ライフサイエンスコーパス: follicular

1 ing diffuse large B-cell (3.29 [1.63-6.62]), follicular (3.01 [1.95-4.63]), marginal zone (1.90 [1.13

2 ology does not allow differentiation between follicular adenoma and carcinoma on Bethesda type IV les

3 and had histopathological results of either follicular adenoma or carcinoma.

4 ealed distinct IRF8 and PU.1 target genes in follicular and activated B cells.

5 lted in severe defects in the development of follicular and germinal center (GC) B cells.

6 e collected 14 days apart to distinguish the follicular and luteal phases of the menstrual cycle, and

7 ucted on 273 female cases (210 papillary, 45 follicular, and 18 not otherwise specified TC tumors) an

8 lected during the development of mature B1a, follicular, and marginal zone B cells.

9 Mice lacking cathepsin B display aberrant follicular architecture, a phenotype associated with eff

10 ndispensable requirement for the survival of follicular B (FOB) cells and Burkitt lymphoma (BL) cells

11 TORC1 attenuation may be necessary for human follicular B cell development.

12 gen and the timing of T cell help may affect follicular B cell fate, including death, survival, anerg

13 ession of target genes vital for maintaining follicular B cell identity and GC development.

14 were normally upregulated in the myeloid and follicular B cell lineages.

15 mice, we evaluated B-1b, marginal zone, and follicular B cell responses to the TI-2 Ag, NP-Ficoll.

16 hat metabolic quiescence was acquired at the follicular B cell stage in both humans and mice.

17 Differentiation to the follicular B cell stage in vitro correlated with surface

18 od studies revealed loss of transitional and follicular B cells in severe disease and accumulation of

19 Follicular B cells increased the abundance of the cell s

20 the IgM and IgD BCR isotypes on mature naive follicular B cells tunes responsiveness to endogenous an

21 plasma cell-inductive signals, we find that follicular B cells up-regulate a wide array of UPR-affil

22 Most follicular B cells were IgM(+) (70-80%), but IgA(+) (and

23 mpared with peritoneal B-2 cells and splenic follicular B cells, respectively.

24 In follicular B cells, the expression of genes involved in

25 tion exhibited a near absence of circulating follicular B cells.

26 l-2 stage paralleling the differentiation of follicular B cells.

27 dings demonstrate B-1b, marginal zone B, and follicular B subsets significantly contribute to the TI-

28 s of three well-defined populations: B1, B2 (follicular B, FOB), and marginal zone B (MZB) cells.

29 Formation of germinal centers (GCs), and follicular B- and T-cell differentiation was reduced, wh

30 gnancy are associated with a higher risk for follicular carcinoma in Bethesda type IV thyroid nodules

31 stologically confirmed cases of papillary or follicular carcinoma, diagnosed from 1993 to 1999, and 4

32 ce of a perinodular hypoechogenic halo) with follicular carcinoma.

33 terize the changes in the metabolism of each follicular cell type (i.e., oocyte, granulosa cells, inc

34 imaging for direct analysis and diagnosis of follicular cell-derived neoplasia tissues and FNA biopsi

35 on of the retrotransposon ZAM in the somatic follicular cells and subsequent germline genome invasion

36 the bi-directional communication between the follicular cells and the oocyte, which is partly mediate

37 Luteal cells were distinguishable from follicular cells by the presence of LDs, LD-associated p

38 t of EVs in mediating the stress response in follicular cells is not fully understood.

39 Follicular cells respond to heat stress (HS) by activati

40 cellular matrix swelling in both stromal and follicular cells.

