ライフサイエンスコーパス: fibroblast growth factor

1 ch as insulin-like growth factor 1 and basic fibroblast growth factor.

2 oss of kinase activity upon stimulation with fibroblast growth factor.

3 larization of the graft bed by agarose-basic fibroblast growth factor.

4 ulated autophagy through IL6, IL8, and basic fibroblast growth factor.

5 Fibroblast growth factor 1 (FGF1) has been shown to reve

6 e intracerebroventricular (icv) injection of fibroblast growth factor 1 (FGF1) induces remission of h

7 e intracerebroventricular (icv) injection of fibroblast growth factor 1 (FGF1), and the mediobasal hy

8 e intracerebroventricular (icv) injection of fibroblast growth factor 1 (FGF1).

9 with increased secretion of adiponectin and fibroblast growth factor 1 from adipose tissue, is expec

10 n vivo revealed that conditional deletion of fibroblast growth factor 10 (Fgf10) from Fgf10-expressin

11 evels of neurite markers betaIII tubulin and fibroblast growth factor 12, with differential effects i

12 t phosphorylation at these two sites impairs fibroblast growth factor 13 (FGF13)-dependent regulation

13 Fibroblast growth factor 14 (FGF14) is a functionally re

14 ein interactions (PPIs) between Na(v)1.6 and fibroblast growth factor 14 (FGF14) leads to impaired ex

15 Here, we show that fibroblast growth factor 14 (FGF14), previously describe

16 c approaches, we identified a motogenic cue, fibroblast growth factor 15 (FGF15), whose expression in

17 Farnesoid X receptor (FXR) induces fibroblast growth factor 15 (FGF15; human ortholog FGF19

18 size, altered BA composition, and increased fibroblast growth factor 15 production.

19 hepatic small heterodimer partner, and ileal fibroblast growth factor 15.

20 Fibroblast Growth Factor-15/19 (mouse FGF15, human FGF19

21 igh content cell-based screen, we identified fibroblast growth factor 16 (FGF16) as a potent inducer

22 (transforming growth factor beta 1), FGF18 (fibroblast growth factor 18), CTSK (cathepsin K), and IL

23 Serum fibroblast growth factor 19 (FGF19) and 7alpha-hydroxy-4

24 whether citrulline (CIT) and the enterokine fibroblast growth factor 19 (FGF19) are associated with

25 Fibroblast growth factor 19 (FGF19) is an ileum-derived

26 Fibroblast Growth Factor 19 (FGF19) is one of the most f

27 Effects of fibroblast growth factor 19 (FGF19) on hepatic transport

28 We show that fibroblast growth factor 19 (FGF19), a gut hormone, is r

29 clear translocation, while bile acid-induced fibroblast growth factor 19 (FGF19), acting via mTOR/ERK

30 excretion over 48 h; n = 4 studies assessed fibroblast growth factor 19 (FGF19).

31 ive feedback regulation by the ileal hormone fibroblast growth factor 19 (FGF19).

32 Postprandial fibroblast growth factor 19 (human FGF19, mouse FGF15) s

33 NGM282, an engineered fibroblast growth factor 19 analogue, rapidly and signif

34 cholic acid test performed better than serum fibroblast growth factor 19 and 7alpha-hydroxy-4-cholest

35 eptor and TGR5, the BA-induced gut hormones, fibroblast growth factor 19 and glucagon-like peptide 1,

36 The fibroblast growth factor 19 gene FGF19 has previously be

37 Aldafermin, an engineered analog of fibroblast growth factor 19, inhibits bile acid synthesi

38 rous genes, including an intestinal hormone, fibroblast growth factor-19 (FGF19; FGF15 in mice).

