ライフサイエンスコーパス: folic acid

1 s (PUFAs) and folate (or the synthetic form, folic acid).

2 rtified foods in an oxidized synthetic form (folic acid).

3 tended negative consequences of supplemental folic acid.

4 containing 0.4 to 0.8 mg (400-800 microg) of folic acid.

5 p=0.931) for those receiving lamotrigine and folic acid.

6 duce renal dysfunction and decrease of serum folic acid.

7 ions who were consuming bread fortified with folic acid.

8 ficiency in the presence of high exposure to folic acid.

9 , independent of embryo genotype and dietary folic acid.

10 rt for reconsidering the basis for the UL of folic acid.

11 oth the inability of Juno to bind vitamin B9/folic acid [1], and why mutations within the flexible re

12 All participants received folic acid, 1 mg/d, 6 days per week.

13 For this purpose, the concentrations of folic acid, 10-formylfolic acid, 5-methyltetrahydrofolat

14 s and status compared with nonconsumption of folic acid (18% of the population).

15 Treatment was combined with folic acid 5 mg/week.

16 adjunctive B-vitamin supplement (containing folic acid [5 mg], B(12) [0.4 mg], and B(6) [50 mg]) or

17 The main contributor to total folate was folic acid, 5-methyl-tetrahydrofolate was the only minor

18 ) a novel drink powder (4 g betaine, 800 mug folic acid, 5.2 mug vitamin B12, and 2.8 mg vitamin B2),

19 )) under B-vitamin supplementation (2.5 mg/d folic acid, 50 mg/d vitamin B6, and 1 mg/d vitamin B12),

20 Folic acid 6 days per week (odds ratio, 0.16; 95% CI, 0.

21 of bacteriophage phi29 motor pRNA to escort folic acid, a fluorescent image marker and BRCAA1 siRNA

22 Folic acid administration 6 or 7 times per week protecte

23 ments containing iron-folic acid versus iron-folic acid alone in 112 953 pregnant women.

24 of omega-3 fatty acids, soy, ginkgo biloba, folic acid alone or with other B vitamins, beta-carotene

25 f the proposed procedure was investigated by folic acid analysis in flour samples and the relative re

26 Men were randomized (1:1) to 5 mg folic acid and 30 mg elemental zinc daily or matching pl

27 an outside clinic) to receive either 5 mg of folic acid and 30 mg of elemental zinc (n = 1185) or pla

28 association between food fortification with folic acid and a reduction in the birth prevalence of sp

29 ervention group had higher recorded iron and folic acid and calcium supplement consumption and mean d

30 317 participants enrolled in the Bangladesh Folic Acid and Creatine Trial (FACT).

31 the effect of maternal intakes of folate and folic acid and fetal genetic variants in folate metaboli

32 ndividually, as well as an imbalance of high folic acid and low vitamin B-12 status, may be associate

33 Folic acid and magnesium may also merit supplementation

34 of periconceptional multivitamins containing folic acid and of folic acid food fortification on conge

35 75%) were enrolled into a programme for free folic acid and penicillin, of whom 36 (88%) completed th

36 (400 MPa for 5 min) induced the transfer of folic acid and phosvitin from the egg yolk granule to th

37 Folic acid and vitamin B-12 from fortified foods and sup

38 d 98 controls) were eligible for analysis of folic acid and vitamin B12 levels.

39 A diet poor in folic acid and vitamin B6 is also associated with a high

40 marketed for male fertility commonly contain folic acid and zinc based on limited prior evidence for

41 % for percentage of DNA fragmentation in the folic acid and zinc group and 27.2% in the placebo group

42 t between treatment groups (404 [34%] in the folic acid and zinc group and 416 [35%] in the placebo g

43 rease in DNA fragmentation was observed with folic acid and zinc supplementation (mean of 29.7% for p

44 These findings do not support the use of folic acid and zinc supplementation by male partners in

45 es seeking infertility treatment, the use of folic acid and zinc supplementation by male partners, co

46 trointestinal symptoms were more common with folic acid and zinc supplementation compared with placeb

47 lacebo-controlled trial to determine whether folic acid and zinc supplementation in men improves seme

48 The Folic Acid and Zinc Supplementation Trial (FAZST) was a

49 up of female partners of men enrolled in the Folic Acid and Zinc Supplementation Trial studying coupl

50 DESIGN, SETTING, AND PARTICIPANTS: The Folic Acid and Zinc Supplementation Trial was a multicen

51 men were randomized during 2013-2017 (1,185 folic acid and zinc, 1,185 placebo); they had a mean age

52 iron and folic acid (IFA; 60 mg Fe + 400 mug folic acid) and 16 clusters received LNS-PLs (20 g/d, 11

53 mega-3 fatty acids, 30-mug selenium, 400-mug folic acid, and 20-mug vitamin D3 plus 100-mg calcium) v

54 nds; dietary factors, including vitamin B12, folic acid, and fish oil; obesity; and stress.