41 ce have no significant difference in ovarian follicular counts and stages, nor in reproductively func

42 , nanoparticles were rapidly shuttled to the follicular dendritic cell (FDC) network and then concent

43 rowing, indolent tumors containing extensive follicular dendritic cell (FDC) networks and recurrent E

44 CAR-T cells can recognize and eliminate the follicular dendritic cell (FDC) reservoir of HIV-bound i

45 -acquired prion strains upon stromal-derived follicular dendritic cells (FDC) within the small intest

46 e reservoir of infectious HIV that exists on follicular dendritic cells (FDCs), persists in vivo duri

47 lymphoid structures that contain T cells and follicular dendritic cells and are particularly rich in

48 resent preferentially in splenic B cells and follicular dendritic cells during the persistent phase o

49 pathway-mediated immune complex delivery to follicular dendritic cells in vivo.

50 cells, high endothelial venules, supporting follicular dendritic cells network, and functional germi

51 opological remodeling of light and dark zone follicular dendritic cells required CXCL12-dependent cro

52 enmeshed with conventional dendritic cells, follicular dendritic cells, and stromal cells, usually l

53 rase-5 (GGT5), which was highly expressed by follicular dendritic cells, metabolized GGG to a form th

54 umbers of target germinal center B cells and follicular dendritic cells, which are the primary reserv

55 n the numbers of germinal center B cells and follicular dendritic cells.

56 d CR2 also mediate immune complex binding to follicular dendritic cells.

57 gnals, such as T follicular helper cells and follicular dendritic cells.

58 Similarly, in the ovary there is a lack of follicular development and lack of corpora lutea formati

59 nsulin family during two distinct windows of follicular development, ovulation, and luteinization.

60 etotrichography, is unique in measuring this follicular electrical activity, with possible applicatio

61 tin-proteasome system in both HTori3 thyroid follicular epithelial cells and follicular thyroid carci

62 Studying the follicular epithelial cells of Drosophila, we discovered

63 tightly organised epithelial monolayer, the follicular epithelium (FE).

64 pate in cell reintegration in the Drosophila follicular epithelium [4].

65 oogenesis, border cells delaminate from the follicular epithelium and migrate.

66 is not required for elongation of the early follicular epithelium, but drives the tissue toward opti

67 amenable Drosophila pupal photoreceptor and follicular epithelium.

68 1), and protein kinase B (Akt or PKB) in the follicular epithelium.

69 e released at significantly higher levels by follicular fibroblasts than by interfollicular subtypes.

70 tion from the follicle and hypothesized that follicular fibroblasts would accelerate skin re-epitheli

71 d the proteome of domestic cat (Felis catus) follicular fluid EVs (ffEV).

72 To determine the influence of follicular fluid EVs on gamete cryosurvival and the abil

73 and the oocyte, which is partly mediated by follicular fluid extracellular vesicles (EVs) released f

74 RNA) changes in PCOS and their expression in follicular fluid has been described, though the number o

75 They are synthesised by follicular granulosa cells as alpha plus betaA/betaB sub

76 te the role of circulating CXCR5(+)PD-1(+) T follicular helper (cT(FH)) and T follicular regulatory (

77 nction of neutrophils, instead inducing iNKT follicular helper (iNKTfh) cells that in turn promote au

78 Peripheral T follicular helper (pTfh) responses to S or N strongly co

79 tential biomarker to draw inferences about T follicular helper (T(FH)) activity within germinal cente

80 esus monkeys (RMs) resulted in more robust T follicular helper (T(FH)) cell responses and GC B cells

81 we concomitantly investigated the role of T follicular helper (T(FH)) cells and B cells during ABMR

82 T follicular helper (T(FH)) cells are a distinct type of C

83 T follicular helper (T(FH)) cells are CD4(+) T cells that

84 T follicular helper (T(FH)) cells are critical in adaptive

85 T follicular helper (T(FH)) cells direct the affinity and

86 lite acetate alter the functional state of T follicular helper (T(FH)) cells in vitro and in vivo, th

87 CD4(+) T follicular helper (T(FH)) cells provide help to B cells

88 ndent manner and induced the maturation of T follicular helper (T(FH)) cells.

