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1 n potentials (adipogenesis, osteogenesis and fibrogenesis).
2 (HSC) activation, which contributes to liver fibrogenesis.
3 ctors that are known to drive diabetic renal fibrogenesis.
4 ely up-stream of TGF-betas in the process of fibrogenesis.
5 in procollagen I secretion in HSCs and liver fibrogenesis.
6 xplored how a PNPLA3 variant impacts hepatic fibrogenesis.
7 ic Chagas disease, favoring inflammation and fibrogenesis.
8 s hepatic stellate cell (HSC) activation and fibrogenesis.
9 activated, they are contractile and promote fibrogenesis.
10 an important role in the reversal of hepatic fibrogenesis.
11 and emerging blood and imaging biomarkers of fibrogenesis.
12 asingly recognized as an important driver of fibrogenesis.
13 during carbon tetrachloride induced hepatic fibrogenesis.
14 dent determinant of chemically induced liver fibrogenesis.
15 the contributions of HIV and HCV to hepatic fibrogenesis.
16 nt, immunoregulation, regeneration, and also fibrogenesis.
17 stress, which play important roles in liver fibrogenesis.
18 egy that blocks TGF-beta signaling and renal fibrogenesis.
19 and tissue resident cell populations invokes fibrogenesis.
20 Hedgehog (Hh) signalling regulates hepatic fibrogenesis.
21 ly targeting apelin, a critical repressor of fibrogenesis.
22 s and identify Tc2 cells as key mediators of fibrogenesis.
23 verify in vivo the relevance of AR in NAFLD fibrogenesis.
24 e deposition of fibrillary collagen in liver fibrogenesis.
25 s a source of myofibroblast formation during fibrogenesis.
26 ohepatitis (NASH) is associated with hepatic fibrogenesis.
27 ription 3 (STAT3) had been involved in liver fibrogenesis.
28 inflammation, but may paradoxically promote fibrogenesis.
29 tricate links between activation of HPCs and fibrogenesis.
30 Ia with IPF HLF suggests its contribution to fibrogenesis.
31 sponses, as well as pattern recognition, and fibrogenesis.
32 in-Ang II-AT1 cascade in promoting pulmonary fibrogenesis.
33 n multiple pathological processes, including fibrogenesis.
34 R), a second portal phenomenon implicated in fibrogenesis.
35 liver sinusoidal endothelial cells abrogated fibrogenesis.
36 ent and progression of diabetic interstitial fibrogenesis.
37 native LYCAT expression by siRNA accentuated fibrogenesis.
38 by Gram-negative bacteria stimulate hepatic fibrogenesis.
39 trogens inhibit stellate cell activation and fibrogenesis.
40 culating markers of hepatocyte apoptosis and fibrogenesis.
41 ameliorates hepatocellular damage and liver fibrogenesis.
42 whether microbiota may interfere with liver fibrogenesis.
43 y the protective effects of estrogen against fibrogenesis.
44 ine models to establish the role of LYCAT in fibrogenesis.
45 hat play vital roles in liver physiology and fibrogenesis.
46 ial cell-derived mesenchymal proteins during fibrogenesis.
47 liferation, apoptosis, oxidative stress, and fibrogenesis.
48 ecto-enzymes CD39 and CD73, promotes dermal fibrogenesis.
49 gests they play an important role in hepatic fibrogenesis.
50 in FFC-fed mice abrogated indices of hepatic fibrogenesis.
51 ignals perpetuating hepatic inflammation and fibrogenesis.
52 l mechanism contributing to inflammation and fibrogenesis.
53 f ongoing parenchymal injury by disease, not fibrogenesis.
54 d myofibroblast activation, thereby limiting fibrogenesis.
55 response in the liver and promoting hepatic fibrogenesis.
56 ism of TLR4-mediated fibroblast responses in fibrogenesis.
57 ation of lung leukocyte subsets in pulmonary fibrogenesis.
58 y diseases associated with TGF-beta-mediated fibrogenesis.
59 tor were used to assess the role of SphKs in fibrogenesis.
60 yet unidentified mechanisms underlying liver fibrogenesis.
61 rise to HSCs and myofibroblasts during liver fibrogenesis.
62 y potential key regulatory proteins of liver fibrogenesis.
63 stitial spaces and are key effectors of lung fibrogenesis.
64 e cell viability, promoting inflammation and fibrogenesis.
65 d glomerular flow is a critical initiator of fibrogenesis.
66 lung development and may be involved in lung fibrogenesis.
67 g initial inflammatory responses and hepatic fibrogenesis.
68 henyleneiodonium was associated with reduced fibrogenesis.
