ライフサイエンスコーパス: frontal

1 n in post-amphetamine [(11)C]CIMBI-36 BP(ND)(frontal).

2 DeltaBP(ND)(frontal) = 1 - (BP(ND)(frontal) post-dose/BP(ND)(frontal

3 areas through parietal multisensory areas to frontal action planning areas.

4 8 +/- 0.5 m d(-1)) and resulted in a glacier frontal advance of 1495 +/- 47 m.

5 toms and attenuated resting-state posterior->frontal alpha connectivity, which were both correlated w

6 (male and female), we find that prestimulus frontal alpha power correlates with the tendency to resp

7 alpha in the encoding of sensory information frontal alpha reflects complementary mechanisms influenc

8 )C]UCB-J V(T) was significantly lower in the frontal and anterior cingulate cortices in schizophrenia

9 We find frontal and anterior temporal regions involved in social

10 Archetypal MPDs (60% of MPDs) had increased frontal and decreased posterior ALFF, and decreased cort

11 associated with static brain connectivity in frontal and default mode networks, whereas age showed po

12 ad significantly lower MMN amplitudes at the frontal and frontocentral electrodes than the healthy co

13 2) Atypical MPDs (40% of MPDs) had decreased frontal and increased posterior ALFF with no associated

14 Our findings emphasize the role of the frontal and limbic areas in the aetiology of psychotic s

15 ol children showed increased connectivity in frontal and limbic hubs over time, children with PME sho

16 ith higher dementia risk scores in parietal, frontal and medial occipital cortices, (3) with higher U

17 nular pyramidal neurons in tissue from human frontal and occipital lobes, in 11 cases with (M(age) =

18 man brains more efficient cross talk between frontal and other high-order associative areas of the te

19 We found that although increases in frontal and parietal activity were associated with incre

20 we investigated how delay-period activity in frontal and parietal cortex, which is known to correlate

21 ention network includes both cortical (e.g., frontal and parietal cortices) and subcortical (e.g., th

22 ad regions of the cortex, most pronounced in frontal and parietal cortices; and (c) a significant dis

23 alculated within-subject correlation between frontal and parietal delay-period activity and whole-tri

24 ft, diffusion explained the most variance in frontal and parietal delay-period activity.

25 y associated with higher MT in the bilateral frontal and parietal lobes.

26 c ejection fraction and GMD across the whole frontal and parietal medial cortex reflecting the conseq

27 direction of the remembered stimulus, as did frontal and parietal regions of the dorsal attention net

28 E epsilon4+group (left cingulate and lateral frontal and parietal regions p<0.01, threshold-free clus

29 cement in NREM delta power especially in the frontal and parietal regions, and (iii) progressive incr

30 distant cortical areas, particularly in the frontal and parietal regions.

31 linked with functional connectivity between frontal and perceptual regions and suggest a compensator

32 ing patterns of functional imbalance between frontal and posterior brain regions, as compared to HC:

33 came unconscious, but later encompassed both frontal and posterior cortex when subjects were more dee

34 developed in recent years over the roles of frontal and posterior cortices in mediating consciousnes

35 early encoding processes that engage lateral frontal and sensory regions to successfully encode event

36 Much is known about frontal and striatal processes that support cognitive co

37 tory-phonological network involving inferior frontal and supramarginal regions; a ventral semantic ne

38 ad smaller changes in oxy-haemoglobin in the frontal and temporal cortices than HCs.

39 nd in ASD compared to TD participants in the frontal and temporal lobes and several sub-lobar regions

40 with key regions of tau accumulation in the frontal and temporal lobes for all phenotypes and key re

41 sure forms by the relative overgrowth of the frontal and temporal lobes over the insula, correspondin

42 acterized by progressive degeneration in the frontal and temporal lobes.

43 Additionally, the degree of left frontal and temporal pole thinning, and clinical score a

44 ystem, and changes in oxy-haemoglobin in the frontal and temporal regions were quantified.

45 that a large set of regions in the parietal, frontal, and insular cortices shows increases in 2-4 Hz

46 ulting behavioral adjustments in dorsomedial frontal, anterior cingulate, and orbitofrontal cortex.

47 men (vs women) had any opacification in the frontal, anterior ethmoid, and sphenoid sinuses.

48 tern showed volume loss of the right ventral frontal area and the left temporal lobe, which represent

49 matter microstructure might be more focal on frontal areas of the brain, as opposed to global differe

50 In contrast, beta-band power within medial frontal areas was selectively attenuated when participan

51 inhibition can be rapidly re-instantiated by frontal areas when movements have to be rapidly cancelle

52 tion and increased beta-bursting over medial frontal areas with movement cancellation.

