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1 oop of the mental nerve mesial to the mental foramen.
2 jor optic nerve blood vessels at the scleral foramen.
3 skull ossification and persistent calvarial foramen.
4 a mean of 11.4 mm from the greater palatine foramen.
5 facial nerve transection at the stylomastoid foramen.
6 sterior crista (PC) and the utriculosaccular foramen.
7 that eventually exited through the nutrient foramen.
8 e meninges and lining the anterior lacerated foramen.
9 to the SN, and the SN in the greater sciatic foramen.
10 induce blood vessels from a small root canal foramen.
11 e the location of the accessory infraorbital foramen (AIOF) with reference to accessible external lan
12 e right atrium may result in widening of the foramen and consequently, cause serious conditions due t
13 pect to verifying the position of the mental foramen and validating the presence of an anterior loop
14 ior medial pterygoid tubercle, mesial mental foramen, and narrow corpus place it closer to early mode
16 terior to, posterior to, or above the mental foramen; and prior to placing an implant anterior to the
23 heterozygous loss of Twist function causes a foramen in the skull vault similar to that caused by los
26 lip sensations are preventable if the mental foramen is located and this knowledge is employed when p
27 position when an anterior loop of the mental foramen is suspected of being present or if it is unclea
28 Ding, Fischer, and co-authors developed the foramen lacerum impingement of trigeminal nerve root (FL
29 syndrome, skull base fractures involving the foramen lacerum, neck soft tissue injury, or neurologica
31 blood flow (CBF) and ventriculostomy defect, foramen magnum (FM), and cerebral aqueduct CSF flow.
32 o assess cerebrospinal fluid movement at the foramen magnum and lateral ventricle during both regular
34 rdiac 1st and 2nd harmonics) at the level of foramen magnum during spontaneous versus yogic breathing
35 e displacement of the cerebellum through the foramen magnum into the spinal canal, is one of the most
42 h tonsils extending more than 5 mm below the foramen magnum were classified by the neurosurgeon as sy
43 death (all intracranial structures above the foramen magnum), cerebral death (all supratentorial stru
44 ntrol of the location and orientation of the foramen magnum, and changes in the breadth of the basioc
46 e length of the clivus, the AP length of the foramen magnum, the AP length of the posterior fossa, th
47 atient group, except of the AP length of the foramen magnum, were statistically significantly lower t
55 the position, number, and size of the mental foramen, mental nerve anatomy, and consequences of nerve
57 men that is deeper than the safety zone, the foramen must be probed to exclude the possibility that a
58 al nerve may be present mesial to the mental foramen needs to be considered before implant surgery to
65 on of the inferior alveolar nerve and mental foramen on panoramic and periapical films prior to impla
67 sensitivity of MR for diagnosing a sublabral foramen or Buford complex was 0.94 (47 of 50 patients, 9
68 and accuracy of MR for depicting a sublabral foramen or Buford complex were calculated along with 95%
69 ), and the presence of a posterior pneumatic foramen or fossa (absent in most tetanurans, but sporadi
70 , 0.95 to 1.21), atrial septal defect/patent foramen ovale (1.04, 0.88 to 1.24), neural tube defect (
72 m-type atrial septal defect (n=12) or patent foramen ovale (n=5) by a totally endoscopic approach, ut
73 ients 18 to 60 years of age who had a patent foramen ovale (PFO) and had had a cryptogenic ischemic s
75 evention of embolism in patients with patent foramen ovale (PFO) and otherwise unexplained ischemic s
76 ischemic attack presumably related to patent foramen ovale (PFO) are at risk for recurrent cerebrovas
79 The main randomized trials evaluating patent foramen ovale (PFO) closure after a presumed PFO-associa
80 dence of atrial arrhythmia (AA) after patent foramen ovale (PFO) closure and whether this complicatio
82 patients who underwent transcatheter patent foramen ovale (PFO) closure for paradoxical embolism.
83 ical devices may be used "off-label." Patent foramen ovale (PFO) closure is indicated to reduce recur
87 ional studies have shown percutaneous patent foramen ovale (PFO) closure to be a safe means of reduci
89 served in up to 25% of patients after patent foramen ovale (PFO) closure, but its long-term influence
90 eft atrial appendage (LAA) occlusion, patent foramen ovale (PFO) closure, transcatheter aortic valve
93 close atrial septal defects (ASD) and patent foramen ovale (PFO) has a number of limitations, includi
96 ss the risk of ischemic stroke from a patent foramen ovale (PFO) in the multiethnic prospective cohor
104 astomoses (IPAVA) in humans without a patent foramen ovale (PFO) is negatively correlated with pulmon
106 Percutaneous transcatheter closure of patent foramen ovale (PFO) is used as an alternative to surgery
108 ectiveness of percutaneous closure of patent foramen ovale (PFO) plus medical therapy versus medical
115 with atrial septal aneurysm (SA) and patent foramen ovale (PFO), and to determine the efficacy of me
116 to determine the association between patent foramen ovale (PFO), atrial septal aneurysm (ASA), and s
117 headache symptoms in patients with a patent foramen ovale (PFO), both of which conditions are highly
118 milder form of atrial septal defect, patent foramen ovale (PFO), exists in about one-quarter of the
119 to evaluate the relationship between patent foramen ovale (PFO), ischemic stroke, and subclinical ce
120 Controversy surrounds the issue of patent foramen ovale (PFO), stroke, and secondary prevention st
125 ifty-four (86%) had effective closure of the foramen ovale (trivial or no residual shunt by echocardi
126 cted symptomatic patients, closure of patent foramen ovale after cryptogenic stroke, treatment of ins
132 aphy identified three patients with a patent foramen ovale and right-to-left shunt flow while breathi
133 cryptogenic embolism in patients with patent foramen ovale are administration of antithrombotic medic
135 e closure (9 secundum ASDs and 4 with patent foramen ovale associated with a cerebral vascular accide
140 Evidence from trials suggests that patent foramen ovale closure is superior to medical therapy alo
141 pparent increased risk for concurrent patent foramen ovale closure or mitral or tricuspid repair.
