ライフサイエンスコーパス: fibroblast growth factor-2

1 ce in their 5' leaders, Gtx itself and FGF2 (fibroblast growth factor 2).

2 ors (serum, insulin-like growth factor I, or fibroblast growth factor-2).

3 scorbic acid, bone morphogenic protein-2, or fibroblast growth factor 2.

4 ls by vascular endothelial growth factor and fibroblast growth factor 2.

5 and invasion, in the presence or absence of fibroblast growth factor 2.

6 owth; Igf2 does so in a manner additive with fibroblast growth factor 2.

7 idermal growth factor-like growth factor and fibroblast growth factor 2.

8 uring cortical explants in medium containing fibroblast growth factor 2.

9 conventional secretion shared by HIV-Tat and fibroblast growth factor 2.

10 mation, and VEGFR-2 signaling in response to fibroblast growth factor-2.

11 ll as intracellular and secreted isoforms of fibroblast growth factor-2.

12 process is mediated at least in part through fibroblast growth factor-2.

13 (EC(50) = 88 pmol/L) induced by 500 ng/ml of fibroblast growth factor-2.

14 matid separation, and regulates secretion of fibroblast growth factor-2.

15 ompanied by an increase in astrocyte-derived fibroblast growth factor-2.

16 y and expression of glutamine synthetase and fibroblast growth factor-2.

17 ed growth factor, platelet concentrates, and fibroblast-growth factor-2.

18 cant increases in expression of collagen and fibroblast growth factor (2.8 and 3.4 fold, p<0.05).

19 e or presence of different concentrations of fibroblast growth factor 2, a known regulator of cortica

20 is is through the regulation of secretion of fibroblast growth factor-2, a guidance factor for migrat

21 ontaining vascular endothelial growth factor/fibroblast growth factor-2 achieved normoglycemia at a h

22 AdtrEGFR did not alter fibroblast growth factor 2 actions on mitogenesis.

23 rough binding to cell-surface tropomyosin in fibroblast growth factor-2-activated endothelial cells (

24 onstrate here that the continued presence of fibroblast growth factor 2 along with N-CAM or brain-der

25 retinoic acid, insulin-like growth factor-1, fibroblast growth factor-2, alpha-thrombin, and interleu

26 FGF2 encodes fibroblast growth factor 2 (also referred to as basic fi

27 ed only in cultures grown in the presence of fibroblast growth factor 2 and either N-CAM or brain-der

28 ound, however, that direct administration of fibroblast growth factor 2 and epidermal growth factor i

29 l growth factor, fibroblast growth factor 7, fibroblast growth factor 2 and hepatocyte growth factor

30 other pro-angiogenic growth factors, namely fibroblast growth factor 2 and hepatocyte growth factor.

31 were differentiated by sequential culture in fibroblast growth factor 2 and human activin-A, hepatocy

32 ectodomain of the core protein and required fibroblast growth factor 2 and stroma-derived factor 1.

33 We have discovered that a combination of fibroblast growth factor 2 and transforming growth facto

34 Importantly, fluorescence analysis of fibroblast growth factor 2 and tumor cell-induced vessel

35 n expression in cardiomyocytes is induced by fibroblast growth factor-2 and accumulates in response t

36 sphorylation and suppressed EC expression of fibroblast growth factor-2 and Bcl-2.

37 our murine retinal angiogenesis model, both fibroblast growth factor-2 and interleukin-6 administrat

38 elial dysfunction, characterized by abnormal fibroblast growth factor-2 and interleukin-6 signaling,

39 nsforming growth factor-beta, in contrast to fibroblast growth factor-2 and interleukin-6, promotes h

40 everal other angiogenesis factors, including fibroblast growth factor-2 and the receptors Flt-1 and F

41 into gene expression cascades revealed that fibroblast growth factor-2 and transforming growth facto

42 Two other Schwann cell mitogens, fibroblast growth factor-2 and transforming growth facto

43 del for two biomedically important proteins, fibroblast growth factor-2 and vascular endothelial grow

44 Finally, angiogenic responses to fibroblast growth factor-2 and vascular endothelial grow

45 (vascular endothelial growth factor), FGF2 (fibroblast growth factor 2), and their receptors VEGFR2

46 of tumor necrosis factor alpha, IL-8, IL-10, fibroblast growth factor 2, and IL-7 remained higher.