69 synthesis of extracellular matrix and liver fibrogenesis.
70 knockdown enhanced Ctgf induction and liver fibrogenesis.
71 GFR significantly decreased TGFbeta-mediated fibrogenesis.
72 intracellular renin receptor that stimulates fibrogenesis.
73 lysaccharide (LPS) are implicated in hepatic fibrogenesis.
74 presence of TGF-beta1, along with decreased fibrogenesis.
75 on of primary human HSC, this a key event in fibrogenesis.
76 XR activation in HSCs, subsequently reducing fibrogenesis.
77 diary between enhanced tissue glycolysis and fibrogenesis.
78 ase the healthy life span of PCLSs and model fibrogenesis.
79 pressing hepatic progenitor cells to biliary fibrogenesis.
80 racellular matrix (ECM) proteins and hepatic fibrogenesis.
81 noninvasively detect and quantify pulmonary fibrogenesis.
82 duced TG2 expression and activity as well as fibrogenesis.
83 rough effects on immune cell recruitment and fibrogenesis.
84 contribute to portal hypertension (PHTN) and fibrogenesis.
85 in the heart and may be relevant to cardiac fibrogenesis.
86 of Ifng(-/-) ILC1s has no effect on adipose fibrogenesis.
87 eptor-1 pathway is implicated in suppressing fibrogenesis.
88 ization and infiltration, and enhanced liver fibrogenesis.
89 ected their transitions and crosstalk during fibrogenesis.
90 ns and may be promising targets for treating fibrogenesis.
91 e acid-induced ductular reaction (DR) drives fibrogenesis.
92 UPR in facilitating collagen I secretion and fibrogenesis.
93 e identified that may be important during PD fibrogenesis.
94 and survival of HSCs and protects from liver fibrogenesis.
98 pha, hydrogen peroxide, hydroxynonenal), and fibrogenesis (alpha-smooth muscle actin, picrosirius red
99 .IMPORTANCE Endoglin plays a crucial role in fibrogenesis and angiogenesis and is an important protei
103 HF/MCD+leptin lean rats, significant hepatic fibrogenesis and cirrhosis, marked portal hypertension,
104 /SMAD genomic circuit that regulates hepatic fibrogenesis and define a role for VDR as an endocrine c
105 ured PCLSs can be successfully used to model fibrogenesis and demonstrate efficacy of antifibrotic th
106 ogically soft silicone substrates suppresses fibrogenesis and desensitizes mesenchymal stem cells (MS
107 may modulate the immune response and inhibit fibrogenesis and discuss the current evidence for their
109 genes that control hepatocyte maturation and fibrogenesis and genes not specifically associated with
112 receptor (EGFR) inhibitor erlotinib on liver fibrogenesis and hepatocellular transformation in three
113 aradigm for the regulation of CCN2-dependent fibrogenesis and identifies fibrotic pathways as targets
114 uncover a new molecular mechanism of tissue fibrogenesis and identify sEphrin-B2, its receptors EphB
115 nding of the roles of vitamin D in pulmonary fibrogenesis and in the treatment of pulmonary fibrosis.
116 the renin-angiotensin system contributes to fibrogenesis and increased hepatic resistance in patient
118 e activation of each homolog modulates renal fibrogenesis and inflammation has not been established.
119 R-21 was protective against TGF-beta-induced fibrogenesis and inflammation in glomerular and intersti
122 idase like-2 (LOXL2) plays a central role in fibrogenesis and is elevated in the serum and liver of p
123 vasculogenesis, wound healing responses, and fibrogenesis and is upregulated in fibroblasts and myofi
125 e cells and in mice following injury reduced fibrogenesis and matrix accumulation; this effect was pa
126 ion state of eosinophils in EoE with altered fibrogenesis and motility of esophageal fibroblasts and
128 ion as a fundamental epigenetic mechanism in fibrogenesis and place Egr-1 upstream in TGF-beta-driven
130 mising therapeutic approach for reduction of fibrogenesis and prevention of HCC in high-risk cirrhosi