55 tituted purine CDK inhibitor conjugates with folic acid as a drug-delivery system targeting folate re

56 ollow-up, pregnant women taking supplemental folic acid at or above the recommended dose, combined wi

57 ology and may include erythropoietic agents, folic acid, B12, and iron preparations.

58 rnicious anemia was treated with high-dosage folic acid before vitamin B12 was widely available in th

59 The potential of the folic acid biosynthesis pathway as a target for the deve

60 pairs, several of which putatively mapped to folic acid biosynthesis, a key pathway in colorectal can

61 that captured recent and habitual intakes of folic acid, both as supplements and as fortified foods,

62 We established that folic acid breaks down into fragments while the pteridin

63 er, carbohydrate, protein, crude fiber, ash, folic acid, Ca, Mg, Fe, Mn, and Zn increased significant

64 rd does not provide compelling evidence that folic acid can potentially cause neurologic complication

65 ternal periconceptional use of the B vitamin folic acid can prevent many NTDs.

66 hybrids with near-infrared gold nanorods and folic acid can serve as molecular high-contrast photoaco

67 of Washington, where her work on the role of folic acid coenzymes in one-carbon metabolism revealed t

68 ) for patients receiving lamotrigine without folic acid compared with 0.12 ([-2.58 to 2.82]; p=0.931)

69 A new micelle-forming material, folic acid-conjugated carboxymethyl lauryl chitosan (FA-

70 We suggest that high folic acid consumption reduces MTHFR protein and activit

71 for other covariates, including supplemental folic acid consumption.

72 olate status by demographic group and use of folic acid-containing supplements.

73 g random-effects models, stratifying by iron-folic acid dose, and exploring individual study effect.

74 ssigned to placebo and 146 (43%) assigned to folic acid during the second surveillance interval (RR:

75 We conducted secondary analyses to evaluate folic acid effects with additional follow-up after treat

76 Total folate content, expressed as folic acid equivalent, was 17-490 mug/100 g in all sampl

77 irth defect research, the mechanism by which folic acid exerts this benefit remains unknown.

78 aired cognitive function may exist when high folic acid exposures are combined with low vitamin B-12

79 with biodegradable copolymer pluronic F-127-folic acid (F-127-FA), RET-BDP molecules can form unifor

80 nts (MMN) or iron + folic acid (IFA), versus folic acid (FA) alone, on risk of spontaneous preterm bi

81 A comparative study is reported where folic acid (FA) and boronic acid (BA) based cytosensors

82 udy compares enzymatic treatments to release folic acid (FA) and endogenous 5-methyltetrahydrofolate

83 This study investigated the effects of folic acid (FA) and/or creatine supplementation on the p

84 New folic acid (FA) coupled three layered micelles (3LM) wer

85 Iron (Fe) and folic acid (FA) fortified parboiled rice was produced by

86 ity, it has been unclear how vitamin B12 and folic acid (FA) function at the molecular level to direc

87 Folic acid (FA) is a key vitamin in the prevention of ma

88 Folic acid (FA) is a synthetic vitamin commonly used for

89 e 1-phosphate, in kidney fibrosis induced by folic acid (FA) or unilateral ischemia-reperfusion injur

90 Maternal folic acid (FA) protects against developmental toxicity

91 (PM) exposure and preterm delivery (PTD) by folic acid (FA) supplementation has not been studied.

92 nt individuals, this process is augmented by folic acid (FA) supplementation, thereby lowering blood

93 ico, wheat and corn flour fortification with folic acid (FA) was implemented in 2001 and mandated in

94 -methyltetrahydrofolate (L-5-MTHF) with free folic acid (FA) when exposed to thermal cooking conditio

95 ed nanogels were surface-functionalized with folic acid (FA) with the help of a PEG spacer without af

96 ful achieved the ultrasensitive detection of folic acid (FA) with the limit of detection (LOD) as 0.8

97 in (TF), 4-carboxyphenylboronic acid (CPBA), folic acid (FA), and hyaluronic acid (HA) molecules, by

98 aling unit) bonded in different positions to folic acid (FA), the folate receptor (FR) targeting unit

99 nt, are pregnant, or are lactating consume a folic acid (FA)-containing supplement.We sought to deter

100 ctive tumor-targeting by a novel Cy3-labeled folic acid (FA)-derivatized DNA dendrimer nanocarrier (3

101 tributions of ferroptosis and necroptosis to folic acid (FA)-induced AKI in mice.

