ライフサイエンスコーパス: floral organ

1 ly acquire specialized functions within each floral organ.

2 least predominantly expressed in one type of floral organ.

3 lls in the Arabidopsis sepal, a reproducible floral organ.

4 atterns to investigate the homology of their floral organs.

5 st after the production of a fixed number of floral organs.

6 inflorescences are fasciated with additional floral organs.

7 tion by miR319a of TCP4 is critical in these floral organs.

8 that form lateral domains of vegetative and floral organs.

9 eptacle, the part of the stem that holds the floral organs.

10 the flavonoid biosynthetic pathway in maize floral organs.

11 logical and patterning defects of leaves and floral organs.

12 gans tested, RING mRNAs are most abundant in floral organs.

13 flux in patterning, initiation and growth of floral organs.

14 hout nectary development as well as in other floral organs.

15 ion-PCR, but CsTFL RNAs were detected in all floral organs.

16 cells and the resulting production of extra floral organs.

17 he AP3 (APETALA3) promoter specific to these floral organs.

18 some of these factors are LEAFY and UNUSUAL FLORAL ORGANS.

19 wering timing and cell-type specification in floral organs.

20 mously from differentiating cells in lateral floral organs.

21 , specified redundantly by LEAFY and UNUSUAL FLORAL ORGANS.

22 almost exclusively in the epidermal cells of floral organs.

23 AYED DEHISCENCE1 mRNA accumulated within all floral organs.

24 gulatory elements that promote activation in floral organs.

25 elopment and in the initiation and growth of floral organs.

26 of floral organ identity as well as loss of floral organs.

27 lates synthesis of red flavonoid pigments in floral organs.

28 nes to specify the identity of each whorl of floral organs.

29 sive transcriptomic data from vegetative and floral organs.

30 f floral organs and the production of mosaic floral organs.

31 nt marginal growth to leaves, cotyledons and floral organs.

32 and timing of fusion, and fusion with other floral organs.

33 hich promotes growth of the distal region of floral organs.

34 lants and are only known to occur on sterile floral organs [3].

35 In Arabidopsis, floral organs abscise after pollination, and this cell s

36 ion with such mutants, hws loses its delayed floral organ abscission ("skirt") phenotype, suggesting

37 s auxin-mediated ovary patterning as well as floral organ abscission and lateral organ lamina outgrow

38 Floral organ abscission and lateral root emergence are b

39 ation of flowering, rosette leaf senescence, floral organ abscission and silique ripening.

40 ole of indoleacetic acid (IAA) in regulating floral organ abscission in Arabidopsis (Arabidopsis thal

41 ARF2 regulated leaf senescence and floral organ abscission independently of the ethylene an

42 the pathway regulating developmentally timed floral organ abscission is conserved in regulating droug

43 ulate auxin activity specifically within the floral organ abscission zone (AZ).

44 We have identified five novel dab (delayed floral organ abscission) mutants (dab1-1, dab2-1, dab3-1

45 STTM160-expressing plants displayed abnormal floral organ abscission, and produced leaves, sepals and

46 2 receptors are redundant in function during floral organ abscission, but during lateral root emergen

47 HAESA functions in developmentally regulated floral organ abscission.

48 tor-mediated signaling pathway that controls floral organ abscission.

49 receptor signaling system known to regulate floral organ abscission.

50 ng time, floral development, senescence, and floral organ abscission.

51 ia (Petunia x hybrida), ethylene produced in floral organs after pollination elicits a series of phys

52 w GA regulates the late-stage development of floral organs after the establishment of their identitie

53 Overexpression of UNUSUAL FLORAL ORGANS also alters C. hirsuta leaf shape in an LF

54 lender phenotypes in cotyledon, leaflet, and floral organs, an elongated ovary, and negatively correl

55 ifying the "floral state" by contributing to floral organ and meristem identity.

56 of its downstream genes that are involved in floral organ and silique growth is still incomplete.

