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1 1 and COP1 controls CO stability to regulate flowering time.
2 hich modulates defense against pathogens and flowering time.
3 ut responses determining seasonal growth and flowering time.
4 ated with altitude were also associated with flowering time.
5 ation, impairs COP1 function in coordinating flowering time.
6 s, and three major loci were found to govern flowering time.
7 is responsible for photoperiodic control of flowering time.
8 1 while the ICCV 96029 form had no effect on flowering time.
9 the early growth stage to jointly determine flowering time.
10 ways exhibit the strongest associations with flowering time.
11 h directly upregulates J/GmELF3a to modulate flowering time.
12 with the environmental sensitivity of maize flowering time.
13 a with allelic perturbations known to affect flowering time.
14 ing germination, vegetative growth rate, and flowering time.
15 Thus, plants face tradeoffs with advanced flowering time.
16 SNPs exhibited high accuracy for predicting flowering time.
17 its, such as grain quality, fruit shape, and flowering time.
18 mperature changes combine to modulate FT and flowering time.
19 nt temperature are major stimuli controlling flowering time.
20 e of the VEG2 gene from pea, associated with flowering time.
21 m4 mutant exhibits slower growth and delayed flowering time.
22 t dosage-dependent effects on ear length and flowering time.
23 pression in the vegetative meristem controls flowering time.
24 e this cold-exposure information to regulate flowering time.
25 i (genes) associated (34% combined PVE) with flowering time.
26 s hypocotyl elongation, root elongation, and flowering time.
27 lored to temperature-dependent plasticity in flowering time.
28 icting the consequences of climate change on flowering time.
29 is was reduced in frequency by selection for flowering time.
30 nd study their association with variation in flowering time.
31 ely been acknowledged as possible drivers of flowering time.
32 trated a role of this gene in the control of flowering time.
33 nds on synchrony between insect activity and flowering time.
34 egulates key physiological responses such as flowering time.
35 -regulatory changes in adaptive variation of flowering time.
36 obiota also altered patterns of selection on flowering time.
37 isms whose abundance in soil correlates with flowering time.
38 LASE6, and play a central role in regulating flowering time.
39 tion of spikelet development and accelerates flowering time.
40 t on major fitness-related traits, including flowering time.
41 sights into the regulation and adaptation of flowering time.
42 promiscuous germination, which then affects flowering time.
43 TaVrn1 had significant epistatic effects on flowering time.
44 , including young leaf serration and altered flowering time.
45 to the promoters of target genes to regulate flowering time.
46 g the thermal plasticity of plant growth and flowering time.
47 e exploration of the genetic architecture of flowering time.
48 iolation, and is also involved in regulating flowering time.
49 regimes, have also been linked to changes in flowering times.
50 e for a role of FRIGIDA in the regulation of flowering times.
51 hat are locally adapted and display distinct flowering times.
54 tle about how biotic interactions can affect flowering times, a significant knowledge gap given ongoi
55 discuss the circadian regulation of growth, flowering time, abiotic and biotic stress responses, and
56 appreciable variation in genetic effects on flowering time across both time and space; the greatest
59 mapping (FOAM) to map the genes that control flowering time, across 22 environments, and identified 1
60 hat phytochrome B (phyB) is able to regulate flowering time, acting in the phloem companion cells, as
61 nts of the photoperiod pathway that regulate flowering time, also control stomatal aperture in a dayl
62 essenger RNA levels generally correlate with flowering time among accessions grown in different photo
64 ronment-specific fitness landscapes based on flowering time and branching architecture, we observed t
65 tion-present conditions, where plasticity in flowering time and early internode lengths was adaptive.
67 d vernalization pathways interact to control flowering time and floret fertility in response to ambie
68 d this warm temperature interruption affects flowering time and floret number and is stage specific.
69 solated from one parent by the difference in flowering time and from the other by habitat adaptation
71 pread concordance of C3 grasses accelerating flowering time and general delays for C4 grasses with in
73 d with yield components, of which seed size, flowering time and harvest maturity traits were stable a
75 CENTRORADIALIS (CEN) is a key regulator of flowering time and inflorescence architecture in plants.
77 gene networks for two major breeding traits, flowering time and oil metabolism, and revealed new cand
82 ine and validated two novel miPs involved in flowering time and response to abiotic and biotic stress
84 unctional protein regulating plant immunity, flowering time and responses to hormones through interac
85 issue is critical and sufficient to regulate flowering time and root growth; control of cotyledon and
88 ed responses to chilling was correlated with flowering time and senescence to create a range of seaso
89 T (FT)), and are associated with seed size, flowering time and soil fertility in dune-adapted sunflo
90 F4 as regulators of endopolyploidization and flowering time and suggest an involvement of cell cycle
91 re reproductively isolated by differences in flowering time and survivorship on soils containing high
92 ation across an altitudinal gradient both in flowering time and the expression and regulation of gene
95 ially contribute to phenotypic plasticity of flowering time and to differential selection observed be
96 rature increases could differentially affect flowering times and pollinator flight periods, leading t
97 nd suggest the possibility of convergence in flowering times and therefore an increase in gene flow a
100 n chromosomes 11 and 15 were associated with flowering time, and four on chromosomes 11 and 16 were a
102 cryptochromes regulate the circadian rhythm, flowering time, and photomorphogenesis in higher plants
103 s genotypic correlations between Delta(13)C, flowering time, and plant height were not significant.