39 t found that prostaglandin E(2) (PGE(2)) and fibroblast growth factor 2 (FGF-2) -mediators known to i

40 of a number of HS-binding proteins including fibroblast growth factor 2 (FGF-2), and the chemokines C

41 he target genes for these regulators include fibroblast growth factor 2 (FGF2) and Ariadne RBR E3 ubi

42 Human fibroblast growth factor 2 (FGF2) and insulin-like growt

43 al concentrations of the neurotrophic factor fibroblast growth factor 2 (FGF2) are negatively associa

44 Using fibroblast growth factor 2 (FGF2) as a model system, we

45 ntly found in rodents that alcohol increases fibroblast growth factor 2 (FGF2) expression in the dors

46 We previously demonstrated that fibroblast growth factor 2 (FGF2) from adipose tissue st

47 Here, we demonstrate that fibroblast growth factor 2 (FGF2) from bone marrow strom

48 Fibroblast growth factor 2 (FGF2) has been shown to inte

49 Fibroblast growth factor 2 (FGF2) has previously been im

50 d that alcohol upregulates the expression of fibroblast growth factor 2 (FGF2) in dorsomedial striatu

51 Fibroblast growth factor 2 (FGF2) is crucial for the dev

52 ted neutrophils expressed substantially more fibroblast growth factor 2 (FGF2) than naive neutrophils

53 s IL-1beta and IL-18, growth factors such as fibroblast growth factor 2 (FGF2), redox enzymes such as

54 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2).

55 we established a role for ASMC-derived basic fibroblast growth factor 2 (FGF2b) and FGF receptor (FGF

56 s showed abnormal HS composition and altered fibroblast growth factor 2 signaling, which was rescued

57 (vascular endothelial growth factor), FGF2 (fibroblast growth factor 2), and their receptors VEGFR2

58 of tumor necrosis factor alpha, IL-8, IL-10, fibroblast growth factor 2, and IL-7 remained higher.

59 gamma-induced protein 10, IL-4, IL-9, IL-10, fibroblast growth factor 2, IL-7, IL-15, and transformin

60 ulated kinase phosphorylation in response to fibroblast growth factor 2, showing that changes in 6-O-

61 scorbic acid, bone morphogenic protein-2, or fibroblast growth factor 2.

62 viously reported that growth factors such as fibroblast growth factor-2 (FGF-2) and bone morphogeneti

63 VWF also binds to VEGF-A and fibroblast growth factor-2 (FGF-2) in human plasma and c

64 In this study, DPCs were treated with fibroblast growth factor-2 (FGF2) for 5-6 consecutive se

65 fference in VAT activity was associated with fibroblast growth factor-2 (FGF2) levels.

66 r (VEGF), brain-derived neurotrophic factor, fibroblast growth factor-2, and ciliary neurotrophic fac

67 demonstrated increased expression levels of fibroblast growth factor-2, transforming growth factor-b

68 of neuroprotective compounds, which include fibroblast growth factor-2, vascular endothelial growth

69 ed growth factor, platelet concentrates, and fibroblast-growth factor-2.

70 Endogenous fibroblast growth factor 20 (FGF20) supports maintenance

71 s a spreading Ectodysplasin A (EDA) wave and Fibroblast Growth Factor 20 (FGF20)-cell aggregate-based

72 t of 263 proteins analyzed at baseline, only fibroblast growth factor 21 (FGF-21) predicted weight lo

73 Greater decreases in plasma fibroblast growth factor 21 (FGF21) after 24-h fasting a

74 Pegbelfermin (BMS-986036), a PEGylated human fibroblast growth factor 21 (FGF21) analogue, has previo

75 h c-Jun NH2-terminal kinase (JNK) inhibiting fibroblast growth factor 21 (FGF21) and activating Bmal1

76 Fibroblast growth factor 21 (FGF21) controls metabolic o

77 Fibroblast Growth Factor 21 (FGF21) elicits an array of

78 Fibroblast growth factor 21 (FGF21) has emerged as an im

79 Fibroblast growth factor 21 (FGF21) induces weight loss

80 Fibroblast growth factor 21 (FGF21) is a hormone that is

81 reported an association between circulating fibroblast growth factor 21 (FGF21) levels and pericardi

82 Fibroblast growth factor 21 (FGF21) regulates energy exp

83 Fibroblast growth factor 21 (FGF21) signaling in the bra

84 circulating concentration of the hepatokine fibroblast growth factor 21 (FGF21) that regulates syste

85 epatic expression of the metabolic regulator fibroblast growth factor 21 (FGF21) was blunted by TCS.