102 sed to receive one of four supplements: iron-folic acid (FeFol; standard of care), multiple micronutr

103 l multivitamins containing folic acid and of folic acid food fortification on congenital heart defect

104 analysis was used to quantify the effect of folic acid food fortification on nonchromosomal CHD subt

105 Folic acid food fortification was associated with lower

106 Associations were primarily present before folic acid food fortification was implemented (RR: 2.03;

107 cioeconomic deprivation, and the presence of folic acid food fortification.

108 face has been covalently functionalized with folic acid for molecular targeting of cancer cells.

109 rived patient safety indicators (PSI) in the Folic Acid for Vascular Outcome Reduction in Transplant

110 traindicated because of reduced GFR from the Folic Acid for Vascular Outcome Reduction in Transplant

111 Using specimens collected during the Folic Acid for Vascular Outcome Reduction in Transplanta

112 ubjects were selected randomly from the 4110 Folic Acid for Vascular Outcome Reduction in Transplanta

113 We conducted a post hoc analysis of the Folic Acid for Vascular Outcome Reduction in Transplanta

114 ceived LDMTX (5-15 mg/week) or placebo (plus folic acid) for 24 weeks and were followed for an additi

115 g ultrasonic treatment and absorption of the folic acid fortificant.

116 Pre-folic acid fortification (1988-1994), risks of deficienc

117 Following mandatory folic acid fortification of dietary grains, a dramatic r

118 In the context of mandatory and voluntary folic acid fortification, the exposure of children to fo

119 red by policy makers who are responsible for folic acid fortification.

120 ve birth rates among a population exposed to folic acid fortification.

121 amins and bone health from a population with folic acid fortification.

122 f nutrient status in a population exposed to folic acid fortification.

123 in Ireland who were exposed to the voluntary folic acid-fortification regimen in place in Ireland.

124 obic deep eutectic solvent for extraction of folic acid from flour samples followed by HPLC determina

125 Mean daily intake of folic acid from fortified foods and supplements was 109

126 tal folate intake (including food folate and folic acid from fortified foods and supplements) or othe

127 urface plasmon resonance (SPR) sensing using folic acid-functionalized gold nanoparticles (FA-AuNP) i

128 inally, we found that QDs cap-exchanged with folic acid-functionalized ligands could effectively targ

129 imaging of three different endocytic ligands-folic acid, galectin-3 (Gal3) and the Shiga toxin B-subu

130 , and inefficient chemotaxis toward cAMP and folic acid gradient.

131 d fortification, the exposure of children to folic acid has been a focus of concern, particularly reg

132 ablets and were treated with additional iron folic acid if they were anemic (hemoglobin <11 g/dL).

133 The women were provided with one daily iron-folic acid (IFA) capsule, one capsule containing multipl

134 regnancy through 6 mo postpartum or iron and folic acid (IFA) capsules during pregnancy and calcium p

135 pring from 6 to 24 mo (LNS-LNS), 2) iron and folic acid (IFA) during pregnancy and the first 3 mo pos

136 th multiple micronutrients (MMN) or iron and folic acid (IFA) in Indonesia.

137 for-age z score (LAZ) at 24 mo than iron and folic acid (IFA) provided to women during pregnancy and

138 ltiple micronutrient (MM) compared with iron folic acid (IFA) supplementation from early pregnancy im

139 enatal MM supplementation compared with iron-folic acid (IFA) supplementation was examined for its im

140 with multiple micronutrients (MMN) or iron + folic acid (IFA), versus folic acid (FA) alone, on risk

141 tandard of care comprising CSB+ and iron and folic acid (IFA).

142 ational weeks; 48 clusters received iron and folic acid (IFA; 60 mg Fe + 400 mug folic acid) and 16 c

143 s for its usage surrounding the synthesis of folic acid in 1945 and vitamin B12 in 1948.

144 The absorbed folic acid in brown rice has 93.53% retention after wash

145 ns in LND fibroblasts depend on the level of folic acid in the culture media.