104 , GNC and GNL control germination, greening, flowering time, and senescence downstream from auxin, cy
106 enarios, changes in carbon metabolism during flowering time are a consequence rather than a cause of
109 tic basis of divergence in floral traits and flowering time associated with mating-system evolution,
111 and down-regulation (>three folds) of eight flowering time-associated genes (including six genes val
113 y of the three panels identified nearly 1000 flowering time-associated SNPs, mainly distributed aroun
114 andidate gene-based association mapping in a flowering time association panel (92 diverse desi and ka
116 ve trait locus controlling the difference in flowering time between maize and its wild ancestor, teos
118 ody perennials involves pathways controlling flowering timing, bud dormancy and outgrowth in response
119 ences on key agricultural traits, especially flowering time but also yield, biomass, and biennial gro
120 Rising temperatures have begun to shift flowering time, but it is unclear whether phenotypic pla
121 pe II TFs regulate floral organ identity and flowering time, but type I TFs are relatively less chara
122 all component of the phenotypic variation in flowering time, but were sufficient to produce a signatu
123 xport, and regulates the circadian clock and flowering time by binding to chromatin of the flowering
124 ed the effect of the floral repressor FLC on flowering time by using constant temperature laboratory
126 s on the regulatory role of MCTP1 (FTIP1) in flowering time control in Arabidopsis, demonstrating tha
127 field data reveal that a cryptic function of flowering time control is to limit seed set of winter an
128 he expression and regulation of genes in the flowering time control network, often independent of FLC
130 oduction in temperate regions depends on its flowering time control, but the underlying molecular mec
131 nctionally characterized in other plants for flowering time control, seed development and pod dehisce
132 the importance of pre-winter temperatures in flowering time control, we artificially simulated climat
135 sea level) in the Swiss Alps to test whether flowering time correlated with altitude under different
137 genome-wide association studies (GWAS) using flowering time data from a switchgrass association panel
138 gene families, including those with roles in flowering time, defense response, flavor, and pigment ac
139 he shoot apex, VRN2 differentially modulates flowering time dependent on photoperiod, whilst its pres
140 association potential (41% combined PVE) for flowering time differentiation in cultivated and wild ch
143 iology, the ways in which genetic studies of flowering time diversity have enriched the field of evol
148 tal of 8, 22, and 47 QTL were identified for flowering time, early vigor, and energy traits, respecti
149 egulation of FLORE, whereas GUS-staining and flowering time evaluation were used to determine its bio
150 size recent findings on the genetic basis of flowering time evolution as a way to begin deciphering w
152 of floral development (APETALA3 and PI) and flowering time (FLC) in the Brassicales and for the regu
153 ons in the control of germination, greening, flowering time, floral development, senescence, and flor
154 of parental isolating major genes related to flowering time from one parent and alleles of major gene
156 for two target traits-heading date (HD) and flowering time (FT)-were identified and positioned on li
158 ed from maize and encompassing ZCN8, a major flowering time gene associated with adaptation to high l
160 e co-expressed with five genes homologous to flowering time genes in Arabidopsis, and Glyma11g15480 w
161 e expression of Brachypodium homologs of key flowering time genes in the photoperiod pathway such as
163 ime-associated expression of eight potential flowering time genes was confirmed in three tulip cultiv
164 the increased transcriptional levels of two flowering time genes with opposing functions, FLOWERING
165 e examined the allelic variation in the four flowering-time genes across the diverse accessions from
166 Here, we review examples of pleiotropy of flowering-time genes and highlight those that also influ
169 ker models identified only 1 of 14 benchmark flowering-time genes, while transcript models identified
170 onomic traits, including disease resistance, flowering time, glucosinolate metabolism and vitamin bio
172 We found that the genomic architecture of flowering time has been shaped by the most recent whole-
173 date, suggesting that temperature control of flowering time has evolved to constrain seed set environ
176 e as traditional labor-intensive measures of flowering time, height, biomass, grain yield, and harves
180 ly been shown to affect natural variation in flowering time in Arabidopsis thaliana, most either show
183 c overexpression of PhFT4 and PhFT5 promotes flowering time in Arabidopsis, and that of PhFT1, PhFT2
186 the genetic control of natural variation in flowering time in Brachypodium distachyon, a nondomestic
187 ironmental and endogenous cues that regulate flowering time in C. hirsuta We found that petal number
189 ngs indicate that climate change is shifting flowering time in complex ways, even across local spatia
191 on of RAV gene function in the regulation of flowering time in monocotyledonous and dicotyledonous pl
192 advance many phenological events, including flowering time in plants and the flight time of insects.
