ライフサイエンスコーパス: fumigatus

1 ence that Gna1 is an antifungal target in A. fumigatus.

2 sis, melanin production, and virulence in A. fumigatus.

3 of the innate immune response to Aspergillus fumigatus.

4 ch may be exploited to selectively target A. fumigatus.

5 the opportunistic human pathogen Aspergillus fumigatus.

6 might harbor triazole-resistant Aspergillus fumigatus.

7 esulting in azole-resistant phenotypes of A. fumigatus.

8 gents Listeria monocytogenes and Aspergillus fumigatus.

9 pounds affected cell wall biosynthesis in A. fumigatus.

10 cial role in propagation and virulence of A. fumigatus.

11 ) pathway in the fungal pathogen Aspergillus fumigatus.

12 es involved in innate responses to viable A. fumigatus.

13 cell transplantation, especially regarding A fumigatus.

14 vailable in 1 patient and showed Aspergillus fumigatus.

15 fection with the fungal pathogen Aspergillus fumigatus.

16 llowing subchronic inhalation exposure to A. fumigatus.

17 ugmented in animals repeatedly exposed to A. fumigatus.

18 the opportunistic human pathogen Aspergillus fumigatus.

19 environmental resistance to triazoles in A. fumigatus.

20 le in antifungal defense against Aspergillus fumigatus.

21 d against GBS serotypes for reactivity to A. fumigatus.

22 antagonist after challenge with Aspergillus fumigatus.

23 in regulating antifungal immunity against A. fumigatus.

24 BAL was analyzed for the presence of A fumigatus.

25 ce factor of the fungal pathogen Aspergillus fumigatus.

26 panel of pathogenic bacteria and Aspergillus fumigatus.

27 f Panama and tested them against Aspergillus fumigatus.

28 pes of the human fungal pathogen Aspergillus fumigatus.

29 y remodelling and inflammation elicited by A fumigatus.

30 t in the human pathogenic fungus Aspergillus fumigatus.

31 portunistic pathogens, including Aspergillus fumigatus (1.4%) and cytomegalovirus (6%), were rare, sc

32 uding real-time visualization of Aspergillus fumigatus (5 d for culturing, 1-2 d for imaging).

33 Twelve bIFIs (Aspergillus fumigatus [5], Aspergillus species [2], Mucorales [2], F

34 Using Aspergillus fumigatus, a respiratory pathogen, we characterize the i

35 Aspergillus fumigatus, a ubiquitous human fungal pathogen, produces

36 Aspergillus fumigatus, a ubiquitous mold, is a common cause of invas

37 Conditioned media (CM) from A. fumigatus, A. niger, and A. flavus cultures also reduced

38 In contrast to Aspergillus fumigatus, A. terreus infections are associated with hig

39 he common fungal airway pathogen Aspergillus fumigatus activates heterologously-expressed PAR-2.

40 mothers, the magnitude of HDM or Aspergillus fumigatus (AF) extract-induced airway hyperresponsivenes

41 istic antifungal effects against Aspergillus fumigatus (AF) in an in vitro human alveolus bilayer mod

42 e sensitised and challenged with Aspergillus fumigatus (Af).

43 globulin E and G4 directed to recombinant A. fumigatus allergens in 55 cystic fibrosis patients witho

44 IgG4 responses to recombinant A. fumigatus allergens were detected in all patients, but n

45 t include immune responses to recombinant A. fumigatus allergens.

46 ngi such as Candida albicans and Aspergillus fumigatus also form biofilms during infection.

47 A fumigatus also induced endothelin-1 in murine lungs, ass

48 ted in the development of azole-resistant A. fumigatus and address the therapeutic options available.

49 lateral branching in pathogenic Aspergillus fumigatus and Aspergillus flavus, and appressorium forma

50 s isolated and evaluated in vitro against A. fumigatus and B. dendrobatidis showing MIC values of 62.

51 have a limited ability to migrate toward A. fumigatus and block the growth of A. fumigatus conidia (

52 Trafficking of TLR9 to A. fumigatus and C. albicans phagosomes requires Dectin-1 r

53 on of innate immune responses to Aspergillus fumigatus and Candida albicans.

54 notation of transcriptional regulators in A. fumigatus and construction of a library of 484 transcrip

55 st commonly by Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, result in more de

56 lturally derived azole-resistant Aspergillus fumigatus and emerging threats such as multidrug resista

57 a-(1,3)-glucan polysaccharide of Aspergillus fumigatus and ensuing CD4+ T-cell polarization are poorl

58 e sensitized and challenged with Aspergillus fumigatus and evaluated ex vivo in tissue culture.

