戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ances from maternal blood and contributes to fetal growth restriction.
2 presence or absence of ultrasonic markers of fetal growth restriction.
3 o trigger delivery in mothers of babies with fetal growth restriction.
4 e outcomes of human pregnancy complicated by fetal growth restriction.
5  complicated with intervillositis, can cause fetal growth restriction.
6 sed to play a role in the pathophysiology of fetal growth restriction.
7 ), several are of pathological relevance for fetal growth restriction.
8 ancies worldwide and is often complicated by fetal growth restriction.
9 oteinuria, and edema) and, in some patients, fetal growth restriction.
10 egnancy disorders including preeclampsia and fetal growth restriction.
11 sk of major birth defects, preterm birth, or fetal growth restriction.
12 ot the PCE area, was associated with LBW and fetal growth restriction.
13 ncluding late miscarriage, preeclampsia, and fetal growth restriction.
14  except for early preterm birth and possibly fetal growth restriction.
15 nce of preeclampsia, placental abruption, or fetal growth restriction.
16  are associated with maternal infections and fetal growth restriction.
17 e the association between race/ethnicity and fetal growth restriction.
18 atory cytokines that have been implicated in fetal growth restriction.
19  IFN-gamma levels at birth may be related to fetal growth restriction.
20 rst half of pregnancy and is associated with fetal growth restriction.
21 mpletely prevented the high altitude-induced fetal growth restriction.
22 a strong predictor of both preterm birth and fetal growth restriction.
23 rates of preterm birth, low birth weight, or fetal growth restriction.
24 esting mechanisms underlying hypoxia-related fetal growth restriction.
25 the placenta, followed several days later by fetal growth restriction.
26  impact on stillbirth, preterm delivery, and fetal growth restriction.
27 s miscarriage, early-onset preeclampsia, and fetal growth restriction.
28 ormin did not correct placental structure or fetal growth restriction.
29 rovides protection against hypoxia-dependent fetal growth restriction.
30 ommon pregnancy complication associated with fetal growth restriction.
31 macological AMPK activation for treatment of fetal growth restriction.
32 ion resulting in placental insufficiency and fetal growth restriction.
33 ences in shaping placenta development and in fetal growth restriction.
34 regnancy disorders such as pre-eclampsia and fetal growth restriction.
35 nanoplastics throughout gestation results in fetal growth restriction.
36 gnaling and nutrient transport, resulting in fetal growth restriction.
37 mes including miscarriage, pre-eclampsia and fetal growth restriction.
38  factor for preterm birth, preeclampsia, and fetal growth restriction.
39 GFR3 signaling contributed to late-gestation fetal growth restriction.
40 ; and previous pre-eclampsia or intrauterine fetal growth restriction.
41 sociated disorders, such as preeclampsia and fetal growth restriction.
42 R-gamma) protects against hypoxia-associated fetal growth restriction.
43 logical characteristics of preeclampsia with fetal growth restriction.
44 ction are associated with poor outcomes like fetal growth restriction.
45 logical conditions, including stillbirth and fetal growth restriction.
46 out resulted in reduced placental weight and fetal growth restriction.
47 acental inflammation and was associated with fetal growth restriction.
48 d amino acid transporter activity and causes fetal growth restriction.
49 omplications, uteroplacental dysfunction, or fetal growth restriction.
50 ies and preterm newborns without evidence of fetal growth restriction.
51 ype in response to hypoxia, a major cause of fetal growth restriction.
52 , and impaired spiral artery remodeling with fetal growth restriction.
53 hi women, especially those at higher risk of fetal growth restriction.
54 l mechanistic link between pre-eclampsia and fetal growth restriction.
55 estational hypoxia such as pre-eclampsia and fetal growth restriction.
56 f gestation; 1.16, 1.01-1.34; I(2)=64%), and fetal growth restriction (1.26, 1.20-1.33; I(2)=1%).
