, lowe
196 significant preservation of bone loss at the femoral neck (HR 1.56, 95% CI 1.21-2.06, P=0.0007).
197 detected signs of impingement on the distal femoral neck (IDFN) in 18 of the 20 patients with SSI (9
201 ion reduced bone loss from the total hip and femoral neck in those who consumed <1.5 servings of dair
204 cement therapy, the rate of bone loss at the femoral neck increased with blood pressure at baseline.
208 s were associated with lower mean BMD at the femoral neck [lowest-to-highest tertiles (95% CI): 0.934
210 Fluid adjacent to the entire length of the femoral neck, measuring at least 5 mm in width, is compa
211 consortium for lumbar spine (n = 31,800) and femoral neck (n = 32,961) BMD, and from the arcOGEN cons
212 indings, including capsular adhesions at the femoral neck, obliteration of the paralabral sulcus, lab
213 mography the distribution of bone in the mid-femoral neck of 77 proximal femurs from people who died
215 neral density T score of -2.5 or less at the femoral neck or spine to receive once-daily lasofoxifene
217 e postmenopausal for 5 years or more, with a femoral neck or total hip bone mineral density T-score b
219 D T score of -2.5 or lower at the total hip, femoral neck, or lumbar spine; and a history of fracture
221 umbar spine (P < 0.001), 4.1 +/- 1.0% at the femoral neck (P < 0.001), and 4.6 +/- 0.8% at the femora
222 the L2-L4 lumbar spine vertebra (P < 0.05), femoral neck (P < 0.01), and trochanter (P < 0.01) compa
225 ols, MGUS patients had decreased aBMD at the femoral neck (P = .05) and total femur (P < .05) but no
226 lationship of severe CAL with the BMD of the femoral neck (P = 0.015), as well as a positive associat
227 2 at the hip (p = 0.005), 0.012 g/cm2 at the femoral neck (p = 0.02), 0.015 g/cm2 at the spine (p = 0
228 calcium supplements at the spine (P=0.012), femoral neck (P=0.02), total femur (P=0.003), and intert
230 uRBP/uCr and DXA T scores (lumbar [P = .03], femoral neck [P < .001], and total hip [P = .002]).
233 e more complexly loaded human and chimpanzee femoral necks probably receive more prevalent/predominan
236 erminate, or spherical), the femoral head-to-femoral neck ratio as an interval measure of femoral hea
237 k of hip OA increased as the femoral head-to-femoral neck ratio decreased (P for trend<0.001) and wit
238 d the prevalence of abnormal femoral head-to-femoral neck ratio in at least 1 hip was 3.70% in contro
239 , the prevalence of abnormal femoral head-to-femoral neck ratio in the unaffected hip was 2 times gre
240 ent angle, acetabular slope, femoral head-to-femoral neck ratio, and the crossover sign) and compared
241 ng of the human femoral neck, the chimpanzee femoral neck reputedly receives relatively simpler loadi
244 l grip deformity, wide femoral neck, altered femoral neck-shaft angle, appear to play an important ro
246 moderately reduced bone loss measured in the femoral neck, spine, and total body over the three-year
247 ssion, respectively, at the lumbar spine and femoral neck, stratified by male, premenopausal women, a
248 to their lowest baseline T score at spine or femoral neck (stratum I: T score at least -1.0; stratum
249 ctures for those found to have osteoporosis (femoral neck T score < or =-2.5), compared with no inter
251 ation of fracture prevalence was best with a femoral neck T-score of -2.0 or less and a value in the
252 alyses showed that type-1 diabetes, baseline femoral neck T-score, interleukin-2 receptor blockade, a
254 ronate therapy for postmenopausal women with femoral neck T-scores better than -2.5 and no history of
257 fracture or low bone mineral density at the femoral neck (T score, lower than -4 or lower than -3 pl
258 sity (BMD) (g/cm(2)), lumbar spine L2-L4 and femoral neck (T-scores) (P = 0.0149, 0.0002 and 0.0139,
259 ratio had a higher rate of bone loss at the femoral neck than did those with a low ratio (P = 0.02)
260 by a T score for bone mineral density at the femoral neck that was more than 4 SD below the mean peak
261 Compared to complex loading of the human femoral neck, the chimpanzee femoral neck reputedly rece
262 walking does not sufficiently load the upper femoral neck, the fragile zones in healthy bones may nee
264 s were measured in five regions of interest: femoral neck, the Ward triangle, trochanter, intertrocha
265 and by 3 and 6 more percentage points in the femoral neck; the 40-microg dose decreased bone mineral
266 had significantly lower areal density at the femoral neck; total, cortical, and trabecular volumetric
268 the anterior-posterior spine, lateral spine, femoral neck, trochanter, and total body were 4.9% (0.6%
269 %) (P=.002) and prevented bone loss from the femoral neck, trochanter, and total body, despite severe
270 of the anterior-posterior and lateral spine, femoral neck, trochanter, radial shaft, and total body a
273 increase in the modified AHA-DLS, BMD at the femoral neck, trochanter, total hip, and lumbar spine (L
274 MD was measured at the proximal right femur (femoral neck, trochanter, Ward's area) with a dual-photo
275 to bone mineral density (BMD) of the spine, femoral neck, trochanter, Ward's triangle, radius, and t
276 density was measured at the proximal femur (femoral neck, Ward triangle, and trochanter) and lumbar
277 mineral density at the lumbar spine (L2-L4), femoral neck, Ward's triangle, and trochanter, both befo
279 es (for example, bone mineral density at the femoral neck was 0.84 g/cm(2), 0.88 g/cm(2), 0.86 g/cm(2
280 en (for example, bone mineral density at the femoral neck was 0.89 g/cm(2) vs. 0.87 g/cm(2), respecti
281 reasing bone mineral density quintile at the femoral neck was 8.35, 5.74, 5.22, 5.00, and 3.38% in wo
282 ule by at least 5 mm along the length of the femoral neck was almost always seen after injection of 5
284 ne mineral density (BMD) of lumbar spine and femoral neck was measured, and tryptase and histamine me
285 patients with osteopenia/osteoporosis of the femoral neck was twice that in patients with normal bone
287 od pressure, and bone-mineral density at the femoral neck were measured at baseline and bone densitom
288 -score of less than -4.0 at the total hip or femoral neck were not eligible unless they were unable o
289 rements of the lumbar spine (L(2-4)) and the femoral neck were obtained through 1998-1999 by using du
290 n markers (n = 2929) at the lumbar spine and femoral neck were performed in perimenopausal and early
291 Results Capsular adhesions at the anterior femoral neck were present in 12 of the 34 patients (35%)
292 ents, and capsular adhesions at the anterior femoral neck were present in 35% of patients in both gro
294 t, trabecular tracts in human and chimpanzee femoral necks were non-orthogonal (mean approximately 70
295 total body, lumbar spine, and hip (total and femoral neck) were evaluated by using dual-energy X-ray
296 At age 18, smaller lesser trochanter and femoral neck width (FNW) in females still remained altho
297 n 5,245 women (mean age 72.6 years), a wider femoral neck with a more medial centroid position was as
299 vertebral fracture was related to BMD at the femoral neck, with an odds ratio of 1.6 for a 1 SD reduc
300 ndardized BMD (sBMD) at the lumbar spine and femoral neck, World Health Organization (WHO) BMD catego