57 3/PI homologs are generally expressed in all floral organs and (iii) AG homologs are expressed in sta

58 More complete homeotic transformation of floral organs and a greater extent of organ loss in all

59 erations in the size and shape of leaves and floral organs and causes male and female sterility.

60 developmental defects, including callus-like floral organs and fasciated shoot apical meristems.

61 ly expressed to ensure proper development of floral organs and fruits, which are essential for genera

62 synergistically in the development of other floral organs and inflorescence architecture.

63 ession of OVATE unevenly reduces the size of floral organs and leaflets, suggesting that OVATE repres

64 AP1 homologs are generally expressed in all floral organs and leaves, (ii) AP3/PI homologs are gener

65 undation for the subsequent morphogenesis of floral organs and success in reproduction.

66 l phenotypes that include reduced numbers of floral organs and the production of mosaic floral organs

67 YUC genes is essential for the formation of floral organs and vascular tissues.

68 ultrapetala flowers contain more floral organs and whorls than wild-type plants, phenotyp

69 cal meristem (SHOOT MERISTEMLESS and UNUSUAL FLORAL ORGANS) and the hypocotyl (KNAT1).

70 floral organs), paleas (putative first whorl floral organs), and floral meristems.

71 rgement, the production of extra flowers and floral organs, and a decrease in floral meristem determi

72 the production of supernumerary flowers and floral organs, and a delay in floral meristem terminatio

73 ng primordia initiation and distal growth of floral organs, and laminar development of leaflets.

74 ral organs including serrated leaves, narrow floral organs, and petals that contain fewer but more el

75 aberrant cotyledon vein patterning, serrated floral organs, and reduced stature, but plants are viabl

76 rotein-tagged cells, released from abscising floral organs, and used to generate a complementary DNA

77 oing growth; in vascular tissues and various floral organs; and in stomata, trichomes, and hydathodes

78 ar to that of wild-type plants, we find that floral organs are converted into leaf-like organs in sep

79 During shoot regeneration, flowers or floral organs are formed directly from root explants wit

80 ed plants, lateral organs such as leaves and floral organs are formed from the flanks of apical meris

81 ents in maize kernel pericarp and cob; these floral organs are greatly modified in size and shape com

82 Leaves and floral organs are polarized along their adaxial-abaxial

83 ronidase, into the mutant reveals that while floral organs are retained it is not the consequence of

84 Floral organs are specified by the combinatorial action

85 In contrast to most eudicots, floral organs are weakly differentiated in Nuphar and Pe

86 ding yeast Saccharomyces cerevisiae, and the floral organ arrangement in Arabidopsis thaliana.

87 nvolved in the formation of the reproductive floral organs as well as in the control of meristem dete

88 rupt auxin signaling specifically within the floral organ AZ cells.

89 loral stem cells produce a defined number of floral organs before ceasing to be maintained as stem ce

90 known about how "floral organ identity" and floral organ-building genes interact to control floral o

91 The "floral organ-building" gene SPOROCYTELESS/NOZZLE (SPL/NZ

92 they are broadly expressed in vegetative and floral organs, but have relatively higher expression in

93 em and transported to root tips, shoots, and floral organs, but not to mature leaves.

94 overlapping transcriptional programs across floral organ categories.

95 iscrete transcriptional programs in distinct floral organs characterize the more recently derived ang

96 psis strains exhibit aberrant development of floral organs, decline of APETALA3 (AP3) expression, and

97 te sterility, and mutant flowers have severe floral organ defects and indeterminacy that resemble los

98 , including late flowering, aerial rosettes, floral organ defects, low fertility, dwarfism, loss of a

99 larly broadly active ancestral ABCE model of floral organ determination in early angiosperms.

100 Floral initiation and floral organ development are both regulated by the phyto

101 is are required for leaf blade outgrowth and floral organ development as demonstrated by severe pheno

102 factors with partially overlapping roles in floral organ development in Arabidopsis thaliana.

103 ionally substitute SGL1 in compound leaf and floral organ development in M. truncatula.

104 plant, and exhibited a delay in recovery of floral organ development under prolonged drought stress.

