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1 ances from maternal blood and contributes to fetal growth restriction.
2 presence or absence of ultrasonic markers of fetal growth restriction.
3 o trigger delivery in mothers of babies with fetal growth restriction.
4 e outcomes of human pregnancy complicated by fetal growth restriction.
5 complicated with intervillositis, can cause fetal growth restriction.
6 sed to play a role in the pathophysiology of fetal growth restriction.
7 ), several are of pathological relevance for fetal growth restriction.
8 ancies worldwide and is often complicated by fetal growth restriction.
9 oteinuria, and edema) and, in some patients, fetal growth restriction.
10 egnancy disorders including preeclampsia and fetal growth restriction.
11 sk of major birth defects, preterm birth, or fetal growth restriction.
12 ot the PCE area, was associated with LBW and fetal growth restriction.
13 ncluding late miscarriage, preeclampsia, and fetal growth restriction.
14 except for early preterm birth and possibly fetal growth restriction.
15 nce of preeclampsia, placental abruption, or fetal growth restriction.
16 are associated with maternal infections and fetal growth restriction.
17 e the association between race/ethnicity and fetal growth restriction.
18 atory cytokines that have been implicated in fetal growth restriction.
19 IFN-gamma levels at birth may be related to fetal growth restriction.
20 rst half of pregnancy and is associated with fetal growth restriction.
21 mpletely prevented the high altitude-induced fetal growth restriction.
22 a strong predictor of both preterm birth and fetal growth restriction.
23 rates of preterm birth, low birth weight, or fetal growth restriction.
24 esting mechanisms underlying hypoxia-related fetal growth restriction.
25 the placenta, followed several days later by fetal growth restriction.
26 impact on stillbirth, preterm delivery, and fetal growth restriction.
27 s miscarriage, early-onset preeclampsia, and fetal growth restriction.
28 ormin did not correct placental structure or fetal growth restriction.
29 rovides protection against hypoxia-dependent fetal growth restriction.
30 ommon pregnancy complication associated with fetal growth restriction.
31 macological AMPK activation for treatment of fetal growth restriction.
32 ion resulting in placental insufficiency and fetal growth restriction.
33 ences in shaping placenta development and in fetal growth restriction.
34 regnancy disorders such as pre-eclampsia and fetal growth restriction.
35 nanoplastics throughout gestation results in fetal growth restriction.
36 gnaling and nutrient transport, resulting in fetal growth restriction.
37 mes including miscarriage, pre-eclampsia and fetal growth restriction.
38 factor for preterm birth, preeclampsia, and fetal growth restriction.
39 GFR3 signaling contributed to late-gestation fetal growth restriction.
40 ; and previous pre-eclampsia or intrauterine fetal growth restriction.
41 sociated disorders, such as preeclampsia and fetal growth restriction.
42 R-gamma) protects against hypoxia-associated fetal growth restriction.
43 logical characteristics of preeclampsia with fetal growth restriction.
44 ction are associated with poor outcomes like fetal growth restriction.
45 logical conditions, including stillbirth and fetal growth restriction.
46 out resulted in reduced placental weight and fetal growth restriction.
47 acental inflammation and was associated with fetal growth restriction.
48 d amino acid transporter activity and causes fetal growth restriction.
49 omplications, uteroplacental dysfunction, or fetal growth restriction.
50 ies and preterm newborns without evidence of fetal growth restriction.
51 ype in response to hypoxia, a major cause of fetal growth restriction.
52 , and impaired spiral artery remodeling with fetal growth restriction.
53 hi women, especially those at higher risk of fetal growth restriction.
54 l mechanistic link between pre-eclampsia and fetal growth restriction.
55 estational hypoxia such as pre-eclampsia and fetal growth restriction.
56 f gestation; 1.16, 1.01-1.34; I(2)=64%), and fetal growth restriction (1.26, 1.20-1.33; I(2)=1%).