146 late Polyp Prevention Study previously found folic acid increased risk of advanced and multiple color

147 Thus, physiological levels of folic acid induce accumulation of 5-aminoimidazole-4-car

148 In mice with folic acid-induced AKI, delayed treatment with Tanshinon

149 PTH secretion after experimental uremia- or folic acid-induced AKI.

150 cisplatin-induced kidney injury, but not to folic acid-induced fibrosis.

151 number of Troy-derived cells increases after folic acid-induced injury.

152 LD4 using specific siRNA protected mice from folic acid-induced kidney fibrosis and inhibited the inc

153 Using RNA sequencing in a mouse model of folic acid-induced nephropathy, we identified ten genes

154 PLD4 inhibition also prevented the folic acid-induced upregulation of this receptor in mous

155 In contrast, folic acid inhibited HPA activity by binding competitive

156 High folic acid inhibited MTHFR activity in vitro, and MTHFR

157 kappaB2 p100 and p52 increased rapidly after folic acid injection, as did DNA binding of RelB and NFk

158 fect the survival rate of either group after folic acid injection.

159 ses in risk were observed for both strata of folic acid intake (<400 microg/day, >/=400 microg/day),

160 cerns of potential adverse effects of excess folic acid intake and/or elevated folate status, with th

161 Impact of folic acid intake during pregnancy on genomic imprinting

162 Moderately high folic acid intake in pregnant women has led to concerns

163 r goal was to investigate the impact of high folic acid intake on liver disease and methyl metabolism

164 tions indicating adverse effects from excess folic acid intake, elevated folate status, and unmetabol

165 and mechanistic premises connecting elevated folic acid intake, UMFA, and/or high folate status to ad

166 s from the 1940s suggesting that high-dosage folic acid intake, used to treat patients with perniciou

167 R653Q, may modulate the effects of elevated folic acid intake.

168 ve consumption groups formed on the basis of folic acid intake.

169 However, the effects of increased folic acid intakes during pregnancy on maternal and cord

170 iconceptional supplementation have increased folic acid intakes in North American pregnant women.

171 Folate and folic acid intakes of 368 Canadian pregnant women were a

172 ires that captured recent and habitual daily folic acid intakes of children grouped as follows: 0-5 y

173 nication allowed the efficient absorption of folic acid into the brown rice kernel up to 5.195 x 10(4

174 Folic acid is an essential component of 1-carbon metabol

175 Although folic acid is essential to numerous bodily functions, re

176 Here, folic acid is hyperpolarized by dissolution dynamic nucl

177 Increased consumption of folic acid is prevalent, leading to concerns about negat

178 Subgroup analyses showed that the folic acid level was 1.66 ng/mL (95% CI, 0.10-3.21) lowe

179 eting ligands to nanocarriers (attachment of folic acid ligand recognized by folate receptors of canc

180 determine whether vitamins B(12), B(6), and folic acid lower homocysteine and improve symptomatology

181 yed treatment effects were not observed, but folic acid may increase SSA/P risk.

182 all took at least 400 mug/d of supplemental folic acid (median, 500; interquartile range, 400-600 mu

183 -transmembrane receptor fAR1 is required for folic acid-mediated signaling events.

184 samples, we observed that six enzymes of the folic acid metabolic pathway were overexpressed in HGS o

185 le of reusing MR1 that had been bound to the folic acid metabolite 6-formylpterin (6-FP).

186 nces regarding potential mechanisms by which folic acid might modify risk through effects on the epig

187 ession, but also to be aware that concurrent folic acid might reduce its effectiveness.

188 pression of these genes was also high in the folic acid model and in patients with biopsy-proven kidn

189 ine serum albumin (HS:BSA) mixtures onto the folic acid modified sensor resulted in a highly anti-fou

190 adenomas were randomly assigned to 1 mg/d of folic acid (n = 516) or placebo (n = 505), with or witho

191 After induction of folic acid nephropathy or unilateral ureteral obstructio

192 oligonucleotides in a mouse model of chronic folic acid nephropathy we can reduce fibrosis by 50% and

193 rs in animal models with lupus nephritis and folic acid nephropathy.