86 ver expression and elevated plasma levels of fibroblast growth factor 21 (FGF21), a hepatokine known

87 es, markers of inflammation, serum levels of fibroblast growth factor 21 (FGF21), and activation of s

88 nsulin resistance with increased circulating fibroblast growth factor 21 (FGF21), elevated Fgf21 mRNA

89 expressed increased levels of E-cadherin and fibroblast growth factor 21 (FGF21), targets of sirtuin-

90 nd various hormone concentrations, including fibroblast growth factor 21 (FGF21), were assessed at ba

91 these effects require the metabolic hormone fibroblast growth factor 21 (FGF21).

92 Human fibroblast growth factor 21 (hFGF21) has been characteri

93 apies based on 3 longevity associated genes (fibroblast growth factor 21 [FGF21], alphaKlotho, solubl

94 KO mice had increased circulating levels of fibroblast growth factor 21 and adiponectin and were res

95 ng hyperglycemia and include augmentation of fibroblast growth factor 21 and glucagon-like peptide 1.

96 ression of genes involved in beta-oxidation: fibroblast growth factor 21 and peroxisome proliferator-

97 Interestingly, fasting induction of fibroblast growth factor 21 in liver was attenuated.

98 A low BCAA diet transiently induces FGF21 (fibroblast growth factor 21) and increases energy expend

99 ceptor gamma coactivator 1-alpha) and FGF21 (fibroblast growth factor 21).

100 the energy balance regulating hormone FGF21 (fibroblast growth factor 21).

101 ity index was 24% (P < 0.01) and circulating fibroblast-growth factor 21 was 21% higher (P < 0.05), w

102 Here, we report fasting-induced Fibroblast Growth Factor-21 (FGF21) signaling activates

103 Circulating levels of fibroblast growth factor-21 (FGF21) start increasing in

104 st growth factor 23 (iFGF23) into C-terminal fibroblast growth factor 23 (cFGF23), elevated levels of

105 hate, calcium, parathyroid hormone (PTH) and fibroblast growth factor 23 (FGF-23) were studied up to

106 e demonstrate systemic and direct effects of Fibroblast growth factor 23 (FGF23) and Klotho, which no

107 (3) elicited the expected increase of plasma fibroblast growth factor 23 (FGF23) and reduction of par

108 The rapid rise in circulating fibroblast growth factor 23 (FGF23) associated with kidn

109 s of the parathyroid hormone (PTH)-vitamin D-fibroblast growth factor 23 (FGF23) axis, creatinine, an

110 genetic analysis or measurement of levels of fibroblast growth factor 23 (FGF23) before treatment.

111 asma levels of the osteocyte-derived hormone fibroblast growth factor 23 (FGF23) have emerged as a po

112 Circulating levels of fibroblast growth factor 23 (FGF23) increase during the

113 Circulating levels of bone-derived fibroblast growth factor 23 (FGF23) increase early durin

114 fibroblast growth factor receptor (FGFR) and fibroblast growth factor 23 (FGF23) indicates that its b

115 f adults with the phosphaturic human hormone fibroblast growth factor 23 (FGF23) induces tubule expre

116 Fibroblast growth factor 23 (FGF23) is a bone-derived ho

117 Fibroblast growth factor 23 (FGF23) is often elevated in

118 Elevated fibroblast growth factor 23 (FGF23) levels, measured at

119 Klotho functions as a co-receptor for fibroblast growth factor 23 (FGF23) signaling, whereas i

120 Fibroblast growth factor 23 (FGF23) was initially charac

121 lcium, phosphate, parathyroid hormone (PTH), fibroblast growth factor 23 (FGF23), 25-hydroxyvitamin D

122 eocyte-derived, phosphate-regulating hormone fibroblast growth factor 23 (FGF23), a risk factor for p

123 iprocal manner by parathyroid hormone (PTH), fibroblast growth factor 23 (FGF23), and 1,25(OH)(2)D(3)

124 e is regulated by parathyroid hormone (PTH), fibroblast growth factor 23 (FGF23), and 1,25(OH)(2)D(3)

125 perphosphatemia is associated with increased fibroblast growth factor 23 (FGF23), arterial calcificat

126 cterised by elevated serum concentrations of fibroblast growth factor 23 (FGF23), hypophosphataemia,