59 d calcium-mediated signalling in Aspergillus fumigatus and found that calcium chelation severely impe

60 ns of the class II and class V myosins in A. fumigatus and found that while the class II myosin (myoB

61 cnn1b-Tg and wild-type mice with Aspergillus fumigatus and house dust mite allergen and compared the

62 pulmonary aspergillosis but sensitized to A. fumigatus and in nine patients with allergic broncho-pul

63 standing of host-pathogen interactions in A. fumigatus and patients with cystic fibrosis and the ongo

64 ide hydrolase that disrupts GAG-dependent A. fumigatus and Pel polysaccharide-dependent Pseudomonas a

65 e domains from these proteins (Sph3h from A. fumigatus and PelAh from P. aeruginosa) were found to de

66 d by the opportunistic pathogens Aspergillus fumigatus and Pseudomonas aeruginosa, respectively.

67 typic bronchiole and cultures of Aspergillus fumigatus and Pseudomonas aeruginosa.

68 ole-resistant and -susceptible strains of A. fumigatus and that it should have important clinical app

69 usceptible to aeroallergens from Aspergillus fumigatus and the house dust mite, resulting in an asthm

70 e early steps in the interactions between A. fumigatus and the human airway epithelium.

71 y result from the interaction of Aspergillus fumigatus and the immune system of its human host.

72 Sensitisation to Aspergillus fumigatus and/or Penicillium chrysogenum was associated

73 ScTOK, AfTOK1 (Aspergillus fumigatus), and H99TOK (Cryptococcus neoformans grubii)

74 um, Trichophyton mentagrophytes, Aspergillus fumigatus, and Candida albicans.

75 fungi including Candida species, Aspergillus fumigatus, and Cryptococcus neoformans.

76 berculosis, Salmonella enterica, Aspergillus fumigatus, and Epstein-Barr virus (EBV) and samples from

77 ung disease caused by the fungus Aspergillus fumigatus, and is a leading cause of invasive fungal inf

78 domonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous mycobacteria.

79 for TLR9 trafficking to beta-1,3 glucan-, A. fumigatus-, and C. albicans-containing phagosomes.

80 cells that are crossreactive to Aspergillus fumigatus antigens can also drive exaggerated airway inf

81 ons caused by triazole-resistant Aspergillus fumigatus are associated with a higher probability of tr

82 infections with fungal pathogen Aspergillus fumigatus are associated with caspofungin prophylaxis.

83 All patients with IgE sensitisation to A. fumigatus are at risk of lung damage irrespective of whe

84 Candida albicans and Aspergillus fumigatus are dangerous fungal pathogens with high morbi

85 th and virulence of the pathogen Aspergillus fumigatus are unknown.

86 s brasiliensis, and occasionally Aspergillus fumigatus, are primary pulmonary pathogens of otherwise

87 lammation in murine AA caused by Aspergillus fumigatus as well as its consequence on the regulation o

88 utcome of human macrophage infection with A. fumigatus, as well as the impact of calcineurin inhibito

89 Accurate and prompt identification of A fumigatus-associated clinical status might allow early a

90 e showed that deletion of asp f3 rendered A. fumigatus avirulent in a mouse model of pulmonary asperg

91 d, necrosis-dependent lateral transfer of A. fumigatus between macrophages as an important host strat

92 e observed frequent cell-cell transfer of A. fumigatus between macrophages, which assists subsequent

93 tan (GAG) is an integral component of the A. fumigatus biofilm matrix and a key virulence factor.

94 Oxygen gradients inevitably arise during A. fumigatus biofilm maturation and are both critical for,

95 creasing oxygen levels toward the base of A. fumigatus biofilms increases antifungal drug resistance.

96 ment of A. fumigatus with Sph3h disrupted A. fumigatus biofilms with an EC50 of 0.4 nM.

97 PelAh treatment also disrupted preformed A. fumigatus biofilms with EC50 values similar to those obt

98 de biosynthesis, can degrade GAG, disrupt A. fumigatus biofilms, and attenuate fungal virulence in a

99 and single-cell metabolic activity within A. fumigatus biofilms.

100 their ability to degrade GAG and disrupt A. fumigatus biofilms.