58 rm birth (aHR, 1.79; 95% CI, 1.37-2.34), and fetal growth restriction (aHR, 2.04; 95% CI, 1.72-2.43).
59 l dilation, intra-amniotic inflammation, and fetal growth restriction, all of which are clinical sign
61 ht into mechanisms and interventions against fetal growth restriction and adult-onset programmed hype
62 ion; PIO prevented approximately half of the fetal growth restriction and attenuated placental insuff
63 uld have broad implications for the study of fetal growth restriction and birth weight, and for the p
64 d with incident CHF, atrial arrhythmias, and fetal growth restriction and complex CHD was associated
65 on effect of diabetes on oogenesis, leads to fetal growth restriction and congenital deformities.
69 s common environmental risk factors for both fetal growth restriction and high blood pressure or whet
71 his may be due to conditions associated with fetal growth restriction and iatrogenic preterm birth.
72 death, whilst heterozygous loss resulted in fetal growth restriction and impaired placental formatio
73 pregnant dams during early pregnancy led to fetal growth restriction and infection of the fetal brai
77 he placental villous tissue occurred in both fetal growth restriction and pre-eclampsia, whereas CD79
79 Placental hypoxia is causally implicated in fetal growth restriction and preeclampsia, with both occ
83 treatment with antioxidants protects against fetal growth restriction and programmed hypertension in
84 also occurred in other pregnancy disorders (fetal growth restriction and recurrent miscarriage), ind
88 posure is a significant mechanism underlying fetal growth restriction and the programming of adverse
89 tudies have suggested an association between fetal growth restriction and the risk of spontaneous pre
90 ove pregnancy outcomes in severe early-onset fetal growth restriction and therefore it should not be
91 he hypothesis that shared factors cause both fetal growth restriction and urogenital anomalies was su
92 centrations are associated with proportional fetal growth restriction and with an increased risk of p
94 he fetus and is associated with fetal death, fetal growth restriction, and a spectrum of central nerv
95 mined low birth weight (LBW), preterm birth, fetal growth restriction, and birth defects among births
98 weight-for-gestational-age, an indicator of fetal growth restriction, and furthermore the authors ob
99 LC-PUFAs is strongly linked with stillbirth, fetal growth restriction, and impaired neurodevelopmenta
101 weight includes babies born preterm and with fetal growth restriction, and not all these infants have
104 ell as pregnancy complications preeclampsia, fetal growth restriction, and preterm birth, which stem
106 obstetric risks, particularly preterm birth, fetal growth restriction, and stillbirth, will need to b
109 rs, such as recurrent hydatidiform moles and fetal growth restriction, and thus improve placental dev
110 37 weeks (AOR, 3.84; 95% CI, 3.15-4.71), and fetal growth restriction (AOR, 3.25; 95% CI, 2.42-4.38)
115 evisited the prevailing hypotheses regarding fetal growth restriction as a risk factor for urogenital
116 revious understanding and interpretations of fetal growth restriction as represented by small for ges
117 men with normal pregnancies, particularly in fetal growth restriction associated with pre-eclampsia.