47 r (VEGF), brain-derived neurotrophic factor, fibroblast growth factor-2, and ciliary neurotrophic fac

48 es, associated with binding to antithrombin, fibroblast growth factor-2, and herpes simplex virus env

49 ulating proteins, including cyclinD1, c-Myc, fibroblast growth factor-2, and ornithine decarboxylase,

50 in-6 and monocyte chemoattractant protein-1, fibroblast growth factor-2, and the potent vasoconstrict

51 enic signaling molecules, including VEGF and fibroblast growth factor-2, are mainly expressed by non-

52 equires O-sulfation in heparin, and involves fibroblast growth factor-2 as well as fibroblast growth

53 (a) vascular endothelial growth factor; (b) fibroblast growth factor 2/basic fibroblast growth facto

54 5(S)-HETE induced the expression of basic fibroblast growth factor 2 (bFGF-2) in a Jak-2- and PI3-

55 ylation, global protein synthesis, and basic fibroblast growth factor-2 (bFGF-2) expression in VSMC.

56 ke growth factor 1 (IGF-1), as well as basic fibroblast growth factor 2 (bFGF2), reportedly astrocyte

57 Surprisingly, basement membrane fibroblast growth factor-2 binding kinetics remained unc

58 FGF2 (fibroblast growth factor 2), but not vascular endothelia

59 (95% confidence interval [CI], 34%-42%) and fibroblast growth factor-2 by 64% (95% CI, 44-85%; P<0.0

60 enhanced endothelial cell proliferation in a fibroblast growth factor-2-dependent manner.

61 Glypican increased fibroblast growth factor-2 expression and chondroitinase

62 -11 also attenuated glutamine synthetase and fibroblast growth factor-2 expression, but did not rever

63 ice is attributable, in part, to upregulated fibroblast growth factor-2 expression, which is inhibite

64 e mitogens epidermal growth factor (EGF) and fibroblast growth factor 2 (FGF 2), and then by serum.

65 We revisited this concept by using fibroblast growth factor 2 (FGF-2) (80 ng) to stimulate

66 t found that prostaglandin E(2) (PGE(2)) and fibroblast growth factor 2 (FGF-2) -mediators known to i

67 Fibroblast growth factor 2 (FGF-2) and cortisol induced

68 ran sulfate to participate in a complex with fibroblast growth factor 2 (FGF-2) and its receptor tyro

69 ave shown previously that fibrin(ogen) binds fibroblast growth factor 2 (FGF-2) and potentiates stimu

70 We have shown previously that fibroblast growth factor 2 (FGF-2) binds with high affin

71 pathway involves the release of pericellular fibroblast growth factor 2 (FGF-2) from the articular ca

72 ntagonizes interleukin-1 beta (IL-1beta) and fibroblast growth factor 2 (FGF-2) in proteoglycan metab

73 Fibroblast growth factor 2 (FGF-2) inhibited ADAMTS-5 ex

74 tion of 0.03% carrageenan and/or 6 pmoles of fibroblast growth factor 2 (FGF-2) into rat knees.

75 Fibroblast growth factor 2 (FGF-2) is an important neuro

76 Fibroblast growth factor 2 (FGF-2) is an important regul

77 Here we show that the mechanism by which fibroblast growth factor 2 (FGF-2) protects small cell l

78 Fibroblast growth factor 2 (FGF-2) signalling-induced S6

79 in(ogen) binding potentiates the capacity of fibroblast growth factor 2 (FGF-2) to stimulate endothel

80 ced apoptosis was associated with suppressed fibroblast growth factor 2 (FGF-2) transcription, as ass

81 We placed fibroblast growth factor 2 (FGF-2) under conditional con

82 e the safety and effectiveness of 3 doses of fibroblast growth factor 2 (FGF-2) when combined with a

83 The interaction of fibroblast growth factor 2 (FGF-2) with heparan sulfate

84 Signaling by fibroblast growth factor 2 (FGF-2), an autocrine stimula

85 of a number of HS-binding proteins including fibroblast growth factor 2 (FGF-2), and the chemokines C

86 -lysine and mouse laminin in the presence of fibroblast growth factor 2 (FGF-2), nerve growth factor

87 eased from PMNs induced de novo synthesis of fibroblast growth factor 2 (FGF-2), which in turn become

88 t pool of the heparin-binding growth factor, fibroblast growth factor 2 (FGF-2), which is bound to th

89 Fibroblast growth factor 2 (FGF-2), which is highly expr

90 esponse to epidermal growth factor (EGF) and fibroblast growth factor 2 (FGF-2), while retaining the

91 aft tumor models as well as suppresses basic fibroblast growth factor 2 (FGF-2)-induced neovasculariz

92 s are surrounded by an extracellular pool of fibroblast growth factor 2 (FGF-2).