198 r understanding of the genetic regulation of flowering time in switchgrass will aid the development o
202 st that loss of PPD function does not affect flowering time in the presence of functional HR, whereas
205 domestication-related traits, shattering and flowering time, in a mapping population derived from a c
215 viable yet show phenotypes in seed dormancy, flowering time, lateral root, and stomata formation-comp
216 haplotyping algorithm, enabled us to map the flowering time locus in the diploid wheat Triticum monoc
217 e resequenced the entire major but conserved flowering time locus Ppd-D1 in just a few such selected
218 sured traits such as leaf area, growth rate, flowering time, main stem branching, rosette branching,
219 id depletion produces non-genetic changes in flowering time, maturation, and growth rate that are her
221 sted this hypothesis by growing large-effect flowering time mutants of Arabidopsis thaliana in multip
222 , day length and vernalization influence the flowering time of 59 genotypes of Arabidopsis thaliana w
224 e long-term (1895-2013) relationship between flowering times of grass species and climate in space an
225 osystems due to a greater advancement in the flowering times of late-flowering species than early-flo
226 ions in crops typically result in changes in flowering time, often involving the PEBP family of genes
229 rthermore, we show that nonlinear changes in flowering times over the 33-year record are obscured by
230 investigate the natural diversity governing flowering time pathways in a nondomesticated grass, the
231 dentified as a system integrator of numerous flowering time pathways in many studies, and its homolog
233 such as inversions, and genes from multiple flowering-time pathways exhibit the strongest associatio
234 transitory starch synthesis and analyze its flowering time phenotype in relation to its altered capa
235 nctional analyses in Arabidopsis resulted in flowering time phenotypes in line with TgTFL1 being a fl
236 lly with its homolog ICA1, alters growth and flowering time plasticity in relation to temperature in
237 report findings from the dissection of rice flowering-time plasticity in a genetic mapping populatio
240 , 6 of these genomic areas appeared to carry flowering time QTL, 1 of which corresponds to Ppd-D1, a
244 en together, eight potential known/candidate flowering time-regulating genes [efl1 (early flowering 1
245 is well appreciated that genetic studies of flowering time regulation have led to fundamental advanc
247 es insights into the evolutionary context of flowering time regulation in the Poaceae as well as eluc
249 differentiation required changes of genes in flowering timing regulation, while transition to floral
250 kout mutants to examine the role of SPL10 in flowering-time regulation and we investigated possible i
252 t the two GATA factors act upstream from the flowering time regulator SUPPRESSOR OF OVEREXPRESSION OF
253 light receptor and well-known photoperiodic flowering time regulator, in cellulose biosynthesis.
254 ort the identification of GmPRR3b as a major flowering time regulatory gene that has been selected du
255 demonstrate the new methods, we analyzed six flowering time related traits in Arabidopsis thaliana an
256 further validate pKWmEB, we re-analyzed four flowering time related traits in Arabidopsis thaliana, a
257 dentified 48 previously reported genes for 7 flowering time-related traits in Arabidopsis thaliana.
259 that miR824 and AGL16 modulate the extent of flowering time repression in a long-day photoperiod.
265 by 7 days, which is similar in magnitude to flowering time shifts over 2-3 decades of climate change
267 tially associated with meristem maintenance, flowering time, stomatal density, WUE, and/or stress res
269 egetative shoot and required for a wild-type flowering time, supporting that TFL1 expression in the v
270 These results show that CmNF-YB8 influences flowering time through directly regulating the expressio
272 nd are thus candidates for the adaptation of flowering time to environmental gradients such as altitu
274 were associated with major fiber quality and flowering time traits in previously published QTL mappin
275 e maize flowering time variants we evaluated flowering time traits using an extremely large multi- ge
278 d quantitative trait locus (QTL) mapping for flowering time variation between two winter annual popul
285 ease or induction by cold and interacts with flowering-time variation to construct different seasonal
286 ent, is involved in endopolyploidization and flowering time via genetic interaction with MOS1, a nega
290 Branching was under stronger selection, but flowering time was more genetically variable, pointing t
295 contrast, more lateral branches and delayed flowering time were observed in SPL13 silenced plants.
296 ganism Arabidopsis thaliana to determine how flowering time (which defines seed-maturation temperatur
297 ly favorable developmental traits, including flowering time, which resulted in the creation of variet
298 on and eliminates the negative regulation of flowering time, while the analogous C76S substitution in
299 ination season affects subsequent growth and flowering time, with significant genotype-by-environment