89 ther successful DAA treatment reconstitute T follicular helper (T(FH))-B cell axis in HCV patients is

90 mouse model of Ehrlichia muris that type 1 T follicular helper (T(FH1)) cells provide help to CD11c(+

91 le for T-B lymphocyte interactions, drives T follicular helper (Tfh) cell development in germinal cen

92 h consequently control naive B and cognate T follicular helper (Tfh) cell interaction and initiation

93 icited potent SARS-CoV-2-specific GC B and T follicular helper (Tfh) cell responses as well as LLPCs

94 re we examined HA stem-specific B cell and T follicular helper (Tfh) cell responses in the context of

95 a 6 (BCL6), a transcriptional regulator of T follicular helper (Tfh) cells and germinal center B cell

96 T follicular helper (Tfh) cells are a specialized T cell s

97 CD4(+) T follicular helper (Tfh) cells are essential for inducing

98 The current view is that CD4(+) T follicular helper (Tfh) cells are the main subset regula

99 CD4+ T follicular helper (Tfh) cells dominate the acute respons

100 T follicular helper (Tfh) cells in germinal centers of sec

101 osed to peanut flour, naive mice developed T follicular helper (Tfh) cells in their lung draining lym

102 T follicular helper (Tfh) cells play a very important role

103 CXCR5(+) T follicular helper (Tfh) cells provide help to B cells, a

104 This factor, SpeA, can induce abnormal T follicular helper (Tfh) cells that are able to kill B ce

105 one germinal center B cells, as well as to T follicular helper (TFH) cells, and directly regulates B

106 7 cells and germinal center (GC)-promoting T follicular helper (Tfh) cells, resulting in cGVHD.

107 TCRs biased mouse naive T cells to become T follicular helper (Tfh) cells, whereas higher-affinity T

108 a specialized CD4(+) T cell subset called T follicular helper (Tfh) cells.

109 ction between antigen-specific B cells and T follicular helper (Tfh) cells.

110 ological impediments, including defects in T follicular helper (Tfh) cells.

111 the GC microenvironment, including CD4(+) T follicular helper (Tfh) cells.

112 the role of lung dendritic cells (DCs) in T follicular helper (Tfh)-cell induction, a T-cell subset

113 controls antibody production by inhibiting T follicular helper (Tfh)-mediated help to B cells.

114 eneic mismatch murine transplant model and T follicular helper (Tfh):B cell co-culture system.

115 e autoantibody production and dysregulated T follicular helper and B cell responses.

116 (GCs) and requires rate-limiting "help" from follicular helper CD4(+) T (Tfh) cells.

117 PD-L2 blockade, unleashed insulin-specific T follicular helper CD4(+) T cells and enhanced their surv

118 echanism, we analyzed the B and T peripheral follicular helper cell (pTfh) responses.

119 Germinal centres (GCs) are T follicular helper cell (Tfh)-dependent structures that f

120 ver that immune checkpoint therapy induces T follicular helper cell activation of B cells to facilita

121 fection induced a partial expansion of the T follicular helper cell compartment, essential for B cell

122 umoral CD8(+) T cell exhaustion and CD4(+) T follicular helper cell development.

123 hat the initial B-cell clonal composition, T-follicular helper cell signaling, increased rounds of pr

124 ected of promoting disease) and a specific T follicular helper cell subset that contributes to IgG4 i

125 regulate steady state interactions between T follicular helper cells (TfH) and B cells to limit mucos

126 autoreactive follicles, B-cells encounter T-follicular helper cells (Tfh) that produce interleukin (

127 we found increased proportions of cytotoxic follicular helper cells and cytotoxic T helper (T(H)) ce

128 vival and differentiation signals, such as T follicular helper cells and follicular dendritic cells.