93 ogen and fibrin, and growth is stimulated by fibroblast growth factor 2 (FGF-2).

94 ural tube-like structures in the presence of fibroblast growth factor 2 (FGF-2).

95 Although exogenous basic fibroblast growth factor 2 (FGF-2/Fgf-2) is commonly use

96 in rabbit corneal fibroblasts; in contrast, fibroblast growth factor-2 (FGF) and heparin led to a de

97 th platelet-derived growth factor-BB (PDGF), fibroblast growth factor-2 (FGF), transforming growth fa

98 vented when the cells were treated with both fibroblast growth factor-2 (FGF-2) and antibodies agains

99 viously reported that growth factors such as fibroblast growth factor-2 (FGF-2) and bone morphogeneti

100 d binds and activates growth factors such as fibroblast growth factor-2 (FGF-2) and FGF-7, which are

101 a detailed study of the intercompetition of fibroblast growth factor-2 (FGF-2) and heparin-binding e

102 Function blocking anti-fibroblast growth factor-2 (FGF-2) antibody enhanced lys

103 /-) mice were not as invasive in response to fibroblast growth factor-2 (FGF-2) as cancer cells isola

104 inase B gene (matrix metalloproteinase-9) by fibroblast growth factor-2 (FGF-2) during angiogenesis,

105 The single-copy gene for fibroblast growth factor-2 (FGF-2) encodes for multiple

106 ocytes treated with U0126 in the presence of fibroblast growth factor-2 (FGF-2) express normal post-i

107 15(S)-HETE induced fibroblast growth factor-2 (FGF-2) expression rapidly vi

108 mined ciliary neurotrophic factor (CNTF) and fibroblast growth factor-2 (FGF-2) expression, because b

109 angiotensin II (AII) receptors activate the fibroblast growth factor-2 (FGF-2) gene through a unique

110 nded or high molecular weight (HMW) forms of fibroblast growth factor-2 (FGF-2) has been shown to aff

111 Additionally, decreased cellular fibroblast growth factor-2 (FGF-2) immunostaining was as

112 VWF also binds to VEGF-A and fibroblast growth factor-2 (FGF-2) in human plasma and c

113 ddition, 14,15-EET induced the expression of fibroblast growth factor-2 (FGF-2) in Src- and PI3K-Akt-

114 we used this model to determine the role of fibroblast growth factor-2 (FGF-2) in the remodeling res

115 Fibroblast growth factor-2 (FGF-2) increases the express

116 rbol 13-acetate (TPA), specifically inhibits fibroblast growth factor-2 (FGF-2) induced proliferation

117 Fibroblast Growth Factor-2 (FGF-2) induces cell prolifer

118 Here, we show that fibroblast growth factor-2 (FGF-2) induces proliferation

119 HMW fibroblast growth factor-2 (FGF-2) inhibits cell migrati

120 inical studies report that the expression of fibroblast growth factor-2 (FGF-2) is decreased in the p

121 Fibroblast growth factor-2 (FGF-2) is one of the best ch

122 d (RA) and epidermal growth factor (EGF) and fibroblast growth factor-2 (FGF-2) mitogens.

123 Both the exogenous administration of fibroblast growth factor-2 (FGF-2) or the induction of m

124 al cell (HUVEC) tube formation stimulated by fibroblast growth factor-2 (FGF-2) or vascular endotheli

125 Other endothelial growth factors, such as fibroblast growth factor-2 (FGF-2) or VEGF-A, may also c

126 Fibroblast growth factor-2 (FGF-2) promotes proliferatio

127 Da) and low (18-kDa) molecular mass forms of fibroblast growth factor-2 (FGF-2) regulate cell prolife

128 human neuroblastoma cell line, SK-N-MC, with fibroblast growth factor-2 (FGF-2) results in induction

