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1 n = 11-12) and IUGR (n = 12) late gestation fetal sheep.
2 cardiovascular development or growth in the fetal sheep.
3 od flow and oxygen consumption rates in IUGR fetal sheep.
4 an hematopoiesis after transfer to preimmune fetal sheep.
5 acid uptake was significantly lower in IUGR fetal sheep.
6 ovascular adaptations in chronically anaemic fetal sheep.
7 evelopment of cerebral OXPHOS in hypothyroid fetal sheep.
8 e adipose tissue development of midgestation fetal sheep.
9 tic uptake following metformin infusion into fetal sheep.
10 ephalic vasculature of the intact, near term fetal sheep.
11 vasculature served by the carotid artery of fetal sheep.
12 factors reduces myofibre hypertrophy in IUGR fetal sheep.
13 we transplanted clonally derived BMSCs into fetal sheep.
14 hydrate metabolism and insulin signalling in fetal sheep.
15 or intrahepatic (n = 6) route into preimmune fetal sheep.
16 ion model in the instrumented 0.65 gestation fetal sheep.
17 -pseudotyped retroviruses for injection into fetal sheep.
18 nsplanted by intraperitoneal injections into fetal sheep.
19 anges of CBF in the chronically instrumented fetal sheep.
20 product of the gene c-fos in late-gestation fetal sheep.
21 resistance artery function of mid-gestation fetal sheep.
22 cortical vascular resistance by ~15 % in the fetal sheep.
23 lanted in the cerebral cortices of near-term fetal sheep.
24 rticotropic hormone (ACTH) in late-gestation fetal sheep.
25 mmunodeficiency (NOD-SCID) mice or preimmune fetal sheep.
26 s engrafted primary, secondary, and tertiary fetal sheep.
27 by improved fetal lung function after 4 d in fetal sheep.
28 ta reported in this study of unanaesthetised fetal sheep (1) show that minute-by-minute analyses of h
31 In unanaesthetized chronically instumented fetal sheep (118-121 days gestation) we investigated the
33 duced for 30 min in chronically catheterized fetal sheep (125 +/- 3 days), with or without ketamine (
34 tact and nine carotid sinus denervated (CSD) fetal sheep (125-128 days gestation) we measured heart r
35 ebral blood flow in chronically instrumented fetal sheep (127-135 days gestation, term approximately
36 was measured in cardiomyocytes isolated from fetal sheep (135 day gestational age) in response to 100
39 VIP immunostaining was extremely robust in fetal sheep adrenal cortical neurofibers and cells while
40 eta-cell responsiveness in hypoglycaemic (H) fetal sheep and ascertain whether a 5 day euglycaemic re
41 sent throughout the adrenal cortices of both fetal sheep and baboons, were heavily innervated by VIP-
42 olypeptide (VIP) and tyrosine hydroxylase in fetal sheep and fetal baboon adrenal cortices and medull
43 t the liver is a site of metformin action in fetal sheep and macaques, given relatively abundant OCT1
44 mboxane mimetic stimulates ACTH secretion in fetal sheep, and that the endogenous production of throm
47 IL-6 mAbs and systemically infused mAbs into fetal sheep at 126 days of gestation after exposure to b
49 med in cerebral cortical cell membranes from fetal sheep at 88, 120, and 136 d gestation (term = 150
51 the endogenous production of new glucose by fetal sheep at a time when the amount of glucose transfe
52 raperitoneal or intrahepatic route (IH) into fetal sheep at concentrations ranging from 1.1-2.6 x 10(
53 er with a fluorescent O2 probe, in near-term fetal sheep at low altitude (n = 8) and those acclimatiz
54 (~10 min) umbilical cord occlusion (UCO) in fetal sheep at ~0.88 gestation on fetal plasma cortisol
55 of measuring cerebral metabolic rate in the fetal sheep based on heat production in a local region o
56 In pancreatic islets isolated from intact fetal sheep, beta cell proliferation in vitro was reduce
58 del, and human cells harvested from chimeric fetal sheep bone marrow have been shown to successfully
59 androgen, estrogen, and progesterone in the fetal sheep brain during the critical period for sexual
62 episode of complete UCO late in gestation in fetal sheep can result in prolonged effects on fetal bra
64 ized that suppression of MEIS1 expression in fetal sheep cardiomyocytes leads to a metabolic switch a
65 nsin II (Ang II) stimulates proliferation in fetal sheep cardiomyocytes when growth is dependent on t
69 normal sera from neonatal and adult, but not fetal, sheep contained IgM and IgG XNA reactive with rat
70 medulla were smaller in diameter compared to fetal sheep cortex (1.22+/-0.13 vs. 2.93+/-0.34 microm,