101 ncreased mortality rates, higher Aspergillus fumigatus burden and reduced neutrophil recruitment into

102 tolerant filamentous fungi, in particular A. fumigatus but not total IgE, is associated with fixed ai

103 fected with a triazole-resistant Aspergillus fumigatus, but a direct comparison with triazole-suscept

104 Recognition of Aspergillus fumigatus by the host immune system leads to activation

105 Thus, A. fumigatus CalA is an invasin that interacts with integri

106 opportunistic pathogenic fungus Aspergillus fumigatus, called aspergillosis.

107 Aspergillus fumigatus can exploit the hypoxic microenvironment in th

108 Aspergillus fumigatus, Candida albicans and Mycosphaerella tassiana

109 12) from human fungal pathogens (Aspergillus fumigatus, Candida albicans, Cryptococcus neoformans and

110 The A. fumigatus cell wall is a complex network of polysacchari

111 al or biological (germination) removal of A. fumigatus cell wall melanin.

112 n CalA is expressed on the surface of the A. fumigatus cell wall, where it mediates invasion of epith

113 ost resistance against pulmonary Aspergillus fumigatus challenge through the regulation of antifungal

114 restingly, induction of type I IFNs after A. fumigatus challenge was only partially dependent on MDA5

115 al activity, and lung fungal clearance in A. fumigatus-challenged mice.

116 dynamic simulations, and mutations of the A. fumigatus CnA/CnB-FK506-FKBP12-complex identify a Phe88

117 Neutrophil/A fumigatus coculture, flow cytometry, and video microscop

118 common fungus in asthmatics was Aspergillus fumigatus complex and this taxon accounted for the incre

119 cs and the most common fungus is Aspergillus fumigatus complex.

120 l wall of the filamentous fungus Aspergillus fumigatus comprises polysaccharides.

121 ward A. fumigatus and block the growth of A. fumigatus conidia (proportion with growth blocked, 69%).

122 ng myeloid cell responses against inhaled A. fumigatus conidia and demonstrates a benefit for systemi

123 , are involved in the killing of Aspergillus fumigatus conidia and hyphae, using neutrophils from pat

124 alpha-(1,3)-Glucan was isolated from A. fumigatus conidia and mycelia cell wall.

125 tin in uptake or killing of intracellular A. fumigatus conidia either in vitro or in a murine model o

126 After phagocytosis, A. fumigatus conidia rapidly escaped from DCs, whereas A. t

127 Furthermore, while A. fumigatus conidia triggered expression of DC activation

128 Specifically, germinating A. fumigatus conidia were associated with Clec7a and were p

129 were protected from antifungals, whereas A. fumigatus conidia were efficiently cleared.

130 with apoptosis-like features in Aspergillus fumigatus conidia, the most prevalent human mold pathoge

131 d survival after pulmonary challenge with A. fumigatus conidia.

132 ubchronic inhalation exposure to Aspergillus fumigatus conidia.

133 cuity, we solved the crystal structure of A. fumigatus Crh5 (AfCrh5) in complex with a chitooligosacc

134 Aspergillus fumigatus culture-positive patients were investigated to

135 A. fumigatus DeltasakA and DeltampkC were virulent in mouse

136 Homology modeling of Aspergillus fumigatus DHODH has identified a predicted binding mode

137 ort a fluorogenic probe to image Aspergillus fumigatus directly in human pulmonary tissue.

138 the SeptiFast assay in detecting Aspergillus fumigatus DNA in whole blood samples from 38 critically

139 A. fumigatus DNAemia was associated with poor outcome.

140 ion by the human-pathogenic mold Aspergillus fumigatus during invasive growth.

141 fumigatus during pathogenesis, a prerequisite to underst

142 Macrophage phagocytosis of A. fumigatus enabled control of 90% of fungal germination.

143 to identify triazole-resistant strains of A. fumigatus, even in mixtures of triazole-resistant and -s

144 In an Aspergillus fumigatus extract-induced inflammation model, IL-10-prod

145 ere we study the function of the Aspergillus fumigatus family of five Crh transglycosylases.

