ライフサイエンスコーパス: food deprivation

1 onal shift to a 'high-drive' condition (18-h food deprivation).

2 crose pellets under 24-hr but not under 0-hr food deprivation).

3 ing neurons increases in response to chronic food deprivation.

4 kness with ad libitum food and after 48 h of food deprivation.

5 y and suppressed 6-h refeeding after 24 h of food deprivation.

6 limited food is presented after a period of food deprivation.

7 bohydrate content of the diet consumed after food deprivation.

8 te for the reduced contribution from chronic food deprivation.

9 nable weanling animals to survive periods of food deprivation.

10 drial beta-oxidation genes were repressed by food deprivation.

11 ct any alterations in ARC NPY mRNA following food deprivation.

12 ed by the refeeding hyperphagia that follows food deprivation.

13 5, 10, 20 and 40 microg) following overnight food deprivation.

14 ther of these responses when the stimulus is food deprivation.

15 in response to systemic/metabolic aspects of food deprivation.

16 ht to mediate the neuroendocrine response to food deprivation.

17 both physiological/psychological stress and food deprivation.

18 no inhibitory effects on feeding after 24 h food deprivation.

19 or SHU9119 to rats before a 24 hr period of food deprivation.

20 elimination of odors as cues, and the use of food deprivation.

21 ody weight, and become hyperphagic following food deprivation.

22 o give rise to interoceptive cues like 24-hr food deprivation.

23 ent to initiate locomotor responses to acute food deprivation.

24 he neurons express 5-HT and are inhibited by food deprivation.

25 substantial meals, particularly those after food deprivation.

26 neurons expressed 5-HT and were inhibited by food deprivation.

27 low whole-body glucose levels within 24 h of food deprivation.

28 ll Period mutant mice was also robust during food deprivation.

29 sweeter nonnutritive sugar after periods of food deprivation.

30 tion during the lengthening period of summer food deprivation.

31 ity in other systems may be downregulated by food deprivation.

32 isual interneurons, an effect not altered by food deprivation.

33 failed to recover normal pumping rates after food deprivation.

34 did not show an altered feeding response to food deprivation.

35 ahepatic tissues during periods of prolonged food deprivation.

36 specifically regulates odr-10 in response to food deprivation.

37 uced fibre atrophy caused by denervation and food deprivation.

38 to exhibit exaggerated locomotor response to food deprivation.

39 o promotes neuroendocrine adaptations during food deprivation.

40 ed by control floxed mice only after 24 h of food deprivation.

41 n than controls under conditions of complete food deprivation.

42 ally contiguous phases: 1-h chow access, 2-h food deprivation, 10-min chow access, and 10-min access

43 In Experiment 1, the impact of food deprivation (24 and 48 h) was examined.

44 ntal area upon feeding responses elicited by food deprivation (24 h), 2-deoxy-D-glucose-induced gluco

45 DAMGO (90% maximal decrease), and 24 h acute food deprivation (60% maximal decrease).

46 eus (Arc) and their synthesis increases with food deprivation, a naturally-occurring stimulus that ma

47 ile males exhibit high odr-10 expression and food deprivation activates odr-10 in adult males [4-6].

48 Both restraint and food deprivation affected select limbic areas associated

49 etailed profile of cognitive consequences of food deprivation, affected by time of day, task demands,

50 Here, we investigate how food deprivation affects olfactory behavior in Drosophil

51 e, and bacterial translocation compared with food deprivation (all p < 0.05).

52 on the GH-IGF-I axis differed from those of food deprivation alone; and b) whether administration of

53 culating FFA for glucose-stimulated IS after food deprivation also applies in the case of nonglucose

54 Food deprivation also challenges the gut microbiota, whi

55 Food deprivation alters the processing of sensory inform

56 Included among these are (1) food deprivation and (2) acute microinjection of the neu

57 Food deprivation and concentration interacted to increas

58 The effects of food deprivation and concentration on burst size (BS), b

59 he mammalian body provide resilience against food deprivation and dietary stress.