127 on evolving concepts regarding the roles of fibroblast growth factor 23 (FGF23), inflammation and sy

128 ar bone osteocytes in Hyp mice overexpressed fibroblast growth factor 23 (Fgf23), its expression in m

129 h concentrations of circulating phosphatonin fibroblast growth factor 23 (FGF23), which causes renal

130 The discovery of fibroblast growth factor 23 (FGF23), which revolutionize

131 unique basal and parathyroid hormone (PTH)-, fibroblast growth factor 23 (FGF23)-, and 1,25(OH)2D3-me

132 D promotes production and cleavage of intact fibroblast growth factor 23 (iFGF23) into C-terminal fib

133 sm (including abnormalities in phosphate and fibroblast growth factor 23 [FGF23]) contribute to adver

134 ), and recent findings suggest that blocking fibroblast growth factor 23 actions may be the most effe

135 e blood of cKO mice had an elevated level of fibroblast growth factor 23 and reduced level of phospho

136 rminal pro-B-type natriuretic peptide level, fibroblast growth factor 23 level, estimated glomerular

137 Although FGF23 (fibroblast growth factor 23) is associated with heart fa

138 ns of certain molecules, such as phosphates, fibroblast growth factor 23, parathyroid hormone, sclero

139 rathyroid hormone and phosphatonins, such as fibroblast growth factor 23, regulate the activity of th

140 ency anemia, but certain formulations induce fibroblast growth factor 23-mediated hypophosphatemia.

141 The bone-derived hormone fibroblast growth factor-23 (FGF-23) activates complexes

142 n (mKL) and recognized as the coreceptor for fibroblast growth factor-23 (FGF23) and a circulating so

143 ercent tubular reabsorption of phosphate and fibroblast growth factor-23 (FGF23) at all CKD stages, a

144 Higher serum phosphate and fibroblast growth factor-23 (FGF23) levels may be modifi

145 related to mineral metabolism/calcification (fibroblast growth factor-23 and OPG [osteoprotegerin]),

146 ake stimulates parathyroid hormone (PTH) and fibroblast growth factor-23 secretion, increasing phosph

147 Fibroblast growth factor-23, 1,25-(OH)2-vitamin D3, and

148 Fibroblast growth factor 4 (FGF4) is the key signal driv

149 As, miR-155 is significantly upregulated and fibroblast growth factor 7 (FGF7) mRNA (target of miR-15

150 miR-155 inhibition increases diabetic wound fibroblast growth factor 7 expression in diabetic wounds

151 a precise and dynamic expression pattern of fibroblast growth factor 8 (Fgf8) in the HAA anlage, whi

152 terior to posterior (AP) patterning, whereas fibroblast growth factor 8 (Fgf8) is produced by the api

153 lls, induction of cellular HD5 expression by fibroblast growth factor 9 (FGF9) significantly inhibite

154 ypoxia-inducible factor 1alpha [HIF-1alpha], fibroblast growth factor 9 [FGF-9], and p53) is still tr

155 treated the graft/host interface with acidic fibroblast growth factor (aFGF) and chondroitinase ABC (

156 We uncovered 16 signaling pathways (such as fibroblast growth factor and Eph/ephrin pathways).

157 fies neural crest and placodes, modulated by fibroblast growth factor and Wnt.

158 study was to test the hypothesis that basic Fibroblast Growth Factor (bFGF) regulates SFD-related CN

159 Basic fibroblast growth factor (bFGF) released from a nanostru

160 at muscle fibers secrete and concentrate the fibroblast growth factor binding protein 1 (FGFBP1) at N

161 at muscle fibers secrete and concentrate the fibroblast growth factor binding protein 1 (FGFBP1) at N

162 vels of the reinnervation-promoting cytokine fibroblast growth factor binding protein 1 (FGFBP1) than

163 Fibroblast growth factor-binding protein 1 (FGFBP1) is a

164 tromal cell-derived factor 1alpha, and basic fibroblast growth factor concentrations in plasma.

165 Glycolytic activity downstream of fibroblast growth factor controls WNT signalling in the

166 ietic KDR(+)CD235a(-) mesoderm in a WNT- and fibroblast growth factor-dependent manner.

