ライフサイエンスコーパス: fetal death

1 l neurologic abnormalities, and intrauterine fetal death).

2 birth weight, small-for-gestational-age, or fetal death.

3 causing defective placental development and fetal death.

4 utrophil activation, trophoblast injury, and fetal death.

5 had no significant effects on prematurity or fetal death.

6 low birth weight (<2500 g), prematurity, and fetal death.

7 different adjuvants fails to protect against fetal death.

8 ed risk of respiratory distress syndrome and fetal death.

9 y or indirectly to abnormal placentation and fetal death.

10 reterm birth but not in inflammation-induced fetal death.

11 pesticides showed no strong association with fetal death.

12 fy the relation between maternal smoking and fetal death.

13 95 (3.9%) of 2420 pregnancies resulted in fetal death.

14 from 12 pregnancies showed half resulted in fetal death.

15 ns during pregnancy can increase the risk of fetal death.

16 causing severe anemia, hydrops fetalis, and fetal death.

17 , reduced late muscle marker expression, and fetal death.

18 sfusions in order to avoid fetal hydrops and fetal death.

19 , neonatal intensive care unit admission, or fetal death.

20 tion, have an important role in asphyxia and fetal death.

21 omatic dengue infection during pregnancy and fetal death.

22 ancy is associated with an increased risk of fetal death.

23 ntal infection although it did not result in fetal death.

24 , and, ultimately, abnormal placentation and fetal death.

25 in all analyses but was most pronounced for fetal death.

26 rombophilic women were at increased risk for fetal death.

27 ncy was associated with an increased risk of fetal death.

28 and human complement induced placentitis and fetal death.

29 ccid paralysis, congenital abnormalities and fetal death.

30 e fetus and an increased incidence of sudden fetal death.

31 entrations of air pollution to each birth or fetal death.

32 s; 18 were spontaneous abortions and 14 were fetal deaths.

33 ns, 1.3% in ectopic pregnancies, and 0.5% in fetal deaths.

34 ancies, including 9,402 livebirths and 3,063 fetal deaths.

35 , vaccinated dams experienced fewer in utero fetal deaths.

36 in the US to identify maternal, infant, and fetal deaths.

37 delayed parturition initiation, dystocia and fetal deaths.

38 cells, and led to placental hemorrhages and fetal deaths.

39 s and stillbirth, hopefully leading to fewer fetal deaths.

40 There were 3 intrauterine fetal deaths (1 woman had used LMWH); 9 cases of preecla

41 2 terminations of pregnancy and intrauterine fetal deaths, 189 fetopsies were available: 16 (8.5%) di

42 gs are that 1) smoking increased the risk of fetal death; 2) regular supplement use either before or

43 3 pregnancies were included in this study of fetal death (249 787 [54.7%] in Canada, 197 913 [43.3%]

44 aths (mean [SD] estimated gestational age at fetal death, 26.3 [8.7] weeks) that were collected from

45 patients, were discovered in 3 intrauterine fetal deaths (3.3% [95% CI, 0.68%-9.3%]).

46 There were no maternal deaths and three fetal deaths (4%).

47 rriage (28.6% versus 9.2%; P<0.001) and late fetal death (7.1% versus 0.7%; P=0.016).

48 he following singleton birth categories: 450 fetal deaths; 782 very low birth weight (VLBW, < 1,500 g

49 s spontaneous abortions, neonatal mortality, fetal deaths, admission to the neonatal intensive care u

50 was a composite of perinatal death (in-utero fetal death after randomisation or known neonatal death

51 There were no fetal deaths after 10 weeks' gestation and no evidence o

52 men that were raised by anecdotal reports of fetal deaths after vaccination.

53 ence of maternal smoking; 3) odds ratios for fetal death among smoking women who regularly used suppl

54 The risks of fetal death and congenital malformation were not related

55 ionising radiation are at increased risk of fetal death and congenital malformation.

56 significantly reduced fusobacterial-induced fetal death and decidual necrosis without affecting the

57 indicate that TF is an important mediator in fetal death and growth restriction and that statins may

58 nancy outcomes that can lead to maternal and fetal death and long-term medical complications.