131 T-cell responses, which may encourage liver fibrogenesis and progression to end-stage liver disease.
133 icate that TRPV4 activity mediates pulmonary fibrogenesis and suggest that manipulation of TRPV4 chan
135 n the kidney and other organs, their role in fibrogenesis and their role in regulation of the microva
138 TCS exposure, especially on enhancing liver fibrogenesis and tumorigenesis, and the relevance of TCS
140 ars critical to the induction of progressive fibrogenesis and, ultimately, the ominous complications
144 athway plays a central role in inflammation, fibrogenesis, and immunomodulation in the HCC microenvir
145 ajor source of extracellular proteins during fibrogenesis, and may directly, or via secreted products
146 iral-mediated gene transfer on angiogenesis, fibrogenesis, and portal hypertension-associated hemodyn
147 ain receptor-2, contributes significantly to fibrogenesis, and promotes resolution of lung inflammati
148 They act as a brake on the processes driving fibrogenesis, and they dismantle and degrade established
150 ia efferocytosis may affect inflammation and fibrogenesis; and insights into HSC and macrophage heter
151 and elucidating the final common pathways of fibrogenesis are critical for the development of efficac
154 matory infiltrate may contribute directly to fibrogenesis as well as influence the fate of the DR hep
156 were protected against CCl(4)-mediated liver fibrogenesis, as evidenced by reduced collagen type I al
157 stellate cells (HSC) play a critical role in fibrogenesis associated with HCC onset and progression,
158 f CAV1 contributed to the hyperactivation of fibrogenesis-associated RUNX2, a transcription factor ge
159 artate aminotransferase (-57% and -52%), and fibrogenesis biomarkers neoepitope-specific N-terminal p
160 ctin EDA isoform (EDA FN) is instrumental in fibrogenesis but, to date, its expression and function i
161 hanges in the expression of HSC microRNAs in fibrogenesis, but suggests a need for caution when trans
163 of liver fibrosis resolution, VEGF promoted fibrogenesis, but was also required for hepatic tissue r
164 activation is required for the EMT and renal fibrogenesis by activation of multiple profibrotic signa
165 estigations demonstrate that TXNDC5 promotes fibrogenesis by enhancing TGFbeta1 signaling through dir
166 adapted to mice, thus allowing the study of fibrogenesis by genetic approaches in transgenic mice.
167 he AT(1) receptor on T cells mitigates renal fibrogenesis by inhibiting Th1 differentiation and renal
168 n hepatic stellate cells (HSCs), could drive fibrogenesis by modulating the HSC pro-fibrogenic phenot
169 SPHK2 plays an important role in kidney fibrogenesis by modulating transforming growth factor-be
170 ithelial cells (AECs) directly contribute to fibrogenesis by secreting mesenchymal proteins, such as
171 source of myofibroblasts that participate in fibrogenesis by way of synthesis of proinflammatory cyto
172 se to kidney injury, resulting in attenuated fibrogenesis, capillary rarefaction, and inflammation.
173 fects of galectin-3 inhibition on myocardial fibrogenesis, cultured fibroblasts were treated with gal
174 and PTEN in the tubulointerstitium promotes fibrogenesis, defining molecular events underlying PPM1A
176 cle actin (alpha-SMA) signaling pathway, and fibrogenesis despite similar fat accretion with diet-ind
177 ates both diet-induced hepatic steatosis and fibrogenesis, despite the observation that L-Fabp parado
178 GTPase activity selectively in HSC enhances fibrogenesis, driven at least in part through up-regulat
180 role in modulating hepatic inflammation and fibrogenesis during NASH progression, suggesting the pos
183 ifferentiation, cell cycle arrest, and renal fibrogenesis evident in vehicle-treated obstructed kidne
184 ribe here a novel method to quantify hepatic fibrogenesis flux rates both directly in liver tissue an
185 out specific cellular contributors mediating fibrogenesis hampers the design of effective antifibroti
189 nto the cellular and molecular mechanisms of fibrogenesis herald the promise of new therapies to slow
193 sts for PC1 and evaluated the role of PC1 in fibrogenesis in adult rat fibroblasts and myofibroblasts
198 mal transition (EndMT) and spontaneous liver fibrogenesis in EC-specific constitutive hemi-deficient
211 that pomalidomide could be used to decrease fibrogenesis in pancreatic cancer and may be ideal as a
213 gest a novel model to account for persistent fibrogenesis in scleroderma, in which activation of fibr
214 edanib, and obeticholic acid therapy limited fibrogenesis in TGFbeta1/PDGFbetabeta-stimulated PCLSs,
215 ent lysine deacetylase, sirtuin 1 (SIRT1) in fibrogenesis in the cell culture, animal model, and huma
216 shows consistent and reproducible allograft fibrogenesis in the context of single-lung transplantati
218 ng cell culture expansion protects MSCs from fibrogenesis in the host wound environment and increases
220 nction; fibroblasts are poised to coordinate fibrogenesis in tissue injury, neoplasia, and aging.
