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1 , paxillin, and talin and dissolution of the focal adhesion complex.
2 f integrin beta4 (ITGB4), a component of the focal adhesion complex.
3 both of which are critical components of the focal adhesion complex.
4 ulin) were associated with modulation of the focal adhesion complex.
5 g its enzyme activity acts on targets in the focal adhesion complex.
6 e GIT2-alphaPIX-PAK complex, an IS-localized focal adhesion complex.
7 RNA to a specific location-in this case, the focal adhesion complex.
8 Cas is a member of the focal adhesion complex.
9 adhesion kinase (FAK) and disassembly of the focal adhesion complex.
10 actor (C3G) and inducibly associate with the focal adhesion complex.
11 oskeletal architecture, and the formation of focal adhesion complexes.
12 hosphorylation of proteins in the associated focal adhesion complexes.
13 associate with cytoskeletal scaffolds within focal adhesion complexes.
14 roundings through modulating the activity of focal adhesion complexes.
15 d spleen tyrosine kinase and paxillin within focal adhesion complexes.
16 hed regulators of the actin cytoskeleton and focal adhesion complexes.
17 ional architecture and nanoscale dynamics of focal adhesion complexes.
18 slocation to the nucleus and localization in focal adhesion complexes.
19 owered by uncharacterized motor proteins and focal adhesion complexes.
20 ne by controlling F-actin polymerization and focal adhesion complexes.
21 d in rapid activation of vinculin-containing focal adhesion complexes.
22 x and the clustering of these receptors into focal adhesion complexes.
23 5 leads to Rho kinase-dependent formation of focal adhesion complexes.
24 lar S-adenosylhomocysteine and disruption of focal adhesion complexes.
25 ctin cytoskeleton, leads to the formation of focal adhesion complexes.
26 lusters a multi-subunit signaling complex at focal adhesion complexes.
27 PAK also fosters loss of focal-adhesion complexes.
28 kinase (FAK) are two major components of the focal adhesion complex, a multiprotein structure linking
29 y be participating in the disassembly of the focal adhesion complex and actively interrupting surviva
30 is associated with the beta1 integrin in the focal adhesion complex and as such is a candidate kinase
31 the focal adhesion kinase and its associated focal adhesion complex and the consequent acquisition of
36 nd the spicules, indicating the formation of focal adhesion complexes and suggesting that the cells s
37 sses Hic-5/ARA55, which is localized both at focal adhesion complexes and within the soluble cytoplas
38 f focal adhesion molecules, formation of the focal adhesion complex, and activation of Rho-GTPases.
39 se (pp125FAK), an important component of the focal adhesion complex, and identify pp125FAK as a novel
40 , RA promotes formation of stress fibers and focal adhesion complexes, and activation of ERK1/2, JNK1
41 ochondrial trafficking, turnover of membrane focal adhesion complexes, and enhanced tumor cell migrat
42 ng the restoration of cell polarity, typical focal adhesion complexes, and longitudinal F-actin stres
45 1/2 phosphorylation, suggesting that intact focal adhesion complexes are required for GPCR-induced m
46 pled with the formation of stress fibers and focal adhesion complexes, are proposed to have a signifi
47 of several proteins of the integrin-mediated focal adhesion complex as they experience intra- and ext
48 HSP20 led to loss of actin stress fibers and focal adhesion complexes as demonstrated by immunocytoch
49 of the time- and site-specific regulation of focal adhesion complex assembly and disassembly required
54 he signaling pathway by which VEGF regulates focal adhesion complex assembly by examining the signali
55 studies define a mechanism for HIV-1 Tat in focal adhesion complex assembly in HBMECs via activation
56 mechanism for the regulatory role of VEGF in focal adhesion complex assembly in HBMECs via activation
57 man skin equivalents, in part by attenuating focal adhesion complex assembly, and prevented and rever
58 monstrated that HGF caused a condensation of focal adhesion complexes at the leading edges of cell pr
60 signaling and cytoskeletal components of the focal adhesion complex; binding of paxillin, but not pp1
62 es in expression of proteins involved in the focal adhesion complex but did exhibit intrinsic focal a
63 6 contributes to the integration of NCAM1 in focal adhesion complexes but, unlike cells lacking PrP,
64 We examine the process of expansion of a focal adhesion complex by which a biological membrane co
65 sactivation of receptor tyrosine kinases and focal adhesion complexes by G-protein-mediated signals,
67 overexpression up-regulates the formation of focal adhesion complexes compared with control cells.