57          An atmosphere of 13% oxygen induced fetal growth restriction (1182 +/- 9 mg, n = 90 vs. 1044
58 rm birth (aHR, 1.79; 95% CI, 1.37-2.34), and fetal growth restriction (aHR, 2.04; 95% CI, 1.72-2.43).
59 l dilation, intra-amniotic inflammation, and fetal growth restriction, all of which are clinical sign
60                             The link between fetal growth restriction and abruption suggests that the
61 ht into mechanisms and interventions against fetal growth restriction and adult-onset programmed hype
62 ion; PIO prevented approximately half of the fetal growth restriction and attenuated placental insuff
63 uld have broad implications for the study of fetal growth restriction and birth weight, and for the p
64 d with incident CHF, atrial arrhythmias, and fetal growth restriction and complex CHD was associated
65 on effect of diabetes on oogenesis, leads to fetal growth restriction and congenital deformities.
66                        After controlling for fetal growth restriction and early delivery, the high ri
67 iety of adverse clinical outcomes, including fetal growth restriction and fetal demise.
68                    Hypoxic pregnancy induced fetal growth restriction and fetal oxidative stress.
69 s common environmental risk factors for both fetal growth restriction and high blood pressure or whet
70 nd JZ+D at GD14 and GD18 in association with fetal growth restriction and higher blood pressure.
71 his may be due to conditions associated with fetal growth restriction and iatrogenic preterm birth.
72  death, whilst heterozygous loss resulted in fetal growth restriction and impaired placental formatio
73  pregnant dams during early pregnancy led to fetal growth restriction and infection of the fetal brai
74  excess of mortality; notably, the effect of fetal growth restriction and infectious diseases.
75                       Here we show that both fetal growth restriction and over-growth are associated
76                           Complications like fetal growth restriction and pre-eclampsia are linked to
77 he placental villous tissue occurred in both fetal growth restriction and pre-eclampsia, whereas CD79
78 in severe pregnancy complications, including fetal growth restriction and pre-eclampsia.
79  Placental hypoxia is causally implicated in fetal growth restriction and preeclampsia, with both occ
80 itigation of pregnancy complications such as fetal growth restriction and preeclampsia.
81 alovirus (HCMV) infections and is a cause of fetal growth restriction and pregnancy loss.
82 tions and by the likelihood of recurrence of fetal growth restriction and preterm birth.
83 treatment with antioxidants protects against fetal growth restriction and programmed hypertension in
84  also occurred in other pregnancy disorders (fetal growth restriction and recurrent miscarriage), ind
85            Placental insufficiency can cause fetal growth restriction and stillbirth.
86              The placental disease can cause fetal growth restriction and stillbirth.
87 ry condition of the placenta associated with fetal growth restriction and stillbirth.
88 posure is a significant mechanism underlying fetal growth restriction and the programming of adverse
89 tudies have suggested an association between fetal growth restriction and the risk of spontaneous pre
90 ove pregnancy outcomes in severe early-onset fetal growth restriction and therefore it should not be
91 he hypothesis that shared factors cause both fetal growth restriction and urogenital anomalies was su
92 centrations are associated with proportional fetal growth restriction and with an increased risk of p
93          Here, we developed a mouse model of fetal-growth restriction and placental insufficiency tha
94 he fetus and is associated with fetal death, fetal growth restriction, and a spectrum of central nerv
95 mined low birth weight (LBW), preterm birth, fetal growth restriction, and birth defects among births
96 ed flow of maternal blood into the placenta, fetal growth restriction, and death.
97 ce significantly increased placental damage, fetal growth restriction, and fetal resorption.
98  weight-for-gestational-age, an indicator of fetal growth restriction, and furthermore the authors ob
99 LC-PUFAs is strongly linked with stillbirth, fetal growth restriction, and impaired neurodevelopmenta
100 rmation has been described in pre-eclampsia, fetal growth restriction, and miscarriage.
101 weight includes babies born preterm and with fetal growth restriction, and not all these infants have
102 hich led to fetal and placenta iron loading, fetal growth restriction, and placentomegaly.