221 halting the progression of liver injury and fibrogenesis in various liver pathogeneses driven by NLR
223 on in HSCs would decrease HSC activation and fibrogenesis in vivo by disrupting receptor tyrosine kin
224 X-2 or primary rat HSCs in vitro and hepatic fibrogenesis in vivo Finally, we identified a critical r
226 , depletion of mononuclear phagocytes during fibrogenesis in vivo resulted in suppressed NF-kappaB ac
227 reviously unidentified regulatory pathway in fibrogenesis in which a macrophage scavenger receptor pr
229 on: Prom1 plays an important role in biliary fibrogenesis, in part through integrin-mediated TGF path
230 , PC1, are required for critical elements of fibrogenesis, including transforming growth factor beta-
231 panied by increased expression of markers of fibrogenesis; incubation of HSCs with 3PO or knockdown o
232 Firstly, we estimated in mice with acute fibrogenesis induced by a single CCl4 injection the half
233 iver injuries and functions to inhibit liver fibrogenesis induced by either carbon tetrachloride into
235 ular matrix proteins and pathways related to fibrogenesis, infection and immune responses, and transc
236 iciency of Fn14 protected mouse kidneys from fibrogenesis, inflammation, and associated vascular inst
237 ve endotoxin producers, may accelerate liver fibrogenesis, introducing dysbiosis as a cofactor contri
239 chestrates the multifaceted program of renal fibrogenesis involving proximal tubular dysfunction, fai
242 g studies suggest that their contribution to fibrogenesis is limited compared with that of hepatic st
245 senchymal features during carcinogenesis and fibrogenesis is regulated by several mesenchymal transcr
251 of GSK-3beta in cardiac fibroblasts leads to fibrogenesis, left ventricular dysfunction, and excessiv
252 of GSK-3beta in cardiac fibroblasts leads to fibrogenesis, left ventricular dysfunction, and excessiv
254 natively activated macrophages contribute to fibrogenesis, pancreatic intraepithelial neoplasia (PanI
255 onger administered, despite normalization of fibrogenesis parameters; these findings confirm reversal
256 athologic angiogenesis, attenuation of liver fibrogenesis partly mediated through inhibition of hepat
259 cells, and myeloid leukocytes contribute to fibrogenesis predominantly by producing key fibrogenic c
260 etected early onset of fibrosis, whereas the fibrogenesis probe Gd-Hyd proved most accurate for detec
261 specific probe EP-3533 and allysine-targeted fibrogenesis probe Gd-Hyd, MR elastography, and native T
264 on of myofibroblasts, which are critical for fibrogenesis, requires both a mechanical signal and acti
268 any cellular processes being associated with fibrogenesis such as cell migration and contraction.
271 on are believed to be the primary drivers of fibrogenesis that ultimately lead to cirrhosis in patien
272 ar imaging probes for detecting fibrosis and fibrogenesis, the active formation of new fibrous tissue
273 idal remodeling is believed to contribute to fibrogenesis, the impact of sinusoidal angiogenesis on t
274 y an important role in diabetic interstitial fibrogenesis; therefore, targeting Hippo signaling may b
275 ponents of the pathophysiological process of fibrogenesis; these target molecules include collagen ty
276 /-) mice by adoptive transfer drives adipose fibrogenesis through activation of TGFbeta1 signaling; h
278 steatosis-induced HSC activation and hepatic fibrogenesis through mitigating inflammation and oxidati
279 an acute insult, macrophages regulate renal fibrogenesis through the release of various factors that
280 enetic confounders in the recipient on graft fibrogenesis, thus explaining significantly different gr
281 ibroblasts correlates with regions of active fibrogenesis, thus representing a pathologically relevan
282 invasively detect and quantify the extent of fibrogenesis using magnetic resonance imaging (MRI).
284 our work supports that controlling pulmonary fibrogenesis via inhibition of miR-497-5p expression may
286 holangiocyte proliferation but also to liver fibrogenesis via the coordinate activation of GalR1 in c
288 lial growth factor (VEGF), angiogenesis, and fibrogenesis was determined through the analysis of huma
289 ecular events underpinning TGF-beta1-induced fibrogenesis, we examined the proteomic profiling of a T
290 from proximal tubule is sufficient for renal fibrogenesis, we generated a novel mouse strain with ind
291 Because TGF-betas play critical roles in fibrogenesis, we initiated efforts to define the role of
292 ta's pleiotropic effects on inflammation and fibrogenesis, we investigated potential effect modificat
294 tential function of E-type cyclins for liver fibrogenesis, we repetitively treated constitutive CcnE1
295 use certain ciliopathies are associated with fibrogenesis, we sought to explore the fate and potentia
296 ematopoietic cell-derived type I collagen to fibrogenesis, we use a double-transgenic system to speci
298 te into myofibroblasts and promote pulmonary fibrogenesis, while inhibition of its expression could e
299 represents a critical signaling node during fibrogenesis with potential implications for the develop