ライフサイエンスコーパス: fluid-phase

1 analogs was similar to that observed in the fluid phase.

2 ssure is the isotropic stress exerted by the fluid phase.

3 uid to superlattice and from superlattice to fluid phase.

4 ),Bb], which normally is present only in the fluid phase.

5 ent IAA cross-link two insulin moieties in a fluid phase.

6 her nonionic detergents than bilayers in the fluid phase.

7 hat graphene be dispersed and processed in a fluid phase.

8 ar volume and area per headgroup in the bulk fluid phase.

9 aximum at the transition between the gel and fluid phase.

10 he effect of enhancing binding to aPL in the fluid phase.

11 rescence microscopy suggests a homogeneously fluid phase.

12 at the interface between the matrix and the fluid phase.

13 ver wild-type in the solid phase but not the fluid phase.

14 consists of a matrix meshwork embedded in a fluid phase.

15 letes interstitial HA and eliminates the gel-fluid phase.

16 onon gap when the lipid transitions into the fluid phase.

17 100 mmHg through the creation of a large gel-fluid phase.

18 ges and bacteria in proximity and out of the fluid phase.

19 oversight of the predominant role of the gel-fluid phase.

20 perties of small unilamellar vesicles in the fluid-phase.

21 ge pathways and viscous coupling between the fluid phases.

22 id bilayer were the same for the gel and the fluid phases.

23 nucleation of microcrystalline solids within fluid phases.

24 h fluidity and internal lipid order into the fluid phases.

25 ase and the POPG partitioned between gel and fluid phases.

26 ycero-3-phosphocholine (DPPC)/cholesterol in fluid phases.

27 ries, and can merge quickly, consistent with fluid phases.

28 be catalyzed by the interface between the 2 fluid phases.

29 tively profile the deformation of nanoscale, fluid-phase 1,2-dioleoyl-sn-glycero-3-phosphocholine (DO

30 toyl-sn-glycero-3-phosphocholine (DPPC), and fluid phase, 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoch

31 1 (p1) and piscidin 3 (p3) are determined in fluid-phase 3:1 phosphatidylcholine/phosphatidylglycerol

32 paramagnetic behavior in either the solid or fluid phase above 200 K and weak antiferromagnetic inter

33 ays was unexpectedly linked and dependent on fluid-phase activation of factor XII.

34 patients with HAE, which assumes a systemic, fluid-phase activation of the contact system to generate

35 24 SCR protein that is secreted with normal fluid-phase activity but marked loss of complement regul

36 peptide-MHC class II complexes derived from fluid-phase Ag processing remains unchanged.

37 entially shielding them from inactivation by fluid phase agents and promoting efficient generation of

38 llectins, we hypothesized the existence of a fluid-phase analog of CL-12 released from cells, which m

39 brane thickness fluctuation amplitude in the fluid phase and a rapid suppression of fluctuations upon

40 ti-constituent mixture comprised of a linear fluid phase and a thrombus (solid) phase.

41 is considered to be a sludgelike mixture of fluid phase and aggregates of rigid clusters.

42 -component homogeneous giant vesicles in the fluid phase and analyses of the domain areas of the fuse

43 excessive complement activation, both in the fluid phase and at host cell surfaces decorated by C3d.

44 xing of the anesthetic drugs in the membrane fluid phase and exclusion from the solid phase.

45 by complement dysregulation occurring in the fluid phase and in the glomerular microenvironment, whic

46 key role in the homeostasis of complement in fluid phase and on cell surfaces.

47 t controls complement activation both in the fluid phase and on specific cell surfaces, thus allowing

48 PGK inhibited C9 polymerization both in the fluid phase and on the surface of sheep erythrocytes.

49 ability of FI to degrade C4b and C3b in the fluid phase and on the surface, irrespective of the cofa

50 ycan hyaluronan, together with the large gel-fluid phase and pressures it generates, were recently id

51 LdRab5a and LdRab5b differentially regulate fluid phase and receptor-mediated endocytosis in Leishma

52 consequences and epitope mapping, using both fluid phase and solid phase approaches, were performed.

53 f matrix-detached cells in a complex ascites fluid phase and subsequent adhesion to the mesothelium l

54 readily forms complexes with plasminogen in fluid phase and such complexes are present in human seru

55 rnative pathway complement activation in the fluid phase and the cell surface in the fH-mutant mice.