142 Assessment of Flecainide to Lower the Patent Foramen Ovale Closure Risk of Atrial Fibrillation or Tac
143 patients who underwent transcatheter patent foramen ovale closure to prevent recurrent cerebrovascul
144 alysis demonstrated that simultaneous patent foramen ovale closure was not associated with an increas
146 left atrial appendage occlusion, and patent foramen ovale closure, profoundly differ with respect to
151 a demonstration of both technologies, patent foramen ovale creation and closure was performed in a sw
152 mainly the adult population, such as patent foramen ovale device closure and closure of postinfarct
153 aware of these variations when accessing the foramen ovale during trigeminal neuralgia interventions.
154 ients, all of whom underwent recordings with foramen ovale electrodes and scalp electroencephalogram.
155 mesial temporal activity using intracranial foramen ovale electrodes in two patients with Alzheimer'
159 benefit associated with closure of a patent foramen ovale in adults who had had a cryptogenic ischem
161 th warfarin or aspirin as part of the Patent Foramen Ovale in Cryptogenic Stroke Study (PICSS), based
162 s (the PRIMA [Percutaneous Closure of Patent Foramen Ovale in Migraine With Aura] and PREMIUM [Prospe
163 hocardiograms in 11 patients showed a patent foramen ovale in one patient but no systemic source of e
164 that occur in the major veins and across the foramen ovale in the circulation of the fetal lamb.
171 e (11%), aortic valve procedure (9%), patent foramen ovale or atrial septal defect closure (23%), car
176 compression sickness with concomitant patent foramen ovale that was successfully closed contrary to s
177 closure of an atrial septal defect or patent foramen ovale to assess how the procedure affected migra
181 cept for 19.6 +/- 2.3% of the cycle when the foramen ovale was closed during atrial contraction.
184 theter closure or medical therapy for patent foramen ovale were required to include at least 10 patie
186 ly assigned to undergo closure of the patent foramen ovale with the Amplatzer PFO Occluder or to rece
187 f the ten patients undergoing semi-invasive (foramen ovale) electrode monitoring reveals that for at
188 d in 47% (including 17% with isolated patent foramen ovale), and 11% had undetermined stroke etiology
191 (TAA), 88% had left-to-right flow across the foramen ovale, 91% had monophasic mitral inflow, and 94%
193 patients had simultaneous closure of patent foramen ovale, and 19 patients had other various cardiac
194 ne prevalence of atrial fibrillation, patent foramen ovale, and arterial stenoses were also similar f
196 persistent pulmonary hypertension, a patent foramen ovale, and free-floating right-heart thrombus ar
197 ion of the inferior vena cava, right atrium, foramen ovale, and left atrium with a guidewire and 1.8F
198 sease, presence of left common trunk, patent foramen ovale, and time for atrial fibrillation diagnosi
199 ccult paroxysmal atrial fibrillation, patent foramen ovale, aortic arch atherosclerosis, atrial cardi
200 has been reported in patients with a patent foramen ovale, carcinoid tumor of the lung, and active c
201 h cryptogenic stroke or TIA who had a patent foramen ovale, closure with a device did not offer a gre
203 en pulmonary embolism coexists with a patent foramen ovale, increased pressure in the right atrium ma
204 rmal LV length, reversed flow in the TAA and foramen ovale, monophasic mitral inflow, and LV dysfunct
205 defect, secundum atrial septal defect/patent foramen ovale, neural tube defect, clubfoot, and oral cl
206 ing of intracranial atheroma, patent cardiac foramen ovale, or elevated levels of antiphospholipid an
207 re of secundum atrial septal defects, patent foramen ovale, patent ductus arteriosus, stent placement
208 patients with cryptogenic stroke and patent foramen ovale-related ischemic stroke to receive either
229 and utilizing the rib as a conduit into the foramen provided an advantage in patients with osteopeni
230 f the nose and the lower wall of the orbital foramen provided comparable results to the standard tech
231 nerve; surgical corroboration of the mental foramen's position when an anterior loop of the mental f
233 s (FMNs) survive axotomy at the stylomastoid foramen (SMF), whereas, before postnatal day 15 (P15), d
235 hylogenetic analyses reveal an autapomorphic foramen that distinguishes it from all other troodontids
236 r of neurons with somata near the esophageal foramen that gave rise to arborizations in the protocere
237 to placing an implant anterior to the mental foramen that is deeper than the safety zone, the foramen
238 ng the cranium through bony canals and large foramens; that central neurons receiving nociceptive inf
239 trally as they progressed toward the scleral foramen (the mouse does not have a lamina cribrosa), and
240 Cdh23 is expressed in the urticulo-saccular foramen,the ductus reuniens, and Reissner's membrane, su
241 portion of the orbit primarily via the optic foramen; they travel only short distances and end blindl
242 noncontact vagal stimulation at the jugular foramen, through the internal jugular veins (extracardia
243 lear how much bone is present coronal to the foramen to establish a zone of safety (in millimeters) f
245 gh the presence of a rounded and conspicuous foramen vasculare distale and the trochlea metatarsi II
246 asal process, a large and deep neurovascular foramen within the perinarial fossa, and a deep perinari