53 erconnecting posterior temporal and inferior frontal areas.

54 ccompanied by beta-power increases over (pre)frontal areas.

55 Centrality of cingulate and orbito-frontal as well as other brain areas was associated with

56 ing white matter tracts corresponding to the frontal aslant tract and the anterior segment of the arc

57 Test (FERT); Mini-Mental State Examination; Frontal Assessment Battery; lexical fluency (F-A-S), cat

58 r 5 pyramidal neurons in the visual (V1) and frontal association cortex (FrA) of 1-month-old mice.

59 ions surviving Bonferroni corrections in the frontal (B = -10.85, 95% CI -17.91, -3.79, p < 0.0125) a

60 tal) = 1 - (BP(ND)(frontal) post-dose/BP(ND)(frontal) baseline) was used as an index of 5-HT release.

61 ping latencies, motor evoked potentials, and frontal beta power (13-20 Hz) did not differ between the

62 is required, whereas the reduction in medial frontal beta-band activity relates to the inhibition of

63 While much is known about how frontal brain regions facilitate cognitive control, less

64 usion, a distributed network of temporal and frontal brain regions supports gamma phase synchronizati

65 nd neocortical (inferior temporal and middle frontal) brain volumes determined using longitudinal Fre

66 ked to decreased alpha oscillation in medial frontal channels.

67 is retrospective study, 22 960 de-identified frontal chest radiographs from 11 153 patients (average

68 x, opacity, nodule or mass, and fracture) on frontal chest radiographs.

69 es in predispositional insula-based striatal-frontal circuits, highlighting the circuit's potential a

70 fiscal interventions decreased the amount of frontal cognitive control required to buy healthy foods

71 Larger decreases in amygdala-frontal connectivity and insula-parietal connectivity we

72 Amygdala-frontal connectivity mediated the relationship between t

73 Amygdala-frontal connectivity was analysed using a whole-brain se

74 atory activity (in both power and posterior->frontal connectivity), given its role in sensory cortica

75 humans is associated with increased extended frontal connectivity, allowing human brains more efficie

76 ss populations of neurons in the dorsomedial frontal cortex (DMFC), DMFC-projecting neurons in the ve

77 This process depends on the medial frontal cortex (MFC) and the medial temporal lobe, but i

78 Influential theories of Medial Frontal Cortex (MFC) function suggest that the MFC regis

79 ise, relative to their precision in superior frontal cortex (replicated across studies, combined n =

80 this cognitive impairment, but not increased frontal cortex 5-HT(2A)R density or psychedelic-induced

81 , including Ruminococcaceae, correlated with frontal cortex 5-HT(2A)R density.

82 Damage primarily to inferior frontal cortex affected the production of syntactically

83 me comparative studies link this to a larger frontal cortex and even larger frontal white matter in h

84 DNA methylation of Th and Bdnf genes in the frontal cortex and hippocampus.

85 s of rodents revealed that FXG expression in frontal cortex and olfactory bulb followed consistent pa

86 odulation pattern that began earliest in the frontal cortex and subsequently flowed downstream to the

87 The frontal cortex and temporal lobes together regulate comp

88 The frontal cortex binding potential (BP(ND)(frontal)) was c

89 HIV infection associates with reduced brain frontal cortex expression of the antioxidant/anti-inflam

90 APOE epsilon4, we examined the dorsolateral frontal cortex from deceased participants within a commu

91 The medial frontal cortex has been linked to voluntary action, but

92 Neuronal oscillations in the frontal cortex have been hypothesized to play a role in

93 ecific cortical thickness differences in the frontal cortex in adults, support previous work emphasiz

94 that C4-dependent circuit dysfunction in the frontal cortex leads to decreased social interactions in

95 nd prevented increases in Csf1r and Cd11b in frontal cortex microglia following CUS.

96 that truncated STMN2 RNA was elevated in the frontal cortex of a cohort of patients with FTLD-TDP but

97 naptic expression of uPA is decreased in the frontal cortex of AD brains and 5xFAD mice, and uPA trea

98 e profiling of DNA hydroxymethylation of the frontal cortex of AD patients from China, emphasizing an

99 -to-process length ratio was observed in the frontal cortex of children and young adults with Down sy

100 ontribute to movement cancellation in medial frontal cortex of macaque monkeys.

101 phenotype previously observed in postmortem frontal cortex of schizophrenic subjects.