129 antibodies increase germinal center B and T follicular helper cells and plasma neutralizing antibodi

130 Bcl6 is required for the development of T follicular helper cells and T follicular regulatory (Tfr

131 es taken prior to treatment suggested that T follicular helper cells and various other immune cell su

132 a strongly promoted the differentiation of T follicular helper cells followed by an enhanced germinal

133 he germinal center (GC) reaction, in which T follicular helper cells interact with GC B cells to prod

134 xpression signature associated with CD4(+) T follicular helper cells that is associated with longer p

135 aive and memory B cells and a reduction in T follicular helper cells with a phenotype suggesting rece

136 SIGNR1(+) dendritic cells (which activate T follicular helper cells) and lymphatic sinus-associated

137 ation of human germinal-centre B cells and T follicular helper cells, and antagonized the induction o

138 of effector T cell populations, including T follicular helper cells, and increases germinal center B

139 2 function, germinal center formation, and T follicular helper cells, especially when the load of the

140 C and signaling-selective variants reduced T follicular helper cells, germinal center formation, immu

141 f IL-21, produced mainly by Th17 cells and T follicular helper cells, has been intensively investigat

142 hallmarks of this effector program become T follicular helper cells, supporting development of B cel

143 toantibody levels in part by inhibition of T follicular helper cells.

144 o drive T cell maturation into CXCR5+PD-1+ T follicular helper cells.

145 ion of B cells and indirectly by promoting T follicular helper cells.

146 on strictly dependent on interactions with T follicular helper cells.

147 ZV-specific cytotoxic T cell (VZV-CTL) and T follicular helper responses to ZVL did not correlate wit

148 ) cells have specialized roles in modulating follicular helper T (T(FH)) cell activation of B cells.

149 er T cells were phenotypically distinct from follicular helper T (T(FH)) cells and lacked BCL6 expres

150 1 (SOSTDC1), secreted by a subpopulation of follicular helper T (T(FH)) cells and T-B cell border-en

151 Follicular helper T (T(FH)) cells are implicated in type

152 tion of inflammatory effector T cells versus follicular helper T (T(FH)) cells that mediate high-affi

153 ation depends upon effective interactions of follicular helper T (T(fh)) cells with germinal center (

154 CD1(+) peripheral helper T (T(PH)) cells and follicular helper T (T(FH)) cells.

155 ectories developed as T helper 1 (T(H)1) and follicular helper T (T(FH)) transcriptomes contracted an

156 show that CD4(+) effector T cells including follicular helper T (Tfh) cells are the major producers

157 ter (GC) is governed by signals delivered by follicular helper T (Tfh) cells to B cells.

158 These defects could be corrected when follicular helper T (Tfh) cells were induced before macr

159 AD, but accumulating evidence has shown that follicular helper T cell (T(FH)), a critical player in h

160 we were able to identify a super-functional follicular helper T cell (Tfh)-like subpopulation in lup

161 ricted CD8 Treg-mediated suppression of host follicular helper T cell-dependent antibody production.

162 Follicular helper T cells (T(FH) cells) have long been i

163 of HIV-infected germinal center (GC) CD4(+) follicular helper T cells (Tfh) after combination antire

164 CD4+ follicular helper T cells (Tfh) are essential for germin

165 Follicular helper T cells (Tfh) play critical roles inst

166 accessibility and gene connectivity in human follicular helper T cells (TFH), a cell type required fo

167 Follicular helper T cells (Tfh), CD4 lymphocytes critica

168 ubsets such as T(H)2, T(H)9, T(H)17, T(H)22, follicular helper T cells and CD28(null) T cells, as wel

169 Despite this, circulating follicular helper T cells from CVID+AIC subjects exhibit

170 e applied it to the profiling of circulating follicular helper T cells implicated in systemic lupus e

171 he population of germinal center B cells and follicular helper T cells in the draining lymph node and

172 oduction by T(H)2 and follicular helper T/ex-follicular helper T cells promotes asthma by inhibiting

173 Follicular helper T cells were the primary source of IL-

174 antly diminished bone marrow PCs, lymph node follicular helper T cells, and memory B cell proliferati

175 We also discuss the role of mTOR in follicular helper T cells, regulatory T cells, and other

176 follicle and steering their interaction with follicular helper T cells.

