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1 tremely small head and a long tail without a fluke.
2 l nematode, and Schistosoma mansoni, a blood fluke.
3 the 363 megabase nuclear genome of the blood fluke.
4  is actively used for schistosomes and other flukes.
5  swimmers have reduced limbs and fleshy tail flukes.
6 d tropical disease caused by parasitic blood flukes.
7 ase caused by infection with parasitic blood flukes.
8 omiasis, a disease caused by parasitic blood flukes.
9 ther's clitorises using snouts, flippers, or flukes(3).
10                 Stable positioning using the fluke and flipper was confirmed by video data for both t
11 h therapeutic effectiveness against specific fluke and tapeworm infections and will help the developm
12 63 family tegument tetraspanins in parasitic flukes and support efforts to target these proteins for
13 l fibrosis, which is a precursor of CCA, the flukes and their microbiota may together drive this dist
14  Clonal relatedness between the actor (brain fluke) and recipients (abdomen flukes) enables kin selec
15 ng hair and hind limbs, and having flippers, flukes, and a streamlined body.
16 rms, related more distantly to tapeworms and flukes, and have been developed as models to study the m
17 s - gastrointestinal nematodes (GINs), liver flukes, and lungworms - and the search for subclinical i
18 errations that drive the pathogenesis of non-fluke-associated intrahepatic CCA, fibroblast growth fac
19                                          The fluke-associated microorganisms included potential patho
20  superbly adapted for marine life, with tail flukes being a key innovation shared by all extant speci
21 icant advances in our understanding of liver fluke biology have been made through in-depth interrogat
22 rotubule-related genes between TCBZ-S and -R flukes, both without drug treatment and in response to t
23 ential vaccine candidates could reduce total fluke burden and egg output by up to 43% and 99%, respec
24 ttle, but it is unclear at what levels liver fluke burdens cause production losses.
25 y is maintained with distantly related blood flukes but we find extreme losses of genes and pathways
26  immature and adult Fasciola hepatica (liver fluke) but widespread resistance to the drug greatly com
27 es TRPM(PZQ) homologs in other PZQ-sensitive flukes, but not Fasciola hepatica.
28 quantel decreases the risk of CCA from liver flukes, but reinfection is common and future vaccination
29 y tract of hamsters and developed into adult flukes, but the infection resulted in reduced pathology
30 ely related to CsRn1 from the Oriental liver fluke Clonorchis sinensis and to kabuki from Bombyx mori
31 It has been established that the Asian liver flukes, Clonorchis sinensis and Opisthorchis viverrini s
32 d resistance to the drug greatly compromises fluke control in livestock and humans.
33                       EVs were identified in fluke culture medium and bile specimens from infected ho
34 mated that cattle classified as having liver fluke damage had on average 10 days greater slaughter ag
35 ting behavior caused by larvae of the lancet fluke Dicrocoelium dendriticum in ants.
36 nzyme-linked immunosorbent assay (ELISA) and fluke egg counts.
37  actor (brain fluke) and recipients (abdomen flukes) enables kin selection of the parasite's host-man
38 terizing the metabolome of the hepatobiliary fluke Fasciola hepatica , using ultra performance liquid
39     Only marginal activity against the liver fluke Fasciola hepatica and Trichostrongylus colubriform
40                                    The liver fluke Fasciola hepatica is a major pathogen of livestock
41 n emerging human disease caused by the liver fluke Fasciola hepatica.
42 ation of gene expression in the common liver fluke, Fasciola hepatica.
43                For instance, the giant liver fluke, Fascioloides magna, is a major parasite of severa
44  dedicated to the dissemination of data from flukes, flatworm parasites of the class Trematoda, phylu
45  roundworm, whipworm, pinworm, Chinese liver fluke, fleas and lice.
46 TCBZ-sensitive (TCBZ-S) and 210 TCBZ-R adult flukes from 146 bovine livers in Cusco, Peru.
47 knockout of the Ov-GRN-1 gene from the liver fluke genome.
48 e of the secreted growth factor termed liver fluke granulin (Ov-GRN-1) in pre-malignant lesions by un
49 gene knockout (KO) demonstrated that a liver fluke growth mediator contributes to disease progression
50 2 (Ov-tsp-2) gene and found that Ov-tsp-2-KO flukes had abnormal tegument biogenesis.
51 st-manipulating behavioral trait, the lancet fluke has a high abundance of clonemates.