167 Endocrine fibroblast growth factors (eFGFs) control pathways that

168 Fibroblast growth factor-extracellular signal-regulated

169 eral plate mesoderm formation while reducing fibroblast growth factor/extracellular signaling-regulat

170 Levels of fibroblast growth factor (FGF) 1, FGF2, and FGF7 were si

171 BA and fibroblast growth factor (FGF) 19 levels (a surrogate fo

172 n human and murine pregnancies, by measuring fibroblast growth factor (FGF) 19/15 protein and mRNA le

173 es in serum phosphate, FC reduced C-terminal fibroblast growth factor (FGF) 23 compared with control.

174 m levels of the phosphate regulating hormone fibroblast growth factor (FGF) 23 have emerged as powerf

175 Fibroblast growth factor (FGF) 23 is a phosphaturic horm

176 uble protein, functions as a co-receptor for Fibroblast Growth Factor (FGF) 23, a known pro-inflammat

177 the patterning of feathers relies on coupled fibroblast growth factor (FGF) and bone morphogenetic pr

178 tenin, bone morphogenetic protein (Bmp), and fibroblast growth factor (Fgf) and its associated recept

179 Fibroblast Growth Factor (FGF) dependent signalling is f

180 For example, fibroblast growth factor (FGF) drives naive pluripotent

181 The three members of the endocrine fibroblast growth factor (FGF) family designated FGF19,

182 Recent evidence highlights the fibroblast growth factor (FGF) family in emotion modulat

183 Members of the fibroblast growth factor (FGF) family play essential and

184 The three members of the endocrine-fibroblast growth factor (FGF) family, FGF19, 21, and 23

185 Here, we show that fibroblast growth factor (FGF) ligands FGF8, FGF17 and F

186 Lapsyn works in concert with the fibroblast growth factor (FGF) pathway to promote branch

187 Fibroblast growth factor (FGF) plays a vital role in the

188 HS interacts physically with canonical fibroblast growth factor (FGF) proteins that signal thro

189 Activating mutations in fibroblast growth factor (FGF) receptor 3 and inactivati

190 lotho, are essential components of endocrine fibroblast growth factor (FGF) receptor complexes, as th

191 These treatments lead to activation of the fibroblast growth factor (FGF) receptor, phospholipase C

192 Fibroblast growth factor (Fgf) receptors have a recogniz

193 Central activation of fibroblast growth factor (FGF) receptors regulates perip

194 atterning defects and define a Hedgehog (Hh)-fibroblast growth factor (FGF) signaling axis required f

195 The fibroblast growth factor (FGF) signaling cascade is a ke

196 show that posttranscriptional attenuation of fibroblast growth factor (FGF) signaling is essential fo

197 Fibroblast growth factor (FGF) signaling is required for

198 scriptomics, we found that components of the Fibroblast Growth Factor (FGF) signaling pathway were en

199 progenitors and myoblasts is mediated by the fibroblast growth factor (FGF) signaling pathway, and ex

200 Fibroblast growth factor (FGF) signaling plays pivotal r

201 Fibroblast Growth Factor (FGF) signaling promotes self-r

202 we investigated how Sonic hedgehog (Shh) and Fibroblast growth factor (Fgf) signaling regulate limb d

203 ner cell mass and trophectoderm cells due to fibroblast growth factor (FGF) signaling.

204 cription of glycolytic enzymes downstream of fibroblast growth factor (FGF) signaling.

205 Fibroblast growth factor (Fgf) signalling cooperates wit

206 Fibroblast growth factor (FGF) signalling in the distal

207 Regulated fibroblast growth factor (FGF) signalling is a prerequis

208 ransforming growth factor beta (TGFbeta) and fibroblast growth factor (FGF) signalling pathways to co

209 ivin maintain self-renewal in the absence of fibroblast growth factor (FGF) signalling.

210 We found that the loss of an intracellular fibroblast growth factor (FGF), FGF13, in the mouse DRG

211 Here, by modulating fibroblast growth factor (FGF), transforming growth fact

212 ollicle patterning, identifying a network of fibroblast growth factor (FGF), wingless-related integra

213 a occurs despite a marked elevation in serum fibroblast growth factor (FGF)-23.