56 up by nonspecific endocytosis as part of the fluid phase and traveled through the endosomal compartme

57 ric multiphase inclusions of crystalline and fluid phases and are characterized by a "crustal" signat

58 ertaining to the adsorption of 34 nm radius, fluid-phase and gel-phase liposomes onto a titanium oxid

59 t morphology and show delayed uptake of both fluid-phase and membrane markers.

60 ) is essential for complement homeostasis in fluid-phase and on surfaces.

61 membrane protein required for trafficking of fluid-phase and receptor-mediated endocytic cargos.

62 nfluence gp120 binding, HIV-1 attachment, or fluid-phase and receptor-mediated endocytosis.

63 plement system is tightly controlled by many fluid-phase and tissue-bound regulators.

64 icated in phagosome maturation, retention of fluid phase, and antigen presentation.

65 e IQC is body-centred, self-assembles from a fluid phase, and in parameter space neighbours clathrate

66 or self-assembly and without an intermediate fluid phase, and in the continuous case, solid-solid rec

67 ive pathway is continuously activated in the fluid phase, and tissue surfaces require continuous comp

68 ibutable to coexistence of gel (L(beta)) and fluid phases are found for ternary mixtures with low cho

69 ion of human Bruch's membrane/choroid to the fluid-phase assay accelerated the C3b cleavage, and this

70 Fluid-phase assays showed reduced FH activities that cor

71 holipid syndrome, using both solid-phase and fluid-phase assays.

72 Fluid-phase assembly is particularly attractive, but the

73 ase diagram, allowing the rational design of fluid-phase assembly processes.

74 ere incubated with C-reactive protein in the fluid phase at physiological concentrations, no associat

75 d model wherein nascent C3b generated in the fluid phase attaches nonspecifically to its targets.

76 in two functional assays (cell surface- and fluid-phase-based) measuring cofactor activity of CFH in

77 A first-order phase transition in the fluid phase between a molecular insulating fluid and a m

78 ) upon varying electrolyte concentration and fluid phase bilayers formed from DMPG/DMPC and POPG/POPC

79 ition of the module's concave surface forces fluid-phase bilayers to bend locally.

80 rimental data and shows that, in the case of fluid-phase bilayers, polymerization arises equally due

81 Published studies utilize solid-phase or fluid-phase binding assays to show that the factor H Y40

82 o determine equilibrium unfolding curves and fluid phase boundaries for solutions of coarse-grained g

83 motion of lipid molecules, especially in the fluid phase, but not the faster internal motion.

84 C3 convertase and facilitates recruitment of fluid-phase C3 convertase to the cell surfaces.

85 activation fragments, including the typical fluid-phase C3(H(2)O), measured by surface plasmon reson

86 likely to explain this because regulation of fluid phase C3b is unaffected by domains 19-20.

87 ive generation and/or defective clearance of fluid-phase C3b:protein complexes may have pathological

88 critical roles in internalizing membrane and fluid phase cargo and in balancing the inflow and outflo

89 and enhances, respectively, the delivery of fluid-phase cargo to lysosomes, without affecting Rab5 a

90 Thus, septins regulate fluid-phase cargo traffic to lysosomes by promoting macr

91 llagen type I was determined with the use of fluid-phase cell attachment assays in HSFs, human foresk

92 yer, 2) the reorganization of unconstrained, fluid-phase CL molecules in concert with Drp1 self-assem

93 lains resistance to warfarin, which inhibits fluid-phase coagulation but not selectins.

94 linear and branched lipids gave rise to gel/fluid phase coexistence at room temperature.

95 leation and growth in membrane mixtures with fluid phase coexistence.

96 ms with liquid-ordered and liquid-disordered fluid phase coexistence.

97 strates the cooperation between membrane and fluid phase complement inhibitors and the body's ability

98 cally, this analysis separated patients with fluid-phase complement activation (clusters 1-3) who had

99 Fluid-phase complement activation caused deposition of C

100 In patients with fluid-phase complement activation, those in clusters 1 a

101 at these C3 glomerulopathies are diseases of fluid-phase complement dysregulation.

102 e hypothesized that the interactions between fluid-phase complement regulators and conserved ricketts

103 ls and cell lines was the time absent from a fluid-phase complement source; therefore, we hypothesize

104 The unsaturation level of the fluid-phase component was varied by lipid choice, i.e.,

105 h respect to saturation under single and two fluid-phase conditions in a micromodel.

106 used to characterize the transport under two fluid-phase conditions.