103 ical manifestations - such as preterm birth, fetal growth restriction, and pre-eclampsia.
104 ell as pregnancy complications preeclampsia, fetal growth restriction, and preterm birth, which stem
105 se obstetric outcomes such as pre-eclampsia, fetal growth restriction, and preterm birth.
106 obstetric risks, particularly preterm birth, fetal growth restriction, and stillbirth, will need to b
107 egnancy complications, such as preeclampsia, fetal growth restriction, and stillbirth.
108 ications of pregnancy, such as preeclampsia, fetal growth restriction, and stillbirth.
109 rs, such as recurrent hydatidiform moles and fetal growth restriction, and thus improve placental dev
110 37 weeks (AOR, 3.84; 95% CI, 3.15-4.71), and fetal growth restriction (AOR, 3.25; 95% CI, 2.42-4.38)
111           Premature labor, fetal demise, and fetal growth restriction are accompanied by indices of i
112                      Increased morbidity and fetal growth restriction are reported in uninfected chil
113                            Pre-eclampsia and fetal growth restriction arise from disorders of placent
114                                   We defined fetal growth restriction as a combination of estimated f
115 evisited the prevailing hypotheses regarding fetal growth restriction as a risk factor for urogenital
116 revious understanding and interpretations of fetal growth restriction as represented by small for ges
117 men with normal pregnancies, particularly in fetal growth restriction associated with pre-eclampsia.
118                                              Fetal growth restriction associates with increased risk
119                              Hypoxia-related fetal growth restriction becomes apparent between 25 and
120 rnal vitamin D deficiency has been linked to fetal growth restriction, but the underlying mechanisms
121 late levels in pregnancy are associated with fetal growth restriction, but the underlying mechanisms
122 ro tobacco exposure has been associated with fetal growth restriction, but uncertainty remains about
123 urely from pregnancies complicated by PE and fetal growth restriction can have low nephron mass, whic
124                           Severe early-onset fetal growth restriction can lead to a range of adverse
125 ces fetal development and metabolism and how fetal growth restriction can result in susceptibility to
126  left ventricles that are independent of the fetal growth restriction condition were identified.
127 ion and re-admissions, treatment compliance, fetal growth restriction, congenital malformations, gest
128  Epidemiological studies have indicated that fetal growth restriction correlates with later disease,
129              Molecular mechanisms underlying fetal growth restriction due to placental insufficiency
130 de is associated with a greater incidence of fetal growth restriction due, in part, to lesser uterine
131 ed rates of pregnancy pathologies, including fetal growth restriction, due at least in part to reduct
132        Household air pollution might lead to fetal growth restriction during pregnancy.
133 e consumption led to placental inefficiency, fetal growth restriction, elevated fetal serum glucose a
134 offspring of alcohol-exposed sires exhibited fetal growth restriction, enlarged placentas, and decrea
135             Later the compensation fails and fetal growth restriction ensues.
136 yncytiotrophoblasts, leading to intrauterine fetal growth restriction, fetal liver hypocellularity, a
137 e availability causes human diseases such as fetal growth restriction, fetal malformations and cancer
138  folate availability causes diseases such as fetal growth restriction, fetal malformations and cancer
139                                              Fetal growth restriction (FGR) affects >200,000 pregnanc
140                                              Fetal growth restriction (FGR) affects 5% to 10% of newb
141                                              Fetal growth restriction (FGR) affects 5-10% of pregnanc
142                                              Fetal growth restriction (FGR) affects around 5% of preg
143 folate and choline status resulted in severe fetal growth restriction (FGR) and impaired fertility in
144                                              Fetal growth restriction (FGR) and maternal supine going
145                                              Fetal growth restriction (FGR) and pre-eclampsia are sev
146                                              Fetal growth restriction (FGR) and preeclampsia (PE) are
147 ed with complications of pregnancy including fetal growth restriction (FGR) and preeclampsia.