72 and dyadic assembly proceed gradually during fetal sheep development, from 93 days of gestational age
73 y proceed gradually in cardiomyocytes during fetal sheep development, from 93 days of gestational age
74 distribution of ligand binding sites in the fetal sheep diencephalon indicated that the highest leve
75 distribution of adenosine A(2A) receptors in fetal sheep diencephalon, we have used a receptor autora
76 ysiological responses to acute hypoxaemia in fetal sheep during and following maternal treatment with
77 ic GHR mRNA has therefore been determined in fetal sheep during late gestation and after experimental
78 The results show that glucogenesis occurs in fetal sheep during late gestation in conditions in which
80 human mesenchymal stem cell population into fetal sheep early in gestation, before and after the exp
84 , transfer of CD34+/CD38+ cells to preimmune fetal sheep generated only short-term human hematopoiesi
85 We tested the hypothesis that high-altitude fetal sheep have evolved cardiovascular compensatory mec
86 tation intrauterine growth-restricted (IUGR) fetal sheep have fewer binucleated cardiomyocytes, refle
87 de that cardiac nerves in the late-gestation fetal sheep have minor influences on plasma renin activi
88 identification and pathway analysis of novel fetal sheep heart transcriptome splice variants is a fir
91 ateral ventricle of chronically instrumented fetal sheep in utero at 128 +/- 1 days gestation (term i
93 nd dexamethasone treatment of late-gestation fetal sheep, in doses similar to those employed clinical
95 , and that growth retardation in hypothyroid fetal sheep is associated with reductions in pancreatic
99 While cortisol is known to reduce growth in fetal sheep, its effects on the uteroplacental handling
100 10 days of umbilicoplacental embolization in fetal sheep, IUGR fetuses had elevated circulating long-
101 10 days of umbilicoplacental embolization in fetal sheep, IUGR fetuses had elevated circulating long-
102 term rise in tri-iodo-l-thyronine (T(3) ) in fetal sheep leads to the inhibition of proliferation and
104 in cerebral vascular response to hypoxia in fetal sheep may not be attributed to changes in vascular
106 PS), we validated this hypothesis in primary fetal sheep microglia cultures re-exposed to LPS in pres
109 during basal and insulin clamp periods in a fetal sheep model of placental insufficiency and IUGR.
110 y demonstrated in a chronically catheterized fetal sheep model that experimentally elevated glucagon
116 ration induced by intraamniotic endotoxin in fetal sheep occurred without an increase in fetal plasma
121 tic hernias were created surgically in seven fetal sheep on gestational day 100 (term = 145 days).
125 have since used the chronically instrumented fetal sheep preparation to investigate the fetal compens
126 harvested from the brains of third trimester fetal sheep previously exposed in the second trimester t
128 Chronically catheterized late gestation fetal sheep received an intravenous infusion of glucagon
129 s were injected into a total of 14 preimmune fetal sheep recipients using the amniotic bubble techniq
130 Prenatal glucocorticoid plus T4 treatment of fetal sheep results in improvements in oxygenation, gas
132 The present study utilized late gestation fetal sheep, stereotaxic methodology and retrograde axon
133 rs occurred in greater (P<0.05) frequency in fetal sheep than in fetal baboons (14.82+/-3.10 vs. 0.84
134 3 mmHg) steady-state conditions in near-term fetal sheep that had undergone either surgical sham or b
135 al cortex (FCTX) and olfactory bulbs (OB) of fetal sheep that were delivered on day 64 of gestation.
138 eriments were carried out in unanaesthetized fetal sheep to evaluate the significance of non-N-methyl
140 133-selected cells engraft successfully in a fetal sheep transplantation model, and human cells harve
141 conclusion, rodent grafts transplanted into fetal sheep undergo HAR, likely through direct activatio
148 tion control (CON) (n = 8) and IUGR (n = 13) fetal sheep were catheterized with aortic and femoral ca
154 latory responses to acute hypoxia, near-term fetal sheep were instrumented with laser Doppler probes
156 Chronically instrumented, late-gestation fetal sheep were prepared to: (1) characterize cardiovas
157 effects on fetal brain functions are unknown Fetal sheep were subjected to umbilical cord occlusion (
159 rticoids correct the pulmonary immaturity of fetal sheep with CDH by physiologic, biochemical, and hi