129 Only fibroblast growth factor-2 (FGF-2) was able to initiate

130 Fibroblast growth factor-2 (FGF-2) was one of the first

131 study to compare the neurotrophic actions of fibroblast growth factor-2 (FGF-2) with the better chara

132 -growth factor (NGF), neurotrophin-3 (NT-3), fibroblast growth factor-2 (FGF-2), and insulin-like gro

133 ivered insulin-like growth factor I (IGF-I), fibroblast growth factor-2 (FGF-2), and/or transforming

134 lar, heparin-binding growth factors, such as fibroblast growth factor-2 (FGF-2), bind to heparan sulf

135 ivator of transcription (Tat), combined with fibroblast growth factor-2 (FGF-2), can induce the dedif

136 h had been genetically engineered to produce fibroblast growth factor-2 (FGF-2), can protect nigrostr

137 o well-characterized lymphangiogenic factors fibroblast growth factor-2 (FGF-2), hepatocyte growth fa

138 Fibroblast growth factor-2 (FGF-2), in the presence of d

139 Addition of fetal bovine serum, fibroblast growth factor-2 (FGF-2), transforming growth

140 ), TNF-alpha, interleukin-1 beta (IL-1beta), fibroblast growth factor-2 (FGF-2), transforming growth

141 n that the gamma chain serves as a depot for fibroblast growth factor-2 (FGF-2), which is likely to p

142 -2 (COX-2), interleukin-1beta (IL-1beta) and fibroblast growth factor-2 (FGF-2), which led to a marke

143 ial dysfunction, is an important mediator of fibroblast growth factor-2 (FGF-2)-induced angiogenesis.

144 n and tube formation as potently as 20 ng/mL fibroblast growth factor-2 (FGF-2).

145 als received intramuscular sustained-release fibroblast growth factor-2 (FGF-2).

146 lasts, associated with impaired responses to fibroblast growth factor-2 (FGF-2).

147 ocytes were exposed to increasing amounts of fibroblast growth factor-2 (FGF-2).

148 ells was dependent on receptor activation by fibroblast growth factor-2 (FGF-2).

149 ect axonal plasticity, adenoviruses encoding fibroblast growth factor-2 (FGF-2/Adts), nerve growth fa

150 Fibroblast growth factor-2 (FGF-2; 250 ng/ml)-treated ex

151 Elevated autocrine fibroblast growth factors 2 (FGF-2) signaling promotes p

152 rowth factors (kit ligand [KL], FLT3 ligand, fibroblast growth factor-2 [FGF-2], vascular endothelial

153 this study, we identified the gene encoding fibroblast growth factor 2 (Fgf2 or basic fibroblast gro

154 that bone morphogenetic proteins (BMPs) and fibroblast growth factor 2 (FGF2) act antagonistically t

155 increases in signaling on coactivation with fibroblast growth factor 2 (FGF2) and a 5-HT1A agonist,

156 he target genes for these regulators include fibroblast growth factor 2 (FGF2) and Ariadne RBR E3 ubi

157 We show that fibroblast growth factor 2 (FGF2) and FGF receptors are

158 Human fibroblast growth factor 2 (FGF2) and insulin-like growt

159 proteoglycans (neoPGs) with affinity for the fibroblast growth factor 2 (FGF2) and introduced them in

160 r heparan and chondroitin sulfate, including fibroblast growth factor 2 (FGF2) and its receptor FGFR1

161 mRNA levels for fibroblast growth factor 2 (FGF2) and mediators of the G

162 Fibroblast growth factor 2 (FGF2) and vascular endotheli

163 line-derived neurotrophic factor (GDNF) and fibroblast growth factor 2 (FGF2) are critical for roden

164 al concentrations of the neurotrophic factor fibroblast growth factor 2 (FGF2) are negatively associa

165 Using fibroblast growth factor 2 (FGF2) as a model system, we

166 el of candidate growth factors we identified fibroblast growth factor 2 (FGF2) as a potent regulator

167 alpha (TNFalpha) as an ototoxic molecule and fibroblast growth factor 2 (FGF2) as an otoprotective mo

168 We also found that increased production of fibroblast growth factor 2 (Fgf2) by HoxA10-overexpressi

169 Sonic hedgehog (SHH) and fibroblast growth factor 2 (FGF2) can both induce neocor

170 Fibroblast growth factor 2 (FGF2) can enhance the prolif

171 ntly found in rodents that alcohol increases fibroblast growth factor 2 (FGF2) expression in the dors

172 peptidase E, CPE), concomitant with enhanced fibroblast growth factor 2 (FGF2) expression, and an inc

173 ippocampal cell proliferation and astrocytic fibroblast growth factor 2 (FGF2) expression.

