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1 sample, where lamin B1 was increased in the frontal lobe.
2 es, temporal lobe, parietal lobe, and medial frontal lobe.
3 umor type arising with high frequency in the frontal lobe.
4 (k(f)) of the creatine kinase enzyme in the frontal lobe.
5 gradients, with particular enrichment in the frontal lobe.
6 of behavior mediated in part by the maturing frontal lobe.
7 rmation is conveyed from the parietal to the frontal lobe.
8 plexity specific to the human lineage in the frontal lobe.
9 ow grade gliomas (LGG) localized in the left frontal lobe.
10 rietal and occipital regions compared to the frontal lobe.
11 the temporal lobes, right basal ganglia and frontal lobe.
12 rguing for an initiation of selection in the frontal lobe.
13 , and culminating as causal inference in the frontal lobe.
14 twork that includes all 7 motor areas in the frontal lobe.
15 s that from any of the premotor areas in the frontal lobe.
16 s have implicated abnormal activities in the frontal lobe.
17 he corpus callosum connecting left and right frontal lobes.
18 redict the level of functional access to the frontal lobes.
19 atients showed increased connectivity in the frontal lobes.
20 he subarachnoid space, particularly over the frontal lobes.
21 that it involved relative enlargement of the frontal lobes.
22 regions predominantly influenced activity in frontal lobes.
23 ignificant hypometabolism in the temporal or frontal lobes.
24 d towards subtle abnormalities in the medial frontal lobes.
25 % of survivors, occurring more frequently in frontal lobes.
26 stro-caudal hierarchical organization of the frontal lobes.
27 the greatest increases in lateral and medial frontal lobes.
28 ng action selection signals generated in the frontal lobes.
29 associated with abnormal connectivity of the frontal lobes.
30 e encephalopathy predominantly affecting the frontal lobes, 1 hemorrhagic shock and encephalopathy, 1
31 elirium was associated with smaller superior frontal lobe (-2.11 cm(3) [-3.89, -0.32]; p = .03) and h
32 er, other atypical regions involved were the frontal lobes (30.4%), temporal lobes (8.69%), basal gan
34 hite matter water diffusion metrics from the frontal lobe accounted for poor performance on both memo
35 esponse characterized by inferior and middle frontal lobe activation (right>left) which bears some si
36 0% and 13.1% of the total effect of inferior frontal lobe activation on angina severity, respectively
39 animal's decision was reliably maintained in frontal lobe activity through a selective state sequence
40 unilateral and unilobar focal lesions in the frontal lobe affecting the cortical grey matter or the c
41 localised disease, frequently located in the frontal lobes, affecting the cortical grey matter and ad
45 olumetric/area measurements obtained for the frontal lobe, amygdala, and hippocampus and subregions o
47 genes differentially expressed within human frontal lobe and a striking increase in transcriptional
48 abnormalities extended rostrally towards the frontal lobe and dorsocaudally towards the superior temp
49 sconsin Card Sorting Test, a classic test of frontal lobe and executive function in humans, to assess
50 rformance on neuropsychological tests of the frontal lobe and executive functioning, the Trail Making
51 pect of the fissure moving anteriorly to the frontal lobe and laterally in the direction of the tempo
53 of 10 regions-of-interest including insula, frontal lobe and putamen in AD compared with controls, b
54 ion for imagined words in the temporal lobe, frontal lobe and sensorimotor cortex, consistent with pr
55 Network disorganization primarily involving frontal lobe and subcortical regions in nonpsychotic rel
57 injection or needle insertion into the right frontal lobe and were euthanized at different times to e
59 th premotor (PM) and motor (M1) areas of the frontal lobe and with other areas of parietal and occipi
60 volume difference of approximately 8% in the frontal lobes and 12% in the occipital lobes-these regio
61 erns of coherence are identified between the frontal lobes and between frontal and distant brain regi
62 puncture after enucleation and biopsied the frontal lobes and optic nerves of a macaque experimental
63 t there are no inhibitory modules within the frontal lobes and that behavioural inhibition is an emer
64 rtical structures including bilateral medial frontal lobes and the right inferior frontal and tempora
65 more likely in subnetworks that include the frontal lobe, and can be opposite in nature depending on
66 in the olfactory epithelium, olfactory bulb, frontal lobe, and lung tissues in cadavers from the city
67 er WMH load, specifically in the subcortical frontal lobe, and periventricular frontal and parietal c
