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1 sion levels in H-HCA, IHCA, b-HCA, UHCA, and focal nodular hyperplasia.
2 recursor, hepatic adenoma, but not in benign focal nodular hyperplasia.
3 atients (42.2%) had adenomas, 29 (19.7%) had focal nodular hyperplasia, 25 (17.0%) had hemangiomas, 1
4    Significant differences were seen between focal nodular hyperplasia and all other lesion types (P
5                                       Benign focal nodular hyperplasia and hepatocellular adenoma cou
6            This finding completely separated focal nodular hyperplasia and malignancies.
7 necrosis, in alcoholic liver disease, and in focal nodular hyperplasia are compared with liver progen
8 s encompassed cysts (70), hemangiomata (37), focal nodular hyperplasia (FNH) (23), adenomata (47), an
9 on-weighted images) to differentiate between focal nodular hyperplasia (FNH) and hepatocellular adeno
10                                              Focal nodular hyperplasia (FNH) and hepatocellular adeno
11                   Nonoperative management of focal nodular hyperplasia (FNH) is an accepted paradigm
12                                              Focal nodular hyperplasia (FNH), hepatocellular adenoma
13 ed with nodular regenerative hyperplasia and focal nodular hyperplasia (FNH), which finally evolved t
14  radiologically indeterminate for adenoma or focal nodular hyperplasia (FNH): (1) continue to observe
15  (CCC), hepatocellular carcinomas (HCC), and focal nodular hyperplasias (FNH).
16                                              Focal nodular hyperplasia had stimulated acoustic emissi
17 nal uptake of SH U 508A is characteristic of focal nodular hyperplasia, is seen in some hemangiomas,
18                           Hepatic abscesses, focal nodular hyperplasia-like in patients treated with
19 cclusive thrombosis, and lack of visibility; focal nodular hyperplasia-like nodules (six [14%] of 42
20 inoma (n = 6), cavernous hemangioma (n = 4), focal nodular hyperplasia (n = 2), hamartoma (n = 1), an
21 liver disease (n = 9), hydatid cyst (n = 6), focal nodular hyperplasia (n = 3), and adenoma (n = 9).
22 ellular carcinoma, n = 4; hemangioma, n = 9; focal nodular hyperplasia, n = 7) received 2.5 g SH U 50
23                Recent evidence suggests that focal nodular hyperplasia of the liver (FNH) may represe