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1 of the mandible and reduced the diameters of foramina.
2 gical symptoms due to obstruction of cranial foramina.
3 shaped rods inserted into the intervertebral foramina.
4 a and maxilla where they are associated with foramina.
8 e spinal cord, vertebral bodies, both neural foramina, and anterior paravertebral soft tissue were si
11 s of the lateral recesses (in mm) and in the foramina (area in mm(2) and smallest diameter in mm) obt
13 ls of the nasal cavity, and the infraorbital foramina change consistently with maxillary transverse g
16 e also register for the first time pneumatic foramina (fo) and fossae connecting camellate structures
18 inear distances between the greater palatine foramina (GPFd), the lateral walls of the nasal cavity (
20 this hypothesis by examining lateral palatal foramina in both filter-feeding and non-filter-feeding c
22 ponsible for multiple exostoses and parietal foramina in PSS, the identity of the gene(s) associated
24 elop postnatally, appearing first around the foramina in the basal parts of the skull and spinal cana
25 haracterized by multiple exostoses, parietal foramina, intellectual disability (ID), and craniofacial
28 rom apes in the lateral shift of the carotid foramina, mediolateral abbreviation of the lateral tympa
29 ed taxa, suggesting that the lateral palatal foramina observed in extant mysticetes are homologous to
31 the cross-sectional areas of the transverse foramina of the atlas and the left carotid canal in StW
39 l were greater than 99%, with reduced neural foramina recall for the DL model (84.5%) and radiologist
41 n show decreases in the lateral recesses and foramina related to the predominance of symptoms while s
42 n show decreases in the lateral recesses and foramina related to the predominance of symptoms while s
44 with deletion of 11p11.2 who have biparietal foramina, support the contention that ALX4 is a candidat
45 t that the positions of the greater palatine foramina, the lateral walls of the nasal cavity, and the