102 ntracellular localization of mGluR2 in mouse frontal cortex pyramidal neurons.

103 us during pregnancy induced up-regulation of frontal cortex serotonin 5-HT(2A) receptor (5-HT(2A)R) d

104 -wide profiles of both 5mC and 5hmC in human frontal cortex tissues from late-onset Chinese AD patien

105 te long-range inputs with local circuitry in frontal cortex to implement top-down attentional control

106 hoices are preferentially represented in the frontal cortex when required.

107 verity, to functional connectivity of orbito-frontal cortex with caudate nucleus and to structural ch

108 nding of increased beta-bursting over medial frontal cortex with movement cancellation in humans is d

109 n to anterior cingulate cortex within medial frontal cortex, a group of subcortical structures includ

110 he supplementary motor area, superior medial frontal cortex, and putamen-brain circuits respectively

111 derived from the substantia nigra, putamen, frontal cortex, and white matter, and were all significa

112 es including being in eQTLs in blood and the frontal cortex, CpG islands and shores, and exons.

113 enriched circRNA abundantly expressed in the frontal cortex, derived from Homer protein homolog 1 (HO

114 Brains were dissected into frontal cortex, hippocampus, cerebellum, and brainstem.

115 f amygdala, basal-ganglia, thalamus, orbital frontal cortex, inferior frontal gyrus and dorsomedial p

116 by the basal ganglia, extended amygdala, and frontal cortex, respectively.

117 ision-making, which in mammals relies on the frontal cortex, specifically the orbitofrontal cortex (O

118 al connectivity profiles of the ventromedial frontal cortex, temporoparietal junction, and precuneus.

119 totemporal junction, the lateral and orbital frontal cortex, the anterior insula, and mesiotemporal s

120 ivity for sound classes emerges first in the frontal cortex.

121 sitive, ubiquitin-positive aggregates in the frontal cortex.

122 mus (PeF), periaqueductal gray, amygdala and frontal cortex.

123 tex, orbitofrontal cortex, and left superior frontal cortex.

124 ustral neurons attenuated SW activity in the frontal cortex.

125 bilateral anterior insula and right inferior frontal cortex.

126 ctivation in S1, with minimal propagation to frontal cortex.

127 network that includes specialized patches in frontal cortex.

128 loid and global cognitive scores (eg, in the frontal cortex: beta = -0.13; 95% CI: -0.23, -0.02; P =

129 notype analyses in postmortem samples of two frontal cortical brain regions from 214 control subjects

130 The results identify a distributed frontal cortical circuit associated with behavioral flex

131 affected structure and activity in the same frontal cortical circuit.

132 onset of psychiatric disorders that display frontal cortical deficits.

133 n an ascending series of ferret auditory and frontal cortical fields, and the dynamics of this repres

134 urons, adult nigral dopaminergic neurons and frontal cortical neurons.

135 , and its pattern of coupling with the other frontal cortical regions was assessed.

136 or insula but were also found in three other frontal cortical regions: lateral orbitofrontal cortex (

137 terations in behavior, striatal BDNF levels, frontal cortical Th total percentage DNA methylation lev

138 sociations between obesity and lower temporo-frontal cortical thickness (maximum Cohen's d (left fusi

139 ion in primary somatosensory cortex (S1) and frontal cortices, including both the whisker region of p

140 points showed abnormal thinning in inferior frontal cortices.

141 s from CheXpert and transfer learning on 314 frontal CXRs from COVID-19 patients.

142 traced the onset of KMT2D and UTX mutant NCC frontal dysfunction to a stage of altered osteochondral

143 Ca(2+) spikes and describe resulting medial-frontal EEG on a male macaque monkey.

144 Results from time-frequency analysis in frontal electrodes showed an increase in theta amplitude

145 the same white or gray matter changes in the frontal-executive and corticolimbic circuitries as those

146 rain alterations in white and gray matter of frontal-executive and corticolimbic circuitries in five

147 ubcortical volumes and cortical thickness in frontal-executive and corticolimbic regions of interest

148 By contrast, the frontal eye field (FEF) and the intraparietal sulcus (IP

149 edial-dorsal nucleus of the thalamus (MD) to frontal eye field (FEF) carries such a CD for saccadic e

150 aparietal [LIP], posterior parietal area 7A, frontal eye field [FEF], and prefrontal cortex [PFC]) wh

151 e cues and the inferred causal structure the frontal eye field and the intraparietal sulcus form a ci

152 The smooth eye movement region of the frontal eye fields (FEF(SEM)) is a critical node in the

153 the lateral intraparietal area (LIP) and the frontal eye fields (FEF) exhibit persistent activity tha

154 S to examine cortical activations within the frontal eye fields (FEF) while initiating vergence eye m

155 the lateral intraparietal cortex (LIP), the frontal eye fields (FEF), and the superior colliculus (S

156 tion factor scores than pure ADNC, and lower frontal factor scores than pure LATE-NC.