107 rred to represent a magmatic-derived aqueous fluid phase, contain significant concentrations of Mo (~

108 model systems, we show that an HA-dependent fluid phase contributes substantially to pressures in ma

109 tory functions of factor H without affecting fluid-phase control of complement.

110 be regulated by a series of steps involving fluid-phase convertases.

111 atic Helfrich Hamiltonian, much unlike their fluid-phase counterparts.

112 We investigated the binding of fluid-phase CRP to six immobilized proteins: complement

113 al point and throughout the metastable fluid-fluid phase diagram.

114 wo mechanisms: cell-free infection following fluid-phase diffusion of virions and by highly-efficient

115 1)) relaxation rates from 0.022 to 21.1 T of fluid phase dipalmitoylphosphatidylcholine bilayers are

116 In fluid-phase DMPC bilayer systems, the peptides interacte

117 lls can also segregate into micrometer-scale fluid phase domains.

118 e POPC/POPG MLVs but is deeply inserted into fluid phase DPPC/POPG vesicles, resulting in immobilizat

119 hat C3b:plasma protein complexes form in the fluid-phase during complement activation.

120 tra-rare renal diseases are characterized by fluid-phase dysregulation of the alternative complement

121 Dense deposit disease is caused by fluid-phase dysregulation of the alternative complement

122 Fluid phase endocytosis accounted for the remainder of a

123 endocytic-dependent degradation and restores fluid phase endocytosis in rvs161 cells.

124 lastomeres exhibit polarized actin-dependent fluid phase endocytosis only on the G(M1), integrin, mic

125 on level and phosphorylation state regulates fluid phase endocytosis via the interaction between the

126 Fluid phase endocytosis was inhibited by expression of a

127 d impedes Lucifer yellow uptake (a marker of fluid phase endocytosis).

128 The ability to take up Ag, measured by fluid phase endocytosis, was comparable between CIITA(-/

129 monstrate that Cav1 overexpression decreases fluid phase endocytosis, whereas silencing of Cav1 enhan

130 that the cell internalizes light chain by a fluid phase endocytosis, which is then modified and ulti

131 esponding sequence in Cav2, did not suppress fluid phase endocytosis.

132 esis that Cav1 can regulate Cdc42-dependent, fluid phase endocytosis.

133 ular uptake of bisphosphonate drugs requires fluid-phase endocytosis and is enhanced by Ca2+ ions, wh

134 Pase Rab5 has previously been shown to block fluid-phase endocytosis and trafficking of plasma membra

135 es suggest that BSA-SWNTs enter NRK cells by fluid-phase endocytosis at a rate of 30 fg/day/cell upon

136 ze dietary protein via receptor-mediated and fluid-phase endocytosis for intracellular digestion and

137 erexpression, CORO1C overexpression restored fluid-phase endocytosis in SMN-knockdown cells by elevat

138 which was confirmed by direct measurement of fluid-phase endocytosis in the presence of these compoun

139 hibited by dansylcadaverine, indicating that fluid-phase endocytosis is involved in the initial inter

140 ound that LdRab5a specifically regulates the fluid-phase endocytosis of horseradish peroxidase and al

141 Fluid-phase endocytosis of LDL by M-CSF human macrophage

142 QDs are internalized by macropinocytosis, a fluid-phase endocytosis process triggered by Tat-QD bind

143 inocytosis is a type of poorly characterized fluid-phase endocytosis that results in formation of rel

144 bulin, co-localize with dextran, a marker of fluid-phase endocytosis, and induce fibrillization of in

145 In fact, through the process of fluid-phase endocytosis, Pgp was redistributed from the

146 mechanism used by GPI-anchored receptors and fluid-phase endocytosis.

147 les through local TLR4 receptors and through fluid-phase endocytosis.

148 ermore, uptake characteristics differed from fluid-phase endocytosis.

149 ial cells is generally believed to occur via fluid-phase endocytosis.

150 ciated with macropinosomes, the vesicles for fluid-phase endocytosis.

151 nd for clathrin-independent, actin-dependent fluid-phase endocytosis.

152 s occurs nonspecifically and originates from fluid-phase endocytosis.

153 atoms from a Cu-Au mixture, leaving behind a fluid phase enriched with Au adatoms that subsequently a

154 C1INH activity levels because inhibition of fluid-phase Factor XIIa and kallikrein requires lower C1

155 es of solute transport (dispersion) in a two fluid-phase filled micromodel, we directly delineated th

156 rder of magnitude larger than that in single fluid-phase flow in the same micromodel.

