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1 n cell lines established from adults than in fetal cells.
2 CTH secretion (alone or with CRH) from human fetal cells.
3  a male fetus or evidence of persistent male fetal cells.
4 nct pathological effects on the maternal and fetal cells.
5 nhibited Zika infection in both maternal and fetal cells.
6 sheathed more host axons per donor cell than fetal cells.
7 ions of neurotrophic factors and implants of fetal cells.
8 se attributable to transplacentally acquired fetal cells.
9 uclear transfer-derived sheep generated from fetal cells.
10 ty requires cytogenetic analysis of amniotic fetal cells.
11 ation process examined in this review, these fetal cells acquire properties of leukocytes and endothe
12                                              Fetal cells acquired during pregnancy provide a potentia
13                                              Fetal cells and cell-free DNA reach the maternal circula
14 rved from yeast to human cells, expressed in fetal cells and most adult tissues, and acting synergist
15  IGFBP2 gene, which is normally expressed in fetal cells and turned off in adult cells, becomes react
16 r is not continuously exposed to circulating fetal cells and, in fact, has the capacity to eliminate
17 e of activated cell types, including tumors, fetal cells, and cells of the immune system.
18 evels in adult erythroid cells compared with fetal cells, and function cooperatively to repress HBG1/
19 e of culture failure, maternal overgrowth of fetal cells, and poor chromosome morphology.
20 tion known to induce iNOS maximally in human fetal cells, and the expression of iNOS was determined b
21                                The engrafted fetal cells are a potential source of growth factors and
22                                      Because fetal cells are a source of placental IFN-gamma and cord
23                                              Fetal cells are known to persist in mothers for many yea
24 s from patients with fragile X syndrome, and fetal cells as early as 8 weeks old.
25 ed in neonatal RBC, will direct parasites to fetal cells, as well as F cells, and less to the approxi
26 gh complex interactions between maternal and fetal cells at distinct tissue sites, collectively terme
27 iRNA increased maximal oxygen consumption in fetal cells but not in postnatal cells.
28 d to kill, they usually do not kill infected fetal cells but promote placentation.
29 c cell sorting followed by isolation of pure fetal cells by microdissection.
30 native strategy, which suggested that intact fetal cells can be found in maternal plasma by use of si
31                                              Fetal cells can be found in the maternal circulation in
32                                         Rare fetal cells can be recovered from maternal blood, which
33 s in humans have shown that during pregnancy fetal cells can enter the maternal circulation and persi
34 lure, our objective was to determine whether fetal cells can migrate to the maternal heart and differ
35                               Rare nucleated fetal cells circulate within maternal blood.
36                                        Human fetal cells contained OGF and the zeta receptor.
37                    Although apoptotic intact fetal cells could contribute to the detection of fetal D
38 em cells, which have several advantages over fetal cell-derived therapies.
39 phenotyping to show how Sp3 regulates murine fetal cell differentiation and proliferation.
40                      The mean number of male fetal-cell DNA equivalents detected in 109 women bearing
41                      The mean number of male fetal-cell DNA equivalents detected in 16-ml blood sampl
42 significantly higher than the number of male fetal-cell DNA equivalents detected in 46,XY fetuses (P
43                      The mean number of male fetal-cell DNA equivalents detected when the fetal karyo
44                      The mean number of male fetal-cell DNA equivalents detected when the fetus was m
45                  We determined the number of fetal-cell DNA equivalents present in maternal whole-blo
46                                         Male fetal-cell DNA-equivalent quantitation was determined by
47 ing of the interactions between maternal and fetal cells during early placentation and highlights nov
48                                              Fetal cells enter maternal blood during pregnancy and pe
49                                              Fetal cells enter the maternal circulation during most p
50                                              Fetal cells exhibited a comparatively short-lived or rap
51  postpartum, the result of an encounter with fetal cells expressing the appropriate MHC/peptide compl
52 er the mechanism by which two populations of fetal cells (fetal cortex derived Gli1-expressing cells
53  and fetal blood sampling are used to obtain fetal cells for genetic diagnosis.
54                               In vivo, eGFP+ fetal cells form endothelial cells, smooth muscle cells,
55            However, recovery and analysis of fetal cells from blood is complex, and sensitivity is lo
56 is method of prenatal diagnosis by enriching fetal cells from maternal blood by magnetic cell sorting
57 nosis of single gene disorders by recovering fetal cells from maternal circulation appears to be a fe
58 iptome analyses of primary cultures of human fetal cells from pregnancies affected with trisomy 21 (t
59 idence existed for persistent circulation of fetal cells from prior pregnancies.
60      Utilizing a mouse model of AD and human fetal cells harboring mutant amyloid precursor protein,
61                                 In addition, fetal cells have been implicated in the development of p
62 recent report that showed calves cloned from fetal cells have longer telomeres than their age-matched
63 s, we demonstrate engraftment of multipotent fetal cells in injury zones of maternal hearts.
64 Sc remains to be elucidated, the presence of fetal cells in internal organs suggests that they could
65 dies have confirmed the residual presence of fetal cells in maternal blood and tissues following preg
66                                              Fetal cells in maternal blood are a noninvasive source o
67 ckgrounds (e.g., circulating tumor cells and fetal cells in maternal blood), detection of cells/parti
68 s possible that persistent microchimerism of fetal cells in maternal circulation may, for some mother
69 tion with these systems for the detection of fetal cells in maternal circulation.
70 etal:maternal microchimerism and the role of fetal cells in maternal health and disease.