59 l studies, revealed significant increases in fetal death and malformation after Q fever during pregna

60 The potential association between fetal death and residential proximity to agricultural pe

61 Outcomes of include abortion, fetal death and respiratory disease in newborn piglets.

62 evel of TNF-alpha was always associated with fetal death and that the sTNFRs may be important for fet

63 ng pregnancy is a major risk factor for late fetal death and the sudden infant death syndrome, we inv

64 d 259 cases were reviewed after exclusion of fetal deaths and deaths of children who were not residen

65 ed 6% of pregnancy outcomes (primarily early fetal deaths and elective terminations) were lost.

66 Singleton term (37-42 weeks) fetal deaths and live births, stratified by sex, served

67 villus sampling, (iv) amniocentesis, and (v) fetal deaths and live births.

68 IKV) infection in pregnant women can lead to fetal deaths and malformations.

69 e pregnancy outcomes including 143 000 early fetal deaths and stillbirths, 62 000 neonatal deaths, 44

70 tment during pregnancy only in case of prior fetal death), and women with negative thrombophilia scre

71 ham-vaccinated controls experienced viremia, fetal death, and abortion postchallenge.

72 f delivery, birth defects, preterm delivery, fetal death, and low Apgar score.

73 o an intensive care unit (ICU), perinatal or fetal death, and maternal death.

74 significant contributor of preterm birth and fetal death, and that these adverse phenotypes are rescu

75 ications (spontaneous abortion, intrauterine-fetal-death, and preterm delivery) and neonatal sequelae

76 h weight (aOR, 1.91; 95% CI, 1.33-2.76), and fetal death (aOR, 2.23; 95% CI, 1.14-4.37).

77 reconceptual monitoring to increased risk of fetal death are equivocal and require further investigat

78 (aRR = 5.9; 95% CI, 1.8-19.7), intrauterine fetal death (aRR = 9.0; 95% CI, 1.2-65.5), and maternal

79 birth weight, small-for-gestational-age, and fetal death as well as microcephaly (i.e., overall and d

80 eral supplement use might reduce the risk of fetal death associated with maternal smoking.

81 medical history of note was an occurrence of fetal death at 12 weeks gestation when she was 35 years

82 haemolytic disease of the fetus that led to fetal death at 16 + 0 weeks or later, those treated ante

83 he main outcomes were stillbirth, defined as fetal death at 20 or more weeks' gestation, and late sti

84 From a case of in utero fetal death at 24+2 weeks of gestation that occurred 7 d

85 ' gestation, and late stillbirth, defined as fetal death at 28 or more weeks' gestation.

86 tions before the 10th week of gestation or 1 fetal death at or beyond the 10th week of gestation.

87 tions before the 10th week of gestation or 1 fetal death at or beyond the 10th week of gestation.

88 Delivery or fetal death before 35 weeks occurred in 41.5% of pregnan

89 The primary study outcome was delivery or fetal death before 35 weeks of gestation.

90 ave significantly smaller litters because of fetal death between 8.5 and 15.5 days postcoitum.

91 th defects and oral clefts in livebirths and fetal deaths between 1997 and 2000 in seven Texas counti

92 ne growth restriction, low birth weight, and fetal death, but findings are limited by suboptimal cont

93 human complement-induced placentitis caused fetal death, but some fetuses were born normal when lowe

94 evere dengue infection increased the risk of fetal death by about five times (4.9, 2.3-10.2).

95 a disruption of placental architecture, and fetal death by E15.5.

96 ural, and erythroid lineages, culminating in fetal death by embryonic day 15.5 (E15.5).