52  Parasitic flatworms from the group Digenea (flukes) have free-living larvae, cercariae, which are re
53  for associations among infection with liver fluke, Helicobacter and hepatobiliary fibrosis.
54                                              Fluke images were uploaded to Happywhale, a global digit
55 and gene-editing tools to the study of liver fluke immunobiology and pathogenesis has accelerated the
56  Disease, caused by the infection with blood flukes in the genus Schistosoma.
57 ts for diagnosing fluke infection as well as fluke-induced CCA, and out-performs parasite crude extra
58  O. viverrini were examined in this model of fluke-induced CCA.
59 f infection and a tissue microarray of liver fluke-induced human cholangiocarcinomas (CCAs).
60 ntibody serodiagnostic tests for human liver fluke infection and associated cancer.
61 ome to identity antibody biomarkers of liver fluke infection and CCA with sera from study participant
62                        Relationships between fluke infection and immune responses were analyzed by us
63 gle recombinant antigen tests for diagnosing fluke infection as well as fluke-induced CCA, and out-pe
64 ent progress in tracking the spread of rumen fluke infection in Europe, and propose some research que
65                                Chronic liver fluke infection induces a distinct mutational landscape,
66                                        Liver fluke infection is a major risk for cholangiocarcinoma (
67  are poorly understood, though chronic liver fluke infection is a risk factor for disease.
68                    In endemic regions, liver fluke infection is associated with CCA, owing to the onc
69 ertainties by estimating the impact of liver fluke infection on UK beef cattle productivity and inves
70   Microbial community analyses revealed that fluke infection perturbed the gastrointestinal tract mic
71 eening in northeastern Thailand, where liver fluke infection rates are particularly severe; however,
72 iated risks for food security posed by rumen fluke infection, it is imperative that we develop a bett
73 ment of treatment guidelines for human liver fluke infection.
74 rrently registered drugs against human liver fluke infection.
75 t cagA-positive H. pylori than those free of fluke infection.
76                                        Liver fluke infections occur in people worldwide.
77  targeting hookworm and liver and intestinal fluke infections through current MDA approaches.
78 nd the prospects for future control of liver fluke infections using an integrated parasite management
79 is - in addition to the intestinal and liver fluke infections, as some of the most important gastroin
80              Fasciola hepatica, common liver fluke, infects cattle and sheep causing disease and prod
81  alone (such as the presence of a crescentic fluke), information on their soft parts has hitherto bee
82 the modulation of the immune response; a key fluke intercession that allows for establishment and dev
83 lineus, a bile duct-dwelling trematode liver fluke is highly endemic.
84 oma mansoni, a multicelluar eukaryotic blood fluke, is a major cause of morbidity worldwide in humans
85                     Following ingestion, the flukes locate to the biliary tre where chronic infection
86 ments are encouraging a renaissance in liver fluke neurobiology that can now support flukicide discov
87  293 and 260 positive and negative for liver fluke O. viverrini eggs, of residents in Northeastern Th
88 ly effective, lacking activity against liver flukes of the Fasciola genus.
89                     The coevolution of blood flukes of the genus Schistosoma and their human hosts is
90 pical disease caused by infection with blood flukes of the genus Schistosoma.
91 is a major parasitic disease caused by blood flukes of the genus Schistosoma.
92 --is a parasitic disease caused by trematode flukes of the genus Schistosoma.
93 between parasitism by the carcinogenic liver fluke Opisthorchis viverrini and this coregulator using
94                                    The liver fluke Opisthorchis viverrini infects 10 million people i
95       Transmission of the carcinogenic liver fluke Opisthorchis viverrini is ongoing across Southeast
96          Infection with the food-borne liver fluke Opisthorchis viverrini is the principal risk facto
97                                    The liver fluke Opisthorchis viverrini resides within the biliary
98                                    The liver fluke Opisthorchis viverrini, highly prevalent in Southe
99 e of granulin from the human parasitic liver fluke Opisthorchis viverrini, known as Ov-GRN-1, induces
100 langiocarcinoma and infection with the liver fluke Opisthorchis viverrini.
101 and there is a direct correlation with liver fluke (Opisthorchis viverrini) infection.
102 sease caused by the carcinogenic Asian liver fluke, Opisthorchis viverrini.