105 ped, and loci related to nitrogen uptake and floral organ development were located within mapped quan

106 ole in shoot apical meristem maintenance and floral organ development, and under intense selection du

107 in functions such as polarity specification, floral organ development, meristem development and auxin

108 regulation of FT2 and transcripts related to floral organ development, phytohormones, and cell cycle

109 cription factors have important roles during floral organ development.

110 ral organ-building genes interact to control floral organ development.

111 classic A-C antagonism of the ABC model for floral organ development.

112 is similar to the Drosophila trx, regulates floral organ development.

113 r relationships, cumulatively contributes to floral organ development.

114 reproduction, including floral meristem and floral organ development.

115 oth genes are responsible for the defects in floral organ development.

116 ntal stages including floral bud initiation, floral organ differentiation and bud outgrowth, and iden

117 hat transition from floral bud initiation to floral organ differentiation required changes of genes i

118 cal adhesion forces between petals and other floral organs during floral development.

119 ing methods, we isolated EXCESSIVE NUMBER OF FLORAL ORGANS (ENO), an AP2/ERF transcription factor whi

120 nt ga1-3 mutant shows retarded growth of all floral organs, especially abortive stamen development th

121 Maize anthers, the male reproductive floral organs, express two classes of phased small-inter

122 AP2 in Arabidopsis, where it is required for floral organ fate.

123 s in photomorphogenesis, auxin response, and floral organ formation, possibly via the regulation of u

124 In addition to cotyledon and floral organ fusions, severe lateral organ fusion is fou

125 e mutant phenotypes, including cotyledon and floral organ fusions.

126 0ox1, -2, and -3 have significant effects on floral organ growth and anther development, and that bot

127 rue homology to eudicot and nongrass monocot floral organs has been a topic of debate.

128 Floral organ identities in plants are specified by the c

129 al cascades controlling the specification of floral organ identities.

130 monstrates that these genes are required for floral organ identity [2].

131 d leaf development, and is also required for floral organ identity and development.

132 logs together with AGL6 encode classical SEP floral organ identity and floral termination functions,

133 The type II TFs regulate floral organ identity and flowering time, but type I TFs

134 al events, including proper specification of floral organ identity and number and the development of

135 bit altered spikelet morphology with changed floral organ identity and number, as well as defective f

136 genes, zfl1 and zfl2, led to a disruption of floral organ identity and patterning, as well as to defe

137 tic networks underlying the determination of floral organ identity are well studied, but much less is

138 ss function specifies second and third whorl floral organ identity as described in the classic ABCE m

139 ower, leading to homeotic transformations of floral organ identity as well as loss of floral organs.

140 mportant regulators not only of meristem and floral organ identity but also of flowering timing and c

141 gest that DFO1 functions in maintaining rice floral organ identity by cooperating with PcG proteins t

142 Elevated miRNA172 accumulation results in floral organ identity defects similar to those in loss-o

143 ription factor subfamilies play key roles in floral organ identity determination and floral meristem

144 The floral organ identity factor AGAMOUS (AG) is a key regul

145 heterodimerization in yeast exhibit partial floral organ identity function in transgenic Arabidopsis

146 more diverse than the well-conserved B and C floral organ identity functions.

147 at the nectary can form independently of any floral organ identity gene but is restricted to the 'thi

148 to transcriptionally co-repress the AGAMOUS floral organ identity gene.

149 The Arabidopsis floral organ identity genes APETALA3 (AP3) and PISTILLAT

150 ETALA1 (AP1), and CAULIFLOWER (CAL), and the floral organ identity genes APETALA3 (AP3) and PISTILLAT

151 s controlling the spatial restriction of the floral organ identity genes are more diverse than the we

152 al epigenetic repressors that regulate these floral organ identity genes have been characterized.