174 doderm cultured in the presence of exogenous fibroblast growth factor 2 (FGF2) fails to initiate expr

175 Upon treatment with fibroblast growth factor 2 (FGF2) for another 10 d, thes

176 We previously demonstrated that fibroblast growth factor 2 (FGF2) from adipose tissue st

177 Here, we demonstrate that fibroblast growth factor 2 (FGF2) from bone marrow strom

178 Fibroblast growth factor 2 (FGF2) functions were analyze

179 Fibroblast growth factor 2 (FGF2) has been shown to inte

180 Fibroblast growth factor 2 (FGF2) has previously been im

181 reported that targeted overexpression of the fibroblast growth factor 2 (FGF2) high molecular weight

182 d that alcohol upregulates the expression of fibroblast growth factor 2 (FGF2) in dorsomedial striatu

183 e transcriptional response of osteocalcin to fibroblast growth factor 2 (FGF2) in MC3T3 osteoblasts.

184 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2) in the development of

185 It is well established that fibroblast growth factor 2 (FGF2) induces lymphangiogene

186 Fibroblast growth factor 2 (FGF2) is a critical mitogen

187 Fibroblast growth factor 2 (FGF2) is a key signaling mol

188 Fibroblast growth factor 2 (FGF2) is a potent mitogen pr

189 Fibroblast growth factor 2 (FGF2) is crucial for the dev

190 This study takes advantage of fibroblast growth factor 2 (FGF2) knock-out mice to dete

191 Fibroblast growth factor 2 (FGF2) positively modulates o

192 H3K9me3 at Glucocorticoid Receptor (GR) and Fibroblast growth Factor 2 (FGF2) promoters differ recip

193 oenvironmental proteins FLT3 ligand (FL) and fibroblast growth factor 2 (FGF2) protect FLT3-ITD+ MOLM

194 ling pathway, which is upregulated, owing to fibroblast growth factor 2 (FGF2) secretion or increased

195 endent association between apelin (APLN) and fibroblast growth factor 2 (FGF2) signaling in pulmonary

196 Fibroblast growth factor 2 (FGF2) signaling plays a pivo

197 protein that has been directly implicated in fibroblast growth factor 2 (FGF2) signaling.

198 onstrate that loss of NG2 glial secretion of fibroblast growth factor 2 (FGF2) suffices to induce the

199 ted neutrophils expressed substantially more fibroblast growth factor 2 (FGF2) than naive neutrophils

200 Fibroblast growth factor 2 (FGF2) was uniquely capable o

201 Despite strong evidence linking fibroblast growth factor 2 (FGF2) with anxiety and depre

202 Fibroblast growth factor 2 (FGF2), a member of the FGF f

203 her show that beta1-integrin cooperates with fibroblast growth factor 2 (Fgf2), a potent growth facto

204 ere, we focus on unconventional secretion of fibroblast growth factor 2 (FGF2), a secretory mechanism

205 otentiating the action of the growth factors fibroblast growth factor 2 (FGF2), hepatocyte growth fac

206 In turn, CAFs produced high levels of fibroblast growth factor 2 (FGF2), initiating a signalin

207 wth was associated with increased binding of fibroblast growth factor 2 (FGF2), phosphorylation of ex

208 lar to what has been reported previously for fibroblast growth factor 2 (FGF2), providing strong evid

209 s IL-1beta and IL-18, growth factors such as fibroblast growth factor 2 (FGF2), redox enzymes such as

210 racking in fibroblast pericellular matrix of fibroblast growth factor 2 (FGF2), stoichiometrically la

211 Tumor necrosis factor alpha and fibroblast growth factor 2 (FGF2), which are abnormally

212 We found that fibroblast growth factor 2 (FGF2), which by itself does

213 Blood vessel growth into fibroblast growth factor 2 (FGF2)-containing Matrigel pl

214 The transition from Q111 pNSCs to fibroblast growth factor 2 (FGF2)-responsive dNSCs was m

215 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2).

