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1 inogen to fibrin and subsequent formation of fibrin degradation products.
2 ng time or platelet count but exhibited less fibrin degradation products (1/6 animals) than did contr
3                    An increase in fibrinogen/fibrin degradation products and a rise in plasminogen ac
4 n degrades the crosslinked fibrin to release fibrin degradation products and expose the D-dimer antig
5 to the fibrin network, breaking it down into fibrin degradation products and releasing the entrapped
6                                 In contrast, fibrin degradation products and the molecular markers th
7 emostatic components (fibrinogen, platelets, fibrin degradation products) and inflammatory mediators
8 on to a 3-BRS comprising C-reactive protein, fibrin degradation product, and heat shock protein-70 im
9 stment for risk factors, C-reactive protein, fibrin degradation product, and heat shock protein-70.
10  thromboplastin time, fibrinogen, fibrinogen-fibrin degradation products, and AT activity.
11 ing levels of biomarkers C-reactive protein, fibrin degradation products, and heat shock protein-70 r
12 ular coagulation (activated prothombin time, fibrin degradation products, and one-step prothombin tim
13   Leukocyte changes, fibrinogen consumption, fibrin degradation products, and vital signs were simila
14 lobal blood coagulation markers (fibrinogen, fibrin-degradation products, APTT), and preserved the en
15 ing decreases in fibrinogen and increases in fibrin degradation products at 3 x 10(12)vp, precluded a
16  Previous studies have shown that fibrin and fibrin degradation products can have biologic effects on
17 , thrombin-antithrombin complex, crosslinked fibrin degradation product D-dimer, and thrombin-activat
18 letion, and production of high levels of the fibrin degradation product D-dimer.
19 and platelet pathways with the production of fibrin degradation products (D-dimer) and consumption of
20                 D-dimer antigen can exist on fibrin degradation products derived from soluble fibrin
21 artial thromboplastin time (PTT), fibrinogen/fibrin degradation products (FDP), and fibrinogen were u
22 s-high-sensitivity C-reactive protein (CRP), fibrin degradation products (FDP), and heat shock protei
23                                              Fibrin degradation products (FDPs) and interleukins 6 an
24 proteolysis of cross-linked fibrin generates fibrin degradation products (FDPs) with multiple biologi
25 e state with increased levels of D-dimer and fibrin degradation products (FDPs).
26                                   D-dimer, a fibrin degradation product, generated following activati
27              METHODS AND C-reactive protein, fibrin degradation product, heat shock protein-70, and s
28 ibrinogen concentrations and the increase in fibrin degradation products observed in the control grou
29 rediction rule were performed, and levels of fibrin degradation products (plasma D-dimers) were asses
30                         D-dimer is a soluble fibrin degradation product that results from ordered bre
31                   Reduced levels of D-dimer (fibrin degradation product) were evident in ticks fed on
32 ies suggest that histones and DNA bind large fibrin degradation products with 191 and 136 nM dissocia
33 e of substantial amounts of fibrin(ogen) and fibrin degradation products within intimal lesions, the