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1 acterized by uteroplacental ischaemia and/or fetal hypoxia.
2 disease in pregnancy complicated by chronic fetal hypoxia.
3 disease in pregnancy complicated by chronic fetal hypoxia.
4 ect fetal growth in pregnancy complicated by fetal hypoxia.
5 by placental insufficiency, which results in fetal hypoxia.
6 acterized by uteroplacental ischaemia and/or fetal hypoxia.
7 genes and an interaction of these genes with fetal hypoxia.
8 characterized by placental insufficiency or fetal hypoxia.
9 d umbilical blood flow can cause significant fetal hypoxia.
10 on in human pregnancy complicated by chronic fetal hypoxia.
11 spring of pregnancies complicated by chronic fetal hypoxia.
12 spring in pregnancies complicated by chronic fetal hypoxia.
13 be given to pregnancies affected by chronic fetal hypoxia.
14 period and whether it is affected by chronic fetal hypoxia.
18 important objective indicator of intrapartum fetal hypoxia and is used to predict neonatal morbidity
19 The authors examined whether a history of fetal hypoxia and other obstetric complications elevated
20 epression in a sex-dependent manner, linking fetal hypoxia and pathophysiological consequences in the
21 mine whether obesity during pregnancy causes fetal hypoxia and to explore potential underlying mechan
22 ss-induced placental pathology, accompanying fetal hypoxia, and neuroproliferative defects in the fet
23 maternal obesity-induced iron deficiency and fetal hypoxia are important mechanisms by which obesity
24 o (3.90+/-0.33 vs. 2.12+/-0.26, P<0.05), and fetal hypoxia (arterial PO2; 12.79+/-0.97 vs. 18.65+/-1.
25 term delivery and elicited further perinatal fetal hypoxia by placing maternal rats in 14% FiO(2) 3 h
30 dult females who had been exposed to chronic fetal hypoxia had significantly reduced somatic ovarian
31 scular system, and the importance of chronic fetal hypoxia in triggering a developmental origin of fu
32 ) spectroscopy for detecting and quantifying fetal hypoxia in utero is demonstrated in a pregnant ewe
33 y take us closer to therapeutic treatment of fetal hypoxia-induced CNS disorders and possibly other f
34 The present study tested the hypothesis that fetal hypoxia-induced methylation of cytosine-phosphate-
45 results provide new insight into how chronic fetal hypoxia may initiate fibrotic and metabolic liver
46 ce a model whereby the neurotoxic effects of fetal hypoxia may lead to an early onset of schizophreni
47 understand the impact of exposure to chronic fetal hypoxia on the developing female reproductive syst
48 hypothesis that iFHRV is affected by chronic fetal hypoxia, one of the most common adverse outcomes o
55 uteroplacental insufficiency and subsequent fetal hypoxia significantly increased cerebral Bax mRNA
58 l volumes were seen in those who experienced fetal hypoxia than in those who did not, a difference no
59 dulthood in gestation complicated by chronic fetal hypoxia, the most common adverse consequence in hu
60 ons of genetic predisposition and history of fetal hypoxia to hippocampal volume in patients with psy
61 complications of pregnancy, such as chronic fetal hypoxia, trigger a fetal origin of cardiovascular
62 complications of pregnancy, such as chronic fetal hypoxia, trigger a fetal origin of cardiovascular