97 Rate of fetal injury deaths, based on fetal death certificates coded with an underlying cause

98 Data were derived from birth and fetal death certificates that were linked for the first

99 acental transmission to the fetus along with fetal death, congenital microcephaly, and/or Central Ner

100 A grade 4 second trimester fetal death, considered possibly related to the MVA-NP+M

101 Using live birth and fetal death data from New Jersey from 1998 to 2004, the

102 The primary outcome was fetal death, defined as either spontaneous abortion or s

103 In conclusion, the risk of fetal death did not change after the implementation of C

104 Fetal death during labor at term is a complication that

105 d management of emergency fetal delivery and fetal death during surgery.

106 y have reduced the risk of influenza-related fetal death during the pandemic.

107 Of 234 492 women with a live birth or fetal death during the study period, 12 976 (5.5%) were

108 There was no difference in the risk of fetal death during the year following pandemic mitigatio

109 Pregnant individuals experiencing births or fetal deaths during the study period were included.

110 ort practice in the event of either imminent fetal death (during or after fetal surgery) or the need

111 sure to higher incidence of miscarriages and fetal death, even at blood lead elevations ( approximate

112 In 3 states, reported crash-related fetal deaths exceeded that of crash-related infant death

113 eterious to the fetus and is associated with fetal death, fetal growth restriction, and a spectrum of

114 nd low platelet count syndrome, intrauterine fetal death, fetal growth restriction, or placental abru

115 Certificates of 1,449,287 live births and fetal deaths filed in Georgia from 1980 through 1992 wer

116 isease Control and Prevention live birth and fetal death files.

117 CHD cases (n=26 598) comprised live births, fetal deaths from 20 weeks gestation, and terminations o

118 ncy outcomes, including rates of stillbirth (fetal death >=24 weeks' gestation), preterm and cesarean

119 We examined 8,510 fetal deaths (>/=20 weeks' gestation) to estimate their

120 It remains unclear whether the rate of fetal death has changed during the COVID-19 pandemic.

121 ly, other serious neurologic conditions, and fetal death, health ministries across the region have ad

122 In addition, 5 intrauterine fetal deaths hosted SCN5A rare nonsynonymous genetic var

123 psy is the standard method for investigating fetal death; however, it requires dissection of the fetu

124 uncontrolled complement activation leads to fetal death in aPL-treated mice.

125 signaling mediates neutrophil activation and fetal death in APS and that statins may be a good treatm

126 t causes defective placental development and fetal death in DBA/2-mated CBA/J mice.

127 hort of all pregnancies with a live birth or fetal death in Florida from 1 March 2020 to 30 April 202

128 Waddlia chondrophila is a possible cause of fetal death in humans.

129 he impact of COVID-19 mitigation measures on fetal death in Sweden (449,347 births), Denmark (290,857

130 tion of pregnancy did not affect the risk of fetal death in the absence of maternal smoking; 3) odds

131 Patients with recurrent early miscarriage or fetal death in the absence of preeclampsia or placental

132 ational comparison, stillbirth is defined as fetal death in the third trimester (>/=1000 g birthweigh

133 n setting, a region with the highest rate of fetal death in the world.

134 placental diseases-the most common causes of fetal death in this at-risk group.

135 The reduced fetal death in TLR4-deficient mice was accompanied by de

136 oup and three in the intervention group) and fetal death in utero (ten in the standard care group and

137 s in key safety outcomes including abortion, fetal death in utero, and congenital anomalies.

138 mplement activation in the placenta leads to fetal death in utero.

139 w birth weight, small-for-gestational-age or fetal death in women who already have gestational comorb

140 on on the risk of specific malformations and fetal death is lacking.

141 Although fetal death is now understood to be a severe outcome of

142 There were 163 fetal death/live-birth different-sex twin pairs.

143 There were 434 fetal death/live-birth same-sex twin pairs.

144 l cleft, low birth weight, preterm delivery, fetal death, low Apgar score, and mode of delivery were

145 l cleft, low birth weight, preterm delivery, fetal death, low Apgar score, and mode of delivery.

146 sure with orofacial cleft, preterm delivery, fetal death, low Apgar score, and mode of delivery.