157 increases in individual slow wave slope and frontal fast oscillation power.

158 Parieto-frontal growth, mainly between 7 and 13 years in FT chil

159 tic-naive patients, NAA was reduced in right frontal gyri (19 voxels, CCLAV = 0.05) and myo-inositol

160 d higher activation in the pSTS and inferior frontal gyri in CM than in AM.

161 al cortex (DLPFC) (i.e., middle and superior frontal gyri) and insula GMVs were associated with incre

162 tients, choline was increased in left middle frontal gyrus (18 voxels, CCLAV = 0.04).

163 evealed in the right superior and precentral frontal gyrus (BA 6) in the patient group, compared with

164 ns' over left anterior or posterior inferior frontal gyrus (IFG) in post-stroke patients with left te

165 and that impairments in a DPFC and inferior frontal gyrus (IFG) system may be important in suicide a

166 Inferior frontal gyrus (IFG), ventral premotor cortex (PMv) and i

167 a (p = 0.010), caudate (p = 0.008), inferior frontal gyrus (p = 0.004), and supramarginal gyrus (p =

168 ns (inferior temporal gyrus [ITG] and middle frontal gyrus [MFG]), we tested associations between bra

169 n the putamen, globus pailldus, and inferior frontal gyrus after donepezil treatment (p < 0.001).

170 itted and observed decisions in the inferior frontal gyrus and a dissociation in the anterior prefron

171 , thalamus, orbital frontal cortex, inferior frontal gyrus and dorsomedial prefrontal cortex (IFG, dm

172 d concepts evoked responses in left inferior frontal gyrus and left anterior temporal lobe that relat

173 tion field potentials from both the inferior frontal gyrus and subthalamic nucleus in 21 subjects.

174 A hyperdirect pathway between the inferior frontal gyrus and subthalamic nucleus is hypothesized to

175 perdirect pathway that connects the inferior frontal gyrus and the subthalamic nucleus.

176 nosynaptic connectivity between the inferior frontal gyrus and ventral subthalamic nucleus.

177 and glutamate concentrations in the inferior frontal gyrus correlated inversely with stop-signal reac

178 ternal-state-related variables in the medial frontal gyrus in both healthy subjects and smokers, sugg

179 e cortex, inferior frontal gyrus, and middle frontal gyrus was significantly increased in the BD grou

180 left dorsolateral prefrontal cortex/superior frontal gyrus, and left medial orbitofrontal cortex.

181 the left anterior cingulate cortex, inferior frontal gyrus, and middle frontal gyrus was significantl

182 yrus rectus, orbital segment of the superior frontal gyrus, and middle temporal gyrus).

183 dicted univariate responses in left inferior frontal gyrus, and multivariate responses in left anteri

184 resonance spectroscopy in the right inferior frontal gyrus, because of its strong association with re

185 tients versus controls in the right inferior frontal gyrus, but not the occipital lobe.

186 insula, right cerebellum, and right superior frontal gyrus, compared with HC.

187 In the left inferior frontal gyrus, however, prior knowledge leads to integra

188 rginal gyrus, right striatum, right inferior frontal gyrus, left thalamus, bilateral insula, right ce

189 conforming as children in the right superior frontal gyrus, right angular gyrus, right amygdala/parah

190 entary motor cortex and left medial superior frontal gyrus.

191 reflected in the activation of the inferior frontal gyrus.

192 cingulate, superior/mid-temporal, and middle frontal gyrus.

193 presenting to the emergency department for a frontal headache, eye pain, emesis, and lethargy.

194 e rated the competence of faces presented in frontal headshots.

195 ence suggested that treatment decreased left frontal inhibition of the left amygdala, and larger decr

196 ng occurs via shear dispersion, generated by frontal instabilities and episodic vertical mixing.

197 he influence of vision on the ability to map frontal, lateral and back space has not been investigate

198 hich the rodent claustrum is closely tied to frontal/limbic regions and plays a role in processes, su

199 han either olfactory epithelium (p = 0.071), frontal lobe (p < 0.001), or lungs (p = 0.037).

