ライフサイエンスコーパス: fetal membrane

1 omprises the maternal decidua, placenta, and fetal membranes.

2 E (sPE) on the smooth chorion portion of the fetal membranes.

3 gulation of proinflammatory cytokines in the fetal membranes.

4 s (MMPs) and prostaglandin (PG) synthesis in fetal membranes.

5 is of preterm labor and premature rupture of fetal membranes.

6 inases (MMPs) and prostaglandin synthesis in fetal membranes.

7 of pregnant women with premature rupture of fetal membranes.

8 chlamydial inclusions in the endometrium and fetal membranes.

9 mmation and preterm premature rupture of the fetal membranes.

10 lating progesterone signalling and weakening fetal membranes.

11 The wound healing capacity of the fetal membranes after spontaneous or iatrogenic membrane

12 ory response (FIR) theories initiated by the fetal membranes (amniochorion) at the choriodecidual int

13 We examined the contribution of the fetal membranes, amnion and chorion, to human embryonic

14 spiked samples was between 95% and 103% for fetal membranes and between 99% and 114% for placenta.

15 al chorioamnionitis (HCA) is an infection of fetal membranes and complicates 5.2% to 28.5% of all liv

16 d by transmission electron microscopy of the fetal membranes and in cells exposed to cigarette smoke

17 skeletal muscle, and, to a lesser degree, in fetal membranes and other tissues of infected fetuses.

18 for placenta and 1.19 + or - 0.68 pmol/g for fetal membranes, and 0.93 + or - 0.28, 0.88 + or - 0.33,

19 Pghs2, and Mpges1 expression in placenta and fetal membranes, and it elevated amniotic fluid IL-1beta

20 roduction of proparturition cytokines to the fetal membranes are reported, cellular level events that

21 Our results suggested that fetal membranes are the probable source of AF sIL-6R and

22 nd senescence-associated inflammation of the fetal membranes as reported mechanisms related to pPROM.

23 ed from the altered (cervix zone) and intact fetal membranes at term and before labour were used.

24 drostenedione induced maternal endocrine and fetal membrane biochemical changes, and alteration of my

25 mediators on E17 in the cervix, uterus, and fetal membranes but not in the placenta.

26 Inflammation can also arise in fetal membrane cells (amnion/chorion) due to OS-induced

27 This review discusses derangements to fetal membrane cells (physiologically and pathologically

28 vity but did not produce preterm delivery or fetal membrane changes.

29 e model, we found that small ruptures of the fetal membrane closed within 72 h whereas healing of lar

30 aternal blood, amniotic fluid, placenta, and fetal membranes collected during Caesarean section (n=14

31 l line and in explant cultures of term human fetal membranes containing the CDI, the prolabor inflamm

32 ed both survivin and Bcl-xL, indicating that fetal membranes could foster persistent viral infection.

33 on arising from cellular derangements at the fetal membrane-decidual interface (feto-maternal interfa

34 labor and with preterm premature rupture of fetal membranes did not achieve statistical significance

35 These alterations occur in normal fetal membranes during late pregnancy, in preparation fo

36 d significantly in the placenta, uterus, and fetal membranes during PGN+poly(I:C)-induced preterm lab

37 iabetes in pregnancy compromise maternal and fetal membrane essential fatty acids (FAs).

38 It is debated whether these fetal membranes evolved in eggs on land as an adaptation

39 In human fetal membranes (FM) collected immediately after birth f

40 mnionitis, inflammation of the placenta, and fetal membranes (FMs) are commonly observed in preterm b

41 Chorioamnionitis, premature rupture of fetal membranes (FMs), and subsequent preterm birth are

42 Chorioamnionitis, inflammation of the fetal membranes (FMs), is a major risk factor and is cha

43 phagy-inducing factor LC3B were performed in fetal membranes from pPROM, term not in labor, and term

44 >= 37 weeks) and preterm (33-36 weeks) human fetal membranes from ruptured and non-ruptured regions.