216 ic mammalian CNS are initially responsive to fibroblast growth factor 2 (FGF2).

217 ascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2).

218 s of these two FGFR2 mutants in complex with fibroblast growth factor 2 (FGF2).

219 thelial cells to immobilized vitronectin and fibroblast growth factor 2 (FGF2).

220 e Na/K-ATPase in unconventional secretion of fibroblast growth factor 2 (FGF2).

221 to induction of an important repair marker, fibroblast growth factor 2 (FGF2).

222 observed, along with massive accumulation of fibroblast growth factor 2 (FGF2).

223 Fibroblast growth factor-2 (FGF2) activates the extracel

224 ced by intraocular injections of insulin and fibroblast growth factor-2 (FGF2) but not by ciliary neu

225 an postmortem studies have demonstrated that fibroblast growth factor-2 (FGF2) expression is decrease

226 In this study, DPCs were treated with fibroblast growth factor-2 (FGF2) for 5-6 consecutive se

227 In vitro, fibroblast growth factor-2 (FGF2) has been implicated in

228 Fibroblast growth factor-2 (FGF2) has neurotrophic effec

229 Single-bolus intracoronary administration of fibroblast growth factor-2 (FGF2) improved symptoms and

230 The role of the 18-kDa isoform of fibroblast growth factor-2 (FGF2) in the maintenance of

231 owever, much less is known about the role of fibroblast growth factor-2 (FGF2) in this process.

232 Fibroblast growth factor-2 (FGF2) is a potent angiogenic

233 Pharmacological studies have suggested that fibroblast growth factor-2 (FGF2) is involved in cardiop

234 fference in VAT activity was associated with fibroblast growth factor-2 (FGF2) levels.

235 Human fibroblast growth factor-2 (FGF2) regulates cellular pro

236 In the case of fibroblast growth factor-2 (FGF2) signaling, heparin/hep

237 ary mammary epithelial cells stimulated with fibroblast growth factor-2 (FGF2) to model mammary branc

238 heregulin-1beta (a ligand for ERBB2/ERBB3), fibroblast growth factor-2 (FGF2), and activin A support

239 r, we used recombinant adenovirus to express fibroblast growth factor-2 (FGF2), nerve growth factor (

240 factor system, including reduced hippocampal fibroblast growth factor-2 (FGF2).

241 ent coacervate of a polycation, heparin, and fibroblast growth factor-2 (FGF2).

242 nd the capacity to self-renew in response to fibroblast growth factor-2 (FGF2).

243 Fibroblast growth factor-2 (FGF2, bFGF) has been propose

244 we established a role for ASMC-derived basic fibroblast growth factor 2 (FGF2b) and FGF receptor (FGF

245 cans and COL2, -9, -10, and -11), receptors [fibroblast growth factor 2 (FGFR2) and parathyroid hormo

246 eta, IL-6, IL-10, IL-12p40, IL-12p70, IL-15, fibroblast growth factor 2, flt-3 ligand, tumor necrosis

247 ne morphogenetic protein 7/human recombinant fibroblast growth factor 2 for 24 hours before induction

248 ry of vascular endothelial growth factor and fibroblast growth factor-2 from HBPA scaffolds significa

249 heparan sulfate 6-O-endosulfatases (Sulfs), fibroblast growth factor 2-, heparin binding epidermal g

250 gamma-induced protein 10, IL-4, IL-9, IL-10, fibroblast growth factor 2, IL-7, IL-15, and transformin

251 e stimulating hormone, stem cell factor, and fibroblast growth factor-2 in skin of mice lacking RXRal

252 nuated increases in glutamine synthetase and fibroblast growth factor-2 in the reactive astrocytes.

253 nhibit the binding and mitogenic activity of fibroblast growth factor-2 in vascular smooth muscle cel

254 tion of platelet-derived growth factor-B and fibroblast growth factor-2 in VEGF-treated wounds, which

255 ndothelial cell basement membrane-associated fibroblast growth factor-2 increased linearly with cultu

256 Initial microarray studies showed that fibroblast growth factor 2 induced a 2.4-fold increase i

257 LIF alone or TGF beta 2 with fibroblast growth factor 2 induced numerous tubules in i

258 acid docosahexaenoic acid, inhibit VEGF- and fibroblast growth factor 2-induced angiogenesis in vivo,

259 angiogenesis model and found that it reduced fibroblast growth factor-2-induced angiogenesis by 85% (

260 Dasatinib also inhibited fibroblast growth factor-2-induced osteoblast proliferat

261 Our previous work showed that fibroblast growth factor-2 induces proliferation and che

262 in rabbits by subretinal lipopolysaccharide/fibroblast growth factor-2 injection.