103 ses worldwide, is caused by flatworms (blood flukes or schistosomes) that live in the bloodstream of
104 cretory-secretory products (ES) of the liver fluke parasite Fasciola hepatica as cargo of extracellul
105 ocean-basin-scale dataset that includes tail fluke photographic identification (photo-ID) and encount
106 pered efforts to understand the evolution of fluke-powered, but forelimb-controlled, locomotion.
107              Adult Schistosoma mansoni blood flukes reside in the mesenteric veins of their vertebrat
108 observation of tsp-2 and tsp-3 dsRNA-treated flukes resulted in phenotypes with increased tegument th
109      We provide empirical data on the lancet fluke's clonal diversity within its ant host.
110                                    The blood fluke Schistosoma haematobium causes urogenital schistos
111 acterised from the parasitic trematode blood fluke Schistosoma haematobium.
112 resent on the surface of the trematode blood fluke Schistosoma has been implicated in the regulation
113 (AChE) on the surface of the parasitic blood fluke Schistosoma is the likely target for schistosomici
114       The taurocyamine kinase from the blood fluke Schistosoma mansoni (SmTK) belongs to the phosphag
115 glabrata, a natural host for the human blood fluke Schistosoma mansoni Granulins are growth factors t
116                                    The blood fluke Schistosoma mansoni is the causative agent of the
117               As an example, the human blood fluke Schistosoma mansoni produces highly fucosylated gl
118               In the case of the human blood fluke Schistosoma mansoni, responsible for intestinal bi
119 trumental in transmission of the human blood fluke Schistosoma mansoni.
120 abrata, intermediate host of the human blood fluke Schistosoma mansoni.
121 , snail intermediate host of the human blood fluke Schistosoma mansoni.
122                                    The blood flukes Schistosoma japonicum and Schistosoma mansoni are
123                                    The blood flukes Schistosoma mansoni and Schistosoma japonicum inf
124 iquine resistance evolved in the human blood fluke (Schistosoma mansoni) in Brazil in the 1970s.
125                          The human parasitic fluke, Schistosoma haematobium hybridizes with the lives
126 inid snails host the most common human blood fluke, Schistosoma haematobium, responsible for approxim
127                 The metazoan parasitic blood flukes, Schistosoma spp., infect over 200 million people
128 itional evidence that O. viverrini and other flukes secrete proteins that directly modulate host cell
129                                              Fluke secreted and tegumental proteins contribute to the
130 with substantial morbidity and several liver fluke species are recognised as biological carcinogens.
131 eus are the three most important human liver fluke species in the Opisthorchiidae family, infecting a
132 ge by weight and different measures of liver fluke status, while accounting for sex, breed, season, y
133  of aquatic locomotion is that cetaceans use fluke strokes to power their swimming while relying on l
134 riptomes of O. viverrini, elucidate how this fluke survives in the hostile environment within the bil
135 lifestyle evolved in each group, such as the fluked tails, dorsal fins and wing-shaped limbs of ichth
136 ain proteins expressed on the surface of the fluke tegument and extracellular vesicles (EVs) derived
137 alization of Ov-TSP-2 and TSP-3 to the adult fluke tegument.
138     Here, we examined the functions of liver fluke tetraspanins, four-transmembrane domain proteins e
139 complement of Schistosoma mansoni, the blood fluke that causes schistosomiasis, ranked among the most
140           Schistosoma haematobium is a blood fluke that causes urogenital schistosomiasis and induces
141           Schistosoma mansoni is a parasitic fluke that infects millions of people in the developing
142 ities and the parasitic tape worms and blood flukes that exert a massive impact on human health.
143 um, S. mansoni and S. haematobium, are blood flukes that have complex life cycles involving a snail i
144 ate hosts (vectors) for parasitic flatworms (flukes) that cause neglected tropical diseases, such as
145 he intricacies of the biology of these blood flukes, their host relationships, and the diseases that
146  technologies to discover that elusive liver fluke vaccine.
147 l model to assess the effectiveness of liver fluke vaccines under simulated field conditions.
148                  The tegument of Ov-tsp-2-KO flukes was increasingly vacuolated and fewer EVs were se
149 S-2 homologues in liver/intestinal foodborne flukes, we propose a new group within the cystatin super
150 0 km/h, a feat accomplished by thrusting the flukes while adjusting attack angle with their flippers
151           The model describes development of fluke within a group of animals and includes heterogenei

 
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