214 Amongst the best studied are the fibroblast growth factor (FGF)-ERK1/2 pathway, PI3K-AKT,

215 Fibroblast growth factor (FGF)-extracellular signal-regu

216 onal glutamate uptake - could be enhanced by fibroblast growth factor (FGF)1 or FGF2.

217 Fibroblast growth factor (FGF)13, a nonsecreted, X-linke

218 Ras-MAPK signaling pathway and that required fibroblast growth factor (FGF)19 signaling via FGF recep

219 ockout of FXR were given daily injections of fibroblast growth factor (FGF)19.

220 Fibroblast growth factors (FGF) act as proangiogenic and

221 has been obscured by other proteins (such as fibroblast growth factors (FGF) or CaM-dependent kinase

222 eening of secreted peptides, we identify two fibroblast growth factors (FGF), FGF6 and FGF9, as poten

223 Here we show that acidic fibroblast growth factor (FGF1) derived from cancer-asso

224 a prototypical Hp/HS binding protein: basic-fibroblast growth factor (FGF2).

225 The fibroblast growth factor FGF21 was labeled with molecula

226 patterning center in the telencephalon, the Fibroblast Growth Factor, FGF8, disperses as a morphogen

227 Fibroblast growth factors (FGFs) 21 and 23 are recently

228 Fibroblast growth factors (FGFs) 9 and 10 are essential

229 Fibroblast growth factors (FGFs) and FGF receptors (FGFR

230 Abnormal levels of fibroblast growth factors (FGFs) and FGF receptors (FGFR

231 Fibroblast growth factors (FGFs) are a family of essenti

232 ensity is tightly regulated by the supply of fibroblast growth factors (FGFs) from lymphatic endothel

233 Most fibroblast growth factors (FGFs) function as receptor li

234 We also demonstrate that fibroblast growth factors (FGFs) position the pineal pro

235 Hepatocyte growth factor, fibroblast growth factors (FGFs)-2 and -13, and type 1 i

236 The basic fibroblast growth factor gradient can accelerate fibrobl

237 How Na(V) -CaM, CaMKII and FGF/fibroblast growth factor homologous factor interactions

238 )1.5 CT and for ternary complexes containing fibroblast growth factor homologous factors (FHF).

239 Fibroblast growth factor homologous factors (FHFs) are i

240 FHFs (fibroblast growth factor homologous factors) are key reg

241 ch were confirmed by increased expression of fibroblast growth factor-inducible 14 (Fn14), Down syndr

242 of apoptosis (TWEAK) and its sole receptor, fibroblast growth factor-inducible molecule 14 (Fn14), b

243 SIMMS(2) analysis of two fibroblast growth factor-inhibiting hexasaccharides iden

244 ng cells draw passive cells from low to high fibroblast growth factor levels, recruiting them to cont

245 factors (brain-derived neurotrophic factor, fibroblast growth factors, platelet-derived growth facto

246 tumors harboring actionable aberration(s) in fibroblast growth factor receptor (FGFR) 1-3 were treate

247 Fibroblast growth factor receptor (FGFR) 2 gene alterati

248 We assessed actions of fibroblast growth factor receptor (FGFR) 2 in urothelium

249 Mechanistically, FGF23 can bind and activate fibroblast growth factor receptor (FGFR) 4 independently

250 ithelial cells resulted in a higher level of fibroblast growth factor receptor (FGFR) activation and

251 m X-ray structure of sKlotho in complex with fibroblast growth factor receptor (FGFR) and fibroblast

252 The fibroblast growth factor receptor (FGFR) family of recep

253 The clinical activity of fibroblast growth factor receptor (FGFR) inhibitors seem

254 ncer subtype is defined by activation of the fibroblast growth factor receptor (FGFR) pathway and thi

255 Aberrant activation of fibroblast growth factor receptor (FGFR) signalling cont

256 yosin-based force transduction or initiating fibroblast growth factor receptor (FGFR)-dependent bioch

257 activation mechanism of the kinase domain of Fibroblast Growth Factor Receptor (FGFR).

258 ch a conventional optogenetic design for the fibroblast growth factor receptor (FGFR).

