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1 ed as evidence for a higher level of dynamic fusimotor activity maintained during active movements th
2                  There was clear evidence of fusimotor activity occurring during active jaw closing s
3 muscle length changes and static and dynamic fusimotor activity to determine primary afferent firing
4                   The underlying patterns of fusimotor activity were deduced by comparing afferent di
5  not therefore be able to respond to dynamic fusimotor activity.
6 was taken to represent the profile of static fusimotor activity.
7 vements repeated passively in the absence of fusimotor activity.
8         However, they were not innervated by fusimotor axons and they did not express glial derived n
9 increased numbers of large sensory and small fusimotor axons.
10 rent gains and/or the integrity of automatic fusimotor control.
11 tate of their parent muscle and by efferent (fusimotor) control, and their discharges represent futur
12 erefore attributed to a modulation of static fusimotor discharge approximately in parallel with alpha
13            This need to control gamma-static fusimotor drive explicitly as a function of muscle lengt
14 red systematically to evaluate the effect of fusimotor drive on the shape of the temporal profile of
15 plied through a complex nonlinear profile of fusimotor drive that is not yet experimentally observed
16 lpha-gamma coactivation as the main types of fusimotor drive that transform the monosynaptic Ia affer
17 ntly required the modulation of gamma-static fusimotor drive to produce increases in physiological tr
18 igate the nature (i.e. static or dynamic) of fusimotor drive to the flexor hallucis longus (FHL) and
19 servations are compatible with the view that fusimotor drive varies in different muscles during locom
20       Simulated physiological changes to the fusimotor drive were not sufficient to reproduce muscle
21 hy humans likely increase their gamma-static fusimotor drive when muscles shorten.
22  (beta-MNs) in mammals, to create a flexible fusimotor ecosystem to enable voluntary movement.
23 ve and active movements, indicating that the fusimotor effects associated with active contractions ha
24 he intrafusal fibres activated by individual fusimotor efferents) were separated by a minimum conduct
25 y selectively operated by static and dynamic fusimotor efferents.
26             This indicated increased dynamic fusimotor firing during active locomotion.
27 iasing activity, presynaptic inhibition, and fusimotor gain).
28  intrafusal muscle fiber differentiation and fusimotor innervation homeostasis.
29 ongus muscle were recorded in the absence of fusimotor input to ramp and hold stretches as well as to
30  signal isometric force and are modulated by fusimotor input.
31                             This human-based fusimotor model and its incorporation into the reflex ar
32 rophic factor (GDNF), which is essential for fusimotor neuron survival.
33                                      Dynamic fusimotor neurone firing appears to be set at a raised l
34 or a variety of active jaw movements, static fusimotor neurone firing is modulated roughly in paralle
35 s, operated separately by static and dynamic fusimotor neurones, and the topological structure of the
36 ion to the concept that the modulated static fusimotor pattern may represent a 'temporal template' of
37 signal matched the predictions of the static fusimotor signal derived from secondary afferents.
38 and separate and combined static and dynamic fusimotor stimulation were recorded in physiological exp
39      This suggests that the way in which the fusimotor system works may differ between the two muscle
40 l not only the skeletal muscles but also the fusimotor system.
41 nable propriospinal circuit in the mammalian fusimotor system.