177 ), and IL-21(+) (V6, P = .003; V8, P = .002) follicular helper T cells.

178 A recently discovered population of T follicular helper T(fh)13 cells regulates the production

179 The resulting enlarged circulating follicular helper T-cell population from CVID+AIC subjec

180 We show that IL-21 production by T(H)2 and follicular helper T/ex-follicular helper T cells promote

181 ive polymerase chain reaction, and GCs and T follicular helper were assessed using immunohistochemist

182 of subsets, including T(H)1, T(H)2, T(H)9, T follicular helper, T follicular regulatory, and regulato

183 uding expanded double-negative, but depleted follicular helper, T-cell compartments and impaired Fas-

184 hese LL37-specific SLE T-cells displayed a T-follicular helper-(T(FH))-like phenotype, with CXCR5/Bcl

185 CD4(+) T-helper type 1 differentiation and T-follicular-helper cell polarization and increased the ab

186 consisted of less T-central-memory cells, T-follicular-helper cells, TGF-beta response, and CD4( +)

187 , and T helper cell type 17 (T(H)17), and of follicular-helper T cells.

188 Moreover, follicular IgA expression associated with expression of

189 Follicular IgA response was also observed in PAO1-infect

190 We also evaluated follicular IgA responses in the lungs from mice infected

191 Fifty-six patients were enrolled, most with follicular lymphoma (43%) or diffuse large B-cell lympho

192 zone B-cell lymphoma (EMZL) (n = 177, 68%), follicular lymphoma (FL) (n = 26, 10%), diffuse large B-

193 a or small lymphocytic lymphoma (SLL) and RR follicular lymphoma (FL) after two or more prior systemi

194 scuss recently published studies centered on follicular lymphoma (FL) and diffuse large B-cell lympho

195 These results extend the seminal study in follicular lymphoma (FL) from the National LymphoCare St

196 lthough the life expectancy of patients with follicular lymphoma (FL) has increased, little is known

197 r-type H+-translocating ATPase (v-ATPase) in follicular lymphoma (FL) highlights a role for the amino

198 Follicular lymphoma (FL) is a low-grade B-cell malignanc

199 A minority of patients with follicular lymphoma (FL) undergo histological transforma

200 have enabled improved risk classification of follicular lymphoma (FL) using, for example, the m7-FLIP

201 nobiology of the 15% to 30% of patients with follicular lymphoma (FL) who experience progression of d

202 Patients with follicular lymphoma (FL) with early relapse after initia

203 We determined the ICT-GEP of Follicular Lymphoma (FL), and compared it with that of t

204 risk of chronic lymphocytic leukemia (CLL), follicular lymphoma (FL), and diffuse large B-cell lymph

205 Cs are also the origin of malignancy, namely follicular lymphoma (FL), GC B cell-diffuse large B cell

206 s (>=18 years) with histologically confirmed follicular lymphoma (grade 1, 2, 3a, or 3b) that had rel

207 th previously treated relapsed or refractory follicular lymphoma (grade 1, 2, or 3a) were included.

208 ly, CBT has been relatively disappointing in follicular lymphoma and diffuse large B-cell lymphoma.

209 f immune surveillance are lacking, including follicular lymphoma and most diffuse large B-cell lympho

210 Follicular lymphoma B cells undergo continuous somatic h

211 ma, primary mediastinal B-cell lymphoma, and follicular lymphoma grade 3B.