146 The filamentous fungal pathogen Aspergillus fumigatus forms biofilms in vivo, and during biofilm gro

147 g infection, the fungal pathogen Aspergillus fumigatus forms biofilms that enhance its resistance to

148 ory also received 46 isolates of Aspergillus fumigatus from COVID-19 patients (including three that e

149 a-glucan surface exposure during Aspergillus fumigatus germination activates an Atg5-dependent autoph

150 combinant hydrolase domains from Sph3, an A. fumigatus glycoside hydrolase involved in GAG synthesis,

151 /34 H1N1 and subsequently challenged with A. fumigatus had increased fungal burden, viral burden, inf

152 Lately, increased azole resistance in A. fumigatus has become a significant challenge in effectiv

153 Triazole antifungal resistance in A. fumigatus has become recognized as a global, yet poorly

154 Azole resistance in Aspergillus fumigatus has emerged as a global health problem.

155 The genome of Aspergillus fumigatus has four arsM genes encoding ArsMs with only t

156 Aspergillus fumigatus has widely evolved resistance to the most comm

157 st of the variants that we uncover within A. fumigatus have been previously hypothesized to contribut

158 itized to airborne fungi such as Aspergillus fumigatus have more severe asthma.

159 ns, Cryptococcus neoformans, and Aspergillus fumigatus have transitioned from a rare curiosity to a l

160 nfection caused predominantly by Aspergillus fumigatus, have increased due to the growing number of i

161 ed increased phosphorylation of SakA, the A. fumigatus Hog1 homologue.

162 allergen ( Alternaria alternata, Aspergillus fumigatus, house dust mite, and ovalbumin) for 4 wk.

163 A. fumigatus hyphae also contain surface structures that in

164 Treatment of A. fumigatus hyphae with either Sph3h or PelAh significantl

165 e an impaired ability to inhibit Aspergillus fumigatus hyphal growth in vitro and in infected corneas

166 y demonstrated enhanced lung clearance of A. fumigatus IL-33 functioned as a negative regulator of mu

167 mily member IL-33 in lung defense against A. fumigatus IL-33 was detected in the naive lung, which fu

168 which further increased after exposure to A. fumigatus in a dectin-1-independent manner.

169 t assessing the activity of neutrophils on A fumigatus in allogeneic HSCT recipients at different pos

170 amples to perform multi-photon imaging of A. fumigatus in ex vivo human tissue.

171 Colonization by Aspergillus fumigatus in patients with cystic fibrosis (CF) can caus

172 Persistent isolation of A fumigatus in sputum is a significant risk factor for A f

173 e against the opportunistic mold Aspergillus fumigatus In this study, we investigated the IL-1 family

174 ole-resistant and -susceptible strains of A. fumigatus In this work, we developed a robust, highly se

175 actors by airway epithelium in response to A fumigatus, in order to propose novel anti-fibrotic strat

176 presence of fungi, in particular Aspergillus fumigatus, in the airway correlated with asthma severity

177 that the peroxiredoxin Asp f3 of Aspergillus fumigatus inactivates ROS.

178 results demonstrate that C. albicans and A. fumigatus induce PANoptosis and that ZBP1 plays a vital

179 ivo increases in fungal surface chitin in A. fumigatus induced by caspofungin that was associated wit

180 to suppress house dust mite- and Aspergillus fumigatus-induced allergic inflammation in murine models

181 cted A549 pulmonary epithelial cells from A. fumigatus-induced cell damage for up to 24 h.

182 A. fumigatus induces NET formation; however, NETs did not c

183 Our study demonstrates that Aspergillus fumigatus induces regulatory T-cells with a TH17-like ph

184 alveolar lavage fluid (BALF) and lungs of A. fumigatus-infected chronic granulomatous disease (CGD),

185 tibody (mAb), JF5, to neutrophil-depleted A. fumigatus-infected mice allowed specific localization of

186 d environmental isolates, indicating that A. fumigatus infection and colonization may originate from

187 n impairment was associated with Aspergillus fumigatus infection and prior lung surgery.

188 y contributed to the host defense against A. fumigatus infection and to endotoxemia.

189 Here, we report that C. albicans and A. fumigatus infection induced inflammatory programmed cell

190 ly control inflammasome activation during A. fumigatus infection.

191 g invasive C. albicans infection, but not A. fumigatus infection.

192 hils, causing susceptibility to pulmonary A. fumigatus infection.

193 release and inflammasome activation upon A. fumigatus infection.

194 role of neutrophils in protection against A. fumigatus infections, we developed an in vitro assay in

195 to enhance protective immune responses to A. fumigatus infections.

196 nt from that employed to control Aspergillus fumigatus infections.

