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1 role of pressure in pathogenesis of diabetic foot ulcers".
2  as it relates to the occurrence of diabetic foot ulcer.
3 icient to prevent the occurrence of diabetic foot ulcer.
4 iological deficit in the nonhealing diabetic foot ulcer.
5 und healing deficiency, typified by diabetic foot ulcer.
6 serts to diabetic patients with a history of foot ulcer.
7 dicare patients hospitalized with a diabetic foot ulcer.
8 ry second, a person with diabetes develops a foot ulcer.
9  nonhealing, compared with healing, diabetic foot ulcers.
10 ove-ankle) leg amputations owing to diabetic foot ulcers.
11 ection are first-line therapies for diabetic foot ulcers.
12 y care are first-line therapies for diabetic foot ulcers.
13 person-years in people with diabetes without foot ulcers.
14 r local galectin-3 therapy to treat diabetic foot ulcers.
15 tic-resistant clinical strains from diabetic foot ulcers.
16  as lumbar degenerative disease and diabetic foot ulcers.
17 MRSA into the care of diabetic patients with foot ulcers.
18 ure in patients with neuroischaemic diabetic foot ulcers.
19  local treatment for neuroischaemic diabetic foot ulcers.
20 diabetes are attributed to deep infection of foot ulcers.
21 phropathy, 4.5% for neuropathy, and 5.7% for foot ulcers.
22  maintained in a commensal state in diabetic foot ulcers.
23 with neuroischaemic (NI) or neuropathic (NP) foot ulcers.
24 lammation predisposing to polyneuropathy and foot ulcers.
25 to develop therapies for nonhealing diabetic foot ulcers.
26 and effective treatment for chronic diabetic foot ulcers.
27 uction studies and occurrence of new plantar foot ulcers.
28 ion and mortality for patients with diabetic foot ulcers.
29                    Most wounds were diabetic foot ulcers (19.0%), venous leg ulcers (26.1%), and pres
30 namics of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabete
31                Current treatment options for foot ulcers, a serious and prevalent complication of dia
32                                     Diabetic foot ulcers affect approximately 18.6 million people wor
33 ality of life, and in diabetic patients with foot ulcers, amputation of the affected limb in 25% of p
34 te model showed these associations: diabetic foot ulcer and increased minor amputation; venous ulcer
35 sease (PAD) in diabetes may lead to diabetic foot ulcer and lower-extremities amputation.
36 conomic status have higher rates of diabetic foot ulcer and subsequent amputation compared with White
37               Because patients with diabetic foot ulcers and impaired wound healing have reduced expr
38 roving diabetic management and prevention of foot ulcers and neurogenic arthropathy.
39 viduals with a high ABI have higher odds for foot ulcers and neuropathy, as well as lower scores on s
40            Advanced DPN can lead to diabetic foot ulcers and non-healing wounds that often necessitat
41                                     Diabetic foot ulcers and other chronic wounds with impaired heali
42 rsors-based therapies of nonhealing diabetic foot ulcers and other pathologies with impaired angiogen
43 crease amputations in patients with diabetic foot ulcers and possibly accelerate closure of venous ul
44 to chronic wounds on earth, such as diabetic foot ulcers and venous leg ulcers, wounds inflicted in s
45 ssification of Diseases codes for rest pain, foot ulcers, and gangrene.
46                                              Foot ulcers are a common and feared complication for peo
47                                              Foot ulcers are more likely to be of neuropathic origin,
48                                     Diabetic foot ulcers are serious and challenging wounds associate
49                                     Diabetic foot ulcers are severe diabetic complications, and promo
50 tant to remember that rates of recurrence of foot ulcers are very high, being greater than 50% after
51                 People with diabetes develop foot ulcers because of neuropathy (sensory, motor, and a
52 tion for people with diabetes because 20% of foot ulcers become infected and lead to a lower extremit
53 ccur in various contexts, including diabetic foot ulcers, burns, and surgical sites.
54 tic neuropathy patients developed a diabetic foot ulcer by the one-year follow-up.
55                           Using the Diabetic Foot Ulcer Challenge 2022 test set, when compared to the
56 ntracellular infection by S. aureus diabetic foot ulcer clinical isolate and S. aureus USA300.
57                  Differentiation of diabetic foot ulcer-derived induced pluripotent stem cells reveal
58    Our study offers a practical solution for foot ulcer detection, particularly in situations where e
59 wounds i.e. venous leg ulcer (VLU), diabetic foot ulcer (DFU) and pressure ulcer (PRU).
60                                     Diabetic foot ulcer (DFU) infections are challenging.
61                                     Diabetic foot ulcer (DFU) is a problem worldwide, and prevention
62  biological properties is vital for diabetic foot ulcer (DFU) treatment.
63 nization to invasion is critical in diabetic foot ulcer (DFU).
64  study of patients with neuropathic diabetic foot ulcers (DFU).
65 d insights for reducing the risk of diabetic foot ulcers (DFU).