153 LOID is an S-linked independent regulator of floral organ identity genes including PvDEF and PvGLO.

154 , and the MADS box gene AGL24, whereas other floral organ identity genes show reduced expression corr

155 at SEPALLATA (SEP) represents a new class of floral organ identity genes since the loss of SEP activi

156 meristem identify gene floricaula (flo) and floral organ identity genes such as deficiens (def) and

157 structures, daffodil orthologues of the ABC floral organ identity genes were isolated and expression

158 mologs act as upstream regulators of the ABC floral organ identity genes, and this along with previou

159 in the expression levels and patterns of two floral organ identity genes, APETALA3 and AGAMOUS.

160 In addition to duplications for floral organ identity genes, TM3-like, StMADS11, ANR1 an

161 relies on the spatial regulation of the ABC floral organ identity genes.

162 ntrolling the expression domains of numerous floral organ identity genes.

163 al reversion regardless of the activation of floral organ identity genes.

164 1/tpc.117.tt1117/FIG1F1fig1A basic model for floral organ identity has been developed using model sys

165 dicating that sex determination occurs after floral organ identity has been established.

166 the homeotic MIKC MADS factors that regulate floral organ identity have been studied in great detail.

167 AGAMOUS, and APETALA are required for proper floral organ identity in Arabidopsis flowers.

168 Floral organ identity in plants is controlled by floral

169 The ABC model of floral organ identity is based on studies of Arabidopsis

170 whorls and organ numbers are reduced and the floral organ identity is compromised.

171 Floral organ identity is controlled by combinatorial act

172 including floral homeotic proteins, by which floral organ identity is determined.

173 terodimerization between the two Arabidopsis floral organ identity MADS proteins APETALA3 (AP3) and P

174 Our findings show that preexisting floral organ identity programs can be partitioned and mo

175 owever, the ABC genes are not sufficient for floral organ identity since ectopic expression of these

176 (FBP6) largely overlap in function, both in floral organ identity specification and floral determina

177 d to repress the transcription of AGAMOUS in floral organ identity specification.

178 flowering and disrupts the specification of floral organ identity when overexpressed in Arabidopsis.

179 However, much less is known about how "floral organ identity" and floral organ-building genes i

180 r developmental programs (e.g. flowering and floral organ identity) as well as stress-related develop

181 eristem identity, a general role for FUL1 in floral organ identity, and a more specific role for FUL2

182 MADS-box genes, known to regulate floral organ identity, are emerging as important regulat

183 2, a gene that is well known for its role in floral organ identity, but whose role in Arabidopsis fru

184 eir roles include control of flowering time, floral organ identity, cell division patterns, and leaf

185 tors and is involved in the specification of floral organ identity, establishment of floral meristem

186 in a combinatorial manner to control proper floral organ identity.

187 er, particularly petal, suggesting a role in floral organ identity.

188 elet meristem identity, and specification of floral organ identity.

189 FBP4, while contributing only moderately to floral organ identity.

190 codes a transcription factor that determines floral organ identity.

191 he B function according to the ABC model for floral organ identity.

192 nes are important regulators of meristem and floral organ identity.

193 er and lower floral meristem, which initiate floral organs in a defined phyllotaxy before being consu

194 s temporally and spatially down-regulated in floral organs in a manner consistent with current models

195 ergradations often observed between adjacent floral organs in basal angiosperms.

196 nditions, and had the largest vegetative and floral organs in both treatments.

197 Here, we characterized floral organs in carpels (foc), an Arabidopsis mutant wi

198 w that leaf-like structures are converted to floral organs in response to LFY activity.

199 MtSUP controls not only the number of floral organs in the inner two whorls, but also in the s

200 eading to partial homeotic transformation of floral organs in the outer two whorls.

201 Petals, defined as the showy laminar floral organs in the second floral whorl, have been show

202 me, however, the developmental nature of the floral organs in these giants has remained a mystery.

203 e GUS reporter gene to fruits and developing floral organs in tomato and Arabidopsis thaliana, sugges

204 netic components that regulate abscission of floral organs, including a pair of related receptor-like

205 etermination of adaxial (dorsal) identity of floral organs, including adaxial stamen abortion and asy

206 ith a shift towards morphologically distinct floral organs, including differentiated sepals and petal

207 ts weak expression of a GUS reporter gene in floral organs, including husk, silk, kernel pericarp, co

208 ction, fertility rate, and the elongation of floral organs, including petals, sepals, and siliques in

209 induce expression of these genes and retain floral organs indefinitely.