212 apy and 77 to the combination (47% poor-risk Follicular Lymphoma International Prognostic Index score

213 arly stage diffuse large B-cell lymphoma and follicular lymphoma suggests better delineation of disea

214 a ranged from five (13%) of 40 patients with follicular lymphoma to seven (35%) of 20 patients with R

215 lenalidomide with rituximab in patients with follicular lymphoma treated in the CALGB 50401 (Alliance

216 e large B-cell lymphoma and 41 patients with follicular lymphoma were eligible for analysis.

217 with previously untreated high-tumor-burden follicular lymphoma were nonrandomly assigned to receive

218 th diffuse large B-cell lymphoma and 42 with follicular lymphoma were recruited between Sept 27, 2012

219 PET can predict prognosis for patients with follicular lymphoma with high tumour burden at the end o

220 pleted patient treated with obinutuzumab for follicular lymphoma with protracted COVID-19 and viremia

221 cytic lymphoma, 13 (33%) of 40 patients with follicular lymphoma, 16 (36%) of 45 patients with diffus

222 ed; 19 had diffuse large B-cell lymphoma, 53 follicular lymphoma, and one marginal zone lymphoma.

223 assessment of diffuse large B-cell lymphoma, follicular lymphoma, and peripheral T-cell lymphoma.

224 om using TarPan Viewer on publicly available follicular lymphoma, breast cancer, and multiple myeloma

225 all lymphocytic lymphoma (del17p or del11q), follicular lymphoma, diffuse large B-cell lymphoma, and

226 kin lymphoma (diffuse large B-cell lymphoma, follicular lymphoma, mantle cell lymphoma, peripheral T-

227 In follicular lymphoma, traditionally viewed as an incurabl

228 resent in approximately 20% of patients with follicular lymphoma.

229 tuximab in patients with relapsed/refractory follicular lymphoma.

230 class, oral EZH2 inhibitor, in patients with follicular lymphoma.

231 ostat is a novel treatment for patients with follicular lymphoma.

232 + R (BR) alone in relapsed/refractory (R/R) follicular lymphoma.

233 treated patients with relapsed or refractory follicular lymphoma.

234 eatment of patients with relapsed/refractory follicular lymphoma.

235 refractory diffuse large B-cell lymphoma and follicular lymphoma.

236 tion in high-grade serous ovarian cancer and follicular lymphoma.

237 es of CBP/p300 loss-of-function mutations in follicular lymphoma.

238 0 loss-of-function mutations was observed in follicular lymphoma.

239 refractory diffuse large B-cell lymphoma and follicular lymphoma.

240 in the alteration of the immune landscape in follicular lymphoma.

241 a platform for response-adapted treatment of follicular lymphoma.

242 Follicular lymphomas (FLs) are slow-growing, indolent tu

243 The genetic background of follicular lymphomas (FLs) diagnosed in advanced clinica

244 BCL2, 11 of 25 DHITsig-positive-transformed follicular lymphomas were classified as HGBL-DH/TH- BCL2

245 In this study, we examined the follicular melanin unit in variably pigmented follicles

246 of nodules with a pathological diagnosis of follicular neoplasm in order to achieve a more conservat

247 when a halo sign lesion is observed, benign follicular neoplasm should be considered.

248 Measurements were taken in the follicular phase (day 3) of the last menstrual cycle in

249 03 nmol/kg) versus KP54 (9.6 nmol/kg) in the follicular phase of healthy women (n = 9), and in women

250 Moreover, during the follicular phase of the estrous cycle in female mice, th

251 The follicular phase signature was characterized by an eleva

252 with village-level trachomatous inflammation-follicular prevalence.

253 ize divergence in ovarian transcriptomic and follicular profiles between alligators originating from

254 edle biopsy findings indicated malignancy or follicular proliferation and versus the stability of the

255 idered histologic findings of malignancy and follicular proliferation positive because both require s

256 5%) were classified as benign, 50 (25.6%) as follicular proliferation, and 7 (3.6%) as malignant.

