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1 mulation have reported to increase speeds of functional recovery.
2 ing of cortical reorganisation that underlie functional recovery.
3 gR1 limits regenerative axonal sprouting and functional recovery.
4 , improved thalamocortical connectivity, and functional recovery.
5 roenvironment that supports regeneration and functional recovery.
6 endogenous neural tissue repair and promote functional recovery.
7 y after stroke for optimal neural repair and functional recovery.
8 The primary endpoint was time to functional recovery.
9 farct size and negatively impacts tissue and functional recovery.
10 to thereby promote motor re-innervation and functional recovery.
11 n regeneration, cutaneous reinnervation, and functional recovery.
12 on of BDNF signaling pathways may facilitate functional recovery.
13 prolonged hemorrhage, and impaired forelimb functional recovery.
14 e to their distal counterparts contribute to functional recovery.
15 rategies that improve nerve regeneration and functional recovery.
16 ng mechanisms that promote kidney repair and functional recovery.
17 eneration, circuit formation, and eventually functional recovery.
18 esion site is pivotal for axon re-growth and functional recovery.
19 t poststroke cortical plasticity and thereby functional recovery.
20 trimental and resulted in delayed repair and functional recovery.
21 rinsic power of myelin plasticity to promote functional recovery.
22 ing altered astrocyte responses and impaired functional recovery.
23 treatment can produce persistent effects on functional recovery.
24 that cell-based interventions may influence functional recovery.
25 ion of individual MAPKs allowed only partial functional recovery.
26 ssessments of muscle tissue regeneration and functional recovery.
27 ry and necessary for axonal regeneration and functional recovery.
28 obal I/R ex vivo and showed improved cardiac functional recovery.
29 but little is known about those who achieve functional recovery.
30 y lacks effective therapy enabling long-term functional recovery.
31 vomarkedly accelerated axon regeneration and functional recovery.
32 5%) were observed for up to 1 year to assess functional recovery.
33 L and OL progenitor replacement, and chronic functional recovery.
34 s of survival to discharge and survival with functional recovery.
35 vived to discharge, and 7176 (7.4%) achieved functional recovery.
36 dendritic plasticity and to induce long-term functional recovery.
37 s, promotes circuit restoration and improves functional recovery.
38 ly distinct subsets of neurons, resulting in functional recovery.
39 ntral nervous system (CNS) damage, promoting functional recovery.
40 compensation in perilesion cortex, enabling functional recovery.
41 trol of corticospinal connections to promote functional recovery.
42 ons that regulates motor behavior capable of functional recovery.
43 ositive long-term impact on neurological and functional recovery.
44 ings were noted in analyses of survival with functional recovery.
45 l repair cell, neuronal death and failure of functional recovery.
46 tive neurogenesis and accelerates vestibular functional recovery.
47 on, neural angiogenesis, muscle atrophy, and functional recovery.
48 ition following spinal cord injury and limit functional recovery.
49 formation of new synaptic contacts to enable functional recovery.
50 and synaptic connections that drive greater functional recovery.
51 ction and an improved in vitro post-ischemic functional recovery.
52 ently accompanied by a degree of spontaneous functional recovery.
53 n targeting spinal synapses further promotes functional recovery.
54 s is safe, reduces infarct size and improves functional recovery.
55 stroke-induced brain damage, and facilitate functional recovery.
56 xonal regeneration in the damaged CNS limits functional recovery.
57 rate their axon after nerve injury to enable functional recovery.
58 er time; their magnitude being predictive of functional recovery.
59 ory responses, neuropathology, and long-term functional recovery.
60 Primary outcome was time to functional recovery.
61 maintaining muscle receptivity and enhancing functional recovery.
62 ults have a higher risk of relapse and worse functional recovery.
63 after injury enhances aged regeneration and functional recovery.
64 ic mechanism, which impedes regeneration and functional recovery.
65 implicated to inhibit neuronal regrowth and functional recovery.
66 lockers correlated with late survival and LV functional recovery.
67 n regeneration after SCI leading to improved functional recovery.
68 manipulations aimed at improving healing and functional recovery.
69 the brain, including remote areas supporting functional recovery.
70 o engage residual neural networks to improve functional recovery.
71 erbated proinflammatory responses, and worse functional recovery.
72 m activity, probably explaining only partial functional recovery.
73 rmally supports mitochondrial biogenesis and functional recovery.
74 h as after stroke, partially contributing to functional recovery.
75 n functional connectivity is associated with functional recovery 1 year after cardiac arrest (CA).
76 . 43.4 +/- 10%, respectively; P = 0.057) and functional recovery (42.4 +/- 6% vs. 32.2 +/- 7%, respec
78 rodent spinal cord, yet they support delayed functional recovery, a finding of great importance in pl
79 in the peri-infarct region, promoted optimal functional recoveries after stroke in male and female, y
80 uced lumbar MN dendritic atrophy and enabled functional recovery after a rostral thoracic contusion.