68 articularly those located in the subcortical frontal lobe, and the periventricular frontal and pariet
69 netic methods reveals that the size of human frontal lobes, and of specific frontal regions, is as ex
71 callosum, occipital, temporal, parietal and frontal lobes, and right hippocampus (p < 0.025 after fa
73 reases of grey matter were identified in the frontal lobes, anterior cingulate gyrus, left temporal l
74 and cerebral microdialysis monitoring (right frontal lobe, apparently normal tissue) combined with ce
75 and that event-related potentials above the frontal lobe are affected by both word reversal and the
76 glutamatergic and GABAergic deficits in the frontal lobe are potential targets for symptomatic drug
77 r cingulate cortex in the medial wall of the frontal lobe are the source of this increased theta powe
79 data have motivated the hypothesis that the frontal lobes are organized hierarchically, such that co
80 butyric acid (GABA), particularly within the frontal lobes, are some of the most reliable alterations
81 al white matter fiber tracts associated with frontal lobe areas critical in cognitive processing has
84 one subtype showing grey matter loss in the frontal lobes as well as loss of the temporal lobes (fro
87 superior temporal gyrus (BA21), dorsolateral frontal lobe (BA9), and dorsal parietal lobe (BA7) of ea
91 ignificant white matter tract changes in the frontal lobe, brainstem and hippocampal regions of the A
92 in four major brain structures (hippocampus, frontal lobe, brainstem, and cerebellum) using a techniq
93 uman primates indicate that the ventromedial frontal lobe carries information about both losses and g
94 orrelated with volumetric differences in the frontal lobes, caudate nucleus and cerebellum, and hyper
95 ical ROI and links were observed in superior frontal lobe, cingulate cortex, fusiform, putamen, and m
96 aboratory, there is also evidence that other frontal lobe circuits have evolved to make other types o
100 ows that humans have a greater proportion of frontal lobe connections compared with monkeys, when nor
101 the disproportionally large volume of human frontal lobe connections is accompanied by a reduction i
102 and control groups, whilst the structure of frontal lobe connections showed relationships with execu
107 y to studies of HCS and the pH values in the frontal lobe correlated negatively with the YMRS values.
108 ded neurons from different somatosensory and frontal lobe cortices of monkeys performing a vibrotacti
110 ling Test clinical measures are sensitive to frontal lobe damage although only the suppression error
111 ds (impulsivity) characterizes subjects with frontal lobe damage and behavioral disorders including s
117 e diseases such as Parkinson's disease (PD), frontal lobe dementia (FLD) and diffuse Lewy-body dement
118 esearch has demonstrated that performance on frontal lobe-dependent tasks is associated with dopamine
119 ive effects of socioeconomic disadvantage on frontal lobe development (with implications for function
120 ject via the occipitofrontal fascicle to the frontal lobe (dorsal area 6, and areas 8Ad, 8B, and 46);
125 n implicated in autosomal dominant nocturnal frontal lobe epilepsy (ADNFLE) and epilepsy of infancy w
127 novel brain pathology in young patients with frontal lobe epilepsy (FLE) that is distinct from focal
128 reported in patients with schizophrenia and frontal lobe epilepsy and may contribute to their associ
130 associated with autosomal dominant nocturnal frontal lobe epilepsy have been found to show spontaneou
134 ions in nAChRs subunits have been related to frontal lobe epilepsy, neurodegenerative diseases, and o
136 ancer and directly inoculated into the mouse frontal lobe, exhibit striking differences in proliferat
137 enrichment of methylation QTLs (P<0.001) and frontal lobe expression quantitative trait loci (eQTLs)
139 l lobe, left posterior cingulate gyrus, left frontal lobe expressive language regions, and left basal
140 lateral, dorsal, and mesial surfaces of the frontal lobe, extending into primary sensory, superior p
142 and pointing HPs are strongly predictive of frontal lobe EZ; cup, politician's fist, and pincer are
145 ulate cortex and orbitofrontal cortex in the frontal lobe from a total of 90 elderly subjects (age ra
147 s in 60 (45%) of 133 patients on the test of frontal lobe function (FAB), and in between 13 (10%) and
148 dence of a positive effect of simvastatin on frontal lobe function and a physical quality-of-life mea
150 unger adults who performed worse on tests of frontal lobe function showed greater FMT signal in right
151 ze data from a classical task used to assess frontal lobe function, the Wisconsin Card Sorting Test.