148 tal exposure to disinfection by-products and fetal growth restriction (FGR) and preterm birth in the
149 eenage motherhood and short birth intervals, fetal growth restriction (FGR) and preterm birth, child
150  pathways involved in the pathophysiology of fetal growth restriction (FGR) and small for gestational
151                                              Fetal growth restriction (FGR) and stillbirth are associ
152 vanced maternal age (AMA) are susceptible to fetal growth restriction (FGR) and stillbirth.
153 ative protection against altitude-associated fetal growth restriction (FGR) and the positive selectio
154  by pre-eclampsia (PE), preterm birth (PTB), fetal growth restriction (FGR) and/or macrosomia resulti
155                        Preeclampsia (PE) and fetal growth restriction (FGR) are associated with impai
156 The placenta of pregnancies whose outcome is fetal growth restriction (FGR) are characterized by abno
157                        Preeclampsia (PE) and fetal growth restriction (FGR) are serious complications
158  was to identify metabolites associated with fetal growth restriction (FGR) by examining early and la
159                BACKGROUNDSevere, early-onset fetal growth restriction (FGR) causes significant fetal
160 nancies complicated by preeclampsia (PE) and fetal growth restriction (FGR) compared with control thi
161                        Preeclampsia (PE) and fetal growth restriction (FGR) complicate 5-10% of pregn
162 n of placental vessel networks in normal and fetal growth restriction (FGR) complicated pregnancies.
163 regnancies complicated by severe early-onset fetal growth restriction (FGR) exhibit diminished vascul
164 ion complicated by chronic fetal hypoxia and fetal growth restriction (FGR) increases a prenatal orig
165 mely delivery of infants suspected of having fetal growth restriction (FGR) is a balance between prev
166                                              Fetal growth restriction (FGR) is a common outcome in hu
167                                              Fetal growth restriction (FGR) is a major determinant of
168                                              Fetal growth restriction (FGR) is a major risk factor fo
169                                              Fetal growth restriction (FGR) is a pregnancy complicati
170                                              Fetal growth restriction (FGR) is a significant risk fac
171                                              Fetal growth restriction (FGR) is associated with a subo
172                                              Fetal growth restriction (FGR) is associated with aberra
173                                              Fetal growth restriction (FGR) is associated with global
174                                              Fetal growth restriction (FGR) is associated with reduce
175 ht is challenging, and the detection rate of fetal growth restriction (FGR) is low.
176 g fetus and may impact postnatal health, and fetal growth restriction (FGR) is often seen co-occurrin
177                                              Fetal growth restriction (FGR) is the major single cause
178                                              Fetal growth restriction (FGR) is the most common risk f
179 addition, elevated Phlda2 was found to drive fetal growth restriction (FGR) of transgenic offspring a
180                      However, placentas from fetal growth restriction (FGR) pregnancies are character
181                                              Fetal growth restriction (FGR) remains one of the main o
182                                              Fetal growth restriction (FGR) results from placental in
183 ce of preterm pre-eclampsia (< 37 weeks) and fetal growth restriction (FGR) was increased (1.8% vs. 0
184 d pre-eclamptic pregnancies complicated with fetal growth restriction (FGR) with and without villitis
185 of the most common and preventable causes of fetal growth restriction (FGR), a condition in which a f
186                                              Fetal growth restriction (FGR), a major risk factor for
187 ntas from pregnancies complicated by PE with fetal growth restriction (FGR), and (2) suppressyn secre
188 m (PAS), PPH, placenta previa, hysterectomy, fetal growth restriction (FGR), and preterm birth (PTB).
189  (PE), spontaneous preterm birth (sPTB), and fetal growth restriction (FGR), and to identify causes o
190                     They are associated with fetal growth restriction (FGR), but previous studies hav
191 d with adverse pregnancy outcomes, including fetal growth restriction (FGR), due in part to reduction
192 e are two pathways to LBW, preterm birth and fetal growth restriction (FGR), with the FGR pathway res
193 s a major cause of antepartum stillbirth and fetal growth restriction (FGR).