81 c syndrome that diminishes the potential for functional recovery after a transcatheter aortic valve r
83 poptosis, reduced infarct size, and improved functional recovery after acute MI relative to wild-type
93 rated from skin or nerve promotes repair and functional recovery after incomplete cervical crush inju
99 are known to inhibit axonal regeneration and functional recovery after mammalian central nervous syst
100 ed the role of MDMs in long-term spontaneous functional recovery after middle cerebral artery occlusi
101 emonstrated significant reduction in time to functional recovery after MIDP compared with ODP, but wa
102 cal innovation, and aimed to compare time to functional recovery after minimally invasive and open di
104 harmacological approach for the promotion of functional recovery after nerve injury.In vitroandin viv
105 oned mouse optic nerves (MONs) showed better functional recovery after OGD than the non-preconditione
108 vel function of HDAC3 inhibitor in promoting functional recovery after SCI by dampening inflammatory
109 ovide a promising new opportunity to enhance functional recovery after SCI or in other disorders.
116 teoglycans and improves axonal sprouting and functional recovery after spinal cord injury in rodents.
121 of late gadolinium-enhancement in predicting functional recovery after ST-segment-elevation myocardia
123 a link between movement-preparatory LFOs and functional recovery after stroke, promoting their releva
128 GDF10 produced axonal sprouting and enhanced functional recovery after stroke; knocking down GDF10 bl
129 esponses previously associated with pain and functional recovery after surgery, including STAT3 and C
130 r neurogenesis and significantly accelerates functional recovery after unilateral vestibular injury.
132 gery, and identified factors associated with functional recovery among older persons who survive a ma
133 second, to identify factors associated with functional recovery among older persons who survive a ma
134 e than nonhemorrhagic infarcts, with lack of functional recovery and adverse LV left ventricular remo
135 eding after an esophagectomy does not affect functional recovery and did not increase incidence or se
139 otic polyneuropathy is characterized by poor functional recovery and impaired nerve regenerative resp
141 following a MIE results in a faster time to functional recovery and lower 30-day postoperative compl
142 enous Nogo receptor antagonist, in promoting functional recovery and neural repair after spinal cord
143 letion of chronically activated microglia on functional recovery and neurodegeneration up to 3 months
144 rophic factor (BDNF) can modulate vestibular functional recovery and neurogenesis in mammals, in this
145 utative mitochondrial protectant, on cardiac functional recovery and potential mechanisms of CsA acti
148 15-minute reperfusion significantly improved functional recovery and significantly increased phospho-
149 mediates S129 phosphorylation) showed better functional recovery and smaller infarcts when subjected
152 of sudden cardiac death and likelihood of LV functional recovery, and has significant potential to gu
153 ith respect to survival, residential status, functional recovery, and quality of life in both hospita
154 oliposomal treatment for 3 consecutive days, functional recovery as indicated by improved neurologic
155 epithelia, was coupled with a corresponding functional recovery, as seen in the suprathreshold ampli
161 y associated with an increased likelihood of functional recovery: being nonfrail (hazard ratio 1.60;
162 erioperatively with riluzole did not improve functional recovery beyond decompressive surgery in pati
163 ssociated with decreased mortality or better functional recovery but being underweight predicted unfa
164 ervention, persisting hyperglycemia prevents functional recovery but promotes beta-cell mass increase
165 I had similar predictive accuracy to SEE for functional recovery but was not assessable in 25% of pat
166 at corticocuneate inputs increase during the functional recovery, but their functional role is uncert
167 nnections, and RhoA-ROCK inhibition enhances functional recovery by blocking this detrimental effect.
168 suggested to be key inhibitory molecules for functional recovery by impeding axonal regrowth/sproutin
169 to assess consequential effects on beta-cell functional recovery by lowering glucose homeostasis and/
170 RvD6si promotes corneal wound healing and functional recovery by restoring corneal innervation aft
171 ence that maximal circuit reorganization and functional recovery can be achieved by combining molecul
174 smaller than 10 mm in rodents, nearly normal functional recovery can be achieved; for longer gaps, ho
177 eived SCs from either source showed improved functional recovery compared with media- or fibroblast-t
179 r, ablated CNS edema, and led to accelerated functional recovery compared with untreated animals.