68 h the expression and/or activation status of focal adhesion complex components such as Src, FAK, and
71 elial cell (HBMEC) integrity and assembly of focal adhesions, complexes comprised of scaffolding and
74 fects on the force transduction layer of the focal adhesion complex, drives glioblastoma motility and
75 VopA also has potent modulating activity on focal adhesion complex (FAC) proteins, where VopA marked
76 glycoprotein that regulates the activity of focal adhesion complexes, facilitating the SMC migration
78 universally relies on spatial regulation of focal adhesion complexes (FAs) connecting the substrate
79 e addressed the relationship of cavin-1 with focal adhesion complexes following nutritional stimulati
80 or the RhoA-regulated actin stress fiber and focal adhesion complex formation and that Rac1 is involv
81 significant change in actin stress fiber or focal adhesion complex formation in response to serum or
86 spread less well, and formed poorly defined focal adhesion complexes in comparison to the X2C2 and X
87 ces the formation of actin stress fibers and focal adhesion complexes in Galpha13-expressing NIH3T3 c
88 data presented indicate that disassembly of focal adhesion complexes in HSCs is pivotal for hepatic
91 n events within proteins that constitute the focal adhesion complex, including focal adhesion kinase
93 eport the NMR structure of an extremely weak focal adhesion complex (K(D) approximately 3 x 10(-3) M)
95 ptors with extracellular matrix (ECM) at the focal adhesion complex may regulate endothelial cell sha
96 kinase and Cas phosphorylation - markers of focal adhesion complex-mediated Crk-dependent signaling
97 filaments (stress fibers) and of associated focal adhesion complexes of adherent monolayer cells in
98 lical vein endothelial cells (HUVECs) formed focal adhesion complexes on RGD- and RGD and bFGF-immobi
99 ants effectively adhered, spread, and formed focal adhesion complexes on type I collagen matrices.
100 ASP during cell spreading may be involved in focal adhesion complex organization and actin dynamics.
101 , immunohistochemical analysis revealed that focal adhesion complexes persist at the distal tips of e
104 tions in fibroblasts and identified specific focal adhesion complex proteins whose tyrosine phosphory
105 cell survival signals via phosphorylation of focal adhesion complex proteins, such as focal adhesion
107 of the membrane proximal signaling layer in focal adhesion complexes, regulating important cellular
108 is of PARP14 shows that it is a component of focal adhesion complexes required for proper cell motili
109 epithelial cells requires components of the focal adhesion complex signaling pathway, focal adhesion
110 lls, Semaphorin 4D promotes the formation of focal adhesion complexes, stress fibers, and the phospho
111 phorylation and association of components of focal adhesion complexes such as the related adhesion fo
112 integrin-associated signal molecules in the focal adhesion complex that are responsible for propagat
113 influence intracellular biochemistry within focal adhesion complexes that form at the site of integr
114 ution of alpha-actinin and vinculin from the focal adhesion complex to the Triton X-100-soluble fract
115 where it associates with the integrin-based focal adhesion complexes to ensure proper ensheathment o
116 of talin-1, which links integrin-containing focal adhesion complexes to the actin cytoskeleton, faci
117 o-induced actin-containing stress fibres and focal-adhesion complexes, to which the ends of the stres
118 s in order to further understand its role in focal adhesion complex turnover and actin organization.
119 ased cellular calcium increases polycystin-1 focal adhesion complexes versus polycystin-1 adherens ju
124 chemical analysis of v-CrkPC12 cells reveals focal adhesion complexes which are formed at the periphe
125 e formation of contractile stress fibers and focal adhesion complexes while a close relative, Rac, in
126 indlin is a largely overlooked member of the focal adhesion complex whose roles in cellular mechanotr
127 on blocked TCDD/EGF stimulation of clustered focal adhesion complexes without affecting either sustai