120 rnal vitamin D deficiency has been linked to fetal growth restriction, but the underlying mechanisms
121 late levels in pregnancy are associated with fetal growth restriction, but the underlying mechanisms
122 ro tobacco exposure has been associated with fetal growth restriction, but uncertainty remains about
123 urely from pregnancies complicated by PE and fetal growth restriction can have low nephron mass, whic
125 ces fetal development and metabolism and how fetal growth restriction can result in susceptibility to
127 ion and re-admissions, treatment compliance, fetal growth restriction, congenital malformations, gest
128 Epidemiological studies have indicated that fetal growth restriction correlates with later disease,
130 de is associated with a greater incidence of fetal growth restriction due, in part, to lesser uterine
131 ed rates of pregnancy pathologies, including fetal growth restriction, due at least in part to reduct
133 e consumption led to placental inefficiency, fetal growth restriction, elevated fetal serum glucose a
134 offspring of alcohol-exposed sires exhibited fetal growth restriction, enlarged placentas, and decrea
136 yncytiotrophoblasts, leading to intrauterine fetal growth restriction, fetal liver hypocellularity, a
137 e availability causes human diseases such as fetal growth restriction, fetal malformations and cancer
138 folate availability causes diseases such as fetal growth restriction, fetal malformations and cancer
143 folate and choline status resulted in severe fetal growth restriction (FGR) and impaired fertility in
148 tal exposure to disinfection by-products and fetal growth restriction (FGR) and preterm birth in the
149 eenage motherhood and short birth intervals, fetal growth restriction (FGR) and preterm birth, child
150 pathways involved in the pathophysiology of fetal growth restriction (FGR) and small for gestational
153 ative protection against altitude-associated fetal growth restriction (FGR) and the positive selectio
154 by pre-eclampsia (PE), preterm birth (PTB), fetal growth restriction (FGR) and/or macrosomia resulti
156 The placenta of pregnancies whose outcome is fetal growth restriction (FGR) are characterized by abno
158 was to identify metabolites associated with fetal growth restriction (FGR) by examining early and la
160 nancies complicated by preeclampsia (PE) and fetal growth restriction (FGR) compared with control thi
162 n of placental vessel networks in normal and fetal growth restriction (FGR) complicated pregnancies.
163 regnancies complicated by severe early-onset fetal growth restriction (FGR) exhibit diminished vascul
164 ion complicated by chronic fetal hypoxia and fetal growth restriction (FGR) increases a prenatal orig
165 mely delivery of infants suspected of having fetal growth restriction (FGR) is a balance between prev
176 g fetus and may impact postnatal health, and fetal growth restriction (FGR) is often seen co-occurrin
179 addition, elevated Phlda2 was found to drive fetal growth restriction (FGR) of transgenic offspring a
183 ce of preterm pre-eclampsia (< 37 weeks) and fetal growth restriction (FGR) was increased (1.8% vs. 0
184 d pre-eclamptic pregnancies complicated with fetal growth restriction (FGR) with and without villitis
185 of the most common and preventable causes of fetal growth restriction (FGR), a condition in which a f
187 ntas from pregnancies complicated by PE with fetal growth restriction (FGR), and (2) suppressyn secre
188 m (PAS), PPH, placenta previa, hysterectomy, fetal growth restriction (FGR), and preterm birth (PTB).
189 (PE), spontaneous preterm birth (sPTB), and fetal growth restriction (FGR), and to identify causes o
191 d with adverse pregnancy outcomes, including fetal growth restriction (FGR), due in part to reduction
192 e are two pathways to LBW, preterm birth and fetal growth restriction (FGR), with the FGR pathway res
200 putative aetiologies in the pathogenesis of fetal growth restriction (FGR); however, the regulating
201 ry transformation occurs in preeclampsia and fetal growth restriction (FGR); these processes are not
203 preterm birth (<37 weeks of gestation), and fetal growth restriction (FGR; liveborn with birthweight
204 maternal inflammation such as preeclampsia, fetal growth restriction, gestational diabetes, and bact
205 SGA have an increased VCDR, suggesting that fetal growth restriction has a lasting impact on optic d
207 6-32 weeks of gestation who had very preterm fetal growth restriction (ie, low abdominal circumferenc
208 Campylobacter rectus infection that induces fetal growth restriction in a mouse model also compromis
214 mplex (OR, 31.8; 95% CI, 4.3-236.3) CHD, for fetal growth restriction in noncomplex (OR, 1.6; 95% CI,
215 ability; Sildenafil does not protect against fetal growth restriction in the chick embryo, supporting
221 uartile, low birth weight increased >5-fold, fetal growth restriction increased >6-fold, and infant b
222 onatal glucose homeostasis and is altered by fetal growth restriction induced by maternal undernutrit
226 ht that severe COVID-19 during pregnancy and fetal growth restriction is associated with heightened v
229 quartiles had offspring with third-trimester fetal growth restriction, leading to a smaller head circ
231 ommon pregnancy complication associated with fetal growth restriction, may initiate fibrotic and meta
232 l-recessive disorder characterized by severe fetal growth restriction, microcephaly, a distinct facia
234 et and provide a potential mechanism for the fetal growth restriction observed in women who use canna
235 and consisted of 1 or more of the following: fetal growth restriction, oligohydramnios, hypertensive
236 ndependent of the hypoxic pregnancy inducing fetal growth restriction or elevations in maternal or fe
237 This study explores associations between fetal growth restriction or excessive fetal growth, alon
239 lve impaired placental function, either with fetal growth restriction or preterm labour, or both.