71        Fetal microchimerism, the presence of fetal cells in maternal tissues, has garnered interest f
72 l microchimerism (FCM) is the persistence of fetal cells in the maternal circulation and organs follo
73              This results in the presence of fetal cells in the maternal circulation, known as fetal
74 ion-independent manner; approximately 40% of fetal cells in the maternal heart express Caudal-related
75 fficking results in long-term persistence of fetal cells in the mother and maternal cells in her offs
76    Proposed hypotheses about the function of fetal cells in the pathogenesis of maternal cancer inclu
77 pes in ways more closely resembling those of fetal cells, including increased LCR-gamma-globin contac
78                                 Maternal and fetal cells intermingle at the site of placental attachm
79 maternal cell trafficking, or the passage of fetal cells into the maternal circulation, is now a well
80 nces its mother's immune response by sending fetal cells into the maternal circulation.
81 lex (MHC) expressed by the trophoblasts, the fetal cells invading the maternal decidua during pregnan
82                                    In vitro, fetal cells isolated from maternal hearts recapitulate t
83  We find that the splanchnic mesenchyme, the fetal cell layer surrounding the endoderm from which the
84 shows that compared with well-differentiated fetal cells, less differentiated embryonal and undiffere
85 atal arrhythmogenesis is associated with the fetal cell-like nature of immature CMs.
86  rodents with TLE, but the scarcity of human fetal cells limits applicability in patient populations.
87                                    The human fetal cell line RPE 28 SV4 has been useful for studies o
88 es established from postnatal donors than in fetal cell lines.
89 ter in adult-derived fibroblasts than in the fetal cell lines.
90              Loss of Ackr2 specifically from fetal cells makes a key contribution to the placental de
91                      These data suggest that fetal cells may be associated with SLE.
92                                              Fetal cells may mimic these events to selectively delete
93 interactions with CD1d expressed on invading fetal cells may play an immunoregulatory role at the mat
94                                              Fetal cells may respond to maternal injury by developing
95                                              Fetal cell microchimerism (FCM) is the persistence of fe
96 gate whether there is an association between fetal cell microchimerism and thyroid disease in women.
97      Our findings suggest a relation between fetal cell microchimerism and thyroid disease.
98        We reviewed all reports of studies on fetal cell microchimerism, defined as male DNA in matern
99 of factors that influence the development of fetal cell microchimerism.
100       The results of this study suggest that fetal cells migrate from the peripheral circulation into
101                                We found that fetal cell migration is not universal but occurs in only
102                     The sixfold elevation of fetal cells observed in maternal blood when the fetus ha
103              Metaphase karyotype analysis of fetal cells obtained by amniocentesis or chorionic villu
104 ononuclear cytotrophoblasts, the specialized fetal cells of the placenta that invade the uterus, play
105 l intrinsic, originate in the replacement of fetal cells, or result from global regulatory mechanisms
106                         The realization that fetal cells pass into the maternal circulation and can s
107                 Recent studies indicate that fetal cells persist in maternal blood for decades after
108 ound in the maternal circulation and if male fetal cells persist in maternal tissues postdelivery.
109                   This finding suggests that fetal cells persisting in the maternal circulation or ti
110 eatures include extended in vitro culture of fetal cells preceding nuclear transfer, as well as in vi
111 nstrated that after inflammatory stimulation fetal cells secreted more IL-8 (LPS, 8-fold; IL-1beta, 2
112                               We report that fetal cells selectively home to injured maternal hearts
113 ammalian oocytes and spermatozoa derive from fetal cells shared by the sexes.
114         Various cell types including porcine fetal cells, stem cells, immortalized cell lines, and ma
115 have been directed towards the enrichment of fetal cells, such as erythroblasts, from maternal blood
116  in originating prenatally from embryonic or fetal cells that are developmentally primed for TP53-dep
117 vasion of the uterine lining by trophoblast, fetal cells that are the major cell type of the placenta
118 h villous and extravillous trophoblasts, the fetal cells that invade the maternal decidua.
119 al tissue specimens suggests the presence of fetal cells that may have multilineage capacity.
120 ly shown the regenerative potential of mixed fetal cells that were naturally found migrating to the i
121 ined by mAb FDC6, and expressed in cancer or fetal cells/tissues, but not in normal adult cells/tissu
122  with a reduced capacity of the TCDD-exposed fetal cells to compete with control cells in a mixed com
123 ghlight the sensitivity of both maternal and fetal cells to heat stress and provide insight into diff
124                                     Maternal-fetal cell transfer during pregnancy can lead to long-la
125 orts the very long-term clinical outcomes of fetal cell transplantation in 2 patients with Parkinson
126                                              Fetal cell transplantation therapies are being developed
127 ntact between maternal cells and specialized fetal cells (trophoblasts) in the human placenta: firstl
128 omical interfaces between maternal cells and fetal cells (trophoblasts): (i) the villous region where
129 lation similar to outer radial glia (oRG), a fetal cell type that expands the stem cell niche in norm
130                   Although many maternal and fetal cell types activate innate immune signalling pathw
131 established, demonstrating that maternal and fetal cell types underwent changes in transcriptomic act
132  brain cell types correspond well to similar fetal cell types.
133                    Complete functionality of fetal cells was also restored; tritiated thymidine incor
134                              The function of fetal cells was better maintained in the air-liquid inte
135 ellular localization of transcripts in human fetal cells, we show that human TSIX antisense transcrip
136 L against fetal Ags, we found that migrating fetal cells were cleared by the maternal immune system.
137                                         More fetal cells were detected in maternal blood when the fet
138                                              Fetal cells were detected in maternal peripheral blood i
139                                       GFP(+) fetal cells were detected in the blood and migrated to a

 
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