60 oth the depletion of energy induced by acute food deprivation and excessive storage of energy by high

61 bling induction of beta-oxidation genes upon food deprivation and facilitating activation of SCD in f

62 t small intestine OEA levels decrease during food deprivation and increase upon refeeding, suggesting

63 y a biological role for AGS-3 in response to food deprivation and indicate the mechanism for its acti

64 havioral shift occurs more rapidly following food deprivation and is modulated by cAMP and insulin si

65 Both food deprivation and MA (600 micromol/kg), but not 2-DG,

66 f the CRR, and thus the ability to withstand food deprivation and maintain euglycemia, is not known.

67 However, the role of food deprivation and metabolic needs in the selection of

68 om environmental energetic stressors such as food deprivation and physical exertion.

69 Acb shell GABA(A) receptor stimulation or by food deprivation and produced a syndrome of forepaw trea

70 (dNPF), an ortholog of mammalian NPY, mimics food deprivation and promotes memory performance in sati

71 g despite normalizing feeding in response to food deprivation and PYY.

72 on the nutritional state-they decreased with food deprivation and recovered after subsequent feeding.

73 ationally related manipulations of the task (food deprivation and reward magnitude) produced some sub

74 cleus (PSTN) is activated by refeeding after food deprivation and several PSTN subpopulations have be

75 The ZI cells were excited by food deprivation and the gut hunger signal ghrelin.

76 multaneous variations in motivational state (food deprivation) and reinforcer magnitude (food present

77 iven to rats in a 'low-drive' condition (2-h food deprivation), and also after a motivational shift t

78 For example, ghrelin levels rise following food deprivation, and ghrelin administration stimulates

79 obility is regulated by caloric restriction, food deprivation, and heat shock.

80 muli, including exposure to dauer pheromone, food deprivation, and high temperature, can induce C. el

81 as markedly increased by lipin 1 deficiency, food deprivation, and obesity, often independent of chan

82 ubjective deprivation, material deprivation, food deprivation, and political deprivation (which was m

83 Twenty-eight peptides were increased >50% by food deprivation, and some of these were increased by 2-

84 eaten, the increase in consumption following food deprivation, and the type of food eaten.

85 tis elegans to alter several behaviors after food deprivation, apparently so that the animals can mor

86 food deprivation, suggesting that effects of food deprivation are mediated by modifying this protein.

87 severe childhood undernutrition (SCU) during food deprivation are not clear.

88 Surprisingly, these responses to food deprivation are not triggered by internal metabolic

89 e increases in food intake following 24 h of food deprivation are reduced by systemic and central adm

90 s water maze without requiring foot shock or food deprivation as motivating factors.

91 Upon food deprivation, attraction-mediating uniglomerular pro

92 Animals were studied after overnight food deprivation, awake, unrestrained, and unstressed; a

93 ate different protein breakdown responses to food deprivation between children with edematous and non

94 tabolic state, we examined whether overnight food deprivation blunts stress-induced recruitment of th

95 The results demonstrated that NPY and food deprivation both produced dose- or deprivation-depe

96 Peptides found to be elevated by food deprivation but not exercise included a number of f

97 Conversely, food deprivation but not NMDA injection produced marked

98 Finally, food deprivation, but not other stressors, stimulated cA

99 ly hatched Caenorhabditis elegans respond to food deprivation by halting development and promoting lo

100 of liquid diet or 10% sucrose solution after food deprivation by rats with APX also were considerably

101 Here we report that this effect of food deprivation can be blocked by leptin, a hormone inv

102 These data indicate that food deprivation can provoke relapse to heroin seeking v

103 the delay was extended beyond 1 h, and that food deprivation could attenuate the degree of impairmen

104 gely on elevated responding to nonreinforced food deprivation cues.

105 Food deprivation decreased drinking latencies, did not c

106 by a state of negative-energy balance (24-hr food deprivation), demonstrating a specific influence of

107 duced rise in hypothalamic malonyl-CoA after food deprivation, demonstrating that leptin was not requ

108 Food deprivation did not affect BS but rather increased

109 esponse of chromatin-associated proteins, as food deprivation did not affect the mobility of heteroch

110 Following recovery from surgery, food deprivation discrimination performance was compared

111 lt mice, leptin was not acutely regulated by food deprivation during the early postnatal period.