194 was induced by intraperitoneal injections of folic acid (nephrotoxic acute kidney injury) or by IM in

195 The recommended therapeutic daily dosage for folic acid of 5-20 mg was unchanged from 1946 through to

196 The effects of folic acid on the liver, the primary organ for folate me

197 key micronutrients (vitamin A, iodine, iron, folic acid) on improving micronutrient status and functi

198 se findings suggest that, in the presence of folic acid, one-carbon cofactors-notably when consumed t

199 l tube defects (NTDs); among nonconsumers of folic acid, only 16% attained optimal RBC folate.

200 vestigating the association between maternal folic acid or folate intake or maternal or neonatal fola

201 The control groups received daily iron and folic acid or multiple micronutrients during pregnancy a

202 n algorithm to lamotrigine or placebo and to folic acid or placebo.

203 al research needed to establish whether high folic acid or total folate intake contributes to disease

204 All patients received 5 mg of folic acid orally once daily.

205 Folic acid package inserts, early editions of hematology

206 Daily folic acid plus vitamin B12 was associated with improvem

207 ious anemia and with the changes in usage of folic acid preparations, including recommended therapeut

208 NTDs in humans, and the mechanisms by which folic acid prevents disease, remain disputed.

209 Folic acid prevents neural tube closure defects (NTDs),

210 We propose that folic acid promotes normal NTC in some embryos by regula

211 a point-of-care sensor for the detection of folic acid protein (FAP) using graphene-based SPR chips.

212 le micronutrient supplements containing iron-folic acid provided significantly greater reductions in

213 pproach, we discovered the long sought after folic acid receptor, fAR1, in D. discoideum.

214 n of FAP is based on the interaction between folic acid receptors integrated through pi-stacking on t

215 ect (NTD) outcomes among mothers meeting the folic acid recommendations.

216 as a significant interaction (p=0.028), with folic acid reducing the effectiveness of lamotrigine at

217 t gastrointestinal AEs more than once-weekly folic acid, regardless of the total weekly dosage.

218 obesity (35 of 270 patients [13.0%]), but a folic acid regimen was not.

219 ic moiety and the aromatic pteridine ring of folic acid remain outside the cyclodextrin cavity, while

220 had total intakes above the ULs for iron and folic acid, respectively.The level of intake of some nut

221 n of reproductive age and reduce the risk of folic acid-responsive neural tube birth defects (NTDs).

222 eficient mice provide a model to investigate folic acid-responsive NTDs wherein disruption of de novo

223 Conversely, a meta-analysis of folic acid revealed a nonsignificant difference from pla

224 e, magnesium, niacin, phosphorus, potassium, folic acid, riboflavin, and vitamins B-12, C, and E, and

225 Folic acid seems to nullify the effect of lamotrigine.

226 propose the mechanism of modification of the folic acid specifically under the hydrothermal treatment

227 and encapsulation techniques led to improved folic acid stability, especially under dry conditions.

228 found in a Higgins test for both omega-3 and folic acid studies; funnel plots also revealed asymmetry

229 High-dose folic acid supplement treatment exacerbated hypomethylat

230 r breast-feeding or maternal multivitamin or folic acid supplement use.

231 n = 59) compared with those who had adequate folic acid supplementation ( >= 400 mug/d) (betaMBP = 0.

232 8) and among children who had lower prenatal folic acid supplementation ( < 400 mug/d) (betaMBP = 1.3

233 mula: see text] 677TT), as well as high-dose folic acid supplementation ([Formula: see text], per gen

234 wer than those for the women with nonoptimal folic acid supplementation (overall birth defects: OR =

235 es compared with male neonates than did iron-folic acid supplementation alone (RR 0.85, 95% CI 0.75-0

236 ed survey to examine the association between folic acid supplementation and birth defects in Northwes

237 The association between folic acid supplementation and birth defects other than

238 Periconceptional folic acid supplementation and food fortification are re

239 n 1984, incidence of neural tube defects for folic acid supplementation compared with trace element s

240 Folic acid supplementation confers modest benefit in sch

241 We investigated the association between folic acid supplementation during pregnancy and loss of

242 lications for birth defect intervention with folic acid supplementation for countries with a high pre

243 To review the evidence on folic acid supplementation for preventing neural tube de

244 l first trimester urine samples and assessed folic acid supplementation from reported intakes.

245 rch is needed to clarify the precise role of folic acid supplementation in prevention of congenital m

246 Daily folic acid supplementation in the periconceptional perio

247 sed the balance of the benefits and harms of folic acid supplementation in women of childbearing age

248 09, the USPSTF reviewed the effectiveness of folic acid supplementation in women of childbearing age

249 rvices Task Force (USPSTF) recommendation on folic acid supplementation in women of childbearing age.