181 ion at acute imaging and odds ratio (OR) for functional recovery decreased with increasing SEE, altho
182 ation in rates of survival and survival with functional recovery (defined as Cerebral Performance Cat
187 greatly reduced and there was no significant functional recovery even in Ryk conditional knockout mic
188 se of optical coherence tomography (SD-OCT), functional recovery evidenced by multifocal-electroretin
189 M residuals, we constructed the FRESH score (Functional Recovery Expected after Subarachnoid Hemorrha
190 ity, since it undergoes near full growth and functional recovery following acute depletion of granule
197 ssor of pial collateral remodeling, CBF, and functional recovery following permanent middle cerebral
200 t mammalian heart is incapable of meaningful functional recovery following substantial cardiomyocyte
201 one's body, may be an important component of functional recovery for individuals with upper limb abse
203 eural circuits and behavior, but this limits functional recovery from brain diseases and dysfunctions
210 were associated with a reduced likelihood of functional recovery: hearing impairment, greater increas
211 efficacy of daidzein on neuroprotection and functional recovery in a clinically relevant mouse model
212 nd PPARgamma agonist administration improved functional recovery in a clinically relevant mouse strok
213 ce cardiac regeneration and left ventricular functional recovery in a swine model of chronic ischemic
214 nce-dependent cortical plasticity as well as functional recovery in adulthood.SIGNIFICANCE STATEMENT
215 stered intravenously induced morphologic and functional recovery in AKI, the Drosha-knockdown counter
216 fter stroke plays a crucial role in limiting functional recovery in an experimental model of diabetes
217 strategies to augment neuroplasticity and/or functional recovery in animal models of SCI that are pus
218 possible to induce partial respiratory motor functional recovery in chronic SCI following 2 weeks of
222 imulation of the spinal cord (TESS) promotes functional recovery in humans with spinal cord injury (S
223 d the potential for molecular, cellular, and functional recovery in mice from the severe disruption o
224 ing the repair phase was reported to enhance functional recovery in mice suggesting that GABA plays a
225 neural regeneration, tissue remodeling, and functional recovery in mice with spinal cord injury.
227 s system (CNS) axons is a major obstacle for functional recovery in patients suffering neurological d
233 oaches promote significant axonal growth and functional recovery in vivo in a spinal cord injury mode
234 to identify the pathways driving spontaneous functional recovery in wild-type and plasticity-sensitiz
236 motion more or less unchanged, but abolished functional recovery, indicating that dI3 interneurons ar
237 on improves coordinated locomotion, and this functional recovery is accompanied by preservation of my
238 nds on acute infarct size, whereas long-term functional recovery is an important outcome in patients.
239 f the role of neuroanatomical plasticity for functional recovery is fundamental for successful transl
243 ion of plasticity in the adult brain impedes functional recovery later in life from brain injury or d
244 our finding implies that microglia-dependent functional recovery may be particularly difficult in tho
245 th the early compensatory mechanisms and the functional recovery mechanisms, with reduced aromatic L-
246 of the ischemic brain regions is crucial for functional recovery, no therapeutics that promote angiog
247 ce extrusion, exchange, or explantation) and functional recovery of 20/200 or better visual acuity at
248 locking and nonblocking Abs synergize in the functional recovery of antigen-specific exhausted CD8 T
249 l incomplete spinal cord injury show limited functional recovery of elbow extensors compared with elb
252 tion, neuromuscular junction maturation, and functional recovery of injured sciatic nerves, and incre
253 of CD200L expression by CNS-resident cells, functional recovery of mice following SCI was impaired.
259 erve CES prior to DNT significantly improved functional recovery of the common fibular nerve and its
260 gnificant discrepancy between structural and functional recovery of the failing myocardium, as only a
261 kidney-pancreas transplant by evaluating the functional recovery of the graft and biochemical markers
262 their activation promotes axon regrowth and functional recovery of the thermonociceptive behavior.
263 erapy or chemotherapy can hardly support the functional recovery of the tongue-particularly, function
264 f contralesional hemispheric compensation to functional recovery of the upper extremity after a unila
265 ouse visual cortical plasticity and promotes functional recovery of visual acuity defects from amblyo
266 ti-IFNAR treatment did not improve long-term functional recovery or decrease TBI neuropathology at 28
267 ngth of stay (LOS), other variables included functional recovery, pain scores, peak flow, vasopressor
268 ostoperative pain scores at 12 and 48 hours, functional recovery, pain treatment-related complication
269 utcome, but there are little data on whether functional recovery post-stroke varies among hospitals.
270 dence interval, 1.32-1.46) and survival with functional recovery (range, 0.8%-21.0%; median odds rati
271 dependent or dead 3 months postacute stroke; functional recovery rates varied considerably among hosp
274 To confirm a mechanistic role for B cells in functional recovery, rituximab was given to human CD20(+
275 brain after stroke underlies the incomplete functional recovery seen in patients and that boosting h
276 ntly acceleratedin vivoaxon regeneration and functional recovery similar to GSK3alpha(S/A)/beta(S/A)
277 ted neuroinflammation after ICH and promotes functional recovery, suggesting that TGF-beta1 may be a
280 rticipants with delirium demonstrated lesser functional recovery than their counterparts without deli
281 c stroke contribute to long-term spontaneous functional recovery through inflammation-resolving activ
291 g the Confusion Assessment Method, and their functional recovery was followed for 18 months thereafte
294 processes are coordinated over the course of functional recovery, we tracked receptive field reorgani
295 peri-infarct area, infarct size, and animal functional recovery were assessed at 1, 2, and 3 weeks a
296 proving patients' level of consciousness and functional recovery were behavioural and brain imaging o
297 gen-glucose deprivation, they show a reduced functional recovery when returned to oxygen-glucose but
298 tive surgery were positively associated with functional recovery, whereas hearing impairment, greater
299 The associative group significantly improved functional recovery with respect to the control group (m
300 mpairment were strongly associated with poor functional recovery within 6 months, whereas higher body