263 d the effects of three serum growth factors, fibroblast growth factor-2, insulin, and platelet-derive

264 7A receptors, matrix proteins, CCN proteins, fibroblast growth factor 2, interleukin 13 receptor comp

265 Moreover, increased levels of these fibroblast growth factor-2 isoforms induced vascular end

266 laque and vessel wall, through inhibition of fibroblast growth factor-2, leading to reduced plaque gr

267 Dendrimer glucosamine 6-sulfate blocked fibroblast growth factor-2 mediated endothelial cell pro

268 vascular endothelial growth factor and basic fibroblast growth factor-2 mRNA transcripts in ischemic

269 Importantly, by using an anti-fibroblast growth factor-2 neutralizing antibody, we att

270 DF differentiation, via multiple cAMP and/or fibroblast growth factor 2 or basic FGF (FGF2)-dependent

271 ort that intranasal administration of either fibroblast growth factor-2 or heparin-binding epidermal

272 egulated kinase pathway after treatment with fibroblast growth factor-2 or heparin-binding epidermal

273 ntation and >/=50 ng/ml (P < 0.01) of either fibroblast growth factor-2 or vascular endothelial growt

274 and migration (hepatocyte growth factor and fibroblast growth factor 2) or transplantation on materi

275 loss of heparanase 2 led to upregulation of fibroblast growth factor 2/phosphorylated extracellular

276 ession patterns of hepatocyte growth factor, fibroblast growth factor 2, platelet-derived growth fact

277 Fibroblast growth factor-2/platelet-derived growth facto

278 instead related to enhanced endothelial cell fibroblast growth factor-2 release and permeability.

279 Cellular fibroblast growth factor-2 release occurred concomitant

280 that neprilysin proteolytically inactivates fibroblast growth factor-2, resulting in negative regula

281 Recombinant fibroblast growth factor-2 (rFGF-2) improves perfusion i

282 effects of the administration of recombinant fibroblast growth factor-2 (rFGF-2) protein on myocardia

283 al stem cell cultures, which is required for fibroblast growth factor 2's (FGF-2) mitogenic activity

284 in-1, matrix metallopeptidase 9 (MMP-9), and fibroblast growth factor 2 serum levels when compared wi

285 ulated kinase phosphorylation in response to fibroblast growth factor 2, showing that changes in 6-O-

286 s showed abnormal HS composition and altered fibroblast growth factor 2 signaling, which was rescued

287 ic protein-4 and inhibitors of activin A and fibroblast growth factor-2 signaling (BAP treatment).

288 diated by alpha4beta1, whereas antagonism of fibroblast growth factor-2-stimulated chemotaxis is not

289 variation, we observed changes in netrin 4, fibroblast growth factor 2, tenascin C, collagen 1, mepr

290 nesis (vascular endothelial growth factor-A, fibroblast growth factor-2), thrombosis (D-dimer, von Wi

291 oss of cleavage and increased the potency of fibroblast growth factor-2 to induce capillary array for

292 lysis of vascular endothelial growth factor, fibroblast growth factor 2, transforming growth factor b

293 demonstrated increased expression levels of fibroblast growth factor-2, transforming growth factor-b

294 free medium with epidermal growth factor and fibroblast growth factor 2, varied in the level of CD133

295 HCECs, like HUVECs, responded to fibroblast growth factor-2, vascular endothelial growth

296 of neuroprotective compounds, which include fibroblast growth factor-2, vascular endothelial growth

297 lucose-induced increase in basement membrane fibroblast growth factor-2 was instead related to enhanc

298 stimulation by EGF and TGF-alpha, but not by fibroblast growth factor 2, was almost completely blocke

299 nd ART1-Leu257 to ADP-ribosylate agmatine or fibroblast growth factor-2 were similar.

300 binding ligands, such as apolipoprotein E or fibroblast growth factor-2, were noted.