210 ese genes suggest that ANT and AIL6 regulate floral organ initiation and growth through modifications

211 greening, hypocotyl elongation, phyllotaxy, floral organ initiation, accessory meristem formation, f

212 of AINTEGUMENTA-LIKE6 at high levels alters floral organ initiation, growth and identity specificati

213 e aspects of floral organogenesis, including floral organ initiation, growth, identity specification

214 ral aspects of flower development, including floral organ initiation, identity specification, growth,

215 serrated rosette leaves, irregular flowers, floral organs inside siliques, reduced fertility, aberra

216 Stepwise conversion of floral organs into leaves in the most severe RNA interfe

217 Patterning of the floral organs is exquisitely controlled and executed by

218 is (Arabidopsis thaliana), the abscission of floral organs is regulated by two related receptor-like

219 Differences in floral organ length determine the pollination efficiency

220 Floral organ lengths were strongly positively correlated

221 s necessary to delay senescence and increase floral organ longevity.

222 Although JAG expression is detected in all floral organs, loss-of-function jag alleles have their s

223 ng defective floral phyllotaxy and increased floral organ merosity, especially supernumerary sepals,

224 veries of the general principles of leaf and floral organ morphogenesis.

225 sewhere on injected plants triggered altered floral organ morphology, including production of multipl

226 ulates the flavonoid biosynthetic pathway in floral organs, most notably kernel pericarp and cob.

227 t mutation in the AtCUL1 gene showed reduced floral organ number and several defects in each of the f

228 NPA has marked effects on floral organ number as well as on regional specification

229 Wildtype floral organ number in early formed flowers is labile, d

230 transcriptional repressor that regulates the floral organ number in the third and fourth floral whorl

231 e most striking phenotypes is an increase in floral organ number, particularly in the sepals and peta

232 dramatically larger meristems and increased floral organ number.

233 live imaging of the germ cell lineage within floral organs of Arabidopsis using light sheet fluoresce

234 All floral organs of B. rapa were strongly correlated, and c

235 the relationship of lodicules to the sterile floral organs of nongrass monocots we have isolated and

236 omics interrogation of gene expression among floral organs of wild type and "formal double" and "anem

237 In contrast to the conservation of floral organ order in angiosperm flowers, nectary glands

238 expressed in ovules, lodicules (second whorl floral organs), paleas (putative first whorl floral orga

239 ointly with ETT in auxin response to promote floral organ patterning and growth.

240 tra petals, suggesting PGX1's involvement in floral organ patterning.

241 opic expression of AIL5 resulted in a larger floral organ phenotype, similar to that resulting from e

242 d symmetry can change, and the ways in which floral organ position can be varied.

243 ant ail6 double mutants show defects in floral organ positioning, identity, and growth.

244 In addition to shedding their floral organs prematurely, nev evr flowers show enlarged

245 er development, and growth and initiation of floral organ primordia is abnormal, suggesting that basi

246 UFO play a role in regulating the number of floral organ primordia, and we discuss possible mechanis

247 em, and early arrest of floral meristems and floral organ primordia.

248 s dynamic and complex expression patterns in floral organ primordia; altering the patterns spatially

249 nts after germination and increased leaf and floral organ production in stm partial loss-of-function

250 cission zone cells did not produce delays in floral organ senescence or abscission.

251 es membrane trafficking and is essential for floral organ shedding in Arabidopsis.

252 conducted a screen for mutations that alter floral organ shedding in Arabidopsis.

253 ere specifically up-regulated at the site of floral organ shedding.

254 es that this gene plays a functional role in floral organ shedding.

255 actor GTPase-activating protein required for floral organ shedding.