200 For every doubling in the inferior frontal lobe activation, angina frequency was increased

201 pect of the fissure moving anteriorly to the frontal lobe and laterally in the direction of the tempo

202 of 10 regions-of-interest including insula, frontal lobe and putamen in AD compared with controls, b

203 Network disorganization primarily involving frontal lobe and subcortical regions in nonpsychotic rel

204 glutamatergic and GABAergic deficits in the frontal lobe are potential targets for symptomatic drug

205 loid burden was significantly reduced in the frontal lobe compared to the placebo group.

206 the disproportionally large volume of human frontal lobe connections is accompanied by a reduction i

207 Abnormalities within frontal lobe gray and white matter of bipolar disorder (

208 the microcircuitry of agranular areas of the frontal lobe involved in cognitive control and responsib

209 The inferior frontal lobe is an important area of the brain involved

210 The frontal lobe is central to distinctive aspects of human

211 hich suggests that structural changes in the frontal lobe may have an indirect influence on age-relat

212 el tractography approach to demonstrate that frontal lobe networks, extending within and beyond the f

213 matter volume in nearly all networks, except frontal lobe networks, where differences in grey matter

214 identical analyses of EEG recorded over the frontal lobe of macaque monkeys (one male, one female) p

215 d morphological relationships primarily with frontal lobe regions, and their network-level alteration

216 Blood flow to the inferior frontal lobe was evaluated as a ratio compared with whol

217 ger resting-state absolute EEG powers in the frontal lobe were associated with wiser advising from th

218 entified a small cluster in the left lateral frontal lobe where children with greater upper-body musc

219 ancer and directly inoculated into the mouse frontal lobe, exhibit striking differences in proliferat

220 er volume changes in both hemispheres of the frontal lobe, which suggests that structural changes in

221 callosum, occipital, temporal, parietal and frontal lobes, and right hippocampus (p < 0.025 after fa

222 be networks, extending within and beyond the frontal lobes, occupy 66% of total brain white matter in

223 ory analysis moreover showed that the mesial frontal lobes, parahippocampal gyrus, and lateral tempor

224 e also observed with rs151174000 and parieto-frontal low theta coherence, rs14429078 and parieto-occi

225 e, optic, dorso-lateral, basal, subvertical, frontal, magnocellular, and buccal lobes.

226 (P < .001) in several regions, including the frontal (mean +/- standard deviation) (left, 2.16 years

227 we interpret the increased presence of the "frontal" microstate 3 as a sign of deeper local deactiva

228 ulness meditation has been shown to modulate frontal midline theta (FMT) and alpha oscillations that

229 The claustrum core projects predominantly to frontal-midline cortical regions, whereas the dorsal and

230 tions indicate that the observed rapid cross-frontal mixing occurs via shear dispersion, generated by

231 otor putamen are targets of projections from frontal motor, inferior parietal, and ventrolateral pref

232 or claustrum, in regions known to project to frontal, motor, and cingulate cortical areas.

233 d fNIRS to examine the brain activity in the frontal, motor, parietal and occipital regions, aiming t

234 ed with hippocampus compared to temporal and frontal neocortex.

235 ques and marmosets, our results suggest this frontal network specialized for social face processing p

236 ward-guided decisions.SIGNIFICANCE STATEMENT Frontal neural networks and the temporal lobes contribut

237 ntactic network, which comprises an anterior frontal node and a posterior temporal node connected by

238 ar to the one containing tau in the anterior frontal node projected to an area similar to the one con

239 fasciculus) and ventrally located (inferior frontal-occipital, uncinate, and middle longitudinal fas

240 regions bordering the Sylvian fissure of the frontal, parietal and temporal lobes.

241 We defined left and right frontal, parietal, and temporal areas as seeds (or regio

242 etal lobes, in addition to scattered lateral frontal, parietal, and temporal areas.

243 ]AV1451 at the white/gray matter junction in frontal, parietal, and temporal brain regions, a typical

244 mostly overlapping brain circuits including frontal, parietal, posterior, and cingulate regions with

245 ion between wave variability and atmospheric frontal passages, and (2) the influence on the SST varia

246 havior had a sex interaction in the amygdala-frontal pathway; weaker connectivity (lower fractional a

247 division of prefrontal cortex - with lateral frontal pole (FPl) supporting the context-dependent mapp

248 task-defined frontal regions to the lateral frontal pole (FPl), an anatomically defined portion of t

249 ectivity between left angular gyrus and left frontal pole predicted better response to CBT in the OCD

250 ex, right inferior temporal gyrus, and right frontal pole.