112 We find that food deprivation elevates excitatory synaptic input, whi

113 lows survival during the frequent periods of food deprivation encountered during evolution.

114 s were tested for reinstatement after 1 d of food deprivation (experiment 1) or exposure to intermitt

115 ic gene expression resulting from short-term food deprivation (fasting) in Caenorhabditis elegans.

116 We found that short-term food deprivation (fasting) potently induced p21 expressi

117 adapted to prolonged periods (1-2 months) of food deprivation (fasting) which result in metabolic cha

118 Intermittent food deprivation (fasting, IF) improves mood and cogniti

119 erian hamster feeding behaviors; (2) whether food deprivation (FD) co-increases agouti-related protei

120 completely blocked the effects of leptin on food deprivation (FD)-induced feeding.

121 EXPERIMENTS 2 AND 3: Following overnight food deprivation, five groups of rats were injected with

122 We found short-term food deprivation for 24 h, already induces SIRT2 express

123 Here, we report that food deprivation for 48 h in mice significantly reduces

124 Food deprivation for 48 hr also increased CRE binding, w

125 Finally, we found that food deprivation for an 8-h period induced gene expressi

126 In contrast, food deprivation had little effect.

127 Food deprivation has been shown to deleteriously affect

128 lth and subjective, economic, political, and food deprivation; health, economic, and political stress

129 bing neurons controls behavioral response to food deprivation in C. elegans.

130 Methods that utilise food deprivation in contrived environments may lack the

131 d tanycytes, were significantly decreased by food deprivation in male mice in a manner that was parti

132 nment in vivo, resulting in overeating after food deprivation in mice.

133 ke and body weight changes following 24 h of food deprivation in rats.

134 vation induced by 2-deoxy-D-glucose (2DG) or food deprivation in rodents.

135 lects calorie-rich foods following prolonged food deprivation in the absence of taste-receptor signal

136 mediating the short-term feeding response to food deprivation in the nucleus accumbens shell as well

137 of food and over an extended time course of food deprivation in wild-type and mutant worms to determ

138 immunoreactivity in this area is enhanced by food deprivation; in contrast, it is reduced by injectio

139 mice at this chronological age, because 24-h food deprivation increased arcuate NPY mRNA in wild-type

140 Food deprivation increased c-fos expression and spike fr

141 context of diet-induced and genetic obesity; food deprivation increased Deptor mRNA in both the ARC a

142 way is involved in hunger-induced analgesia: Food deprivation increases DA release in the ACC and con

143 Food deprivation increases hypothalamic syndecan-3 level

144 In Drosophila larvae, food deprivation increases locomotor speed via octopamin

145 easure of eating is modulated by satiety and food deprivation increases the reward value of food, the

146 We also establish that food deprivation increases the worm's willingness to cro

147 MTII administration for 24 h prevents food deprivation-induced alterations in hypothalamic neu

148 he other hand, TRB3 knockout mice ameliorate food deprivation-induced atrophy compared with WT litter

149 TRB3 regulates skeletal muscle mass in food deprivation-induced atrophy.

150 nfused into the basolateral amygdala blocked food deprivation-induced changes in sucrose-related acti

151 In addition, IV-SUnSET detected food deprivation-induced decreases in PS in the heart, k

152 ition of ER stress in C2C12 myotubes reduces food deprivation-induced expression of TRB3 and muscle-s

153 These similarities between NPY-induced and food deprivation-induced feeding are consistent with a s

154 oraging and food hoarding were not affected, food deprivation-induced increased food hoarding was sur

155 suggests an intact Arc is not necessary for food deprivation-induced increases in food foraging and

156 ole in skeletal muscle mass regulation under food deprivation-induced muscle atrophy and TRB3 could b

157 uscle-specific TRB3 transgenic mice increase food deprivation-induced muscle atrophy compared with wi

158 Like obesity, prolonged food deprivation induces severe hepatic steatosis; howev

159 Rats were trained to solve a food deprivation intensity discrimination problem in whi

160 ability of an animal to withstand periods of food deprivation is a key driver of invasion success (bi

161 lating concentration of FFA that accompanies food deprivation is a sine qua non for efficient GSIS wh

162 ce of energy and nutrient homeostasis during food deprivation is accomplished through an increase in

163 circulating FFA levels after 24 and 48 h of food deprivation is critically important for pancreatic

164 -specific GHR ablation to save energy during food deprivation is impaired, leading to increased fat l

165 In addition, a brief period of junk-food deprivation is needed for the synaptic insertion of

166 he chemotaxis behavior of aged animals under food deprivation is shown to be dependent on daf-16.

