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1 sion associated with the haplotype of the F7 founder line.
2 lines derived from eight genetically diverse founder lines.
3 although this was more variable in different founder lines.
4 orter gene and used to create six transgenic founder lines.
5 variable levels of expression in individual founder lines.
6 cDNA and utilized this transgene to generate founder lines.
7 tissues, including the thymus in some of the founder lines.
8 GIC) population for lettuce using 16 diverse founder lines.
9 ric bud causes reduced branching in multiple founder lines.
10 lies; however, allelic effects differ across founder lines.
12 tween 3- and 50-fold higher depending on the founder line, and this occurred within 24-48 h after dos
13 rsensitive sites, is expressed in only a few founder lines, and expression is dysregulated in CD8(+)
14 sed across the 26 nested association mapping founder lines, and this variability positively correlate
15 derived from two independent TetOp-DeltaFosB founder lines but not in single NSE-tTA transgenic contr
32 2 generations of free recombination among 60 founder lines, following modification of the mating syst
33 DNA integrations in our genomic engineering founder lines for the purpose of generating complex knoc
34 ere human CD8 beta+ varied, depending on the founder line, from 4 to 88%, whereas the percentage amon
36 ckcrossed homozygous transgenic animals from founder line I had plasma PAI-1 levels of 23+/-12 ng/mL.
39 in thymic lymphomas from multiple transgenic founder lines, including one line in which 15 of 15 prim
40 is produced in nonlymphoid tissues from all founder lines, including tissues that do not express end
43 6 -> Ser) was used to generate 15 transgenic founder lines of hA-IImon mice, that contained plasma hA
44 tively transferred into different transgenic founder lines of mice expressing HA as a peripheral self
46 ge number of contrasting RSA traits among 25 founder lines of the maize nested association mapping po
47 and B cells from three independently derived founder lines of transgenic mice bearing a recombined he
48 hematopoietic disease, we developed separate founder lines of transgenic mice, with the tetracycline
50 In contrast, in the highest overexpressing founder line, TgB, alphabeta T-cell development was dist
52 We generated two independent transgenic mice founder lines that express a stable variant of active hu
55 itively correlate with SPS in multiple MAGIC founder lines under field conditions as well as in trans
56 evealed that one 570 kb YAC, in two separate founder lines, was associated with distinct learning def