194 ral artery remodeling (ISAR) with or without fetal growth restriction (FGR).
195 esponses, both of which were associated with fetal growth restriction (FGR).
196 erapeutic strategies to treat and/or prevent fetal growth restriction (FGR).
197 4 cases of preeclampsia (PE) and 56 cases of fetal growth restriction (FGR).
198  that may be indicative of local ischemia or fetal growth restriction (FGR).
199  normal pregnancies and those complicated by fetal growth restriction (FGR).
200  putative aetiologies in the pathogenesis of fetal growth restriction (FGR); however, the regulating
201 ry transformation occurs in preeclampsia and fetal growth restriction (FGR); these processes are not
202                                              Fetal growth restriction (FGR, <0.46 g) was defined as f
203  preterm birth (<37 weeks of gestation), and fetal growth restriction (FGR; liveborn with birthweight
204  maternal inflammation such as preeclampsia, fetal growth restriction, gestational diabetes, and bact
205  SGA have an increased VCDR, suggesting that fetal growth restriction has a lasting impact on optic d
206                                              Fetal growth restriction has also been proposed, althoug
207 6-32 weeks of gestation who had very preterm fetal growth restriction (ie, low abdominal circumferenc
208  Campylobacter rectus infection that induces fetal growth restriction in a mouse model also compromis
209 us infection increases fetal resorptions and fetal growth restriction in a mouse model.
210                   Placental sO2 was lower in fetal growth restriction in an angiotensin-converting en
211 o the air pollutant ozone is associated with fetal growth restriction in humans and rodents.
212 , fetal and placental weights, and increased fetal growth restriction in IL33 deficient rats.
213 cific transcript alone (Igf2P0(+/-)) lead to fetal growth restriction in mice.
214 mplex (OR, 31.8; 95% CI, 4.3-236.3) CHD, for fetal growth restriction in noncomplex (OR, 1.6; 95% CI,
215 ability; Sildenafil does not protect against fetal growth restriction in the chick embryo, supporting
216 ereas, placental growth restriction precedes fetal growth restriction in the Igf2P0(+/-) mouse.
217  cells is supported by Hmox1 and ameliorates fetal-growth restriction in Hmox1 deficiency.
218                                  Sequelae of fetal growth restriction include metabolic disease as we
219                                   Markers of fetal growth restriction included biometric ratios, uter
220                         Perinatal stress and fetal growth restriction increase the risk of neonatal h
221 uartile, low birth weight increased >5-fold, fetal growth restriction increased >6-fold, and infant b
222 onatal glucose homeostasis and is altered by fetal growth restriction induced by maternal undernutrit
223                                              Fetal growth restriction is a leading cause of stillbirt
224                                              Fetal growth restriction is a major determinant of adver
225                                              Fetal growth restriction is associated with a smaller av
226 ht that severe COVID-19 during pregnancy and fetal growth restriction is associated with heightened v
227 e on critical exposure windows and timing of fetal growth restriction is limited.
228                                 Intrauterine fetal growth restriction (IUGR) is often associated with
229 quartiles had offspring with third-trimester fetal growth restriction, leading to a smaller head circ
230            Epidemiological data suggest that fetal growth restriction, maternal factors such as smoki
231 ommon pregnancy complication associated with fetal growth restriction, may initiate fibrotic and meta
232 l-recessive disorder characterized by severe fetal growth restriction, microcephaly, a distinct facia
233                     Screening procedures for fetal growth restriction need to identify small babies a
234 et and provide a potential mechanism for the fetal growth restriction observed in women who use canna
235 and consisted of 1 or more of the following: fetal growth restriction, oligohydramnios, hypertensive
236 ndependent of the hypoxic pregnancy inducing fetal growth restriction or elevations in maternal or fe
237     This study explores associations between fetal growth restriction or excessive fetal growth, alon
238 ternal physiology, metformin did not prevent fetal growth restriction or placental ageing.
239 lve impaired placental function, either with fetal growth restriction or preterm labour, or both.