256 suggest that the miR156/SPL2 pathway affects floral organs, silique development and plant fertility,

257 genetically decoupled, correlations between floral organ size and both vegetative and life-history t

258 These plants display changes in floral organ size and morphology that are associated wit

259 ss the various ways in which flower size and floral organ size can be modified, the means by which fl

260 ANT:gAIL6 can rescue the floral organ size defects of ant mutants when AIL6 is ex

261 utant protein caused quantitative changes in floral organ size including elongation of pistils and sh

262 * Assuming GA1 causally affects floral organ size, it is interesting that adjacent petal

263 quantitative-genetic and QTL architecture of floral organ sizes, vegetative traits, and life history

264 ein, Dob1p (Mtr4p), HUA ENHANCER2 may affect floral organ spacing and identity through the regulation

265 Moreover, genes with known floral organ-specific expression patterns were correctly

266 emble loss-of-function mutants in E-function floral organ specification genes.

267 rsweet, such as floral transition in spring, floral organ specification, low temperature-mediated flo

268 ss AG and plays an additive role with AP2 in floral organ specification.

269 vasculatures, expanding rosette leaves, and floral organs suggesting a focal role for growth.

270 GmExo70J6 and GmExo70J7 increases greatly in floral organ-supporting receptacles during the developme

271 TCP1, a gene thought to be involved in floral organ symmetric control, was identified as a gene

272 f APETALA1 and 3, SEPALLATA3, LEAFY, UNUSUAL FLORAL ORGANS, TERMINAL FLOWER1, AGAMOUS-LIKE24, and SUP

273 The upper floral meristem initiates extra floral organs that are often mosaic or fused, while the

274 ed receptor mutants generate extra fruit and floral organs that are proposed to arise from enlarged f

275 ation of adjacent embryonic, vegetative, and floral organs, thus implicating miR164 as a common regul

276 gnals may explain the different abilities of floral organs to form fleshy fruit.

277 The differential competence of floral organs to respond to fertilization signals may ex

278 he genetic programs of putatively homologous floral organs traces to those operating in gymnosperm re

279 e F-box protein UFO in AP3 activation and in floral organ transformation.

280 ide with the development of a distinct fifth floral organ type, the staminodium.

281 erent, the same general organization of four floral organ types arranged in concentric whorls exists

282 als, stamens and carpels, with each of these floral organ types having a unique role in reproduction

283 We show that UNUSUAL FLORAL ORGANS (UFO) forms a SCF(UFO) complex, which is a

284 irrhinum gene Fimbriata (Fim) and of UNUSUAL FLORAL ORGANS (UFO) from Arabidopsis.

285 The UNUSUAL FLORAL ORGANS (UFO) gene is required for multiple proces

286 ely regulated by the LEAFY (LFY) and UNUSUAL FLORAL ORGANS (UFO) genes.

287 istem identity genes LEAFY (LFY) and UNUSUAL FLORAL ORGANS (UFO) in Gerbera hybrida, we show that GhU

288 / COMPOUND INFLORESCENCE (S) and the UNUSUAL FLORAL ORGANS (UFO) ortholog DOUBLE TOP (DOT)/ANANTHA (A

289 idopsis, mutations in the F-box gene UNUSUAL FLORAL ORGANS (UFO) result in a number of defects in flo

290 (AP3) gene requires the activity of UNUSUAL FLORAL ORGANS (UFO), an F-box component of an SCF ubiqui

291 n the same pathway and downstream of UNUSUAL FLORAL ORGANS (UFO).

292 es: APETALA1 (AP1), LEAFY (LFY), and UNUSUAL FLORAL ORGANS (UFO).

293 oncentrations of NaCl from the initiation of floral organs until 3 d after pollination.

294 postpones the development of cold-sensitive floral organs until the spring.

295 fferential gene expression in vegetative and floral organs was evident within the clades as well as a

296 By combining silicone flower parts with real floral organs, we created chimeras that identified the m

297 etween the Genomosperma lobed integument and floral organs, we propose that the cupule, integument an

298 in the number, arrangement, and structure of floral organs, whereas the evolutionarily derived monoco

299 Floral organs, whose identity is determined by specific

300 blishes boundaries between most P. axillaris floral organs, with the exception of boundaries between