257 Trials were conducted in quasi-follicular (qF) and quasi-luteal (qL) phases in dry (DRY

258 +)PD-1(+) T follicular helper (cT(FH)) and T follicular regulatory (T(FR)) cells following grass poll

259 T follicular regulatory (Tfr) cells are a specialized subs

260 T follicular regulatory (TFR) cells limit Ab responses, bu

261 velopment of T follicular helper cells and T follicular regulatory (Tfr) cells that regulate germinal

262 The circulating T follicular regulatory cell subset and CXCR5(+) CD8(+) T

263 Follicular regulatory T (T(FR)) cells have specialized r

264 (GC) responses potentiate the generation of follicular regulatory T (T(FR)) cells.

265 Follicular regulatory T (Tfr) cells are a regulatory T c

266 ons, including RORgammat(+) T(reg) cells and follicular regulatory T cells, were c-Maf dependent.

267 Induction of FoxP3(+), follicular regulatory T, and IL-10(+) regulatory B cells

268 T(H)1, T(H)2, T(H)9, T follicular helper, T follicular regulatory, and regulatory T cells.

269 Specifically, CXCL13(+) follicular reticular cells form a small-world network of

270 Follicular skewing correlated with IgA deficiency-associ

271 We have discovered them in the follicular spherical epithelium of ascidians that are em

272 markers of junctional zone and upper isthmus follicular stem cells.

273 from LDs in other bovine tissues, including follicular steroidogenic cells.

274 Estrogen, follicular stimulating hormone (FSH), and luteinizing ho

275 Estrogen (E2), follicular stimulating hormone (FSH), and luteinizing ho

276 e continued to develop plasma cells, splenic follicular structure was restored, and renal pathology w

277 Our study thus highlights the diversity of follicular T cell subsets that contribute to the breakdo

278 Follicular T helper (TFH) cells provide B-cell help and

279 ucing Th2 cells, IL-17-producing Th17 cells, follicular T helper (Tfh) cells, or regulatory T (Treg)

280 ls with activated extrafollicular CD4(+) and follicular T helper (Tfh) cells.

281 of the follicular T helper cell program from follicular T helper (Tfh)-IL-17 to Tfh-IFN-gamma.

282 pite infants exhibiting significantly higher follicular T helper cell (Tfh) and germinal center B cel

283 ed splenic B-cell reconstitution and reduced follicular T helper cell development.

284 XD2-p19(-/-) ) mouse leads to a shift of the follicular T helper cell program from follicular T helpe

285 pulation of HCV-specific CD4+ T cells with a follicular T helper cell signature that is maintained af

286 typic and transcriptional characteristics of follicular T helper cells increasingly shaped the circul

287 a is associated with early commitment to the follicular T-cell lineage in IgA-deficient CVID subjects

288 ols with total differentiated, papillary, or follicular TC.

289 were observed for papillary TC, but not for follicular TC.

290 he transmission of trachomatous inflammation-follicular (TF) and discontinue mass drug administration

291 stricts where the prevalence of trachomatous follicular (TF) is >=10% in children aged 1-9 years.

292 Vanuatu, the sign trachomatous inflammation-follicular (TF) is common, but ocular infection with Chl

293 lar clinical sign "trachomatous inflammation-follicular" (TF) among children aged 1-9 years within po

294 identifies a cluster-independent increase of follicular (TFH) cells potentially driving the known exp

295 nd adaptive IFNgamma production, and inhibit follicular Th cell development.

296 isoform-specific in vivo data are limited in follicular thyroid cancer (FTC), a PI3 kinase-driven tum

297 d for papillary thyroid cancer (n = 341) and follicular thyroid cancer (n = 25) patients, sex, length

298 ori3 thyroid follicular epithelial cells and follicular thyroid carcinoma 133 (FTC133) cells.

299 e in T-bet(+) T(H1) cells and aberrant extra-follicular TNF-alpha accumulation.

300 ling and prevents their differentiation into follicular Treg and tissue-resident Treg cells.