153 ry modality and information domain influence frontal lobe functional organization, they also demonstr
155 ntial underlying mechanism for impairment of frontal lobe functions in both patients and siblings, an
156 22q11.2DS subjects compared with controls in frontal lobe (g = -0.47; p < 0.001), temporal lobe (g =
157 an developmental in vivo evidence confirming frontal lobe GABA maturation, which was linked to impuls
158 ystem, also matures during adolescence, with frontal lobe GABA receptors reaching adult levels late i
159 diffusivity [RD, AxD, MD, respectively]) of frontal lobe, genu, and splenium of the corpus callosum
161 gray matter, total neocortical white matter, frontal lobe gray matter, frontal lobe white matter, and
164 how state desirability signals decoded from frontal lobe hemodynamics, as measured with near-infrare
170 of Interest) was defined in 7 locations: the frontal lobe in the right and left hemispheres, the basa
172 niqueness should therefore focus less on the frontal lobes in isolation and more on distributed neura
173 king circuits working in parallel within the frontal lobes in order to make different types of decisi
174 ion revealed a pathological mass in the left frontal lobe, in close topographical relationship to the
175 ciated with decreased GM density in the left frontal lobe, in the right frontoparietal cortex and in
176 ymmetric reduction in CTh/CSA in most of the frontal lobes, in addition to large temporoparietal area
178 spontaneous long-term recovery from isolated frontal lobe injury and isolated frontoparietal injury.
179 the microcircuitry of agranular areas of the frontal lobe involved in cognitive control and responsib
180 studies using drug cue paradigms have shown frontal lobe involvement in this contextual influence on
182 rcuits control executive function within the frontal lobe involving the dorsolateral prefrontal corte
186 A fundamental challenge in studying the frontal lobe is to parcellate this cortex into "natural"
187 bs of high connectivity (many located in the frontal lobe) is associated with amyloid accumulation.
188 CC), which lies on the medial surface of the frontal lobes, is important in regulating cognitive cont
189 disorder with dysfunction and atrophy of the frontal lobes leading to changes in personality, behavio
190 male Vietnam combat veterans consisting of a frontal lobe lesion group with focal penetrating head in
192 ation approaches developed for patients with frontal lobe lesions and pharmacotherapy approaches desi
195 ue studies in an experiment with humans with frontal lobe lesions, asking whether behavioral impairme
199 hich suggests that structural changes in the frontal lobe may have an indirect influence on age-relat
200 l spatial codes are used in conjunction with frontal lobe mechanisms to plan routes during navigation
201 by considering the role of the anterior and frontal lobes, mesolimbic connections and the right hemi
203 subplate visualization [complete + partial]: frontal lobe, n = 243 vs n = 117; temporal lobe, n = 244
204 rily located in the posterior portion of the frontal lobe, near the controlling site, and non-linear
205 el tractography approach to demonstrate that frontal lobe networks, extending within and beyond the f
206 matter volume in nearly all networks, except frontal lobe networks, where differences in grey matter
207 otheses that (i) cerebellar efferents target frontal lobe neurons involved in forming action represen
208 g firing rates and correlated variability of frontal lobe neurons while monkeys perform a vibrotactil
209 the temporal lobe with predilection for the frontal lobes.New type III is one of the above dysplasia
210 brain has principal and arcuate sulci of the frontal lobe not seen in the stem catarrhine Aegyptopith
211 be networks, extending within and beyond the frontal lobes, occupy 66% of total brain white matter in
212 identical analyses of EEG recorded over the frontal lobe of macaque monkeys (one male, one female) p
213 is procedure to 17 injections, placed in the frontal lobe of nine marmosets as part of earlier studie
214 t parietal and occipital regions, the mesial frontal lobe of the right hemisphere, and the cuneus and
215 ng direct inoculation of M002 into the right frontal lobes of A. nancymae; (iii) there was no histopa
216 stone acetylation in the white matter of the frontal lobes of aged subjects and in patients with chro
217 cid (DHA) accumulates in the hippocampus and frontal lobes of the fetal brain during the last trimest
218 lower FA and significantly higher RD in the frontal lobes of the RHU group, relative to the TS group
220 of the basal ganglia, thalamus, hippocampus, frontal lobe, orbitofrontal cortex, and gyrus rectus.