251 DeltaBP(ND)(frontal) = 1 - (BP(ND)(frontal) post-dose/BP(ND)(frontal) baseline) was used as

252 This suggests that frontal pyramidal neurons use a different integration sc

253 Specifically, left frontal regions and their underlying white matter tracts

254 We corroborate the locations of these frontal regions by demonstrating functional and structur

255 ysis suggest that intrinsic communication of frontal regions engaged in executive functions and emoti

256 e connectivity, localized those task-defined frontal regions to the lateral frontal pole (FPl), an an

257 This bundle connected frontal regions to the STN.

258 the lateral temporal, lateral parietal, and frontal regions were inversely correlated with amyloid b

259 r SCZ, in left supramarginal gyrus and right frontal regions with risk for BD, and in right striatum

260 ray matter hypoplasia was observed in medial frontal regions, the inferior olives, and the cerebellum

261 tcome was significantly correlated with left frontal regions.

262 between the groups in the regions examined, frontal release signs were found more commonly when pati

263 We found that frontal RFs require spectral cues and lateral RFs requir

264 h global (Spearman rho = 0.27; P = .004) and frontal (rho = 0.27; P = .005) amyloid beta load and glo

265 rtical parcellation includes 234 priors from frontal, sensorimotor, parietal, temporal, occipital, ci

266 rception, it only provides information about frontal sound sources.

267 the idea that azimuthal spatial bisection in frontal space requires visual calibration, while detecti

268 Thresholds for both tasks were similar in frontal space, lower for the MAA task than for the bisec

269 sfunction, indicating the involvement of the frontal-subcortical circuit in the pathogenesis of front

270 ing patients with normal cognition (n = 18), frontal-subcortical dysfunction (n = 12) and memory impa

271 dysfunction (n = 12) and memory impairment + frontal-subcortical dysfunction (n = 18), we further inv

272 dentify the underlying pathological basis of frontal-subcortical dysfunction in multiple system atrop

273 We also examined topographic correlates of frontal-subcortical dysfunction with other clinical symp

274 Twelve of them had pure frontal-subcortical dysfunction, defined as the presence

275 found more commonly when patients developed frontal-subcortical dysfunction, indicating the involvem

276 l-subcortical circuit in the pathogenesis of frontal-subcortical dysfunction.

277 emory impairment: n = 6; memory impairment + frontal-subcortical dysfunction: n = 12) had more neuron

278 syndrome and progressive supranuclear palsy-frontal subtypes) and 20 healthy control subjects were i

279 ing the superior temporal gyrus and inferior frontal sulcus.

280 eprived adoptees showed lower right inferior frontal surface area and volume but greater right inferi

281 measures, NfL and grey matter volume of the frontal, temporal and parietal lobes, insula and whole b

282 regulation was associated with activation in frontal, temporal, and parietal regions, whereas both im

283 Critically, increase in frontal theta during viewing of a video was associated w

284 Frontal theta power (3-6 Hz) significantly increased dur

285 uent top-down influences including increased frontal theta power (7-9 Hz, ~120 ms) in mid-anterior ci

286 al differences in the magnitude of change in frontal theta power were related to concurrent nonverbal

287 rtially statistically mediated by changes in frontal theta power.

288 n contrast, social narratives showed greater frontal theta, an index of negative feedback and emotion

289 ht precuneus and right but not left superior frontal thickness), suggesting an interplay of cerebella

290 orical seismicity, whereas south of the Main Frontal Thrust (MFT), the foreland basin is typically po

291 st (MHT) into the foreland basin as an outer frontal thrust, and provide a modern snapshot of the dev

292 Frontal top-down cortical neurons projecting to sensory

293 The simian-human differences in proportional frontal tract volume were significant for projection, co

294 al (BP(ND)(frontal)) was calculated as (V(T)(frontal)/V(T)(cerebellum)) - 1.

295 Our findings suggest that frontal versus posterior functional imbalance as measure

296 ent method [TDL-BAAM]) was trained on 15 129 frontal view pediatric trauma hand radiographs obtained

297 ed for UV-bright prey capture in their upper frontal visual field, which may use the signal from a si

298 amine administration, [(11)C]CIMBI-36 BP(ND)(frontal) was reduced by 14 +/- 13% (p = 0.002).

299 The frontal cortex binding potential (BP(ND)(frontal)) was calculated as (V(T)(frontal)/V(T)(cerebell

300 s to a larger frontal cortex and even larger frontal white matter in humans compared with other prima