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1 ulae the keel is perforated by open windows (fenestrae).
2 , its snout and lower jaw show large cranial fenestrae.
3 arly signs of endothelial injury and loss of fenestrae.
4 morphology and ontogeny of the portulae and fenestrae.
5 cytoplasmic protrusions through endothelial fenestrae.
6 rentiated LSECs is the presence of permeable fenestrae.
7 networks were reduced and their SECs lacked fenestrae.
8 organelles and the diaphragms of endothelial fenestrae.
9 role in the passage of proteins through the fenestrae.
10 phragms and increased density of hypophyseal fenestrae.
11 We also collected data on the life span of fenestrae.
12 O mice had a significantly reduced number of fenestrae.
13 associated with a change in the diameter of fenestrae and can be interpreted as a component of the "
14 rocess leading to the formation of apertured fenestrae and channels are also described in fenestrated
15 Notch1(lox/lox) mice had reduced endothelial fenestrae and developed portal hypertension and hepatic
17 entation, we map protein distribution at SPB fenestrae and interrogate protein-protein interactions w
18 ultures of LSECs prevented the loss of their fenestrae and maintained the expression levels of Gata4
20 to monitor the position, size, and number of fenestrae and sieve plates using four-dimensional AFM (X
21 ecognizes specifically the diaphragms of the fenestrae and the stomatal diaphragms of caveolae and tr
24 eticulum (ER) connects daughter cells across fenestrae, and as the cell plate matures, fenestrae cont
32 agms, and swollen endothelial cells with few fenestrae as revealed by transmission electron microscop
34 though decades of studies have characterized fenestrae at the ultrastructural level, little is known
35 Through metaphase, the SPBs remain in their fenestrae, bound to the polar ends of spindle MTs; at ab
37 e span, formation, and disappearance of LSEC fenestrae; by doing so, we also gathered evidence on thr
39 ss fenestrae, and as the cell plate matures, fenestrae contract, causing the plasma membrane (PM) to
42 eolae formation, glomerular endothelial cell fenestrae formation in vivo does not require caveolin-1,
43 ells (IOCs) autonomously deposit collagen at fenestrae, forming rigid, tension-resisting grommets.
47 d gain, particularly in relation to bars and fenestrae, have led to a variety of hypotheses concernin
50 s from the dermis to the epidermis via small fenestrae in the sub-epidermal collagen fibril layer; mo
52 Previous reports indicate that endothelial fenestrae in vitro can form by fusion of caveolae or cav
53 her formation of glomerular endothelial cell fenestrae in vivo similarly involves caveolae and caveol
56 of VEGF and TGF-beta resulted in the loss of fenestrae on CP vasculature and thickening of the otherw
57 out the previous hypothesis that endothelial fenestrae represent fused caveolae, at least for glomeru
58 ull, including the antorbital and mandibular fenestrae, serrated teeth, and closed lower temporal bar
61 mall upper and ventrally open lower temporal fenestrae, supporting the hypothesis of diapsid affiniti
63 ctural evidence on single laying and grouped fenestrae, thereby elucidating their dynamic nature from
64 cytosol, ions traverse one of four external fenestrae to enter the rotundal vestibule and then cross
72 tinct diapsids comprise an open framework of fenestrae (window-like openings) separated by bony strut
73 thelial cells, had CD31 immunoreactivity and fenestrae with diaphragms, but they had a branched pheno
75 ct the prevailing concept that biogenesis of fenestrae would be PLVAP-dependent, and reveal previousl