167 After food deprivation, leptin induced an increase in phosphor

168 Results suggest that food deprivation may act as a novel stress, thereby incr

169 Upon food deprivation, odr-10 is directly activated by DAF-16

170 er infusion of milk to assess the effects of food deprivation on sleep.

171 We evaluated the influence of food deprivation on the reproductive fitness and behavio

172 However, when challenged by food deprivation or harsh environmental conditions, many

173 Lipid stores are mobilized in the case of food deprivation or high energy demands--for example, du

174 ntaining neurons or fibers are unaffected by food deprivation or MA, and the antimetabolite 2-DG has

175 ine [TMT]), a paradigm that does not involve food deprivation or operant performance.

176 Here, we show in mice that food deprivation or optogenetic activation of AgRP neuro

177 hich can be altered by manipulations such as food deprivation or prior exposure to MOP receptor agoni

178 stimuli (footshocks, shock-predictive cues, food deprivation, or reward omission) and inhibitions af

179 learned schedules of food intake rather than food deprivation per se.

180 Instead, prolonged food deprivation potentiates temperature responses in th

181 at another environmental stressor, acute 1 d food deprivation, potently reinstates heroin seeking in

182 ess NMDA-elicited feeding by PTK inhibitors, food deprivation readily drives PTK activity in vivo.

183 Acute food deprivation reinstated heroin seeking, an effect th

184 corticosterone levels were reduced, whereas food deprivation resulted in significantly enhanced plas

185 In Caenorhabditis elegans, pairing odor with food deprivation results in aversive olfactory learning,

186 actic acid bacteria Lactobacillus reuteri or food deprivation selectively maintained the chemotaxis a

187 in which stimuli produced by 0-hr and 24-hr food deprivation served as discriminative cues for the d

188 Here, we show that after food deprivation, SIRT1 levels fall dramatically in type

189 ternation in a T-maze and on a task in which food-deprivation state was used as a contextual cue.

190 effects on food intake both under normal and food deprivation states.

191 ine or sucrose, it effectively blocked acute food deprivation stress-induced reinstatement of cocaine

192 results independent from changes related to food deprivation stress.

193 change in its biochemical fractionation upon food deprivation, suggesting that effects of food depriv

194 , whereas nutrient supplementation following food deprivation suppressed it; the former and latter co

195 Food deprivation suppresses animal growth and developmen

196 Food deprivation suppresses sleep, presumably to increas

197 Following food deprivation, the expression and acetylation of the

198 In response to food deprivation, the pumping rate rapidly declines by a

199 pamine in aminergic neurons, is increased by food deprivation, thus selecting between antagonistic am

200 responses that are otherwise associated with food deprivation; thus, unlike forced dieting, cessation

201 Food-deprivation transiently activates male odr-10 expre

202 ncy of these 'unassigned' fish was higher in food deprivation treatments, but lower in warm treatment

203 at an increase in locomotor speed induced by food deprivation was accompanied by an activity- and oct

204 In addition, food deprivation was tested as a possible modulating fac

205 portance of considering prolonged periods of food deprivation when assessing chemical risks posed to

206 ach 4-min session that took place under 0-hr food deprivation, whereas no pellets were delivered duri

207 Food deprivation which enhances aPVN GAL produces a mark

208 other, naturally occurring stressors such as food deprivation, which is being exacerbated by global w

209 Food deprivation, which transiently favors feeding over

210 /DA and HVA/DA ratios were normalized by age/food-deprivation while that of 3MT/DA was not.

211 ice, otherwise apparently normal, respond to food deprivation with markedly reduced reflex hyperphagi

212 crose palatability induced by an increase in food deprivation without affecting the performance of su