45 PGE2 content was increased significantly in fetal membranes from thrombin-injected animals.

46 We investigated premature senescence of fetal membranes in women with pPROM and spontaneous PTB

47 Senescence in placenta/fetal membranes is a normal phenomenon linked to term pa

48 The integrity of human fetal membranes is crucial for harmonious fetal developm

49 Chorioamnionitis, inflammation of fetal membranes, is an important cause of preterm birth

50 ibrillar collagens imparting strength to the fetal membranes, is expressed by leukocytes and chorioni

51 rgan-on-a-chip (FMi-OOC), which contains the fetal membrane layers.

52 strate significant effects of the mother and fetal membranes on pregnancy outcome, with possible impl

53 CI): 1.1, 4.9), preterm premature rupture of fetal membranes (OR = 3.6, 95% CI: 1.5, 8.7), and cervic

54 At term, fetal membranes overlying the cervix, the future site of

55 rived conditioned media supernatant weakened fetal membranes (P < 0.05), which MPA inhibited.

56 ntrations were found in placenta compared to fetal membranes (P<0.0001), in umbilical vein compared w

57 Mechanical and inflammatory signals in the fetal membrane play an important role in extracellular m

58 Fetal membranes possess distinctive properties that can

59 Preterm, prelabor rupture of the human fetal membranes (pPROM) is involved in 40% of spontaneou

60 Preterm, prelabour rupture of the fetal membranes (pPROM) is the commonest antecedent of p

61 has linked preterm premature rupture of the fetal membranes (PPROM) to placental abruption.

62 ies were 42 and 36% for the placenta and the fetal membranes, respectively.

63 The human fetal membrane restricted wild-type and LAV replication,

64 RNA sequencing (RNA-seq) analysis of fetal membranes revealed that specific pathways involved

65 The mechanisms of normal and pathologic fetal membrane rupture are not well understood.

66 Thus, fetal membrane rupture is likely to be the result of bio

67 Premature fetal membrane rupture occurs not infrequently and is as

68 hemical and endocrine changes accompanied by fetal membrane rupture, cervical dilatation and live del

69 taglandin (PG) E(2) and F(2alpha), promoting fetal membrane rupture, cervical ripening, and uterine c

70 receptor 4 (TLR4), an actor in pathological fetal membrane rupture, is controlled.

71 th, second trimester loss, preterm premature fetal membrane rupture, or a history of a cervical proce

72 ometrial contraction, cervical dilation, and fetal membrane rupture.

73 V-1 from mother to infant increases when the fetal membranes rupture more than four hours before deli

74 t membranes (<34 weeks) and the inducibility fetal membrane senescence phenotype by oxidative stress

75 Comparison with a prior cohort of fetal membranes shows that acute inflammation only takes

76 Interstitial collagen gives fetal membranes tensile strength, and membrane rupture h

77 unknown role for the gene in the yolk sac, a fetal membrane that is the site of hematopoiesis and is

78 of the cervix and biochemical changes in the fetal membranes that lead to rupture.

79 In the placenta and fetal membranes, the rs2280964 major allele homozygotes

80 Fetal membranes usually rupture during the process of la

81 The amniotic egg with its complex fetal membranes was a key innovation in vertebrate evolu

82 ry cytokine genes Il1b, Il6, Tnf and Il10 in fetal membranes was suppressed by (+)-naloxone, and cyto

83 receptor (CSF2R) in trophoblasts, promoting fetal membrane weakening and abruption-associated PPROM.

84 ved conditioned media supernatant effects on fetal membrane weakening were analyzed.

85 essed CSFR2, contributes to thrombin-induced fetal membrane weakening, eliciting abruption-related PP

86 Term fetal membranes were exposed to cigarette smoke extract

87 Sheep villi and fetal membranes were highly permissive to RVFV infection

88 lli, which are bathed in maternal blood, and fetal membranes, which encapsulate the amniotic cavity.