157 is an evolutionarily-conserved, large-scale, fluid-phase form of endocytosis that has been ascribed d

158 In the fluid phase, GM1 was shown to strongly soften the bilaye

159 inal region of AspA was deficient in binding fluid-phase gp-340, and L. lactis cells expressing AspA

160 been long appreciated, the less mobile, gel-fluid phase has been largely ignored for historical and

161 Data from fluid-phase HRP electron tomography showed that fibricar

162 ation of the performance of a nonradioactive fluid phase IAA assay in relation to standard IAA radioa

163 d samples (n = 477 from 78 women) by 11-plex fluid-phase immunoassay.

164 methods such as comprehensive phage display, fluid-phase immunoassays, and antigen microarrays have b

165 ere we report a simple, robust and ultrafast fluid-phase immunocapture method for clinical measuremen

166 ntroduced by electrospray injection from the fluid phase in ambient conditions into vacuum, and are s

167 Capillary trapping of a nonwetting fluid phase in the subsurface has been considered as an

168 e focused on coexisting optically resolvable fluid phases in simple lipid mixtures.

169 , temperature-induced transition from gel-to-fluid phase increases the lateral diffusion of the lipid

170 t a hyaluronan-rich, relatively immobile gel-fluid phase induces vascular collapse and hypoperfusion

171 urthermore, three-dimensional binding in the fluid phase is biologically and physically distinct from

172 These mixtures consist of a fluid phase lipid component (1,2-dilauroyl-sn-glycero-3-

173 chain unsaturation on the interaction of the fluid-phase lipid and cholesterol.

174 In the case of fluid-phase lipid bilayers, the actin adsorbs to form a

175 With increasing fluid-phase lipid fraction, these hybrid, supported memb

176 somal and emulsion based CAFs with solid and fluid phase lipids, with or without the TLR agonists R84

177 or calculating areas for gel-phase lipids to fluid-phase lipids and obtained agreement to within 5% o

178 We observed that while the fluid phase liposome formulations showed the highest in

179 Using fluid-phase luciferase immune precipitation (LIPS) assay

180 extracellular milieu through a nonsaturable, fluid phase macropinocytic mechanism that is distinct fr

181 te that PTD-mediated transduction occurs via fluid-phase macropinocytosis involving an intracellular

182 cumulation can occur by nonreceptor mediated fluid-phase macropinocytosis when macrophages are differ

183 ntry of several molecules through a putative fluid-phase macropinocytotic mechanism.

184 nelles by colocalization of the internalized fluid phase marker dextran with both mepacrine and trans

185 egalin-cubilin ligand albumin as well as the fluid phase marker dextran.

186 These tubules were labelled by the fluid phase marker horseradish peroxidase (HRP), and wer

187 Endocytosis of a fluid phase marker is unaffected by TbVps34 RNAi, but re

188 imaging of endogenous proteins along with a fluid-phase marker to address these issues.

189 Uptake of the fluid-phase marker, dextran, was only modestly affected.

190 inocytic compartments during the uptake of a fluid-phase marker, dextran.

191 the secretion and trafficking of VEGF and a fluid-phase marker, methylpolyethylene glycol (mPEG).

192 ly in intracellular vesicles with dextran, a fluid-phase marker.

193 live-cell fluorescent imaging of intravirion fluid phase markers to monitor HIV-1 uncoating at the in

194 ke of Ig-opsonized targets, latex beads, and fluid phase markers, and it was accompanied by activatio

195 of reducing binding to a panel of aPL in the fluid phase (mean +/- SD inhibition 14 +/- 18.5% versus

196 tion are similar to the fluctuation modes of fluid phase membranes, highlighting the importance of un

197 In one-component fluid-phase membranes, the probe has the expected spectr

198 ediabetic patients compared with our current fluid phase micro-IAA radioassay (mIAA; 44 and 74%, resp

199 and suppresses GxcU-mediated activity during fluid-phase micropinocytosis.

200 ge of transport hydrodynamics as a result of fluid-phase occupancy.

201 ng glass formation is the development in the fluid phase of local structures that somehow prevent cry

202 t reduce complement regulation in the actual fluid phase of plasma, it reduces regulation on HDL part

203 COPD, as judged by reduced chemotaxis to the fluid phase of sputum from subjects with COPD compared w

204 ains rich in Cer coexist with the continuous fluid phase of the membrane.

205 >99.9% of the microorganisms inhabiting the fluid phase of this particular fracture.