259 rrelations of diseases caused by variants in Fibroblast Growth Factor Receptor 1 ( FGFR1) and report

260 (EMT), which included enhanced expression of fibroblast growth factor receptor 1 (FGFR1) and axonal g

261 whole-genome CRISPR screening and identified fibroblast growth factor receptor 1 (FGFR1) as the top t

262 Hypoxia induced increased fibroblast growth factor receptor 1 (FGFR1) expression i

263 Mechanistically, fibroblast growth factor receptor 1 (FGFR1) inhibition a

264 CRISPR/Cas9-mediated deletion of fibroblast growth factor receptor 1 (FGFR1) or pretreatm

265 d growth factor receptor alpha (PDGFRA), and fibroblast growth factor receptor 1 (FGFR1) to cell prol

266 tor receptor 2 (VEGFR2), EPHA2-VEGFR2, EPHA2-fibroblast growth factor receptor 1 (FGFR1), EPHA2-FGFR2

267 thelial growth factor receptor 2) and FGFR1 (fibroblast growth factor receptor 1) after ischemic stro

268 cinomas (ICCs) express constitutively active fibroblast growth factor receptor 2 (FGFR2) fusion prote

269 The regulatory mechanism of one such RTK, fibroblast growth factor receptor 2 (FGFR2) kinase, is s

270 Fibroblast growth factor receptor 2 (FGFR2) might have a

271 Activating mutations of fibroblast growth factor receptor 3 (FGFR3) are common i

272 Fibroblast growth factor receptor 3 (FGFR3) coimmunoprec

273 y modified, we reported that tyrosine kinase fibroblast growth factor receptor 3 (FGFR3) complexes wi

274 Other anti-fibroblast growth factor receptor 3 (FGFR3) compounds ar

275 We recently found that the fibroblast growth factor receptor 3 (FGFR3) interacts wi

276 Y) 138 of HPV-31 E2 is phosphorylated by the fibroblast growth factor receptor 3 (FGFR3) kinase.

277 We recently discovered that fibroblast growth factor receptor 3 (FGFR3) phosphorylat

278 Fibroblast growth factor receptor 3 (FGFR3) regulates HP

279 Fibroblast growth factor receptor 3 (FGFR3) was also ide

280 is caused by a gain-of function mutation in fibroblast growth factor receptor 3 (FGFR3).

281 ions affecting insulin-like growth factor 1, fibroblast growth factor receptor and WNT signalling are

282 sstalk between Target of Rapamycin (TOR) and Fibroblast growth factor receptor b (Fgfrb) signaling in

283 Activation of the fibroblast growth factor receptor FGFR4 by FGF19 drives

284 he ability to promote cell proliferation via fibroblast growth factor receptor signalling, with only

285 last growth factor receptors (FGFRs) recruit Fibroblast Growth Factor Receptor Substrate 2 (Frs2) and

286 mutations severely disrupt PM association of fibroblast growth factor receptor substrate 2alpha but d

287 tein and lipidation-deficient mutants of the fibroblast growth factor receptor substrate 2alpha.

288 ly, L1-DeltaTM-induced angiogenesis requires fibroblast growth factor receptor-1 signaling, implying

289 rs of the estrogen receptors alpha and beta, fibroblast growth factor receptor-1, protein kinase C, a

290 In contrast, fibroblast growth factor receptors (FGFRs) recruit Fibro

291 olid tumors harboring genetic alterations in fibroblast growth factor receptors (FGFRs) to determine

292 study the hetero-interactions between three fibroblast growth factor receptors-FGFR1, FGFR2, and FGF

293 We identified bFGF (basic fibroblast growth factor) released by ECs as inducer of

294 Fibroblast growth factor signaling is essential for glan

295 Fibroblast growth factor signaling was found to regulate

296 hed in response to a morphogenic gradient of fibroblast growth factor signalling.

297 enerate mMSCs by utilizing hypoxia and basic fibroblast growth factor supplementation.

298 Sonic hedgehog, bone morphogenetic protein, fibroblast growth factor, transforming growth factor bet

299 mple tests to quantify angiogenesis factors (fibroblast growth factor, vascular endothelial growth fa

300 The epidermal growth factor and the basic fibroblast growth factor were entrapped within the poly