147 owing outcomes were considered: maternal and fetal death; malformations; preterm delivery; small for

148 nience sample of 91 unexplained intrauterine fetal deaths (mean [SD] estimated gestational age at fet

149 netic evaluation of 91 cases of intrauterine fetal death, missense mutations associated with LQTS sus

150 during pregnancy almost doubled the odds of fetal death (mOR 1.9, 95% CI 1.6-2.2).

151 dies (44 publications) with more than 10,147 fetal deaths, more than 16,274 stillbirths, more than 43

152 mains of reproductive toxicity: intrauterine fetal death, morphologic teratogenicity, growth impairme

153 imary outcome was a composite of intrapartum fetal death, neonatal death, an Apgar score of 3 or less

154 le autoantibodies, Raynaud's phenomenon, and fetal death occurred at 20 weeks gestation.

155 Fetal death occurred in 12 907 (7.9%) of 163 810 pregnan

156 .0% (95% CI, 15.2% to 23.2%) of pregnancies; fetal death occurred in 4%, neonatal death occurred in 1

157 egnancy resulted in p-aHUS, one intrauterine fetal death occurred, and seven pregancies were uneventf

158 30 fetal deaths occurred among women assigned multivitamins

159 re often seen in children, and abortions and fetal deaths occurred in pregnant women infected with VE

160 Stillbirths (fetal deaths occurring at >/=20 weeks' gestation) are ap

161 e that linked hospital records to births and fetal deaths occurring between October 1, 2002, and Marc

162 of all trauma during pregnancy, with 82% of fetal deaths occurring during these automobile accidents

163 -specific prevalence of cerebral palsy after fetal death of the co-twin is much higher than that repo

164 lete disruption of the N-myc gene results in fetal death on the first day of renal organogenesis.

165 There were two spontaneous fetal deaths (one in each group) without obvious cause a

166 d in 5 states among individuals delivering a fetal death or a live birth between 22 and 44 weeks.

167 Individuals delivering a fetal death or a live birth with gestational age between

168 se serious consequences include miscarriage, fetal death or an infant born with birth defects (i.e.,

169 We defined three primary outcomes: fetal death or birth before 34 weeks and 0 days gestatio

170 or neonatal death and 3 secondary outcomes: fetal death or delivery at or before 28+0 weeks, develop

171 nnsylvania, and South Carolina) delivering a fetal death or live birth.

172 Intrauterine fetal death or stillbirth occurs in approximately 1 out

173 f early preterm birth, gestational diabetes, fetal death or stillbirth, perinatal death, or admission

174 onal age at cART initiation with the risk of fetal death or stillbirth.

175 OR = 0.34, 95% CI 0.21-0.54) or mothers with fetal deaths (OR = 0.36, 95% CI 0.19-0.67).

176 ancies were followed up until preterm birth, fetal death, or 37 completed weeks of gestation.

177 are diagnosed with CHD die as a TOFPA, late fetal death, or early neonatal death.

178 failure (anembryonic gestation, embryonic or fetal death, or incomplete or inevitable spontaneous abo

179 32 live-birth controls and 3,328 live-birth, fetal-death, or electively terminated cases had exposure

180 sis utilized 319 cases of selected causes of fetal death other than congenital anomalies and 611 non-

181 ferences between the 2 groups in the risk of fetal death (P = 0.99) or early infant death (P = 0.19).

182 regnancy and assuming no further spontaneous fetal deaths (P=0.28).

183 A peak rate of 9.3 fetal deaths per 100 000 live births was observed among

184 leading trauma mechanism (82% of cases; 2.3 fetal deaths per 100 000 live births), followed by firea

185 tic fetal injury deaths were identified (3.7 fetal deaths per 100 000 live births).