250 English-language studies of folic acid supplementation in women.

251 Periconceptional folic acid supplementation is a primary care-relevant pr

252 supplementation, and the effects of optimal folic acid supplementation on birth defects were assesse

253 We investigated the effects of moderate folic acid supplementation on reproductive outcomes and

254 fortification in the United States in 1998, folic acid supplementation provided protection against n

255 that the harms to the mother or infant from folic acid supplementation taken at the usual doses are

256 Optimal folic acid supplementation was associated with a decreas

257 Increased risk of SSA/P with extended folic acid supplementation was statistically significant

258 ystem defect rate for the women with optimal folic acid supplementation were lower than those for the

259 were used to match 9,293 women with optimal folic acid supplementation with 9,293 women with nonopti

260 ch can attenuate the measured association of folic acid supplementation with neural tube defects.

261 Newborn Health and Child Health (along with folic acid supplementation, a key reproductive health in

262 lementation with 9,293 women with nonoptimal folic acid supplementation, and the effects of optimal f

263 d a protective association consistently with folic acid supplementation, with ORs ranging from 0.93 t

264 ed new evidence on the benefits and harms of folic acid supplementation.

265 re mitigated by first trimester-of-pregnancy folic acid supplementation.

266 ted potential effect modification by sex and folic acid supplementation.

267 thfd1S(+/+) and Mthfd1S(+/-) mice were fed a folic acid-supplemented diet (FASD) (5-fold higher than

268 ale mice were placed on control diet (CD) or folic acid-supplemented diet (FASD) throughout mating, p

269 Folic acid-supplemented diet (FASD, 10-fold higher than

270 The benefits of the use of folic acid supplements (FASs) during the periconception

271 unders (maternal alcohol consumption, use of folic acid supplements, age, body size, education, and e

272 ications for individuals consuming high-dose folic acid supplements, particularly those who are MTHFR

273 s suggested that high circulating folate and folic acid (synthetic form of folate) supplement use may

274 All women received routine daily iron folic acid tablets and were treated with additional iron

275 of the women reported not consuming the iron folic acid tablets during pregnancy.

276 The consumption of folic acid through fortified foods at low, medium, and h

277 ablish programs to fortify staple foods with folic acid to prevent neural tube birth defects.

278 nd selected micronutrients including B12 and folic acid] to high-intensity interval aerobic fitness a

279 ored concentrations of fasting unmetabolized folic acid (UFA) in the circulation of children living i

280 te status was defined as unmetabolized serum folic acid (UMFA) > 1 nmol/L or serum total folate > 74.

281 e breast-milk total folate and unmetabolized folic acid (UMFA) contents and their relation with FA-su

282 d concentrations of folate and unmetabolized folic acid (UMFA) in a cohort of pregnant Canadian women

283 e, elevated folate status, and unmetabolized folic acid (UMFA) remain inconclusive; the data do not p

284 yl-THF, 5,10-methenyl-THF, and unmetabolized folic acid (UMFA) were measured using liquid chromatogra

285 association between cord blood unmetabolized folic acid (UMFA), 5-methyl tetrahydrofolate (THF), and

286 ing) were evaluated for the encapsulation of folic acid using both a whey protein concentrate (WPC) m

287 le micronutrient supplements containing iron-folic acid versus iron-folic acid alone in 112 953 pregn

288 Periconception maternal folic acid (vitamin B9) supplementation can reduce the p

289 Mixed results were found for zinc, folic acid, vitamin C, and tryptophan, with nonsignifica

290 Rs for vitamin A, vitamin B-6, vitamin B-12, folic acid, vitamin C, vitamin D, vitamin E, and multivi

291 ation of ultrasound-treated milled rice with folic acid was also efficient affording 6.559 x 10(4) mu

292 Folic acid was associated with lower risk for stroke (RR

293 1998 a Tolerable Upper Intake Level (UL) for folic acid was established based on case reports from th

294 Folic acid was not superior to placebo.

295 Fluorescein labeled folic acid was used for flow cytometric detection of the

296 Leveraging the addiction of ALL to folic acid, we conjugated folate to an alcohol derivativ

297 Recoveries of added folic acid were 112%-114%.

298 red lipid carriers (NLC) functionalized with folic acid were optimized through a Box-Behnken design i

299 ated to interactions between the protein and folic acid which favoured the incorporation of the bioac

300 capsulation efficiency, and stability of the folic acid within the capsules under different storage c