223 magnetic resonance imaging with an executive frontal lobe paradigm, we investigated cortical activati
224 ory analysis moreover showed that the mesial frontal lobes, parahippocampal gyrus, and lateral tempor
225 ial diffusivity, and radial diffusivity from frontal lobes, parietal lobes, and in the frontostriatal
227 ing associated cognitive abilities, in other frontal lobe pathologies; however, in clinical trials, M
229 rnally using brain stimulation, and that the frontal lobes play a causal role in mind-wandering behav
230 ng in 19% of the whole cortex and 24% of the frontal lobes, primarily in the right superior, medial o
232 in spatial working memory while the ventral frontal lobe processes language and communication signal
233 nsisted of significantly increased CT within frontal lobe regions and reduced SA in the orbitofrontal
234 ormation about the complex interplay between frontal lobe regions in mediating these processes and ac
235 antly smaller gray matter volume was seen in frontal lobe regions in patients homozygous for the A al
236 focus on recent evidence that suggests four frontal lobe regions make distinct contributions to rewa
237 phase coding in four temporal lobe and four frontal lobe regions of the human brain using local fiel
238 receptive aphasia; (iii) widespread temporal/frontal lobe regions of the left hemisphere and expressi
241 brain regional analysis demonstrated only 3 frontal lobe regions with an SUV estimation bias of 5% o
242 d morphological relationships primarily with frontal lobe regions, and their network-level alteration
243 activity was augmented in the left temporal/frontal lobe regions, as well as left inferior-parietal
244 g the middle and anterior temporal lobe with frontal lobe regions, including the pars triangularis.
246 s under dynamic conditions, with the lateral frontal lobe required for selecting the relevant dimensi
247 associate with reward, and the ventromedial frontal lobe required to learn the reward association it
250 lated with oral naming speed in both lateral frontal lobes (rho = 0.48 and 0.47 for right and left fr
251 yzed ictal semiology of 489 temporal lobe or frontal lobe seizures recorded over a 6-year period at t
252 y- and deoxyhemoglobin concentrations in the frontal lobe show differences in response to both primar
253 s were recorded from temporal, parietal, and frontal lobe sites in 12 pediatric patients undergoing e
254 processes mediated by anatomically distinct frontal lobe subregions are both critical for adaptive c
255 we tested the critical involvement of human frontal lobe subregions in a probabilistic, multidimensi
256 , we examined the necessary contributions of frontal lobe subregions in attributing feedback to relev
258 results revealed regions of occipital lobe, frontal lobe, supplementary motor area, cingulate cortex
261 and may occur in other locations such as the frontal lobes, thalami, basal ganglia and brainstem.
264 theta band, involving mainly regions in the frontal lobe that were sending information to a more dis
265 gions, there is extensive involvement of the frontal lobes that has been missed by past ROI analyses.
268 tion of the right hemisphere, especially the frontal lobe, to reading comprehension performance on th
270 speech information from the temporal to the frontal lobe utilizes two different streams and conducti
272 ome evidence from patients with ventromedial frontal lobe (VMF) damage argues against a very general
274 conomic models propose that the ventromedial frontal lobe (VMF) supports multiattribute decisions by
275 ypes were characterized by a large amount of frontal lobe volume loss, with one subtype showing grey
276 ion, and smaller medial, lateral and orbital frontal lobe volumes showed faster rates of decline.
280 feedback attribution, while the ventromedial frontal lobe was necessary for learning the value of fea
282 comprising the precuneus, cingulate & middle frontal lobe was significantly higher in psALS and affec
284 thinning in children primarily in the right frontal lobes was associated with exposure to prenatal m
285 ger resting-state absolute EEG powers in the frontal lobe were associated with wiser advising from th
286 ration change and the beta band power in the frontal lobe were found to differ the most between the t
289 ated by anatomically distinct regions of the frontal lobes when reinforcement contingencies are assur
290 entified a small cluster in the left lateral frontal lobe where children with greater upper-body musc
292 wed that microstate 3 encompassed the medial frontal lobe, whereas microstate 4 involved the occipita
293 sions with a hypoechogenic halo in the right frontal lobe, which could correspond to brain abscesses.
294 er volume changes in both hemispheres of the frontal lobe, which suggests that structural changes in
295 a predilection for iron accumulation in the frontal lobes, which when combined with the subcortical
296 ical white matter, frontal lobe gray matter, frontal lobe white matter, and the hippocampus in a cros
298 and document increases in activity in medial frontal lobe with decreased activity in the dorsolateral
300 , various cortical areas in the parietal and frontal lobes work together effortlessly to analyze obje