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1 d microtubule cytoskeletons were examined by fluorescence staining.
2 etermined by dihydroethidium (hydroethidine) fluorescence staining.
3 uorescence in situ hybridization and G-actin fluorescence staining.
4 lopment that avoids clinically impermissible fluorescence staining.
5  4-N(3)-PET-8-biotin-11-cGMP RPTs confirming fluorescence staining.
6                                              Fluorescence staining, along with gene expression measur
7 of Abeta as determined by thioflavin T (ThT) fluorescence staining and atomic force microscopy (AFM)
8                                 Furthermore, fluorescence staining and confocal microscopy revealed a
9 tures for culturing organoids in addition to fluorescence staining and imaging without the need for s
10 tance was analyzed using immune blot, immune fluorescence staining and immunohistochemistry.
11 inguishable from native cell surface WI-1 by fluorescence staining and restored adhesivity to the kno
12 croscopy-based nanoindentation measurements, fluorescence staining, and histologic examination of the
13 histochemistry-based 3D imaging, whole-mount fluorescence staining, and real-time quantitative PCR.
14 verified by both electron spin resonance and fluorescence staining assay.
15 y H(2)O(2) and O( minus sign, dot below )(2) fluorescence staining assays in JB6 Cl 41 cells.
16  was measured further by H(2)O(2) and O(-.2) fluorescence staining assays.
17 ite the extensive efforts along the lines of fluorescence staining, autoradiography, and mass spectro
18     Western blotting and immunohistochemical fluorescence staining demonstrates that EphB1 receptor p
19 ividual EVs during 5 cycles of multi-channel fluorescence staining for 15 EV biomarkers.
20 reased: the proportion of laboratories using fluorescence staining for acid-fast microscopy has incre
21 ned by transmission electron microscopy, and fluorescence staining for adhesion site protein expressi
22 n by quantitative immunogold staining and by fluorescence staining for lysosomotropic agents.
23 k and corneal endothelium--revealed positive fluorescence staining for peroxidized carbonyl products.
24                                              Fluorescence staining further revealed that OY phytoplas
25                                          The fluorescence staining identified the same particles as t
26 cell segmentation using light microscopy and fluorescence staining images.
27 ver, tracking polarization requires invasive fluorescence staining, impermissible in the in vitro fer
28      Subcellular colocalization studies with fluorescence staining indicate SV can colocalize with AR
29                                      PIKfyve fluorescence staining largely coincided with trans-Golgi
30                                            A fluorescence staining method was used to show that ChrS
31  a multiplexing approach using a three-color fluorescence staining method, which allowed for up to se
32 ator acridine orange (AO) identified intense fluorescence staining of acidic cytoplasmic vesicles wit
33 d actin depolymerization assays in vitro and fluorescence staining of actin filaments in cells, we sh
34                                       Direct fluorescence staining of Api m 1 and Lol p 1 tetramers h
35 dual NSCs and glial cells were compared with fluorescence staining of cell nuclei and cytoplasm to sh
36 ific cellular uptake of LPS was suggested by fluorescence staining of cells along the abrasion site.
37 maging for up to 13 generations with in situ fluorescence staining of primary human hematopoietic pro
38 ed for vasoreactivity study, dihydroethidium fluorescence staining of superoxide, immunohistochemical
39                                              Fluorescence staining of the cells with rhodamine-conjug
40           These data and the results of dual-fluorescence staining of the endogenous RBCs and syncyti
41 translocation of NF-kappaB was determined by fluorescence staining of the NF-kappaB Rel A (p65) subun
42 midino-2-phenylindole (DAPI) staining and by fluorescence staining of the nuclei of membrane-compromi
43 ntified at various periods after exposure by fluorescence staining of the nuclei of membrane-compromi
44                                   Dual-label fluorescence staining of these sediments showed a fivefo
45           The lectin MAA produced an intense fluorescence-staining pattern only at the J1/J2 interfac
46 alized in bright field by phase imaging, and fluorescence-staining patterns indicate they contain mem
47 es by the use of time-lapse and quantitative fluorescence staining procedures.
48 hilic cationic protein (ECP) was detected by fluorescence staining techniques and confocal microscopy
49 morphogenesis of the MGC by means of various fluorescence staining techniques and laser-scanning conf
50 ated by confocal microscopy following immune fluorescence staining techniques.
51                          As suggested by the fluorescence staining, the deformation lines appeared to
52  and in cholesterol accumulation (by filipin fluorescence staining) throughout the cerebral neocortex
53                                              Fluorescence staining using different lectins with high
54          The use of paper point sampling and fluorescence staining was shown to be a rapid method, ab
55  transplanted into their brains, post-mortem fluorescence staining with a mannose-specific lectin sho
56  adapter was developed to seamlessly combine fluorescence staining with Nile Red and Raman spectrosco