66                                     Diabetic foot ulcers (DFUs) and subsequent amputation incur enorm
67 g over time, fueling an epidemic of diabetic foot ulcers (DFUs) and subsequent risk of leg amputation
68                          Nonhealing diabetic foot ulcers (DFUs) are a common and costly complication
69                                     Diabetic foot ulcers (DFUs) are a common complication of diabetes
70                                     Diabetic foot ulcers (DFUs) are a debilitating complication of di
71                                     Diabetic foot ulcers (DFUs) are a leading cause of high morbidity
72                                     Diabetic foot ulcers (DFUs) are a life-threatening disease that o
73                                     Diabetic foot ulcers (DFUs) are a major complication of diabetes,
74                                     Diabetic foot ulcers (DFUs) are a severe complication of diabetes
75                          Nonhealing diabetic foot ulcers (DFUs) are characterized by low-grade chroni
76  air insole on reducing the risk of diabetic foot ulcers (DFUs) in healthy individuals through microc
77 , the epidermis surrounding chronic diabetic foot ulcers (DFUs) is clinically hypertrophic and nonmig
78                                     Diabetic foot ulcers (DFUs) represent a clinically burdensome and
79                                     Diabetic foot ulcers (DFUs) threaten limbs and prompt hospitaliza
80                                     Diabetic foot ulcers (DFUs), a leading cause of amputations, affe
81 hysiology and optimal management of diabetic foot ulcers (DFUs), a leading complication associated wi
82                                     Diabetic foot ulcers (DFUs), a life-threatening complication of d
83 ic, nonhealing skin wounds, such as diabetic foot ulcers (DFUs), are common in patients with type 2 d
84  its potential role in treatment of diabetic foot ulcers (DFUs).
85 utic approach for the management of diabetic foot ulcers (DFUs).
86 effect of NETosis on the healing of diabetic foot ulcers (DFUs).
87 otential of S. aureus isolated from diabetic foot ulcers (DFUs).
88 the development and failure to heal diabetic foot ulcers (DFUs).
89 mmation are hallmarks of nonhealing diabetic foot ulcers (DFUs).
90  people worldwide are affected by a diabetic foot ulcer each year, including 1.6 million people in th
91 icantly predicted the occurrence of diabetic foot ulcer, even after controlling for demographic varia
92  to new foot complication, including chronic foot ulcer, foot infection, or foot amputation.
93                Critical limb ischemia (CLI), foot ulcers, former amputation, and impaired regeneratio
94  increases the chance of developing diabetic foot ulcers, gangrene and even lower limb amputation.
95 s and a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (
96 care behavior on the development of diabetic foot ulcer has received little empirical investigation.
97                                     Diabetic foot ulcers have been neglected in health-care research
98         Approximately 30% to 40% of diabetic foot ulcers heal at 12 weeks, and recurrence after heali
99 pair, which contributes to impaired diabetic foot ulcer healing.
100 graphic variables and the number of diabetic foot ulcer hospitalizations, however, the effect was non
101 ors (neuropathy severity, number of diabetic foot ulcer hospitalizations, insulin treatment, and peri
102 antly associated with the risk of developing foot ulcers (HR=1.04, 95% CI=1.01-1.07, p=0.004).
103 utic potential for the treatment of diabetic foot ulcers if it is applied topically after the surgica
104 apeutic option for the treatment of diabetic foot ulcers if it is applied topically after the surgica
105 tated wound image dataset consisting of 1109 foot ulcer images from 889 patients to train and test th
106 ehavior predicted the occurrence of diabetic foot ulcers in diabetic patients with neuropathy.
107 ascia (PF) contributes to the development of foot ulcers in diabetics.
108  S. aureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophage, ROSA-l
109                    Management of neuropathic foot ulcers in patients with diabetes (DFUs) has changed
110 e base for many aspects of the management of foot ulcers in people with diabetes is weak, and good-qu
111 ial resistance patterns of infected diabetic foot ulcers in sub-Saharan Africa.
112 ative bacteria were associated with diabetic foot ulcers in sub-Saharan Africa.
113 in the prevention and management of diabetic foot ulcers, including studies that focus on off-loading
114  data on the regional prevalence of diabetic foot ulcer infecting bacteria and their antimicrobial re
115                                     Diabetic foot ulcer is a critical complication of diabetes, but t
116 rtality rate for individuals with a diabetic foot ulcer is approximately 30%, exceeding 70% for those
117  The mortality rate for people with diabetic foot ulcers is 231 deaths per 1000 person-years, compare
118 t, and while its pathogenic role in diabetic foot ulcers is difficult to establish, it may be a previ
119               Treatment for chronic diabetic foot ulcers is limited by the inability to simultaneousl
120 eviously showed that aspects of the diabetic foot ulcer microbiota were correlated with poor healing
121                                     Diabetic foot ulcer microbiota were found to exist in one of four
122 s advocate race as a risk marker of diabetic foot ulcer outcomes, likely reflecting underlying dispar
123  ABI group had significantly higher odds for foot ulcers (p < 0.005) and borderline associations with
124                       The number of diabetic foot ulcer patients is substantially increasing, with th
125                Importantly, in both diabetic foot ulcer patients, metagenomic sequencing identified p
126 al in a timely manner, for example, diabetic foot ulcers, pose a health, economic, and social problem
127 lling for demographic variables and diabetic foot ulcer predictors (neuropathy severity, number of di
128 rtant lessons for future studies on diabetic foot ulcer prevention.
129                                     Diabetic foot ulcers represent a significant source of morbidity
130                 When infection complicates a foot ulcer, the combination can be limb or life-threaten
131 th single-cell data from venous and diabetic foot ulcers uncovers a link between failed keratinocyte
132 in and as a possible participant in diabetic foot ulcers, we used a selective medium to culture both
133 bs from ten patients diagnosed with diabetic foot ulcers were collected and 32 clinical strains compr
134 r findings demonstrate that healing diabetic foot ulcers were significantly enriched with distinct fi
135 e-associated infections, as well as infected foot ulcers, which often lead to amputation.
136 ved wound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 weeks of t
137 ar has been implicated as a primary cause of foot ulcers, yet research is limited on the efficacy of

 
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