197 human inhales hundreds of conidia daily, A. fumigatus invasive infections primarily affect the immun

198 SidA) ornithine hydroxylase from Aspergillus fumigatus is a fungal disease drug target involved in th

199 by triazole-resistant strains of Aspergillus fumigatus is a growing public health concern, as is the

200 Aspergillus fumigatus is a human opportunistic fungal pathogen whose

201 saccharide galactosaminogalactan (GAG) of A. fumigatus is a PAMP that activates the NLRP3 inflammasom

202 Aspergillus fumigatus is a ubiquitous fungal pathogen capable of cau

203 Aspergillus fumigatus is an environmental fungus that can cause inva

204 Azole resistance in Aspergillus fumigatus is an increasing problem.

205 Aspergillus fumigatus is an opportunistic fungal pathogen that cause

206 Aspergillus fumigatus is an opportunistic fungal pathogen that invad

207 Aspergillus fumigatus is an opportunistic human pathogen responsible

208 ion against fungal infections by Aspergillus fumigatus is essential but not fully understood.

209 esistance in the fungal pathogen Aspergillus fumigatus is limited.

210 While A. fumigatus is reliant upon deficiencies in the host to fa

211 TR46/Y121F/T289A azole-resistant Aspergillus fumigatus is reported.

212 attenuated virulence of melanin-deficient A. fumigatus is restored in Atg5-deficient macrophages and

213 Aspergillus fumigatus is the causative agent of allergic broncho-pul

214 The ubiquitous fungal pathogen Aspergillus fumigatus is the primary cause of opportunistic mould in

215 Neosartorya fumigata (Aspergillus fumigatus) is the most common cause of invasive aspergil

216 ed in medicine and agriculture to control A. fumigatus, is complicating the treatment of patients.

217 Aspergillus fumigatus isolate from the patient was more susceptible

218 Based on 3885 samples, 394 A. fumigatus isolates (383 environmental and 11 clinical) w

219 d TR46 Y121F T289A mutations in confirmed A. fumigatus isolates collected in institutions in the Unit

220 c diversity of clinical and environmental A. fumigatus isolates during a demolition period.

221 Heterogeneity among Aspergillus fumigatus isolates results in unique virulence potential

222 Third, primary environmental A. fumigatus isolates that rapidly germinate under airway c

223 s as well as azole resistance in Aspergillus fumigatus Its performance has been validated on bronchoa

224 tion; however, NETs did not contribute to A. fumigatus killing.

225 are both critical for, and the result of, A. fumigatus late-stage biofilm architecture.

226 novel probe for noninvasive detection of A. fumigatus lung infection based on antibody-guided positr

227 omonas aeruginosa and the fungus Aspergillus fumigatus M-CSF treatment during engraftment or after in

228 es and a structural model of the Aspergillus fumigatus mtTyrRS showed that the overall topology of th

229 and BALB/c mice were exposed to Aspergillus fumigatus, O3, or both (3 ppm for 2 hours).

230 ous activation of conidiation in Aspergillus fumigatus or Aspergillus flavus.

231 le-resistant disease are due to resistant A. fumigatus originating from the environment.

232 However, the Aspergillus fumigatus PAMPs that are responsible for inflammasome ac

233 Our data support the hypothesis that A. fumigatus phenotypic variation significantly contributes

234 n in vivo mouse disease model of aspergillus fumigatus pneumonia.

235 epicutaneous sensitization with Aspergillus fumigatus protein extract.

236 ity and gene expression to categorize the A. fumigatus putative in vivo secretome.

237 y aspergillosis is one of the most severe A. fumigatus-related diseases due to possible evolution tow

238 Aspergillus fumigatus remains the most common species in all pulmona

239 he fungal opportunistic pathogen Aspergillus fumigatus Sch9 homologue (SchA).

240 lays in vivo efficacy against a strain of A. fumigatus sensitive to the azole class of antifungals an

241 tients with cystic fibrosis (CF) can cause A fumigatus sensitization and/or allergic bronchopulmonary

242 We sought to identify A fumigatus sensitization in patients with CF by using the

243 rgic broncho-pulmonary aspergillosis from A. fumigatus sensitization with good negative and positive

244 Itraconazole treatment did not affect A fumigatus sensitization.

245 in sputum is a significant risk factor for A fumigatus sensitization.

246 BAT was used to identify A fumigatus sensitization.

247 th CF and ABPA when compared with those in A fumigatus-sensitized and nonsensitized patients with CF

248 pulmonary function and body mass index in A fumigatus-sensitized but not nonsensitized patients with

249 The BAT discriminates A fumigatus-sensitized from nonsensitized patients with CF

250 IgE, but not IgG, levels are increased in A fumigatus-sensitized patients with CF and ABPA when comp

251 ents with CF into 3 groups: nonsensitized, A fumigatus-sensitized, and ABPA.