186 ctive toxicity in five domains (intrauterine fetal death, physical malformations, growth impairment,

187 evere fetal outcomes, including miscarriage, fetal death, preterm birth, intrauterine growth restrict

188 The primary outcome was delivery or fetal death prior to 37 weeks.

189 aborative Research Network Initial Causes of Fetal Death protocol from the Eunice Kennedy Shriver Nat

190 IL-10 attenuated the LPS-induced fetal death rate (to 22%) and growth restriction (P<0.05

191 The fetal death rate was 45.6 per 1000 births, the neonatal

192 In vivo, the fetal death rate was significantly reduced in TLR4-defic

193 This ecologic study notes that fetal death rates (FDR) during the Washington DC drinkin

194 Fetal death rates per 1000 live births decreased from 6.

195 irths identified in MAX 1999-2013 to Florida fetal death records (FDRs) to obtain clinical estimates

196 nattainment O3 levels, we obtained birth and fetal death records from 2008-2013 and estimated materna

197 ough linkage of death records with birth and fetal death records, and 47% (n = 116) through review of

198 ta of reproductive-age women, live birth and fetal death records, and medical examiner records in Mar

199 death, thrombophilic women experienced less fetal death recurrences, less preterm births and preecla

200 41 weeks) at E17.5 had fewer pups, more late fetal deaths, reduced fetal weight, increased placental

201 tor vehicle crashes are the leading cause of fetal deaths related to maternal trauma.

202 h increased risk of perinatal (RR = 0.72) or fetal death (RR = 0.86).

203 0.49, 1.05; P = 0.09] and increased risk of fetal death (RR = 1.58; 95% CI = 0.95, 2.63; P = 0.08),

204 osure during pregnancy and increased risk of fetal death, spontaneous abortion, stillbirth, or major

205 s into consideration to reduce the burden of fetal death, stillbirth, and infant death.

206 that maternal obesity increases the risk of fetal death, stillbirth, and infant death; however, the

207 l BMI were associated with increased risk of fetal death, stillbirth, and neonatal, perinatal, and in

208 Fetal death, stillbirth, and neonatal, perinatal, and in

209 ng adjusted relative risk (RR) estimates for fetal death, stillbirth, or infant death by at least 3 c

210 live births of 160 infants (4 twin pairs), 1 fetal death/stillbirth, 11 spontaneous abortions, and 1

211 dysfunction is associated with intrauterine fetal death, sudden infant death syndrome, cardiac arrhy

212 Among women with prior fetal death, thrombophilic women experienced less fetal

213 y RR per 5-unit increase in maternal BMI for fetal death was 1.21 (95% CI, 1.09-1.35; I2 = 77.6%; n =

214 hereditary TTP became plasma-refractory and fetal death was imminent, weekly injections of recombina

215 clinical diagnosis of influenza, the risk of fetal death was increased (adjusted hazard ratio, 1.91;

216 The risk of fetal death was reduced with vaccination during pregnanc

217 ion between the presence of the mutation and fetal death, we performed additional studies with ZIKV w

218 CI, 0.91 to 1.11; P=0.87), and the rates of fetal death were 4.3% and 5.0%, respectively (relative r

219 Rates of fetal death were 7% in both groups; overall adverse outc

220 0, absolute risks per 10,000 pregnancies for fetal death were 76, 82 (95% CI, 76-88), and 102 (95% CI

221 The leading immediate causes of fetal death were antepartum hypoxia (35.7%) and fetal in

222 Causes of fetal death were assigned by using the Stillbirth Collab

223 imicrobial treatment, 1 pregnancy loss and 1 fetal death were reported.

224 When fetal deaths were counted as treatment failures, the per

225 induced abortions, ectopic pregnancies, and fetal deaths were obtained by using data from the Center

226 No birth defects or fetal deaths were reported in any of the live births.

227 4 untreated patients, 1 pregnancy loss and 3 fetal deaths were reported, including one where F. tular

228 proof that live vaccination protects against fetal death, whereas immunization using whole-tachyzoite

229 rder to prevent congenital abnormalities and fetal death which can result from both excessive and ins

230 report multiple incidences of intra-uterine fetal death, which were also observed in an eighth famil

231 ard ratios were 0.91 (95% CI, 0.67-1.23) for fetal death with time-dependent ondansetron exposure dur

232 ression models to estimate hazard ratios for fetal death, with the gestational day as the time metric

233 scarriages in the first trimester, and 1 had fetal death, with the macerated stillborn showing diffus