240       However, it is not clear whether it is fetal growth restriction or the accelerated postnatal gr
241 ant pain, headache with visual disturbances, fetal growth restriction, or abnormal maternal blood tes
242 et count syndrome, intrauterine fetal death, fetal growth restriction, or placental abruption who had
243 come defined as preeclampsia, preterm birth, fetal growth restriction, or placental abruption.
244  gestation without congenital malformations, fetal growth restriction, or severe postnatal morbidity.
245  seizure, medically indicated preterm birth, fetal-growth restriction, or perinatal death.
246 by AICAR partially prevented hypoxia-induced fetal growth restriction (P < 0.01), due in part to incr
247 p for trend < 0.001) and a decreased risk of fetal growth restriction (p for trend < 0.05).
248 tric complications, including preterm birth, fetal growth restriction, placental abruption, and still
249 d by complications such as pre-eclampsia and fetal growth restriction, placental ageing is notably ac
250 cause of maternal hypertension in pregnancy, fetal growth restriction, premature birth, and fetal and
251 gnancy, and neonatal complications including fetal growth restriction, preterm birth and stillbirth.
252 22% of cases) included respiratory distress, fetal growth restriction, preterm birth, AIHA of the new
253 omposite adverse perinatal outcome including fetal growth restriction, preterm birth, low Apgar score
254  explored the associations of abruption with fetal growth restriction, preterm delivery, and perinata
255 tin are associated with an increased risk of fetal growth restriction, preterm delivery, and preeclam
256 s include alloimmunization, opioid exposure, fetal growth restriction, preterm delivery, and stillbir
257 ing that cardiovascular disease is linked to fetal growth restriction rather than to premature birth.
258 GT/GT) mice died perinatally associated with fetal growth restriction, reduced hepatic glycogen store
259 in mice to determine whether hypoxia-induced fetal growth restriction reduces placental PPAR-gamma pr
260 on in pregnancies complicated by hypoxia and fetal growth restriction remain poorly understood.
261 ow that fetal growth acceleration as well as fetal growth restriction, resulting from between-breed e
262 his correlation to recurrent miscarriage and fetal growth restriction, revealing the common mechanism
263 UtA) blood flow and relative protection from fetal growth restriction seen in altitude-adapted Andean
264 nancy complications such as preeclampsia and fetal growth restriction, since defects in maturation of
265 t undernutrition in the aggregate--including fetal growth restriction, stunting, wasting, and deficie
266  of abnormalities detected prenatally and of fetal growth restriction than the control cases.
267                     We used a mouse model of fetal growth restriction, the placental-specific Igf2 kn
268  psychoactive ingredient in cannabis, causes fetal growth restriction, though the mechanisms are not
269 e than placental malaria per se, might cause fetal growth restriction, through impaired transplacenta
270 ly identification of factors contributing to fetal growth restriction to minimize detrimental outcome
271 and 6 days' gestation and severe early-onset fetal growth restriction to receive either sildenafil 25
272   Maternal exposure to Cd is associated with fetal growth restriction, trace element deficiencies, an
273 in placental villous tissue are increased in fetal growth restriction vs. placentas from women with n
274                               In this study, fetal growth restriction was induced by maternal inhalat
275 eatures with human recurrent miscarriage and fetal growth restriction, we identified tissue factor (T
276 show that lowland mice experience pronounced fetal growth restriction when challenged with gestationa
277                 Here, using a mouse model of fetal growth restriction where maternal protein intake i
278       Maternal undernutrition contributes to fetal growth restriction, which increases the risk of ne
279 osure resulted in pups born with symmetrical fetal growth restriction, with catch up growth by post-n
280  C57Bl/6J sires and CD-1 dams do not exhibit fetal growth restriction, with male fetuses developing s
281  anomalies, suggesting an effect of relative fetal growth restriction within families.
282  example, maternal smoking (Z) is a cause of fetal growth restriction (X), which subsequently affects

 
Page Top