ライフサイエンスコーパス: freundii

1 ogenes, Morganella morganii, and Citrobacter freundii.

2 rs specific for the ampC gene of Citrobacter freundii.

3 es from Enterobacter cloacae and Citrobacter freundii.

4 ia coli, Klebsiella oxytoca, and Citrobacter freundii.

5 , Klebsiella oxytoca (2.7%), and Citrobacter freundii (2.0%).

6 Citrobacter koseri (22 [46%] of 48) and C freundii (20 [42%]) were the predominant CPC species.

7 s aeruginosa (40% identity), and Citrobacter freundii (38% identity).

8 abilis, 10 Citrobacter koseri, 9 Citrobacter freundii, 8 Klebsiella oxytoca, 5 Klebsiella aerogenes,

9 rnatants from Proteus mirabilis, Citrobacter freundii and Enterobacter agglomerans [cyclo(DeltaAla-L-

10 (HBMEC) to study the interaction between C. freundii and HBMEC.

11 carbapenem-resistant isolates of Citrobacter freundii and Klebsiella oxytoca recovered from different

12 ndent glycerol dehydratases from Citrobacter freundii and Klebsiella pneumoniae.

13 ects in heme biosynthesis for E. coli and C. freundii and lipoic acid biosynthesis in E. hormaachei w

14 leotide variants in the E. coli SCV, 5 in C. freundii, and 8 in E. hormaechei.

15 Gram-negative Escherichia coli, Citrobacter freundii, and Enterobacter aerogenes, as well as Gram-po

16 co-isolates of Escherichia coli, Citrobacter freundii, and Enterobacter hormaechei.

17 roduction (Enterobacter cloacae, Citrobacter freundii, and Klebsiella aerogenes only) or for third-ge

18 ed by Salmonella enterica Typhi, Citrobacter freundii, and some soil bacteria belonging to the Burkho

19 ription from the tpl promoter of Citrobacter freundii ATCC 29063 (C. braakii).

20 Citrobacter rodentium (formerly Citrobacter freundii biotype 4280 and Citrobacter genomospecies 9) w

21 Citrobacter rodentium (formally Citrobacter freundii biotype 4280) is a highly infectious pathogen t

22 umans and Citrobacter rodentium (formerly C. freundii biotype 4280)-mediated transmissible colonic hy

23 e have previously shown that the Citrobacter freundii BMC associated with 1,2-propanediol utilization

24 regation was time dependent and seen with C. freundii but not with noninvasive E. coli HB101 and meni

25 Comparative analysis found C freundii carried significantly higher median numbers of

26 rtant determinants of the pathogenesis of C. freundii causing meningitis and brain abscess may relate

27 ation of the intestinal tract by Citrobacter freundii, Clostridium species, Enterobacter cloacae, Ent

28 Neither the class C Citrobacter freundii CMY-2 AmpC beta-lactamase nor the class A TEM-1

29 a number of species (Acinetobacter spp., C. freundii, E. aerogenes, K. pneumoniae, P. aeruginosa, an

30 SAs (AmpC beta-lactamases of M. morganii, C. freundii, E. coli, and E. cloacae).

31 The tpl gene of Citrobacter freundii encodes an enzyme that catalyzes the conversion

32 , such as the E. cloacae P99 and Citrobacter freundii enzymes, the ES GC1 beta-lactamase is able to r

33 gregated after HBMEC came in contact with C. freundii; furthermore, the microtubule aggregation was t

34 All carbapenemase-producing Citrobacter freundii genomes from the NCBI GenBank database were inc

35 tree of 726 global carbapenemase-producing C freundii genomes showed that ST22 (227 [31.3%]) represen

36 B-2), Bacillus subtilis (GFB-3), Citrobacter freundii (GFB-4), and P. aeruginosa (GFB-5) were isolate

37 f the phosphonate with both ES GC1 and WT C. freundii GN346 beta-lactamases have been determined to h

38 lla oxytoca, Citrobacter koseri, Citrobacter freundii group, Enterobacter spp., and Serratia marcesce

39 tyrosine phenol-lyase (TPL) from Citrobacter freundii have been examined.

40 ies such as Escherichia coli and Citrobacter freundii in real-time PCR assays.

41 The increased rate of Citrobacter freundii infections is a public health concern and there

42 Tyrosine phenol-lyase (TPL) from Citrobacter freundii is a pyridoxal 5'-phosphate (PLP)-dependent enz

43 ast to other meningitis-causing bacteria, C. freundii is able to replicate within HBMEC.

44 Tyrosine phenol-lyase (TPL) from Citrobacter freundii is activated about 30-fold by monovalent cation

45 In this study, we show that C. freundii is capable of invading and trancytosing HBMEC i

46 Tyrosine phenol-lyase (TPL) from Citrobacter freundii is dependent on monovalent cations, K(+) or NH(

47 anediol utilization enzymes from Citrobacter freundii is fully functional when cloned in Escherichia

48 interactions between AmpR (from Citrobacter freundii), its DNA operator, and repressor UDP-MurNAc-pe

49 was also observed in strains of Citrobacter freundii, Klebsiella pneumoniae, Enterobacter agglomeran

50 8), Serratia marcescens (n = 6), Citrobacter freundii (n = 4), and Klebsiella aerogenes (n = 2).

51 reement rates were >90% with exception of C. freundii, S. lugdunensis, E. faecalis, S. anginosus and

52 carried out to characterise the high-risk C freundii sequence type (ST) 22 clone.

53 with PS biodegradation including Citrobacter freundii, Serratia marcescens, and Klebsiella aerogenes.

54 ion structure of CPC species and highlight C freundii ST22 as a prominent high-risk international clo

55 ty of these ligands for E. coli, Citrobacter freundii, Staphylococcus epidermidis were 100%, 2.6% and

56 eus, Pseudomonas aeruginosa, and Citrobacter freundii to ensure the species specificity of the select

57 eus, Pseudomonas aeruginosa, and Citrobacter freundii, to ensure the species-specificity of the selec

58 -lyase and to wild type and Y71F Citrobacter freundii tyrosine phenol-lyase was investigated in the c

59 Invasion of HBMEC by C. freundii was determined to be dependent on microfilament

60 Among the 20 CNSC isolates, Citrobacter freundii was the predominant species identified (60%).

61 acteria (Salmonella typhimurium, Citrobacter freundii, Yersinia enterocolitica, Serratia marcescens,