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1  most common malformations identified in the fetal stage.
2 nhibit upstream Vgamma rearrangements at the fetal stage.
3 ssion of the gamma-gene was recruited to the fetal stage.
4  results in hematopoietic defects during the fetal stages.
5 ng in the development of the prostate during fetal stages.
6 tinct stages, marking the entire CCS by late fetal stages.
7 an exquisitely folded columnar epithelium in fetal stages.
8 mRNA content of IGF system components during fetal stages.
9 a in the early embryonic CNS compared to the fetal stages.
10 ells persisted on in vivo differentiation to fetal stages.
11  maternal allele in various mouse tissues at fetal stages.
12 ayer of the small intestine from early human fetal stages.
13 uration process from early fetal to late mid-fetal stages.
14 Hnf4 gene loci to elevate HNF4 expression at fetal stages.
15                         In contrast, at late fetal stages (18.5 dpc), cells that were Shh-responsive
16 hybridization studies in human embryonic and fetal stages (35 days post-ovulation to 9 weeks post-con
17 icate that ciliogenesis is initiated in late fetal stages after neuroblast migration, when the mother
18 s retain their undifferentiated state during fetal stage and become adult Leydig cells in post-pubert
19 s subjected to severe food restriction since fetal stage and controls were transferred to a moderatel
20  findings on white matter development at the fetal stage and in infancy as well as DTI applications f
21 the family manifests with early onset at the fetal stage and is associated with neonatal sudden death
22 gamma2 gene segment predominates in the late fetal stages and beyond, in cells destined for the secon
23     To address the function of Pax3 in later fetal stages and in specific adult tissues, we generated
24 ich expressed Myf5 for the first time at the fetal stage ( approximately E15).
25 he development and growth of prostate during fetal stages as well as during prostate carcinogenesis a
26 ollagen content increased from early to late fetal stages but was subsequently unchanged, whereas ela
27                 Proliferation and subsequent fetal stages can also be disrupted by maternal immune ac
28 ld cells that typically show the behavior of fetal stage cells.
29 tors and dramatic reductions in mTECs during fetal stages, consistent with defects in mTEC specificat
30                 In contrast to embryonic and fetal stages, deletion of the DRR resulted in a pronounc
31                              However, during fetal stage development, investigating further biologica
32                           During mid to late fetal stages (embryonic day 15.5-18.5), epithelial diffe
33                The abundance of glutamate at fetal stages, especially in the ventricular zone, is con
34 ature of ASD and provides the first in vitro fetal-stage explanation for in vivo early brain overgrow
35 tor element, inhibit inappropriate embryonic/fetal stage expression of the beta-globin gene.
36 yte-derived signals are required during late fetal stages for continued development and maintenance o
37 n factor motifs are the most abundant in the fetal stages (>E16.5).
38                           In the mid-to-late fetal stage, hepatocytes have few metabolic functions as
39                       At later embryonic and fetal stages, however, no defect in MyoD expression was
40 age I or II hepatoblastoma without 100% pure fetal stage I or small-cell undifferentiated histology;
41                   Thymic hypoplasia at later fetal stages in compound mutants was associated with inc
42 esulted in a hypoplastic thymus-evident from fetal stages into adulthood-in which a dramatic increase
43  monocytes arrive in the lungs during a late fetal stage, maturing to alveolar macrophages through a
44 ternal allele becomes hypermethylated during fetal stages, methylation of the maternal allele begins
45 ly from cells present in the brain since the fetal stage of development, or if there is further input
46  the control of gamma-gene expression in the fetal stage of development.
47 rom embryonic stem cells mimics an embryonic/fetal stage of hematopoiesis.
48          We show here that circumventing the fetal stage of hematopoietic stem cell (HSC) development
49 lly expressed at high levels only during the fetal stage of human development.
50 ose expression is normally restricted to the fetal stage of ventricular development.
51  bud precursors, and heart tube, but by late fetal stages of development, MNF is down-regulated withi
52 tion and cell proliferation at embryonic and fetal stages of heart development.
53 e nucleus represents the early embryonic and fetal stages of lens development, we infer that BHMT exp
54 ired for normal progression through the late fetal stages of lung development that culminate in alveo
55 id progenitor expansion during adult but not fetal stages of murine development.
56  the myogenic determination gene Myf5 during fetal stages of myogenesis.
57 rant morphology in deep-layer neurons during fetal-stage organoid development.
58 ss the ability to mature beyond mid- to late-fetal stages potentially limits their utility.
59  the aging of the brain is programmed at the fetal stage remains untested.
60                            Specifically, new fetal stage-specific contacts were uncovered between a r
61 f DS fetal progenitors, revealing a complex, fetal stage-specific regulatory network.
62 f the human brain occur in the embryonic and fetal stages-stages that are difficult to access and inv
63 opment, and that they retain this state from fetal stages through meiosis and gametogenesis.
64 orebrain and hindbrain during early and late fetal stages, through an exogenous source of 5-HT which
65 alyze changes in pelvic morphology from late fetal stages to adulthood in a known-age/known-sex foren
66 ha and NRXN1-beta, in prefrontal cortex from fetal stages to aging.
67               Notably, immune cells in early fetal stages were predominantly M2-type macrophages, und
68 systems begin to develop, and finally to the fetal stage where growth and physiological maturation oc
69  in the most caudal part of the hindbrain at fetal stages, where it is maintained until adulthood.
70 Fs) are specifically up-regulated in the mid-fetal stage, which may have contributed to the evolution
71 tream Vgamma3 and Vgamma4 genes in the early fetal stage, which switches to a preference for rearrang