241 ant pain, headache with visual disturbances, fetal growth restriction, or abnormal maternal blood tes
242 et count syndrome, intrauterine fetal death, fetal growth restriction, or placental abruption who had
244 gestation without congenital malformations, fetal growth restriction, or severe postnatal morbidity.
246 by AICAR partially prevented hypoxia-induced fetal growth restriction (P < 0.01), due in part to incr
248 tric complications, including preterm birth, fetal growth restriction, placental abruption, and still
249 d by complications such as pre-eclampsia and fetal growth restriction, placental ageing is notably ac
250 cause of maternal hypertension in pregnancy, fetal growth restriction, premature birth, and fetal and
251 gnancy, and neonatal complications including fetal growth restriction, preterm birth and stillbirth.
252 22% of cases) included respiratory distress, fetal growth restriction, preterm birth, AIHA of the new
253 omposite adverse perinatal outcome including fetal growth restriction, preterm birth, low Apgar score
254 explored the associations of abruption with fetal growth restriction, preterm delivery, and perinata
255 tin are associated with an increased risk of fetal growth restriction, preterm delivery, and preeclam
256 s include alloimmunization, opioid exposure, fetal growth restriction, preterm delivery, and stillbir
257 ing that cardiovascular disease is linked to fetal growth restriction rather than to premature birth.
258 GT/GT) mice died perinatally associated with fetal growth restriction, reduced hepatic glycogen store
259 in mice to determine whether hypoxia-induced fetal growth restriction reduces placental PPAR-gamma pr
261 ow that fetal growth acceleration as well as fetal growth restriction, resulting from between-breed e
262 his correlation to recurrent miscarriage and fetal growth restriction, revealing the common mechanism
263 UtA) blood flow and relative protection from fetal growth restriction seen in altitude-adapted Andean
264 nancy complications such as preeclampsia and fetal growth restriction, since defects in maturation of
265 t undernutrition in the aggregate--including fetal growth restriction, stunting, wasting, and deficie
268 psychoactive ingredient in cannabis, causes fetal growth restriction, though the mechanisms are not
269 e than placental malaria per se, might cause fetal growth restriction, through impaired transplacenta
270 ly identification of factors contributing to fetal growth restriction to minimize detrimental outcome
271 and 6 days' gestation and severe early-onset fetal growth restriction to receive either sildenafil 25
272 Maternal exposure to Cd is associated with fetal growth restriction, trace element deficiencies, an
273 in placental villous tissue are increased in fetal growth restriction vs. placentas from women with n
275 eatures with human recurrent miscarriage and fetal growth restriction, we identified tissue factor (T
276 show that lowland mice experience pronounced fetal growth restriction when challenged with gestationa
279 osure resulted in pups born with symmetrical fetal growth restriction, with catch up growth by post-n
280 C57Bl/6J sires and CD-1 dams do not exhibit fetal growth restriction, with male fetuses developing s
282 example, maternal smoking (Z) is a cause of fetal growth restriction (X), which subsequently affects