206 uantitative structures of the fully hydrated fluid phases of dimyristoylphosphatidylcholine (DMPC) an

207 mbotic or antifibrinolytic mediators in the "fluid phase" of the blood can also predispose toward ACS

208 measurements of the bending rigidity of the fluid phase only, whereas electrodeformation and fluctua

209 Similarly, plasmin either in the fluid phase or attached to surfaces inhibited complement

210 elopment, physical forces originating from a fluid phase or from cells pulling on their environment c

211 tion or expression of P1, in the presence of fluid-phase or adsorbed saliva or salivary agglutinin pr

212 s of Ags such as OVA and transferrin but not fluid-phase or phagocytic Ag uptake.

213 phase Darcy flow in porous media is that the fluid phases organize into separate flow pathways with s

214 n by preference of heavier Mo isotopes for a fluid phase over rutile, the dominant mineral host of Mo

215 level of PSGL-1 and had enhanced binding to fluid-phase P- and E-selectin compared with Ly-6C(lo) mo

216 Pentraxin 3 (PTX3) is a fluid-phase pattern recognition molecule and a key compo

217 Pentraxin 3 (PTX3) is a fluid-phase pattern recognition receptor of the humoral

218 ar effect of KL(4) on the orientation of the fluid phase PG headgroups is observed, with similar chan

219 Macrophages that show high rates of fluid-phase pinocytosis also show similar high rates of

220 sions can take up LDL-sized nanoparticles by fluid-phase pinocytosis and indicate that fluid-phase pi

221 tive unmodified LDL by receptor-independent, fluid-phase pinocytosis converting these macrophages int

222 Fluid-phase pinocytosis has been demonstrated by macroph

223 ticles) can serve as models of LDL uptake by fluid-phase pinocytosis in cultured human monocyte-deriv

224 The mechanisms of regulation of fluid-phase pinocytosis in macrophages and, specifically

225 We therefore sought to determine whether fluid-phase pinocytosis occurs in vivo in macrophages in

226 Fluid-phase pinocytosis of LDL by macrophages is regarde

227 by fluid-phase pinocytosis and indicate that fluid-phase pinocytosis of LDL is a mechanism for macrop

228 e phenotypes demonstrate either constitutive fluid-phase pinocytosis or inducible fluid-phase pinocyt

229 Thus, targeting macrophage fluid-phase pinocytosis should be considered when invest

230 mounts of native LDL by receptor-independent fluid-phase pinocytosis, either constitutively or in res

231 itutive fluid-phase pinocytosis or inducible fluid-phase pinocytosis.

232 res, KL(4) is more peripheral and dynamic in fluid phase POPC/POPG MLVs but is deeply inserted into f

233 f large molecules is a receptor-independent, fluid-phase process that takes place by dynamin-dependen

234 We have developed a multianalyte fluid-phase protein array technology termed high-through

235 membrane attack complex (MAC) assembles from fluid-phase proteins to form pores in lipid bilayers.

236 )-macroglobulin and occurred primarily via a fluid-phase, rather than receptor-mediated, uptake pathw

237 a three-dimensional (3D) technique in which fluid-phase receptors and ligands are removed from their

238 athogenic leptospires bound factor H, a host fluid-phase regulator of the alternative complement path

239 Factor H (FH) is the most important fluid-phase regulator of the alternative pathway of the

240 Allotypic variants of a key complement fluid-phase regulatory protein, complement factor H (CFH

241 nd quantify the principal mechanism of CO(2) fluid phase removal in nine natural gas fields in North

242 data for the phospholipid probes in the DOPC fluid phase require two components (fast and slow).

243 Fluid-phase salivary agglutinin and, to a lesser extent,

244 mmunoreactive and were readily aggregated by fluid-phase salivary agglutinin.

245 To quantitatively understand how the fluid-fluid phase separation affects the crystal nucleation, w

246 nitiated by aqueous mixing followed by fluid-fluid phase separation, such as coacervation.