252 S signaling was driven by dsRNA from live A. fumigatus serving as a key vitality-sensing pattern reco

253 is, Cryptococcus neoformans, and Aspergillus fumigatus (SigmaFICI = 0.05-0.50).

254 sms affect the function of representative A. fumigatus SM gene clusters, such as those involved in th

255 Serum was assessed for A fumigatus-specific IgE and total IgE.

256 Serologic (total and A fumigatus-specific IgE), pulmonary function, and body ma

257 Total and A fumigatus-specific IgE, but not IgG, levels are increase

258 Administration of a [(64)Cu]DOTA-labeled A. fumigatus-specific monoclonal antibody (mAb), JF5, to ne

259 Aspergillus fumigatus specifically caused production of endothelin-1

260 owing repeated inhalation of dry Aspergillus fumigatus spores aerosolized at concentrations potential

261 ay epithelial cells and in mice exposed to A fumigatus spores or secreted fungal factors.

262 Aspergillus fumigatus spores were delivered to the lungs of naive BA

263 Germinating A. fumigatus spores were observed in lungs along with persi

264 optimal antifungal neutrophil killing of A. fumigatus spores.

265 Levels of the A fumigatus-stimulated basophil activation marker CD203c i

266 hich analysed the effect of etanercept on A. fumigatus-stimulated macrophages at the gene expression

267 Using A. fumigatus strains containing deletions of genes for cell

268 Attempts to correlate Aspergillus fumigatus strains from clinical infection or colonizatio

269 with triazole-resistant and -susceptible A. fumigatus strains.

270 res containing only 1% triazole-resistant A. fumigatus strains.

271 IRGB10 causes hyphae damage, modifies the A. fumigatus surface and inhibits fungal growth.

272 d contains a number of species, primarily A. fumigatus, that cause mold-associated disease in humans.

273 ole-resistant and -susceptible strains of A. fumigatus The method had high efficiency and sensitivity

274 and M220I mutations in the cyp51A gene of A. fumigatus The method is based on the widely used TaqMan

275 ken together our data demonstrate that in A. fumigatus the regulatory hierarchy governing alkaline to

276 In the human pathogen Aspergillus fumigatus, the fungal-specific bZIP-type transcription f

277 rulence of the pathogenic fungus Aspergillus fumigatus, the leading etiology of invasive aspergillosi

278 We used Aspergillus fumigatus to induce EoE in TRAIL-sufficient (wild-type)

279 thelium responds to apical stimulation by A. fumigatus to promote the transepithelial migration of ne

280 a novel stage-specific susceptibility of A. fumigatus to zinc and manganese chelation by neutrophil-

281 trophils into the airspace in response to A. fumigatus Together, these data provide critical insight

282 mplex network of interactions amongst the A. fumigatus TOR, SakA and SchA pathways.

283 were exposed via nose-only inhalation to A. fumigatus viable conidia, heat-inactivated conidia (HIC)

284 that methionine synthase is essential for A. fumigatus virulence, defining the biosynthetic route of

285 ion to animal dander, pollen, or Aspergillus fumigatus was associated with asthma.

286 present by qPCR in 86% of patients, while A fumigatus was detectable by qPCR in 46%.

287 Aspergillus fumigatus was the most commonly identified species.

288 which mice rendered allergic to Aspergillus fumigatus were co-infected with influenza A virus and St

289 nteractions between human neutrophils and A. fumigatus were observed in real time, at single-cell res

290 a, Streptococcus pneumoniae, and Aspergillus fumigatus when mice were heavily engrafted with leukemia

291 inst the widely distributed mold Aspergillus fumigatus, which is a major threat for immunocompromised

292 ilent gene cluster of the fungus Aspergillus fumigatus, which is activated by the bacterium Streptomy

293 actosaminogalactan (GG) found in Aspergillus fumigatus, which is the most important airborne human fu

294 ct triazole-resistant clinical strains of A. fumigatus with a variety of cyp51A gene mutations, as we

295 ulbs were positive for triazole-resistant A. fumigatus with CYP51A mutations.

296 Treatment of A. fumigatus with Sph3h disrupted A. fumigatus biofilms wit

297 ance in a panel of 30 clinical strains of A. fumigatus within about 6 h.

298 ereas healthy people can inhale spores of A. fumigatus without developing disease, neutropenic patien

299 We also found that an A. fumigatus zafA mutant, which demonstrates deficient zinc

300 g." In Talaromyces marneffei and Aspergillus fumigatus zebrafish in vivo infections, live imaging dem