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1  location of obstruction, and in identifying filling defects.
2  were depicted at cholangiography as rounded filling defects.
3 of ceramide synthesis rescues the airway gas-filling defects.
4 ind between human observers of intravascular filling defects.
5  for strictures, dilatation, and intraductal filling defects.
6  for strictures, dilatation, and intraductal filling defects (all choledocholithiasis) were 86% (40 o
7 nt resembled the ccpp-1Delta mutant with dye-filling defects and B-tubule breaks.
8  of intimal dissections, absence of residual filling defects, and normal (TIMI grade 3) flow in the s
9 ing asymmetric aortic contour abnormalities, filling defects, and various morphologic patterns, such
10 , severe calcium, lesion irregularity, large filling defect, angulated >/= 45 degrees plus calcium, a
11 adioscaphocapitate ligament can be seen as a filling defect at radiocarpal arthrography.
12                    Echocardiography showed a filling defect at the apex of the right ventricle (RV).
13                               Thus, a mobile filling defect cannot be assumed to be residual fecal ma
14 coglycan mutant mice and found microvascular filling defects consistent with arterial vasospasm.
15                              The presence of filling defects consistent with central or peripheral PE
16                       Sometimes, no definite filling defect could be found by cholangiogram (ERC) dur
17    In five women who had a focal intraductal filling defect, immediate stereotactic vacuum-assisted d
18 Urethrography (VCUG), i.e. linear incomplete filling defect in the penile urethra and associated mild
19  that can automatically detect intravascular filling defects in fluorescein angiogram images.
20               Fluorescein angiography showed filling defects in retinal and choroidal circulations an
21 he detection and assessment of intravascular filling defects is important, because they may represent
22 cluding: indeterminate ductal strictures and filling defects, marginal chronic pancreatitis, treatmen
23 pared with 8% of patients with influenza had filling defects on CT pulmonary angiography (p = 0.0001)
24  without evidence of migration, intraluminal filling defects or side branch occlusion.
25 ilation required (1) complete occlusion, (2) filling defects, or (3) signs of intravascular webs.
26  useful to induce matrix stabilization after filling defect spaces.
27 mportant for starch synthesis, and the grain-filling defect was alleviated by overexpression of AGPas
28 ic vacuum-assisted directional biopsy of the filling defect was performed; results were a benign intr
29                      PE are best depicted as filling defects when displayed with a modified window re
30 ltiphase PMCT angiography showed "unspecific filling defects," which were not reported by medical aut
31                   Thrombus was depicted as a filling defect within the blood pool on bright-blood ima
32  was identified as an occlusion or prominent filling defect within the vessel.