247 Fluid phase separations of racemates are difficult becau

248 acropinocytosis delivered small, fluorescent fluid-phase solutes into endolysosomes sufficiently fast

249 analysis of the interaction between LPS and fluid-phase supported lipid bilayer assemblies (sLBAs),

250 mbrane attack complex and its by-product the fluid-phase terminal complement complex and relate these

251 on occurs via a pathway more consistent with fluid-phase than receptor-dependent endocytosis and is s

252 ctrum of disorders, ranging from exaggerated fluid-phased thrombosis dependent on prothrombotic agent

253 ting in the wedge, and transport of the melt/fluid phase to a reservoir in the crust beneath Mt Raini

254 concentration gradient, all delivered in the fluid phase to eliminate variability associated with air

255 in the silicate melt causes unmixing of the fluid phase to form an H2O-rich vapour and a hydrosaline

256 ic model linking chaotic-mixing rates in the fluid phase to the generic structural properties of gran

257 ution of distances from random points in the fluid phase to the nearest fiber.

258 antifying the contributions of free- and gel-fluid phases to hydraulically transmitted pressures in a

259 luid pressure measurement to capture the gel-fluid phase, together with a dependence on xenograft and

260 We used fluorescent dextrans as fluid phase tracers and observed the cholecystokinin-eli

261 Fluid phase tracers were colocalized with cleaved BZiPAR

262 Here, we used fluid-phase tracers in murine models and determined that

263 that include endocytosis and degradation and fluid-phase transcytosis in the apical-to-basal directio

264 compartments in part via diffusion-limited, fluid phase transfer through the cytosol, suggesting tha

265 approximately 45 degrees C, with the gel to fluid phase transition (L(beta)-L(alpha)) occurring at a

266 n of a membrane bilayer following the gel-to-fluid phase transition in a pure phospholipid vesicle wa

267 pectroscopy, we are able to trigger a gel-to-fluid phase transition in lipid vesicles and monitor in

268 sity and diffusivity change across the fluid-fluid phase transition increases with motif dimension.

269 te NMR and revealed that the lamellar gel-to-fluid phase transition occurs below 0 degrees C, reflect

270 onitoring (QCM-D) to directly detect the gel-fluid phase transition of a SPB.

271 ty of phospholipid membranes at their gel-to-fluid phase transition, where the release of encapsulate

272 Fluid phase transitions inside single, isolated carbon n

273 e temperature-driven fluid-to-gel and gel-to-fluid phase transitions of the fatty acids within the br

274 markedly alter lipid physical properties in fluid phases underscores the need to study the function

275 ium will be present in their interior due to fluid phase uptake from the extracellular space.

276 results reveal a novel and key role for the fluid phase uptake of extracellular calcium and its subs

277 ester hydroperoxides, induce TLR4-dependent fluid phase uptake typical of macropinocytosis.

278 ly internalize exogenous protein antigens by fluid-phase uptake and receptor-mediated endocytosis.

279 f the quantum dots allowed for non-targeted, fluid-phase uptake by macrophage cells.

280 ine the fundamental roles of endocytosis and fluid-phase uptake for the maintenance of the glomerular

281 red for proper cell migration, spreading and fluid-phase uptake in both established cell lines and hu

282 lize and degrade extracellular protein via a fluid-phase uptake mechanism termed macropinocytosis(1).

283 Nonsaturable fluid-phase uptake of LDL by macrophages converts the ma

284 rophage phenotype demonstrating constitutive fluid-phase uptake of native LDL leading to macrophage c

285 ive endosomal compartments, endocytosis, and fluid-phase uptake were severely disrupted in Vps34-defi

286 gocytosis, receptor-mediated endocytosis, or fluid-phase uptake), or costimulatory molecule expressio

287 lizes to actin-rich structures implicated in fluid-phase uptake, including tubular membranes containi

288 As bound to albumin and respond by enhancing fluid-phase uptake.

289 Measurements of these parameters in fluid phase using soluble molecules (i.e., three-dimensi

290 te transport in a micromodel filled with two fluid phases using direct, real-time imaging.

291 r of magnitude larger than the corresponding fluid phase values.

292 nts measured for gel-phase vesicles than for fluid-phase vesicles, even in the absence of anionic lip

293 he endothelial surface, and incorporation of fluid-phase VWF into VWF fibers.

294 nger strands and inhibited the attachment of fluid-phase VWF onto vessel wall strands.

295 binding of either factor H variant from the fluid phase was observed.

296 of problems concerns the composition of the fluid phase, which includes both dispersed/dissolved par

297 her decreasing the lateral interactions, the fluid phase will dominate throughout the 0.18-0.26 inter

298 ystems proceeds via a high-density precursor fluid phase with prenucleation motifs in the form of clu

299 d discontinuous transitions between distinct fluid phases with widely differing viscosities.

300 The regions represent high-viscosity fluid phases, with a size determined by the distance bet